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   视网膜神经元的 的翻译结果: 查询用时:0.475秒
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视网膜神经元的
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  retinal neurons
     Apoptosis of rabbits′ retinal neurons was investigated by TdT-mediated dUTP-biotin nick-end labeling (TUNEL).
     凋亡原位染色 (Td T- mediated d UTP- biotin nick- end labeling,TUNEL)方法观察家兔视网膜神经元的细胞凋亡。
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     Objective To investigate the expression of induced heat shock protein (HSP) 70 in rat′s retinal neurons (RNs) and Müller cells, and evaluate the protective effect of HSP 70 on RNs injured with glucose deprivation and glutamate.
     目的 评价大鼠视网膜神经元 (RNs)和 Müller细胞中热休克蛋白 (HSP) 70的诱导表达 ,及其对低糖和谷氨酸损伤的视网膜神经元的保护作用。
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     Conclusion The decrease in Bcl-2 expression may lead to the apoptosis of retinal neurons after ON crushing with microforceps.
     结论视神经钳夹伤后视网膜Bcl-2表达的下降,导致了视网膜神经元的凋亡。
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     Objective To investigate the expression of induced heat shock protein (HSP) 27 in rat's retinal neurons (RNs) and Muller cells cultured in vitro, and evaluate the protective effect of HSP27 on RNs injured with glutamate.
     目的:评价体外培养的大鼠视网膜神经元(RNs)和Müller细胞中热休克蛋白27(HSP27)的诱导表达,及其对谷氨酸损伤的视网膜神经元的保护作用。
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     Relation between Müller cells and retinal neurons
     Mlüler细胞与视网膜神经元的关系
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     Conclusion Apoptosis is an import ant mechanism of retinal neuron death i n laser-injured retina,and the increased expression of GFAP by Muller cells is probably related to repair of the injury.
     结论激光损伤诱发了视网膜神经元 的细胞凋亡,伤后Muller细胞GFAP表达增加与损伤修复有关。
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     Photoreceptor necrosis and apoptosis was assessed by the method of HE staining and TUNEL.
     苏木精-伊红(HE)染色和TUNEL法检测观察EPO组及生理盐水组视网膜神经元的存留。
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     Conclusions The reduced neuron expression of NT-3 and the astrocyte proliferation in neuroretina exist in early DR , APP17-mer peptide may clearly improve the neuroretina expression , and is therefore neuroprotective.
     结论 在糖尿病视网膜病变早期存在视网膜神经细胞 NT- 3表达减少及星形胶质细胞增生 ,APP17肽可明显改善早期糖尿病大鼠视网膜神经元的表达 ,提示对神经视网膜具有保护作用
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     HSP27 antibody can reduce the viability of cultured RNs and partial block the protection of heat shock preconditioning against incitant toxic injury by exitotoxicity.
     HSP27抗体可以减低体外培养的视网膜神经元的细胞活力,并可以部分阻断热休克预处理对谷氨酸兴奋性损伤的保护作用。
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     The retinas and optic nerves from the other 2 rats were processed for histopathologic investigation under electronic microscope and light microscope.
     各组其余 2只大鼠取视神经作半薄切片 ,光镜下观察是否有轴索退行性变 ,电镜观察同一眼视网膜神经元的超微结构改变。
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  相似匹配句对
     DEVELOPMENT OF NITRIC OXIDE SYNTHASE POSITIVE NEURONS IN TRANSPLANTED RETINA
     移植视网膜NOS阳性神经元的发育
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     The Study of Parvalbumin Immunoreactive Neurons in the Retina of Rats
     大鼠视网膜Parvalbumin免疫反应神经元的研究
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     Retinal Vasculitis
     视网膜血管炎
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     Retinal Detachment
     视网膜脱离
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     Mechanism of Neuronal Migration.
     神经元的迁移机制
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  retinal neurons
1 immunostaining is expressed at many parts of Müller cells, especially in the membrane domains of Müller cells that contact retinal neurons, i.
      
The association of NeuroD predominantly with postmitotic precursors in early as well as late neurogenesis suggests that NeuroD expression plays an important role in the terminal differentiation of retinal neurons.
      
On the contrary, FGFR4 mRNA could not be detected in primary cultures of retinal Müller glia or pigment epithelium, indicating specific enrichment in retinal neurons.
      
However, teleost retinal neurons are post-mitotic and do not further divide under normal situation.
      
However, the position of the affected cell, side by side with retinal neurons, suggests that the glycogen deposits were within Müller cells.
      
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The discovery of S-potentials was the prelude to the intracellular study of retinal neurons.Data from dye-injection experiments show that S-potentials aregenerated by horizontal cells.The present review deals with recent advancesin this topic,with particular reference to the studies of receptor inputs to varioustypes of horizontal cells and interactions among them.The role of horizontalcells in retinal information processing is discussed as well.

S-电位的发现揭开了视网膜神经元细胞内研究的序幕。细胞内染色技术已证明 S-电位起源于水平细胞。本文评述这一领域的主要进展,特别是结合外网状层神经元回路的研究,讨论对各种水平细胞的感受细胞输入及其相互作用。也将论及水平细胞在视信息处理过程中的作用。

The uptake and the realease of 3 H-glycine and 3H-aspartic acid in 22 specimens of fresh human embryonic retina were examined by in vitro experimental methods of a radioisotope tracing and an autoradiography so as to understand the emerging time for functional differentiation of neurons of human embryonic retina.

本文应用同位素示踪和放射自显影技术,观察了新鲜人胚胎22例视网膜神经元体外摄取和释放~3H-甘氨酸和~3H-天门冬氨酸功能的出现。结果表明,14~15周人胚胎视网膜已具有摄取和K~+刺激、Ca~(++)依赖性释放外源性~3H-甘氨酸和~3H-天门冬氨酸的能力。摄取和~3H-甘氨酸的细胞是视网膜内核层最深层的细胞,摄取和蓄积~3H-天门冬氨酸的细胞是内核层中部和外核层的部份细胞,根据细胞形态和所在位置,我们认为这些细胞分别是发育中的无长突细胞,双极细胞和视锥细胞。此外,在~3H-天门冬氨酸的放射自显影图像中,可见视网膜的外核层、内网层和神经纤维层中有条索状银粒分布,这可能是Müller细胞被标记的结果。

The distribution and changing in the development of two kinds of calcium binding proteins, parvalbumin (PV) and calbindin-D28K (CaBP) in retina from 19 cases of developinghuman fetus were investigated immunocytochepeically. The result showed that the subpopulations of amacrine, horizontal, and gahglion cells were PV and CaBP positive. CaBP mightalso locate in photoreceptors. PV-immunoreactive (PV-Ⅰ) neurons and CaBP-immunoreactive (CaBP-Ⅰ) neurons chiefly developed in mid gestation period. In 14-week fetus,...

The distribution and changing in the development of two kinds of calcium binding proteins, parvalbumin (PV) and calbindin-D28K (CaBP) in retina from 19 cases of developinghuman fetus were investigated immunocytochepeically. The result showed that the subpopulations of amacrine, horizontal, and gahglion cells were PV and CaBP positive. CaBP mightalso locate in photoreceptors. PV-immunoreactive (PV-Ⅰ) neurons and CaBP-immunoreactive (CaBP-Ⅰ) neurons chiefly developed in mid gestation period. In 14-week fetus, PV-Ⅰneurons and CaBP-Ⅰ neurons appeared in different regions of the retina, each having theirowe developing mule. PV-Ⅰneurons were identified in ganglion cell layer (GCL) and nervefiber layer (NFL), while CaBP-Ⅰneurons were mainly observed in neuroblast layer(NBL). The distinct distribution pattern of both PV-Ⅰ and CaBP-Ⅰ neurons were basicallyestoblished in 27-week fetus. The most inprotant fidning in this study was that the densityof PV-Ⅰ neurons and. intensity of PV immuno-stain in temporal retina were definitely higherthan that in nasal retina during development. We also found that the development of PV-Ⅰand CaBP-Ⅰ neurons was closely related to the cell migration, laminar differentiation, andformation of synaptic connection of the latinal neurons. these results suggest that PV andCaBP may play important roles in the development of retinal structure.

本实验采用免疫细胞化学方法观察19例人胎视网膜内Parvalbumin(PV)和Calbi-din-D28k(CaBP)免疫反应神经元的分布和发育,对它们在人胎视网膜发育中的演变规律进行了研究。结果显示,PV和CaBP免疫反应神经元属于视网膜水平细胞,无长实细胞和节细胞的不同亚群,CaBP免疫反应神经元还可能分属于视细胞。PV和CaBP免疫反应神经元的发育主要是在胚胎中期,胎14周时它们已分别出现于视网膜内的不同部位,其各自的演变规律不同,至胚胎27周时初步建立了各自的分布模式。在整个发育过程,颞测机网膜内PV免疫反应神经元的密度及反应强度均高于鼻侧视网膜内的PV免疫反应神经元,PV和CaBP免疫反应神经元的发育与视网膜的组织发生也有着密切的联系。本文结果提示,PV和CaBP可能在视网膜神经元的分化、迁移及突触形成中分别起着重要的作用。

 
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