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   组织超微结构 的翻译结果: 查询用时:0.023秒
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组织超微结构
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  tissue ultrastructure
     Related indexes were detected at 6 h after operation in normal group, the changes in cerebral tissue ultrastructure were observed under electron microscope at 6, 24 h and 3, 7 d in the other 3 groups.
     除正常组于术后6h检测相关指标外,其余3组分别于术后6、24h、3、7d(均n=8)电镜观察脑组织超微结构改变。
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     The changes of lung tissue ultrastructure were observed by electron microscope. The changes of the expression of CD31,CD61 and CD62p on platelet membrane were observed by flow cytometry.
     应用电镜和流式细胞术,分别观察大鼠肺组织超微结构以及血小板膜糖蛋白CD31、CD61和CD62p表达的变化。
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     Influence of Nao Xue Kang Tablet on Brain Tissue Ultrastructure in Acute Intracterebral Hemorrhage Rat
     脑血康对急性脑出血大鼠脑组织超微结构的影响
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     At 3 days,brain tissue ultrastructure in the NXKTP was much more improved than the ICH model group,and the effect was better than 24 hours. At 7 days,the NXKTP showed much more improved brain tissue ultrastructure than the ICH model group.
     脑血康6h组较同期模型组大鼠脑组织超微结构有较明显改善,24h治疗组与同期模型组对比有较好治疗效果,3d治疗组较同期的模型组脑组织超微结构有进一步改善,并优于24h治疗组,7d治疗组与同期模型组相比有明显改善,且优于3d治疗组,但仍未能恢复如正常组的超微结构状态。
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     Influence of ChuanAn on pulmonary tissue ultrastructure in asth marat
     喘安对哮喘大鼠肺组织超微结构的影响
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  “组织超微结构”译为未确定词的双语例句
     After DHCA for 90 min,the ATP content in cerebral tissue in RSA group (2.02± 0.19) μmol/g was significantly higher than that in SVC group [(1.72± 0.21) μmol/g, P< 0.05].
     DHCA90min后,RSA组脑组织超微结构缺血、缺氧改变轻于SVC组。 且RSA组脑组织ATP含量[(2.02±0.19)μmol/g]显著高于SVC组[(1.72±0.21)μmol/g,P<0.05]。
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     Expression of Bax, Bcl-2, p53 and ultrastructural change in lung after small intestine ischemia-reperfusion
     小肠缺血再灌注后肺Bax,Bcl-2,p53的表达和组织超微结构改变(英文)
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     Results (1) Ultrastructural change of lung in MP1 group and MP2 group were slighter than that in the model group at 120 min, and the change in MP2 group was slighter than that in MP1 group.
     结果(1)MP1组与MP2组肺组织超微结构变化均较模型组轻。 MP2组120min时肺组织超微结构变化较MP1组轻。
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     The ratsin group B and C were intraperioneally treatedwith LPS (1 mL/kg) and LPS (1 mL/kg) plus rITF(5 g/L, 0.1 mL), respectively. Intestinal tissueswere collected at 2, 6, 24, and 72 h for the exami-nation of NO, MDA productions, iNOS mRNA expression and intestinal histological changes.
     以生理盐水(1mL/kg),EcoliO55:B5(1mL/kg)、5g/LrITF(0.1mL)ip后2,6,24,72h处死动物,留取肠组织生化法检测NO,MDA含量,PCR法检测诱导型一氧化氮合酶(iNOS)mRNA表达,同时作电镜观测肠组织超微结构变化.
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     Effect of CS 2 on the ultrastructure of nerve tissue and calcium distribution in rats
     CS_2 对大鼠神经组织超微结构及钙分布的影响
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  相似匹配句对
     Small testis tissue ultrastructure changed significantly.
     组织超微结构变化显著。
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     Organization
     组织
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     Ultrastructure changes of electrical injury in rats
     电击伤组织超微结构观察
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     Organising Committee
     组织委员会
     Study on Ultrastructure of Benign and Malignant Pheochromocytoma
     嗜铬细胞瘤的超微结构
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  tissue ultrastructure
A similar conclusion was reached from electron microscopic study of the tissue ultrastructure.
      
Glycine also drastically decreases changes in the tissue ultrastructure, particularly of mitochondria, arising under anoxia.
      
There were no observable changes in the tissue ultrastructure of liver and kidney as examined by electron microscopy in any of the treatment groups.
      
There were no observable changes in the tissue ultrastructure as examined by electron microscopy for both the adults and the kids from either ration treatment.
      
Changes in gill tissue ultrastructure included an increase in number of vesicles and a decrease in number and size of mitochondria in nymphs exposed to alkaline pH of 11.75.
      
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Pyquiton, namely praziquantel or Embay 8440, is a new kind 6f antischistoso-mal drugs. It has been demonstrated that pyquiton displays the advantages of lower toxicity, shorter course of treatment and higher efficacy in the treatment of schistosomiasis japonica in both experimental animals and human cases.The present paper reports the histopathological damages of Schistosoma japo-nicum and host liver caused by pyquiton therapy.The resulte are summarized as follows:1.When infected mice were treated with pyquiton...

Pyquiton, namely praziquantel or Embay 8440, is a new kind 6f antischistoso-mal drugs. It has been demonstrated that pyquiton displays the advantages of lower toxicity, shorter course of treatment and higher efficacy in the treatment of schistosomiasis japonica in both experimental animals and human cases.The present paper reports the histopathological damages of Schistosoma japo-nicum and host liver caused by pyquiton therapy.The resulte are summarized as follows:1.When infected mice were treated with pyquiton at a single oral dose of 300 to 500mg/kg, numerous dead worms were odserved in the liver sections as early as 24 hours after the medication under the optical microscopic examination. The histological changes consisted mainly of degeneration and necrosis of the integument and intestinal epithelial cells of the worms. Besides numerous inflammatory cells invading the parenchyma of the worms via integument were also detected.The ultrastructural observations on the integument of worms perfused out from infected mice 24 hours after treatment with pyquiton at a single oral dose of 500 mg/kg, were made by transmission electron microscopy.Main lesions of the integument indued expansion and rupture of the cytoplasmic process, emergence of large vacuoles containing residual sudstance, ovoid bodies and myelin-like structures change of touch junction of the plasmic membrane, formation of bridge-like structures of cytoplasmic process, decrease in rod-like, discoid and spherical secretory bodies as will as appearance of numerous vesicles in the latter.2.Histochemical observations showed that in schistosomes maintained in culture medium containing 3-10 μg/ml of pyquiton for 6 to 48 hours, or worms recovered from mice treated with the drug at a single oral dose of 300mg/kg, the contents of glycogen, AKP and RNA were markedly decreased or even disappeared. On the contrary, ACP and alkalin protein reactions were stronger than those in the control. No apparent chasges were seen in the content and distribution of DNA, tyrosine, tryptophan, histidine, protein bound α-amino groups and SH groups, and phenolase as compared with the controls.3. After administration of pyquiton to the mice and rabbits infected with Schisto-soma japonicum at a single oral dose of 300 mg/kg and 60 mg/kg, respectively, no apparent histopathological lesions were observed in their lives except for the fact that a few focal areas of coagulation necroses could be seen occasionally in the mouse livers 1 to 3 days after the medication.Meanwhile, the hepatic cell division phase was somewhat increased.lt could return to normal 1 week later. 3-4 weeks after the treatment, a few surviving female worms resumed oviposition, but the cellular reaction around the living eggs was rather mild; the fibrosis of the liver containing dead or degenerated eggs was markedly decreased or completely resolved. It is worthy to note that 4-8 weeks after the termination of treatment, a few Iw bloody cysts ( 5-7mm in diameter ) containing male worms which were still living but encapsulated by connective tissue, had been observed along the liver margin of some rabbits.

实验证明,感染血吸虫的小鼠一次口服吡喹酮300~500mg/kg24小时后,肝内即出现很多死虫,其体表及肠管上皮变性坏死,并有炎细胞侵入虫体。经吡喹酮作用后,两性血吸虫体表组织超微结构的变化主要为:细胞质突起膨大、破裂和形成桥样结构;细胞质内出现含有残余体样及髓鞘样结构的巨大空泡或卵园形小体;杆状、盘状及球状分泌体明显减少,特别是球状分泌体内形成很多空泡,以及体表浆膜有紧密连结变化。体内、体外试验结果表明,经吡喹酮作用后,两性血吸虫体内糖原,碱性磷酸酶及RNA均明显减少或消失;酸性磷酸酶及碱性蛋白质均显著增加,而DNA、酪氨酸、色氨酸、组氨酸,结合α-氨基蛋白质、巯基及酚酶等组成,则无明显变化。 感染血吸虫的小鼠及家兔,于一次口服吡喹酮各为300及60mg/kg后,肉眼观察时,只在少数鼠肝内见有少量的点状凝固性坏死灶,并未见有斑块坏死病变。服药3~4周后,残留的雌虫恢复产卵,但活卵周围的细胞反应很轻,同时,死亡虫卵和变性虫卵周围的纤维结缔组织亦明显减少或消散。此外,服药4~8周后少数兔肝边缘查见血性囊泡,内含被结缔组织包围的活的雄虫。

1-4°C potassium-magnesium cardioplegic solution was perfused for myocardial pro-

本文报道临床应用钾镁能量心脏停跳液结合降温保护心肌法80例。从心脏复跳、复苏后升压药及强心剂的应用等方面分析,本法优于局部冰屑深低温法及局部深低温加主动脉根部灌注冰平衡液法。对其中4例心肌组织超微结构的观察,显示主动脉血流阻断75~100分钟后,心肌结构与阻断血流前基本相似。认为本法能使心肌组织得到良好的保护。

The myocardium of 20 chronic heart fa(?)ure (CHF) rabbits caused by adriamycin was studied by electron microscopy. The main morphological alterations of CHF heart were as follows: changes in biological membrane, including seperation of intercalated disks, degeneration of mitochondria, dilatation of sarcoplasmic reticulum; atrophy, disruption and lysis of myofibrils; edema of cardiomycytes and interstitial fibrosis.Compensated and decompensated CHF were classified according to the general manifestations of the...

The myocardium of 20 chronic heart fa(?)ure (CHF) rabbits caused by adriamycin was studied by electron microscopy. The main morphological alterations of CHF heart were as follows: changes in biological membrane, including seperation of intercalated disks, degeneration of mitochondria, dilatation of sarcoplasmic reticulum; atrophy, disruption and lysis of myofibrils; edema of cardiomycytes and interstitial fibrosis.Compensated and decompensated CHF were classified according to the general manifestations of the rabbits. The main morphologic differences of myocardium between them were that in the former, the lesions were focal and mitochondriosis was appeared, in the later, the lesions were more diffuse and severe. A close relationship bettween morphologic alterations and heart function was observed.

本文用电镜观察实验性心力衰竭时心肌组织的超微结构改变,分析了心功能代偿与失代偿阶段病理形态的差异,认为心功能状态与心肌受损的程度和范围有关。

 
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