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室纤维化
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  ventricular fibrosis
     Effect of Compound Danshen Dripping Pills Combined with Fosinopril on Left Ventricular Fibrosis of Spontaneously Hypertensive Rats
     复方丹参滴丸与福辛普利联用对自发性高血压大鼠左室纤维化的影响
短句来源
     Objective This study was to investigate effects of both compound Danshen dripping pills (DSP) and the combined use of DSP with fosinopril on left ventricular fibrosis of spontaneously hypertensive rats.
     目的 探讨复方丹参滴丸 (DSP)与福辛普利联用对自发性高血压大鼠 (SHR)左室纤维化的影响。
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  “室纤维化”译为未确定词的双语例句
     Results compare with the control group,①DSP can decrease left ventricle collage and LVHI of SHR(P<0.01 or P<0.05)and enhance the anti-left-ventricular -fibrosis effect of fosinopril.
     结果 与对照组比较 ,DSP可明显减轻左室肥厚 (P <0 .0 1) ,改善左室纤维化 (P <0 .0 1或P <0 .0 5 ) ,并与福辛普利联用时可进一步提高后者的抗心肌纤维化效应 ;
短句来源
     During the 6 months the serum PⅢNP, BNP and echocardiography were examined in all patients to assess myocardial fibrosis, LV function and volume.
     在6个月干预期内检测两组血清Ⅲ型前胶原氨基端肽(PⅢNP)、脑钠肽(BNP)及超声心动图,以评价左室纤维化、左室功能和左室容积。
短句来源
  相似匹配句对
     Echocardiographic Feature of Right Ventricular Endomyocardial Fibrosis
     右心内膜心肌纤维化的超声心动图特征
短句来源
     4. collective chamber;
     收集;
短句来源
     Pancreatic fibrosis
     胰腺纤维化
短句来源
     Bubble Chamber
     气泡
短句来源
     interstitial fibrosis.
     间质纤维化
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  ventricular fibrosis
Impairments in coronary circulation and ventricular fibrosis, which are an essential part of hypertensive disease, contribute to that increased risk.
      
The effect of therapy on hypertension-induced ventricular fibrosis and impairment in coronary hemodynamics and the risk associated with these changes are also discussed.
      
Progressive right ventricular fibrosis, ventricular hypertrophy, and chronic hypoxia are avoided, which may reduce the incidence of late ventricular arrhythmias.
      
Converting enzyme inhibition reduces in parallel arterial hypertension, cardiac hypertrophy, and ventricular fibrosis; it prevents ventricular premature beats and normalizes heart rate variability.
      
Ventricular premature beats are more frequent in SHR than in age-matched controls; they disappear after converting enzyme inhibition (CEI) relative to the reduction of both cardiac hypertrophy and ventricular fibrosis.
      
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AIM: To study the distribution, location and quantity of myocardial collagen(Col) in different phases after myocardial infarction(MI). METHODS: The method of immunohistochemical technique combined with automatic image analysis was used to investigate the changes of collagen Ⅰ and Ⅲ. RESULTS:Contents of Col Ⅰ, Col Ⅲ and the ratio of Ⅰ/Ⅲ were all increased in infarcted area (IA)after MI,and the increase of Col Ⅰ was higher, the collagen fibers still construct a network in acute phase, in chronic stage, all...

AIM: To study the distribution, location and quantity of myocardial collagen(Col) in different phases after myocardial infarction(MI). METHODS: The method of immunohistochemical technique combined with automatic image analysis was used to investigate the changes of collagen Ⅰ and Ⅲ. RESULTS:Contents of Col Ⅰ, Col Ⅲ and the ratio of Ⅰ/Ⅲ were all increased in infarcted area (IA)after MI,and the increase of Col Ⅰ was higher, the collagen fibers still construct a network in acute phase, in chronic stage, all necrosis myocytes were replaced by collagen, the fibers were dense, and paralelled with cardiac cavity surface.In the septum,noninfarcted area, contents of collagen type Ⅰ and Ⅲ, the ratio of Ⅰ/Ⅲ were increased.In acute phase, the increase of type Ⅲ is dominant,with in chronic phase, type Ⅰ is. Right ventricle(RV) has no significant change in acute phase,untill chronic phase,it changed like septum,the increase in collgen is mainly type Ⅰ. CONCLUSION: The collgen remodeled after myocardial infarction were reparative fibrosis in IA phase, and reactive fibrosis in NIA phase. Remodeling occured later in RV than in septum.

目的 :对心肌梗塞不同时期心脏各部位胶原重建进行定性、定位、定量研究。方法 :应用免疫组化技术结合自动图象分析方法观察Ⅰ、Ⅲ型胶原分布及含量的变化。结果 :心肌梗塞后坏死区Ⅰ、Ⅲ型胶原含量及Ⅰ Ⅲ比值均增加 ,以Ⅰ型胶原为主 ,急性期胶原仍交织成网状 ,至恢复期坏死心肌全部被胶原取代 ,呈致密束状与心腔表面平行排列。室间隔 (未梗塞区 )两型胶原含量及比值均增加 ,急性期以Ⅲ型胶原增加明显 ,恢复期以Ⅰ型胶原增加为主。右室在急性期胶原含量无明显改变 ,恢复期变化同室间隔 ,也以Ⅰ型胶原为主。结论 :心肌梗塞后胶原重建 ,梗塞区呈修复性纤维化 ,非梗塞区呈反应性纤维化 ,右室纤维化较左室晚。

Objective This study was to investigate effects of both compound Danshen dripping pills (DSP) and the combined use of DSP with fosinopril on left ventricular fibrosis of spontaneously hypertensive rats. Methods 48 eight-week-old male spontaneously hypertensive rats (SHRs) were assigned randomly into six groups: low dose DSP (SHRdl); high dose DSP (SHRdh);fosinopril (SHRf); combination use of low dose DSP with fosinopril (SHRdl+f); combination use of high dose DSP with fosinopril (SHRdh+f) and placebo group...

Objective This study was to investigate effects of both compound Danshen dripping pills (DSP) and the combined use of DSP with fosinopril on left ventricular fibrosis of spontaneously hypertensive rats. Methods 48 eight-week-old male spontaneously hypertensive rats (SHRs) were assigned randomly into six groups: low dose DSP (SHRdl); high dose DSP (SHRdh);fosinopril (SHRf); combination use of low dose DSP with fosinopril (SHRdl+f); combination use of high dose DSP with fosinopril (SHRdh+f) and placebo group with distilled water (SHRc). Drugs and distilled water were given to each group for 8 weeks. Arterial systolic blood pressure (SBP) and left ventricular hypertrophy index (LVHI) were measured. Contents of angiotensin Ⅱ(AngⅡ) and aldosterone (Ald) were detected by radioimmunoassay.Myocardial collage was stained and collage volumefraction was determined in the left ventricle by quantitative morphometry. Results compare with the control group,①DSP can decrease left ventricle collage and LVHI of SHR(P<0.01 or P<0.05)and enhance the anti-left-ventricular -fibrosis effect of fosinopril.②DSP can decrease the concentration of plasma AngⅡ and Ald and the concentration of AngⅡin myocardium (P<0.01 or P<0.05). Conclusion DSP and combined use of DSP and fosinopril can decrease left ventricular fibrosis.

目的 探讨复方丹参滴丸 (DSP)与福辛普利联用对自发性高血压大鼠 (SHR)左室纤维化的影响。方法 将 48只8周龄雄性SHR随机分为 6组 :DSP小剂量组 ;DSP大剂量组 ;福辛普利组 ;DSP小剂量与福辛普利联用组 ;DSP大剂量与福辛普利联用组 ;SHR对照组。 6组分别用灌胃法给药8周 ,测大鼠尾动脉收缩压、左室肥厚指数 ;局部心肌 /血浆血管紧张素Ⅱ、血浆醛固酮浓度 ;通过天狼星红染色进行心肌胶原定性和半定量分析。结果 与对照组比较 ,DSP可明显减轻左室肥厚 (P <0 .0 1) ,改善左室纤维化 (P <0 .0 1或P <0 .0 5 ) ,并与福辛普利联用时可进一步提高后者的抗心肌纤维化效应 ;DSP尤其与福辛普利联用时可降低血浆AngII、Ald及局部心肌AngII浓度 (P <0 .0 1或P <0 .0 5 )。结论 DSP与福辛普利联用可改善SHR心肌纤维化(MF)。

Objective To investigate the effect of spironolactone on left ventricular remodeling (LVRM) in patients with acute myocardial infarction. Methods In this multicentric, randomized, controlled study, spironolactone 40 mg/d was randomly administered in addition to the routine treatment for patients with AMI. During the 6 months the serum PⅢNP, BNP and echocardiography were examined in all patients to assess myocardial fibrosis, LV function and volume. Results A total of 88 AMI patients entered the study came...

Objective To investigate the effect of spironolactone on left ventricular remodeling (LVRM) in patients with acute myocardial infarction. Methods In this multicentric, randomized, controlled study, spironolactone 40 mg/d was randomly administered in addition to the routine treatment for patients with AMI. During the 6 months the serum PⅢNP, BNP and echocardiography were examined in all patients to assess myocardial fibrosis, LV function and volume. Results A total of 88 AMI patients entered the study came from 4 hospitals in Shijiazhuang. There were 43 patients with anterior MI and 45 with inferior MI. In anterior MI group 23 patients received spironolactone and 20 accepted the routine treatment. In inferior MI group 23 received spironolactone and 22 accepted the routine treatment. In anterior MI gsoup: ①At 3rd, 6th month PⅢNP and BNP serum levels were significantly lower in!the spironolactione group compared with those in control group[PⅢNP (260.2±59.9) vs (328.0±70.3) ng/L, P=0.001, (197.1±46.3) vs (266.7±52.4) ng/L, P<0.001],[BNP (347.4±84.0) vs (430.1±62.9)ng/L, P<0.001, (243.7±79.7) vs (334.6±62.8) ng/L, P<0.001]; ②There were smaller LVEDD and LVESD in spironolactone group compared with those in control group after 6 months intervention[(51.0±5.5) vs (55.6±4.5)mm, P=0.005, (35.7±4.6) vs (39.1±5.6)mm, P=0.046]. However, in inferior MI group: ①There were no significant differences in PⅢNP and BNP values between the two groups after 6 months intervention; ②There were no significant differences in the LVEDD, LVESD, LVEF after 6 months treatment.Conclusion ①In patients with anterior MI, spironolactone combined with the routine treatment could inhibit myocardial fibrosis and left ventricular dilation and prevent LVRM. ②In patients with inferior MI, no significant difference in prevention of LVRM was found between the spironolactone combined with the routine treatment and the routine treatment alone.

目的 探讨急性心肌梗死(AMI)患者应用螺内酯干预对于左室重构(LVRM)的影响。方法 4家医院共入选AMI患者88例,采用多中心、随机、对照的方法,对46例AMI患者在常规治疗的基础上加用螺内酯40mg/d(螺内酯组),对照组(n=42)常规治疗。在6个月干预期内检测两组血清Ⅲ型前胶原氨基端肽(PⅢNP)、脑钠肽(BNP)及超声心动图,以评价左室纤维化、左室功能和左室容积。结果 88例中,急性前壁心肌梗死患者43例,螺内酯组23例、对照组20例;急性下壁心肌梗死患者45例,螺内酯组23例、对照组22例。急性前壁心肌梗死组在治疗3、6个月时螺内酯组与对照组相比,血清PⅢNP和BNP明显降低[PⅢNP分别为( 260 .2±59. 9 )ng/L比( 328 .0±70 .3 )ng/L, P=0 .001, ( 197 .1±46 .3 )ng/L比( 266. 7±52 .4 )ng/L, P<0. 001 ,BNP分别为( 347 .4±84 .0)ng/L比(430 .1±62 .9)ng/L, P<0 .001, (243 .7±79. 7)ng/L比(334. 6±62. 8)ng...

目的 探讨急性心肌梗死(AMI)患者应用螺内酯干预对于左室重构(LVRM)的影响。方法 4家医院共入选AMI患者88例,采用多中心、随机、对照的方法,对46例AMI患者在常规治疗的基础上加用螺内酯40mg/d(螺内酯组),对照组(n=42)常规治疗。在6个月干预期内检测两组血清Ⅲ型前胶原氨基端肽(PⅢNP)、脑钠肽(BNP)及超声心动图,以评价左室纤维化、左室功能和左室容积。结果 88例中,急性前壁心肌梗死患者43例,螺内酯组23例、对照组20例;急性下壁心肌梗死患者45例,螺内酯组23例、对照组22例。急性前壁心肌梗死组在治疗3、6个月时螺内酯组与对照组相比,血清PⅢNP和BNP明显降低[PⅢNP分别为( 260 .2±59. 9 )ng/L比( 328 .0±70 .3 )ng/L, P=0 .001, ( 197 .1±46 .3 )ng/L比( 266. 7±52 .4 )ng/L, P<0. 001 ,BNP分别为( 347 .4±84 .0)ng/L比(430 .1±62 .9)ng/L, P<0 .001, (243 .7±79. 7)ng/L比(334. 6±62. 8)ng/L, P<0. 001]。治疗6个月时螺内酯组较对照组左室舒张末期内径、左室收缩末期内径明显降低[分别为(51. 0±5 .5)mm比(55. 6±4 .5)mm, P=0 .005, (35 .7±4 .6)mm比(39 .1±5 .6)mm, P=0 .046]。急性下壁心肌梗死组在治疗6个月时螺内酯组与对照组相比血清PⅢNP、BNP水平无统计学意义,(P>0 05),并且左

 
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