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病理效应
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  pathological effects
     Receptors mediating the action of CRH have been identified as CRH-R1,CRH-R2 and CRH-R3.In the process of stress reaction,CRH mainly interacts with CRH-R1 and CRH-R2,producing multiple physiological and pathological effects.
     目前已知的CRH受体有CRH-R1、CRH-R2、CRH-R3 3种。 应激时,CRH主要通过CRH-R1、CRH-R2产生一系列生理、病理效应
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     Stressful pathological effects of hippocamal in chronic restraint stress mice
     慢性束缚应激小鼠海马应激性病理效应
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     TH has physiological and pathological effects on the developing and developed brain through regulating its response genes expression.
     甲状腺激素不仅影响神经系统的发育及功能成熟,对成熟脑组织亦有重要作用,它主要通过调节相应的基因表达来实现其生理或病理效应
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     Here the diverse physiological functions and pathological effects of HMGB1 are reviewed.
     本文拟就高迁移率族蛋白B1的基本特征及其广泛的生理作用与病理效应做一综述。
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  “病理效应”译为未确定词的双语例句
     Anti-RAGE antibody and sRAGE are generally accepted blocking reagents.
     因此,利用抗RAGE抗体或sRAGE阻断RAGE介导的病理效应,将会为这些疾病的治疗提供新方法。
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     Based on HSV-tk/NAS gene therapy for brain tumor,the system for detection of replication-competent adenovirus(RCA) was studied. It included PCR for E1 region of adenovirus and cytopathological effect observation. The sensitivity of PCR is denoted by the copies of E1 region in one 293 cell.
     在腺病毒介导的HSV-tk/NAS系统脑肿瘤基因治疗研究过程中,建立了野生型腺病毒(RCA)的检测系统,主要包括腺病毒E1区的PCR以及野生型病毒的细胞病理效应观察这两种方法,PCR的灵敏度为1个293细胞所含有的E1片段数。
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     Methods: Endostatin gene was cloned by RT PCR and then the replication competent (CNHK200 hE) or replication deficient (Ad hE) adenovirus carrying endostatin gene were reconstructed in 293 cells. Their cytopathic effect and endostatin expression were compared when they were used to infect HT 29 cells.
     方法 :通过 RT- PCR克隆内皮抑素基因 ,2 93细胞内分别重组携带内皮抑素基因的增殖型 (CNHK2 0 0 - h E)和增殖缺陷型 (Ad- h E)腺病毒 ,分别体外感染结肠癌细胞 HT- 2 9后比较两者在细胞病理效应、病毒增殖和内皮抑素表达量方面的差异。
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     ATR-AA is one important factor in the pathogenesis of hypertension and losartan can block this pathological effect.
     ATR-AA是参与抗体阳性难治性高血压的发病的因素之一,氯沙坦可阻断其病理效应
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     DATA SYNTHESIS:NO influenced the physical activities of organism at different concentration by different way.
     资料综合:由于一氧化氮在机体中浓度的不同积累,通过不同的作用方式,发挥着生理、病理效应,影响着机体的生理活动。
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     Pathophysiological effect of excess iodine on the thyroid
     碘过剩对甲状腺的病理生理效应
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     Pathological examination proved that E.
     病理证实E .
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     Gravitational Lens Effect
     引力透镜效应
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     Stressful pathological effects of hippocamal in chronic restraint stress mice
     慢性束缚应激小鼠海马应激性病理效应
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     ‘DaVinci Effect’ in the Electronics Industry
     “达芬奇”效应
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  pathological effects
Although some alterations were common to all three, they each possessed distinctive pathological effects.
      
Pathological effects of the parasite Malameba locustae in males of the migratory grasshopper Melanoplus sanguinipes and its inte
      
Propagation of tomato spotted wilt virus in Frankliniella occidentalis does neither result in pathological effects nor in transo
      
The advanced knowledge of the stereochemistry permits to explain the relationships between structure, function and stability in normal and abnormal hemoglobin, as well as the pathological effects of mutations on the integrity of the red cell.
      
These effects are enhanced following intermittent exposure to high glucose, indicating that short lived excursions in glycaemic control have important pathological effects on the human tubulointerstitium.
      
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The effect of high dos; dexamethasone(5mg/kg wt, intravenous injection)to prevent and treat the secondary pathological damage of craniocerebral injury was studied in animal models of craniocerebral injury caused by high-velocity missiles in dogs.We observed ths physiological changes, analyzed the value of serum and. cerebrospinal fluid lactate dehydrogenase, estimated the permeability of blood brain barrier (BBB) and studied brain pathology by light and electron microscopy.The results suggest high dose dexamethasone...

The effect of high dos; dexamethasone(5mg/kg wt, intravenous injection)to prevent and treat the secondary pathological damage of craniocerebral injury was studied in animal models of craniocerebral injury caused by high-velocity missiles in dogs.We observed ths physiological changes, analyzed the value of serum and. cerebrospinal fluid lactate dehydrogenase, estimated the permeability of blood brain barrier (BBB) and studied brain pathology by light and electron microscopy.The results suggest high dose dexamethasone can help to restore ths structure and function of BBB, protect brain cells as well as lessai the secondary pathological damage in tha respiratory and circulatory systems and reduce th production of lipoptrox-ides(LPO).

本实验用犬制作高速弹颅脑火器伤模型,研究了大剂量地塞米松(5mg/kg,致伤后5min静脉注射在防治高速弹颅脑火器伤继发病理损害中的作用。通过生理指标、血和脑脊液酶学、血脑屏障通透性定量指标对比和距伤道的不同距离的光镜和电镜观察研究,证明应用大剂量地塞米松能在一定程度上保护和恢复血脑屏障的结构和功能;保护脑细胞的结构;减轻呼吸和循环系统的继发病理效应,并可减少脑的脂质过氧化物产生等。

It is known that incontrollahle inflammation and activated complement are implicated in the pathogehesis of multiple organ failure.Recently,the role of gut originated infection resulting from the combined action of bacterial translocation due to the functional failure of gut mu-cosal barrier and endotoxin in the pathogenesis of multiple organ failure receives increasing attention.In order to clarify the relationship between incontrollable inflammation and gut-originated infection,zymosan,an activation agent...

It is known that incontrollahle inflammation and activated complement are implicated in the pathogehesis of multiple organ failure.Recently,the role of gut originated infection resulting from the combined action of bacterial translocation due to the functional failure of gut mu-cosal barrier and endotoxin in the pathogenesis of multiple organ failure receives increasing attention.In order to clarify the relationship between incontrollable inflammation and gut-originated infection,zymosan,an activation agent for complement and inflammatory response,was injected to normally nourished mice (NNM) and protein deficient malnourished mice (MNM).It was found that a sublethal dosage of zymosan (0.1mg/kg) could damage the gut mucosal barrier to result in bacterial invasion,which was limited to the mesenteric lymph nodes in MNM.Protein-deficient malnutrition enhanced the pathological effect of zymosan to result in a bacterial spreading to the liver,the spleen and blood stream and a potentially lethal gut originated infection.The severity of infection and the mortality rate of the animals increased with the severity of malnitrition.On the basis of the findings it is belived that there is a synergic pathological effect of incontrollable inflammation and gut-originated infection especially in protein-deficient conditions.

失控的炎性反应和补体活化参与多脏器功能衰竭的发病过程。而因肠粘膜屏障功能衰竭造成的大量肠道细菌和内毒素侵入体内协同作用形成的肠源性感染在多脏器功能衰竭发病中所起的作用又日益受到关注。本研究给动物注射一种补体活化和炎性反应的活化因子——酵母多糖,观察炎症和感染两者间的内在关联性。结果发现,亚致死量的酵母多糖(0.1mg/g)可损伤肠粘膜屏障而导致肠道细菌侵入体内。而蛋白质营养不良却显著强化了酵母多糖的上述病理效应,使动物形成致死性的全身性肠源性感染,感染的严重性和动物的病死率随营养不良的程度进行性增高。结果表明,在体内失控的炎性反应和肠源性感染有协同致病效应。

Replication of spodoptera litura nuclear polyhedrosis virus in spodoptera littoralis cell line (Semipermissive cell) was studied. After infected by slNPV cells produced limited cytothic effect. Surface of infected cells was rough and infected cells exhibited hypertrophy. 120hr postinfection electron dense virogenic stroma and nucleocapsides were observed in nucleus of infected cells by electron microscope. Neither enveloped virus particles nor PIBS were observed at any time during the slNPV infection of S. Li...

Replication of spodoptera litura nuclear polyhedrosis virus in spodoptera littoralis cell line (Semipermissive cell) was studied. After infected by slNPV cells produced limited cytothic effect. Surface of infected cells was rough and infected cells exhibited hypertrophy. 120hr postinfection electron dense virogenic stroma and nucleocapsides were observed in nucleus of infected cells by electron microscope. Neither enveloped virus particles nor PIBS were observed at any time during the slNPV infection of S. Li cells. S. Li cells could not support slNPV comlet replication. Replication of slNPV in S. Li cell was restricted. The restriction perhaps occured at virion assambly stage, slNPV could normaly replicate in its homologous spodoptera litura cell line (sL-1).

斜纹夜蛾NPV感染半受纳细胞——莲纹夜蛾细胞后,细胞产生一定程度的病理效应,受染细胞表面粗糙,细胞核肿胀膨大。超薄切片电镜观察显示受染细胞核中形成电子致密的病毒发生基质与大量分布在病毒发生基质中间及周围的核衣壳,但并没观察到具囊膜的、成熟的病毒粒子和多角体。莲纹夜蛾细胞不能提供斜纹夜蛾NPV完全复制,病毒在半受纳细胞中的复制与增殖受到限制,限制可能发生在病毒粒子装配阶段。而斜纹夜蛾NPV能在其同源细胞中正常复制。

 
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