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肝硬化肝脏
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  cirrhotic liver
     (2) Tie2 receptor expression was negative in control, scarcely positive in the cirrhotic liver (11 3±8 7/HP) and strongly positive in the microvascular endothelium of HCC specimens (52 4±16 7/HP, P<0 01).
     (2 )Tie2受体在正常肝脏无表达 ,在肝硬化肝脏偶见 (1 1 3± 8 7/HP) ,在肝癌微血管内皮大量表达 (52 4± 1 6 7/HP ,P <0 0 1 )。
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     NO could inhibit the expression of P selectin in hepatocytes of the cirrhotic liver of rat during I/R and alleviate the hepatic I/R injury.
     结论 :肝硬化肝脏 I/ R损伤与肝细胞 P-选择素的表达有关 ,NO能抑制肝细胞 P-选择素的表达 ,减轻肝脏 I/ R损伤程度
短句来源
     Objective:To investigate the effect of nitric oxide (NO) on the expression of P selectin in hepatocytes of the cirrhotic liver of rat during ischemia reperfusion (I/R) .
     目的 :探讨一氧化氮 (nitric oxide NO)对肝硬化肝脏缺血再灌注 (ischemia- reperfu-sion I/ R)损伤后肝细胞 P-选择素表达的影响及保护作用。
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     Objective To measure the hepatic lobes with 16 slices helical CT, and discuss the relationship between the changes of the cirrhotic liver volume and the severity of the disease.
     目的采用16层螺旋CT对肝叶进行体积测量,探讨肝硬化肝脏体积变化的规律及其与肝硬化严重程度之间的关系。
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     Results (1) CD34 was not expressed in the control liver but scarcely expressed in the cirrhotic liver and significantly expressed in HCC (P<0 01).
     结果  (1 )CD34在正常肝脏无表达 ,在肝硬化肝脏偶见 ,在肝癌表达明显增加 (P <0 0 1 ) ;
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  “肝硬化肝脏”译为未确定词的双语例句
     Conclusion: ET-1、CGRP、TNF-α and IL-8 were important indexs which reflected the degree of liver damage and estimated the prognosis in posthepatitic cirrhosis.
     结论:ET-1、CGRP、TNF-α和IL-8水平是反映肝炎后肝硬化肝脏损害程度,可作为预后判断的重要指标。
短句来源
     Conclusion HO-1 plays an important role in protecting liver cirrhosis against ischemia reperfusion injury by increasing MnSOD enzyme level and decreasing expression of caspase-3 and NF-κB.
     结论HO-1能有效地减轻肝硬化肝脏缺血再灌注损伤,其机制可能与提高MnSOD水平、降低caspase-3及NF-κB表达有关。
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     To Study Liver and Portal Vein's CT Anatomy of Cirrhosis and Its Clinical Significance
     肝硬化肝脏及门静脉CT解剖学及其临床意义
短句来源
     Hilar plate and hepatic vein in cirrhosis: Anatomy and clinical application
     肝硬化肝脏肝门板和肝静脉的解剖及其临床应用
短句来源
     Objective:To investigate the effects of ischemia preconditioning(IPC) on liver ischemiareperfusion(IR) injury in rats with cirrhosis and the influence of IPC on Pselectin expression in hepatocytes.
     目的 :探讨缺血预处理 (IPC)对肝硬化肝脏缺血再灌注 (I R)损伤的保护作用以及 IPC对 P 选择素表达的影响和作用。
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  相似匹配句对
     Effect of cirrhosis on the expression of UGT mRNA in liver tissue
     肝硬化肝脏UGTmRNA表达的影响
短句来源
     It is a feature that volume of liver is no reduced in patient with early cirrhosis.
     肝脏体积不缩小是早期肝硬化的特征。
短句来源
     Cirrhotic myocardiopathy.
     肝硬化心肌病
短句来源
     PRIMARY CARCINOSARCOMA OF THE LIVER:REPORT OF A CASE
     肝脏癌肉瘤
短句来源
     Hepatic amyloidosis
     肝脏淀粉样变性
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  cirrhotic liver
Thus, multimodality combination treatment with subsequent resection might prolong survival significantly for some patients with unresectable HCC particularly confined in right lobe of a cirrhotic liver.
      
Methods: Suppression subtractive hybridization was used to isolated a panel of genes that are differentially expressed in hepatocellular carcinoma as compared with cirrhotic liver.
      
The classification of cirrhotic liver disease by Child and Turcotte was initially utilized to predict mortality in patients undergoing surgically placed shunts for portal hypertensive bleeding.
      
It was found that the cirrhotic liver tissue's collagen area and collagen average grey degree were notably higher than those in normal men (P>amp;lt; 0.
      
It suggests that the collagen content in cirrhotic liver tissue increased obviously, the average density of the increased collagen was lower than that in the normal liver tissue and the density of increased collagen was not homogeneous.
      
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In the present study, lymphocyte subpopulations, plasma cells and macrophages infiltrating hepatocellular carcinoma (HCC) and peritumor liver tissues from 78 patients were identified and characterized using a specific immunofluorescent technique with anti-human T lymphocyte globulin(ATG), anti-human B lymphocyte globulin (ABG), antihuman IgG, IgM, IgA serum,and α-naphthyl esterase staining method. The liver specimen from 9 patients with cirrhosis and 19 healthy persons were selected as control groups. Ultrastructural...

In the present study, lymphocyte subpopulations, plasma cells and macrophages infiltrating hepatocellular carcinoma (HCC) and peritumor liver tissues from 78 patients were identified and characterized using a specific immunofluorescent technique with anti-human T lymphocyte globulin(ATG), anti-human B lymphocyte globulin (ABG), antihuman IgG, IgM, IgA serum,and α-naphthyl esterase staining method. The liver specimen from 9 patients with cirrhosis and 19 healthy persons were selected as control groups. Ultrastructural morphology was electromicroscopically observed sometime. The majority of cells infiltrating cancer tissue were T cells (74%) which selectively accumulated in the zone between cancer and noncancer liver tissue and arranged around cancer tissue in a row way. T cells might kill cancer cells in different ways. This results indicate that the local lymphocyte and macrophage infiltration is a specific immune reaction against HCC.

本研究应用特异性抗T、B淋巴细胞血清和抗IgG、IgM、IgA血清重复第一抗体荧光技术,α-萘酯酶组化染色对78例原发性肝癌局部浸润的淋巴细胞亚群、浆细胞、巨噬细胞进行了分类测定,以正常人和肝硬化的肝脏做为对照组,同时结合电镜观察和临床资料探讨了这些细胞在机体抗肝癌免疫中的作用。肝癌组织中浸润的细胞主要是T细胞(74%),其高度选择性集中在癌与无癌肝交界处,分别较肝硬化肝、正常肝高4.8倍和39.5倍。这些T细胞以不同方式攻击、杀伤癌细胞,局部淋巴细胞、巨噬细胞浸润是机体抗肝癌的特异性免疫反应。

This is a basic pharmacodynamic research on the home products of Malotilate. The authors demonstrate tha.t Malotilate can stimulate the synthesis of coagulating factors in rabbits suffering from acute hepatotoxicity due to earbontetrachloride (CCl_4), and also inhibit fatty degeneration and fibrosis in rats with chronic hepatotoxicity caused by CGl_4. On the basis of these facts and pathophysiological changes during the formation of hypoproteinemia in liver cirrhosis the implication of Malotilate therapy and...

This is a basic pharmacodynamic research on the home products of Malotilate. The authors demonstrate tha.t Malotilate can stimulate the synthesis of coagulating factors in rabbits suffering from acute hepatotoxicity due to earbontetrachloride (CCl_4), and also inhibit fatty degeneration and fibrosis in rats with chronic hepatotoxicity caused by CGl_4. On the basis of these facts and pathophysiological changes during the formation of hypoproteinemia in liver cirrhosis the implication of Malotilate therapy and other treatment is discussed and evaluated.

肝硬化肝脏的蛋白质功能低下引起的低蛋白症,单纯采用高蛋白饮食或输注白蛋白制剂常不能得到满意解决。实验证实国产马洛替酯对CCl_4急性肝损害家兔,可促进其凝血因子合成;对CCl_4慢性肝损害大鼠,有明显的抑制肝细胞脂肪变性和纤维化形成的作用,从而避免发生肝硬化。该药的合成和研制为治疗肝硬化及其低蛋白症提供了一个新的途径。

To ascertain the role of hepatie fibrosis in the formation of portal hyper-tension in cirrhosis, the content of liver collagen was measured by semiautomatic imageanalysis system. The results showed that hepatic collagen area in cirrhotic rat was muchhigher than the normal rat (32390.16±11879.98 vs. 3206.30±864.26μm~2, P<0 .001).The surface-density of hepatic collagen was 1.58±0.43% in the normal, and 15.97±5.86% in the cirrhotics. In the cirrhotic rat, there was a positive correlation betweenportal pressure...

To ascertain the role of hepatie fibrosis in the formation of portal hyper-tension in cirrhosis, the content of liver collagen was measured by semiautomatic imageanalysis system. The results showed that hepatic collagen area in cirrhotic rat was muchhigher than the normal rat (32390.16±11879.98 vs. 3206.30±864.26μm~2, P<0 .001).The surface-density of hepatic collagen was 1.58±0.43% in the normal, and 15.97±5.86% in the cirrhotics. In the cirrhotic rat, there was a positive correlation betweenportal pressure gradient and the surface density of portal tracts (r=0.44, P<0.05 ), orcollagen of hepatic lobule (r=0.79, P<0.01) or the sum of both (r=0.76, P<0.01).These results concluded that hepatic fibrosis is a main factor producing portal hyperten-sion in the rats with CCl_4-induced cirrhosis.

为证实肝纤维化在肝硬化门脉高压形成中的作用,我们用半自动图象分析仪测定了大鼠CCl_4肝硬化的肝脏胶原含量,同时用检压计测定了同组动物的门脉压力,分析了两者的相关件。结果表明,正常大鼠肝内胶原面积为3206.30±864.62μm~2,肝硬化大鼠为32390.16±11879.98μm~2,差异极显著(P<0.001)。前者与参考面积的比值为1.58±0.43%,后者为15.97±5.86%。正常鼠肝脏胶原面积与门脉压力梯度没有相关性,肝硬化鼠血管周围面积(r=0.44,P<0.05)、肝小叶内胶原面积(r=0.79,P<0.01)及肝内胶原总面积(r=0.76,P<0.01)均与门脉压力梯度呈显著正相关。结果提示,在CCl_4肝硬化大鼠,肝纤维化是导致门脉高压的重要原因之一,支持门脉高压形成的后向血流学说。

 
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