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诱导肿瘤
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  inducing tumor
     Study on the Mechanism of Inducing Tumor Cell Differentiation by N-Acetyl-1, 6-diaminohexane Using Liquid Chromatography
     用液相色谱法研究N-乙酰-1,6-二氨基己烷诱导肿瘤细胞分化的机理
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     Objective: To investigate the expression of TNF-relat-ed apoptosis inducing ligand (TRAIL)、DCR2/TRAILR4、DR4/TRAILR1、Caspase-3 and NF-κB in gastric carcinomasurrounding tissues and normal stomachic tissues, and to probe into the possible mechanism of TRAIL inducing tumor cells’apoptosis but not in normal stomachic tissu-es 。
     目的:检测人肿瘤坏死因子相关凋亡诱导配体( TNF related apoptosis inducing ligand,TRAIL)及其受体DcR1 和DR4、以及Caspase-3 、NF-κB 在胃癌、癌旁组织及正常组织中的表达,分析表达差异可能的原因,探讨1.TRAIL 诱导肿瘤细胞凋亡以及正常细胞逃避损伤的机制;
短句来源
     Cloning, expression and purification of human interleukin 24 gene and study on its activity of inducing tumor cells apoptosis in vitro
     人IL-24基因的克隆、表达、纯化及体外诱导肿瘤细胞凋亡的研究
短句来源
     Conclusion SRBD has anti-tumor effect on the growth of bearing cancer mice S_(180) cell, the mechanism may be related with inducing tumor cell apoptosis and inhibiting the proliferation cycle of tumor cells.
     结论SRBD对小鼠肉瘤S180的生长具有抑制作用,其机制可能与诱导肿瘤细胞凋亡及抑制癌细胞增殖周期有关。
短句来源
     and (3)inducing tumor cell differentiation.
     (3)诱导肿瘤细胞分化。
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  “诱导肿瘤”译为未确定词的双语例句
     The Construction of Expression Vector of Fas/RAR Alpha Fusion Gene Containing Retinoic Acid Response Elemennt, and Its Effect Induced by Retinoic Acid on the Apoptosis of Tumor Cells
     视黄酸依赖Fas/RARα融合基因表达载体构建及其诱导肿瘤细胞凋亡的研究
短句来源
     Studies on the Mechanisms of Shikonin-induced Tumor Cell Apoptosis
     紫草素诱导肿瘤细胞凋亡作用机制的研究
短句来源
     Molecular Mechanism of Tripchlorolide (TC) Induced Apoptosis of Tumor Cells
     雷公藤氯内酯醇诱导肿瘤细胞凋亡的分子机制探讨
短句来源
     Molecular Mechanisms and Signaling Transduction Pathways of Cell Cycle Arrest Induced by Lidamycin in Human Cancer Cells
     力达霉素诱导肿瘤细胞周期阻滞的分子机制及其信号传导通路研究
短句来源
     Studies on the Mechanisms of Norcantharidin-induced Tumor Cell Apoptosis
     去甲斑蝥素诱导肿瘤细胞凋亡的机制研究
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  相似匹配句对
     and (3)inducing tumor cell differentiation.
     (3)诱导肿瘤细胞分化。
短句来源
     (4) to induce tumor cells differentiation;
     (4)诱导肿瘤细胞分化;
短句来源
     Tumor lymphangiogenesis
     肿瘤淋巴管生成
短句来源
     malignant tumor group:I.
     肿瘤组 :I.
短句来源
     in troducing stimulus;
     诱导刺激;
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  inducing tumor
The manners of VP3 proteinin vivo, inducing tumor cell death were identified by using TUNEL assay.
      
Autologous dendritic cells (DCs) loaded with tumor antigens are particularly potent at inducing tumor antigen-specific immune responses.
      
Considering that tumor cell vaccines are aimed at inducing tumor-specific immune responses, our in vitro observation would rather argue against the in vivo application of such a tumor cell modification in colorectal cancer.
      
These data indicate that clinoptilolite treatment might affect cancer growth by attenuating survival signals and inducing tumor suppressor genes in treated cells.
      
Crude 3 M KCl extracts of the methylcholanthrene-induced fibrosarcoma of C3H/HeJ mice, MCA-F, were demonstrated to contain two fractions, one inducing tumor resistance and the other facilitating the outgrowth of neoplastic cell challenge.
      
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In this paper, a simple and reliable technique of fusion of single parent anucleate cells is established. This technique gives experimental result of the rate of cell fusion and the survival and the purity of cybrids. The cybrids cad noly reproduce and generate in vitro (It has been reptoduced over fourty generations.) but most of them can still express heamoglobin gene. All these result have provided a new technique of normal direction of differentiation for further study of the gene expression and decancerization...

In this paper, a simple and reliable technique of fusion of single parent anucleate cells is established. This technique gives experimental result of the rate of cell fusion and the survival and the purity of cybrids. The cybrids cad noly reproduce and generate in vitro (It has been reptoduced over fourty generations.) but most of them can still express heamoglobin gene. All these result have provided a new technique of normal direction of differentiation for further study of the gene expression and decancerization of the tumor cells. The rate of inducement of reticulocytes and the rate of cell fusion are discussed.

本文介绍一种简便可靠单亲无核的细胞融合技术:其融合率和杂交细胞成活和纯度均达实验要求。这种杂交细胞能在体外试管中繁殖和传代(BWXR巳传40几代,而且部分细胞仍能连续表达合成基因产物(血红蛋白),为基团表达调控和诱导肿瘤细胞向正常方向分化的研究提供一种有效的新方法技术,文中对网质红细胞诱发率及细胞融合等进行讨论。

A comparision of the ECa 109 esophageal epithelial carcinoma cells and normal esophageal epithelial cells was made. Electron microscopically these two cells revealed remarkable differences. The normal esophageal epithelial cells had narrow intercellular space, fewer microvilli, more desmosomes and tonofilaments, with small and round nucleus. No polyribosome could be seen. The ECa 109 cells had wider intercellular space, more microvilli, fewer desmosomes and tonofilaments, larger and irregular nucleus, large...

A comparision of the ECa 109 esophageal epithelial carcinoma cells and normal esophageal epithelial cells was made. Electron microscopically these two cells revealed remarkable differences. The normal esophageal epithelial cells had narrow intercellular space, fewer microvilli, more desmosomes and tonofilaments, with small and round nucleus. No polyribosome could be seen. The ECa 109 cells had wider intercellular space, more microvilli, fewer desmosomes and tonofilaments, larger and irregular nucleus, large and distinct nucleolus. Besides, the appearance of polyribosome was characteristic of cancer cells. Thus, the following characteristics were used as makers of cell differentiation: 1. less microvilli; 2. abundant tonofilaments; 3. more desmosomes; 4. monoribosomes; 5. round nucleus; 6. small nucleus/cytoplasma ratio.Upon addition of 2% DMSO ECa 109 under went striking morphological changes resembling the terminal differentiation stage of normal esophageal epithelial cells.Inaddition, after DMSO treatment, circular Golgi complexes surrounded by small vesicles could be seen. Small vesicles and primary and secondary lysosomesappeared too. Electron-dense particles with halo were easily seen in the nuclei.

肿瘤细胞的特点之一是分化异常。二甲基亚砜可诱导某些肿瘤细胞的分化,为肿瘤细胞分化的研究提供了一种方便的途径。 ECa 109细胞株是一种具有恶性上皮细胞特点的食管癌细胞。经固定、包埋将癌细胞超薄切片后进行透射电镜观察。与正常食管上皮细胞相比较,发现了显著的差异。正常食管上皮细胞具有较少的微绒毛、较多的桥粒与张力原纤维。在胞质中不见聚核蛋白体,而为单核蛋白体。核小而圆。而ECa 109食管癌上皮细胞则有较多微绒毛、较少桥粒与张力原纤维。胞质中均为聚核蛋白体。核大而不规则。由于正常细胞与肿瘤细胞的差别在于食管癌细胞是未分化的细胞,我们根据上述观察提出了下列食管上皮细胞分化的指标:1.微绒毛少;2.丰富的张力原纤维;3.较多的桥粒;4.单核蛋白体;5.较规则的细胞核;6.核/质比例小。加入2%DMSO后,ECa 109食管癌细胞出现与正常食管上皮细胞相似的超微结构,并有胞质分化早于胞核的特点。由此表明ECa 109食管癌上皮细胞可被DMSO诱导分化出现正常食管上皮细胞的特点。为应用DMSO等具有分化作用的药物作为治疗药物提供了线索。讨论了可诱导肿瘤细胞分化的药物。 DMSO还可使ECa 109食管癌上皮...

肿瘤细胞的特点之一是分化异常。二甲基亚砜可诱导某些肿瘤细胞的分化,为肿瘤细胞分化的研究提供了一种方便的途径。 ECa 109细胞株是一种具有恶性上皮细胞特点的食管癌细胞。经固定、包埋将癌细胞超薄切片后进行透射电镜观察。与正常食管上皮细胞相比较,发现了显著的差异。正常食管上皮细胞具有较少的微绒毛、较多的桥粒与张力原纤维。在胞质中不见聚核蛋白体,而为单核蛋白体。核小而圆。而ECa 109食管癌上皮细胞则有较多微绒毛、较少桥粒与张力原纤维。胞质中均为聚核蛋白体。核大而不规则。由于正常细胞与肿瘤细胞的差别在于食管癌细胞是未分化的细胞,我们根据上述观察提出了下列食管上皮细胞分化的指标:1.微绒毛少;2.丰富的张力原纤维;3.较多的桥粒;4.单核蛋白体;5.较规则的细胞核;6.核/质比例小。加入2%DMSO后,ECa 109食管癌细胞出现与正常食管上皮细胞相似的超微结构,并有胞质分化早于胞核的特点。由此表明ECa 109食管癌上皮细胞可被DMSO诱导分化出现正常食管上皮细胞的特点。为应用DMSO等具有分化作用的药物作为治疗药物提供了线索。讨论了可诱导肿瘤细胞分化的药物。 DMSO还可使ECa 109食管癌上皮细胞的高尔基复合器呈环形,外周围之以散在小泡,胞质中有散在大小不等的初级、次级溶酶体。核内易见高电子致密度的带晕颗粒。

Polyactin B (Pb) has been proved to have the effect of considerable tumor suppression. Recently,we used murine S_(180) Sarcoma as a model and observed the effect of Pb on the tumor—infiltrating immunocompetent cells, and also the infiltrating neutrophils and the alteration of small blood vessels within the tumor tissue. Compared with control: in Pb—treated group, there were more L_3T_4~+and Lyt_2~+ lymphocytes infiltrating in the periphery of the tumor ,and also within the tumor. In addition, the tumors had...

Polyactin B (Pb) has been proved to have the effect of considerable tumor suppression. Recently,we used murine S_(180) Sarcoma as a model and observed the effect of Pb on the tumor—infiltrating immunocompetent cells, and also the infiltrating neutrophils and the alteration of small blood vessels within the tumor tissue. Compared with control: in Pb—treated group, there were more L_3T_4~+and Lyt_2~+ lymphocytes infiltrating in the periphery of the tumor ,and also within the tumor. In addition, the tumors had more prominant hyperemia, micro—thrombosis and neutrophil infiltration around the necrotic areas. The present findings suggest that the tumor—suppression effect of Pb might be mediated through TNF produced by immunocompetent cells.

曾经证明多抗乙素有明显的抑瘤作用。本文以小鼠实验性S_(180)肉瘤为模型观察了多抗乙素对肿瘤组织内和肿瘤周围Mφ、L_3T_4~- 及Lyt_2~-细胞的影响,同时观察了肿瘤坏死组织周围中性粒细胞浸润及小血管内血栓形成的改变。结果提示:Pb治疗组,肿瘤周围及肿瘤内浸润的L_3T_4~-和Lyt_2~-细胞显著增多,说明Pb可诱导肿瘤组织中淋巴细胞浸润;肿瘤组织的坏死灶周围明显充血、小血管血栓形成显著增加,并有带状中性粒细胞浸润。这些改变与TNF介导的肿瘤组织损伤相符,提示Pb的抑瘤作用主要是通过免疫活性细胞产生的TNF实现的。

 
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