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角膜血管形成
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  cornea neovascularization
     The rat cornea neovascularization was used to evaluate the effects of HGF/SF and VEGF on angiogenesis in vivo .
     用大鼠角膜血管形成实验检测 HGF/ SF和 VEGF对大鼠体内血管形成的影响。
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  “角膜血管形成”译为未确定词的双语例句
     Results The rates of angiogenesis in the corneas induced by implantation of carcinoma tissue were 12.2% in the treatment eyes, and 91.8% in the control eyes (P< 0.01) .
     结果实验组兔角膜血管形成率为12.2%,对照组为91.8%,两组比较差异有极显著性(P<0.01)。
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     Additionally, recent studies in vitro have demonstrated that adiponectin inhibits endothelial cell proliferation and migration, in the chick chorioallantoic membrane and the mouse corneal angiogenesis assays, adipoenectin remarkably prevents new blood vessel growth, and adiponectin serves as a negative regulator of angiogenesis.
     有体外实验研究表明,脂联素抑制血管内皮细胞的增殖和迁移,通过鸡绒毛膜尿囊膜和小鼠角膜血管形成的体内实验发现,脂联素显著抑制新生血管的形成,因此,研究认为脂联素是血管形成的负调节因子。 另有研究发现,脂联素刺激体外培养的人脐静脉内皮细胞分化成毛细管状结构,在体内刺激新生血管形成,因此,研究认为脂联素是血管形成的正调节因子。
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     The correlation between the grade of angiogenesis induced by carcinoma tissues and the clinical stage of carcinoma were analyzed.
     观察两组角膜血管形成情况,以及实验组MPA对孕激素受体阳性/阴性癌组织诱导血管形成的抑制作用。
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  相似匹配句对
     Vascularization regulation factor and corneal neovascularization
     角膜新生血管形成机制研究进展
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     A morphological study of experimental corneal neovascularization
     实验性角膜新生血管形成的形态学研究
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     Apoptosis in cornea
     角膜细胞凋亡
短句来源
     Progress in preservation of cornea
     角膜保存的进展
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     ⑤the formation of new-born blood vessel.
     ⑤新生血管形成
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  cornea neovascularization
Evaluation of antiangiogenic effects of human angiostatin in the cornea neovascularization assay.
      


bjectives To investigate if the

目的探讨妇科恶性肿瘤组织诱导兔角膜血管形成分级可否作为判断患者预后的一项监测指标,并对妇科三种恶性肿瘤之间的生物学行为进行评价。方法分析11例卵巢恶性肿瘤,10例乳腺癌,9例子宫内膜癌诱导兔角膜血管形成分级与临床预后因素之间的关系,并对三种恶性肿瘤诱导兔角膜血管生成活性进行比较。结果Ⅱ级血管形成在临床Ⅱ~Ⅳ期,淋巴结阳性或有转移灶的病例明显高于临床Ⅰ期,淋巴结阴性或无转移灶的病例,差异有极显著性(P<0.01)。血管形成分级与组织学分级及肿瘤直径之间差异无显著性(P>0.05)。血管形成活性卵巢恶性肿瘤为“+++”,乳腺癌和子宫内膜癌分别为“++”。结论瘤组织诱导兔角膜血管形成分级可作为判断肿瘤患者预后指标之一。血管形成活性在这三种恶性肿瘤排列顺序与他们的生物学行为相一致,其预后卵巢恶性肿瘤最差。

Objective To investigate the effects of HGF/SF and VEGF on vascular endothelial cells proliferation and capillary sprouts formation. Methods Endothelial cells proliferation test and capillary sprouts formation test were used to evaluate the effects of HGF/SF and VEGF on human umbilical artery endothelial cells (HUAECs) proliferation and capillary sprouts formation. ELISA was used to measure the contents of HGF/SF and VEGF in the media. The rat cornea neovascularization was used to evaluate the effects of...

Objective To investigate the effects of HGF/SF and VEGF on vascular endothelial cells proliferation and capillary sprouts formation. Methods Endothelial cells proliferation test and capillary sprouts formation test were used to evaluate the effects of HGF/SF and VEGF on human umbilical artery endothelial cells (HUAECs) proliferation and capillary sprouts formation. ELISA was used to measure the contents of HGF/SF and VEGF in the media. The rat cornea neovascularization was used to evaluate the effects of HGF/SF and VEGF on angiogenesis in vivo . Results HGF/SF and VEGF had obvious effects on angiogenesis and endothelial cells proliferation and capillary sprouts formation. The VEGF was found in HGF/SF media. Conclusion The combination use of HGF/SF and VEGF has cooperative effects on angiogenesis in tumor, HGF/SF may have the effect to stimulate vascular endothelial cells to produce VEGF.

目的 :探讨肝细胞生长因子 /扩散因子 (HGF/ SF)和血管内皮细胞生长因子 (VEGF)联合应用对肿瘤血管再生的影响。方法 :用血管内皮细胞增殖和毛细血管形成实验检测 HGF/ SF和 VEGF对培养皿中人脐动脉血管内皮细胞 (HUAECs)增殖和血管形成的影响。采用酶联免疫吸附测定 (EL ISA)法检测培养基中 HGF/ SF和 VEGF的含量 ,检测这两种细胞因子是否相互影响。用大鼠角膜血管形成实验检测 HGF/ SF和 VEGF对大鼠体内血管形成的影响。结果 :HGF/ SF和 VEGF对血管内皮细胞增殖和毛细血管形成均有明显的促进作用 ,并且含 HGF/ SF的培养基中检测到 VEGF的存在。结论 :HGF/ SF和 VEGF的联合应用对肿瘤血管形成有协同作用 ,HGF/ SF可能具有刺激血管内皮细胞产生 VEGF的作用

Objective To explore the inhibitoion of medroxyprogesterone acetate (MPA) on angiogenesis induced by carcinoma tissue of progestin receptor (PR) positive or negative. Method Thirty two tumor tissues were obtained from operative specimens in patients with ovarian or endometrial carcinoma. Within three hours each specimen was implanted into the six corneas of three rabbits. The right eyes were the control (received no treatment) compared to the left eyes treated with MPA. The angiogenesis rate and the grade of...

Objective To explore the inhibitoion of medroxyprogesterone acetate (MPA) on angiogenesis induced by carcinoma tissue of progestin receptor (PR) positive or negative. Method Thirty two tumor tissues were obtained from operative specimens in patients with ovarian or endometrial carcinoma. Within three hours each specimen was implanted into the six corneas of three rabbits. The right eyes were the control (received no treatment) compared to the left eyes treated with MPA. The angiogenesis rate and the grade of angiogencsis in the corneas of the rabbits were examined. The correlation between the grade of angiogenesis induced by carcinoma tissues and the clinical stage of carcinoma were analyzed. And in the treatment eyes, the effects of MPA on PR positive or negative in carcinoma tissue were compared. Results The rates of angiogenesis in the corneas induced by implantation of carcinoma tissue were 12.2% in the treatment eyes, and 91.8% in the control eyes (P< 0.01) . There was a significant difference between them. In the treatment eyes, the inhibitory rate by MPA to angiogenesis induced by PR positive or negative in carcinoma tissue were 84.5% and 93.7% (P>0. 05). In the control eyes a positive correlation was found between clinical stage of the carcinoma and the angiogenesis, and there was significant difference (P< 0.01) . The later stage of the carcinoma, the more of grade Ⅱ angiogenesis. Conclusion MPA has inhibitory effect on neovascularization induced by carcinoma tissues whether PR positive or negative. The higher the clinical stage, the higher grade of angiogenesis.

目的探讨孕激素甲羟孕酮(MPA)对孕激素受体阳性/阴性癌组织诱导血管形成的抑制作用。方法将手术获取的32例妇科癌组织种植于兔角膜。每例癌组织3h内种植于3只兔的6只角膜,设定所有兔的右眼为对照组,左眼为实验组。在种植癌组织之前,实验组角膜放入MPA,对照组角膜不给予任何药物。观察两组角膜血管形成情况,以及实验组MPA对孕激素受体阳性/阴性癌组织诱导血管形成的抑制作用。考察对照组癌组织诱导血管形成分级与临床分期的关系。结果实验组兔角膜血管形成率为12.2%,对照组为91.8%,两组比较差异有极显著性(P<0.01)。实验组中MPA对孕激素受体阳性和阴性癌组织诱导血管形成的抑制率分别为84.5%和93.7%,差异无显著性。对照组中,Ⅱ级血管形成与临床分期间存在正相关,且差异有极显著性(P<0.01),即临床肿瘤组织分期越晚,角膜形成Ⅱ级血管越多。结论MPA对孕激素受体阳性/阴性癌组织诱导血管形成有明显抑制作用。MPA抗肿瘤血管生成作用与孕激素受体无关。且癌组织临床分期越晚,血管形成分级越高。

 
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