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p组
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  p group
     Gray level (GL, the more higher GL, the more decreasing expression) of bcl-2 in Group P, Group IR, Group IP and Group DP were 114.5±4.2, 135.2±5.8, 124.5 ± 5.1, 122.8±6.6 respectively.
     bcl-2免疫组化的灰度值(gray level,GL)在P组、IP组、IP组和DP组分别为114.5±4.2,135.2±5.8,124.5±5.1,122.8±6.6。
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     had no effect on pulmonary neutrop hil accumulation from R group(1.8± 0. 5)to P group(1.7±0.3)(P>0.05);
     肺内中性粒细胞聚集量,P组与R组(1.7±0.3vs1.8±0.5)无显著差异(P>0.05);
短句来源
     (2)After reperfusion 6 h,p ropof ol inhibited conspicuously transmigrated neutrophils metabolic activity fro m R group(1.76± 0.09)to P group(1.32± 0.04 )(P<0.01);
     (2)再灌注后6h肺内中性粒细胞的代谢活性,P组较R组(1.32±0.04vs1.76±0.09)显著下降(P<0.01);
短句来源
     and obvious attenuation of pulmonary mi crovascular permeability attenuated lung microvascular injury f rom R group(0.44±0.10)to P group(0.32±0.06)(P<0.05).
     肺毛细血管通透性,P组较R组(0.32±0.06vs0.44±0.10)明显降低(P<0.05)。
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     MIP-2:compare with R grroup (31.5±2.4), There was no statistically significant difference in P group(35.4±5.3), the elevation in M group(44.7±6.9, t=7.85, P=0.04), there was significantly elevated in N group( 167.7± 11.8, t=27.779, P=0.000).
     MIP-2:P组(35·4±5·3)与R组(31·5±2·4)比较,差异无统计学意义,M组(44·7±6·9)升高(t=4·382,P=0·04),N组(167·7±11·8)明显升高(t=27·779,P=0·000)。
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  “p组”译为未确定词的双语例句
     There were no significant differences in the three treatment groups (P>0.05).
     F组、P组及F加P组间各指标差异无统计学意义(P>0.05)。
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     Methods:A total of 102 ACS cases were randomly divided into two groups: pravastatin group (50 cases,treated with pravastatin);
     方法:102例ACS患者被随机分成两组:普伐他汀组(P组)和阿托伐他汀组(A组),两组均予氯吡格雷300mg顿服后,75mg/d维持。
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     (2)To compare with baseline the PAR after treatment in either group significantly decreased(P<0.05),but there were no difference in patients between two groups after treatment;
     (2)P组或A组PAR在治疗后和基线相比,差异有显著性(P<0.05);
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     Compared with ketamine group,MDA level was less and SOD was higher in propofol group (P<0.05). Conclusion:Ketamine and propofol sedation attenuate lipid peroxidation markers in total knee replacement with tourniquet application.
     再灌注期(T3)3组病人MDA进一步升高,SOD活性则较T1时点显著降低(P<0.01),K组和P组MDA值低于C组,而SOD值均明显高于C组(P<0.05或P<0.01),且P组MDA值低于K组,SOD值高于K组(P<0.05)。
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     fluorescent expression in liver of group P+U was significantly higher than those in other groups(P<0.001).
     P+U组降低较P组显著,P<0.001;
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     Group E is the highest.
     B
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     Group B: SD to Wistar rat heart tarnsplantation;
     B
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     In every group,peripheral nerve was performed end-to-side anastomosis.
     C:ATP;
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     group B, TPN;
     B,TPN;
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  p group
As shown by both methods, APE1 binds with the highest efficiency to DNA substrate containing 5'-sugar phosphate group in the nick/gap, whereas DNA polymerase β binds to DNA duplex with a mononucleotide gap flanked by the 5'-p group.
      
In particular, it is shown that, as in the abstract case, a pro-p group can have only 0, 1, 2, or infinitely many ends; pro-p groups with two ends are classified and a sufficient condition for a pro-p group to have precisely one end is obtained.
      
Results There were 35 CDM-positive patients (P group, 40.2%) and 52 CDM-negative patients (N group , 59.8%).
      
The NIHSS at entry were significantly lower in the P group, but there was no difference in the outcome at three months measured by the modified Rankin Scale.
      
Clinical features of nosocomial bacteremias due to methicillin-resistantStaphylococcus aureus (MRSA) (M group, n=71) andPseudomonas aeruginosa (P group, n=25) in a university hospital during 1982-1993 were retrospectively analyzed.
      
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A stereologic study of lamellar bodies within pulmonary alveolar type II cells in dogs with experimental acute hemorrhagic pancreatitis(group P)and in dogs inje- cted muscularly with choloroquine before inducing pancreatitis (group PI)was carried out.Our findings showed that volumerical density (Vv), surface density (Sv)and numerical density(Nv)of lamellar bodies were decreased by 29.06%,30.28% and 40.75% in the group P as compared with those in the control group (group N), respectively, and their Sv and Nv...

A stereologic study of lamellar bodies within pulmonary alveolar type II cells in dogs with experimental acute hemorrhagic pancreatitis(group P)and in dogs inje- cted muscularly with choloroquine before inducing pancreatitis (group PI)was carried out.Our findings showed that volumerical density (Vv), surface density (Sv)and numerical density(Nv)of lamellar bodies were decreased by 29.06%,30.28% and 40.75% in the group P as compared with those in the control group (group N), respectively, and their Sv and Nv were decreased by 23.8% and 30.26% in comparison with those in the group PI.The results suggest that the lung injury resulting from acute hemorrhagic pancreatitis was characterized by alveolar surfactant System damage.Dramatical decrease in the number of the lamellar bodies affected the surface tension of alveoli in a certain degree, leading to atelectasis.There was a relationship between lung injury and the effect of PLA2(phospholipidase A2).Choloroquine might play a role in maintaining the number of lamellar bodies.

本文报道了对犬实验性急性出血性胰腺炎(P组)与加用氯喹(PI组)时Ⅱ型肺泡上皮细胞的板层小体体视学形态定量的研究。结果发现:P组板层小体的体积密度(Vv),表面积密度(Sv)及数密度(Nv)分别较对照组下降29.06%,30.28%,及40.75%;其Sv及Nv分别较PI组减少23.8%和30.26%。结果提示:并发于急性出血性胰腺炎的肺损伤以肺泡表面活性物质系统受损为一重要的表现形式。板层小体数目显著减少,一定程度影响到肺泡的表面张力,从而造成肺不张。肺损伤与磷脂酶A_2的作用有关;氯喹在维持板层小体的数量方面可能有一定的作用。

Results of a randomized double blind trial comparing the therapeutic efficacy of single dosos of levo-praziquantel (L-P) and praziquantel (P) respectively in the treatment of 139 matched pairs of proven cases of schistoeomiasis Japonica are reported. 268 were chronic early cases and 10 were late cases. All the patients were treated with doaes of L-P 20mg/kg or P 40mg/kg. 4 and 6 months after treatment, the stool-ova negative conversion rates were 94.85% and 96.27% for the L-P group, and 97.06% and 94.03% for...

Results of a randomized double blind trial comparing the therapeutic efficacy of single dosos of levo-praziquantel (L-P) and praziquantel (P) respectively in the treatment of 139 matched pairs of proven cases of schistoeomiasis Japonica are reported. 268 were chronic early cases and 10 were late cases. All the patients were treated with doaes of L-P 20mg/kg or P 40mg/kg. 4 and 6 months after treatment, the stool-ova negative conversion rates were 94.85% and 96.27% for the L-P group, and 97.06% and 94.03% for the P group, respectively (P>0.05). For mild and moderately infected cases L-P in a single dose of 20 mg/kg is as effective as a single dose of P 40mg/kg, more over, it produces less side effects. It is thus deemed worthy of popularization.

本文采用随机双盲对照法以左旋吡喹酮(Levopraziquantel,L-P)与吡喹酮(Praziquantel,P)一剂治疗粪检阳性的日本血吸虫病278例(139对配对),其中慢性早期(慢血)268例,晚期(晚血)10例。L-P组剂量为20mg/kg,P组为40mg/kg,一次顿服,治后4~6个月,L-P组粪检阴转率分别为94.85%、96.27%;P组粪检阴转率分别为97.06%、94.03%,(P>0.05),L-P20mg/kg治疗轻、中度感染是可行的,疗效与P40mg/kg相仿。L-P剂量小、毒副反应轻、安全、简便、有利于大规模治疗的开展。

Seventy eight New Zealand rabbits were divided into 8 groups. NE, M, and control group were given norepinephrine (1mg/kg), methoxamine or saline, respectively. The other 5 groups were given regitine (R group), propranolol (P group), metaprolol (MP group), yohimbine (Y group) and prazosine (PR group) respectively before NE infusion.Myocardial injury was estimated with a semi-quantitative histological scoring system, and 11 intermediate metabolites in glycolysis and ATP, ADP, AMP, CP in myocardium were measured....

Seventy eight New Zealand rabbits were divided into 8 groups. NE, M, and control group were given norepinephrine (1mg/kg), methoxamine or saline, respectively. The other 5 groups were given regitine (R group), propranolol (P group), metaprolol (MP group), yohimbine (Y group) and prazosine (PR group) respectively before NE infusion.Myocardial injury was estimated with a semi-quantitative histological scoring system, and 11 intermediate metabolites in glycolysis and ATP, ADP, AMP, CP in myocardium were measured. Results showed that myocardial injury was observed in NE group (P<0.01),as well as in P, MP, Y and M groups; group R and PR tended to have less myocardial injury in comparison with NE group. Metabolic dysfunction was found in NE and MP group, while group R showed a nearly normal glycolysis and energy metabolizm. Conclusion: α-adrenoceptor, especially α_1 receptor, plays the key role in development of NE-induced myocardial injury. As for its mechanism we put forward a new hypothesis.

新西兰免78只分成8组。试验组、M组和对照组分别静滴去甲肾上腺素(NE)1毫克/公斤、美索克新明或生理盐水。其余5组在NE之前分别给予酚妥拉明(R组)、心得安(P组)、美多心安(MP组)、育亨宾(Y组)以及(口派)唑嗪(PR组)。以半定量方法分析心肌病理损害程度,并测定心肌中糖酵解中间产物11项以及能量代谢产物。结果发现NE组心肌损害显著(P<0.01);P组、MP组、Y组和M组心肌损害类似于NE组;R组和PR组则较NE组减轻(P<0.01)。NE组和MP组心肌尚有明显的代谢障碍,而R组则较接近正常。提示NE所致心肌损害中α受体,尤其α_1受体起了主要作用。作者等对其发生机理进行讨论,并提出新的假设。

 
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