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   小管坏死 的翻译结果: 查询用时:0.491秒
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小管坏死
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  tubular necrosis
     The results showed that BFAR was 0 685±0 777 in normal cases,0 433±0 176 in acute rejection,0 434±0 137 in acute tubular necrosis,0 365±0 097 in cytomacrovirus infection,and 0 150±0 139 in chronic rejection.
     BFAR平均值正常者0.685±0.077,急性排斥0.433±0.176,急性肾小管坏死0.43±0.137,巨细胞病毒感染0.365±0.097,慢性排斥最低,仅0.150±0.139。
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     The urine volume recovery time were from 7 days to 5 months and in the acute renal tubular necrosis group was (15.4±8.3) days, in the acute rejection group was (25.7±4.3) days.
     尿量恢复时间,最短术后7d,最长术后5个月,急性肾小管坏死组尿量恢复(15.4±8.3)d,急性排斥组尿量恢复(25.7±4.3)d。
短句来源
     Results The reasons that led to oliguria or anuria early after transplantation were acute rejective reaction(55.9%), acute renal tubular necrosis (38.2%) and accelerated rejective reaction(5.9%).
     结果:移植肾术后早期少尿或无尿患者原因,急性排斥反应55.9%,急性肾小管坏死38.2%,加速排斥反应5.9%。
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     Result The renal all grafts of group of retrieval of kidneys alone have alower rate of acute tubular necrosis and lower serum cretonne at 4 days post-transplantation (0.7% vs. 10.2% , P = 0.002; 162 μmol/L vs.262 μmol/L, P < 0. 001).
     结果 肝肾联合切取组受者术后急性肾小管坏死发生率为10.2%,术后第4天血肌酐值平均为262 μmol/L,肾脏单独切取组受者分别为0.7%和162μmol/L,两组间比较差异有显著性。
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     Objective:To study the value of urinary cystatin C in predicting the need of renal placement therapy(RRT)in nonoliguric acute tubular necrosis(ATN).
     目的:探讨尿胱抑素C预测非少尿型急性肾小管坏死(acute tubular necrosis,ATN)近期是否需要肾替代治疗(renal replacement therapy,RRT)的临床应用价值。
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  “小管坏死”译为未确定词的双语例句
     Urinary TNF-αlevels raised significantly in patients with ATN (P<0.01).
     急性肾小管坏死(ATN)组尿液TNF-α升高显著(P<0.01)。
短句来源
     2.The acute rejection rate was 18.1% and ATN rate was 18.1%;
     急性排斥反应、急性肾小管坏死发生率各为18.1%;
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     Objective: Acute renal failure (ARF) is the rapid breakdown of renal function that characterized by a decrease of glomerular filtration rate (GFR) and high levels of blood urea nitrogen (BUN) or serum creatinine (SCr).
     目的:急性肾功能衰竭(acute renal failure, ARF)是肾小球滤过率(GFR)急剧降低、血尿素氮(BUN)和血清肌酐(SCr)增高的恶性症候群,最为常见的病因为缺血和中毒所致急性肾小管坏死(acute tubular necrosis, ATN)。
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     (6) BMT+Glycerol i.m group: Glycerol i.m on day 56 after bone marrow transplantation, to observe the role of bone marrow stem cells in the recovery of ATN.
     (6)骨髓移植+甘油注射组:骨髓移植后第56d甘油肌肉注射建立急性肾小管坏死模型,观察骨髓来源干细胞在急性肾小管坏死修复中的作用。
短句来源
     Conclusion Monitoring of the concentration of urine soluble interleukin-2 receptor are helpful in early diagnosis of acute rejection in renal transplant patients,and it's also a good reference in differentiating acute rejection,acute renel tabular necrosis and infection.
     术后发生急性排斥及感染时 ,可溶性白介素Ⅱ受体浓度显著高于平稳患者 (P <0 .0 5 )。 结论 尿液可溶性白介素Ⅱ受体浓度监测可为移植肾急性排斥的早期诊断提供依据 ,对肾移植术后急性排斥反应、急性肾小管坏死及感染的鉴别诊断有较好的参考价值
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  相似匹配句对
     necrosis of renal tubule was lesser;
     肾小管坏死数量较少 ;
短句来源
     degeneration and necrosis of the epithelia of uriniferous tubules.
     肾小管上皮细胞变性、坏死
短句来源
     Bone Marrow Necrosis
     骨髓坏死
短句来源
     intestinal epithelium cells necrosis;
     神经细胞变性、坏死
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     Nursing Patients During Anastomosis of Broken Lachrymal Canaliculus
     泪小管吻合术的护理
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  tubular necrosis
Multiple reasons are known: surgical complications, obstruction, acute tubular necrosis with delayed graft function, acute rejection, nephrotoxicity of drugs and immunsuppressive agents.
      
Allograft biopsy is mandatory to differentiate acute rejection, acute tubular necrosis or calcineurin inhibitor toxicity.
      
A renal biopsy shows acute tubular necrosis suggesting a toxic reaction secondary to high CBZ plasma levels but not a fibrotic process.
      
Daily intramuscular injections of 8 mg/kg in rats and dogs resulted in renal tubular necrosis which increased in severity with higher doses.
      
In this article, we discuss the etiology, pathogenesis, preventative therapies, and renal replacement strategies in patients with acute tubular necrosis, the most common form of hospitalized ARF.
      
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The main targets of acute rejection (AR) in transplanted kidneys had been proved to be vascular endothelial cells and tubular epithelial cells (TEC) in clinical and animal experiments during the last 14 years. The easiness of technique, harmless to the transplanted kidneys and fastness to report the results (2 hours) by counting TEC in 10—15 cc urine sediment provide a better method for its rapidity and realiability for the diagnosis of AR than biopsies of the transplanted kidneys.

14年来移植肾急性排异(AR)的靶位经临床和动物试验研究已明确为脉管内皮细胞和肾小管上皮细胞(TEC)。在10~15毫升尿沉渣做TEC计数是一种简便、无害且在2小时可出结果的技术,此法较移植肾活检安全、快速且可靠是其优点。从1977年10月至1978年9月尿TEC研究结果如下:(1)23例健康人都无TEC,13例移植前尿毒症仅0~5 TEC;(2)在移植后病人尿TEC数少于15为阴性,15~25为可疑,26及以上为阳性;(3)移植直后,肾如受急性肾小管坏死损伤TEC阳性时并非AR;(3)一年间共测知54次AR,其中51次在TEC阳性前3天至后2天出现临床AR;有三次AR未被TEC测知。故此TEC所诊断的AR率为94%。

The renal histopathological findings of the 71 cases with complete clinical histories were observed, 40 cases developed clinically hepatorenal syndrome, including 11 cases with functional renal failure and 29 cases with acute tubular necrosis.Moreover the former was liable to convert into the latter in coures of time.It was suggested that hepatorenal syndrome should be defined as a complication of advanced liver diseases such as extensive liver cirrhosis, hepatoma, fulminant heptitis,etc.,characterised by acute...

The renal histopathological findings of the 71 cases with complete clinical histories were observed, 40 cases developed clinically hepatorenal syndrome, including 11 cases with functional renal failure and 29 cases with acute tubular necrosis.Moreover the former was liable to convert into the latter in coures of time.It was suggested that hepatorenal syndrome should be defined as a complication of advanced liver diseases such as extensive liver cirrhosis, hepatoma, fulminant heptitis,etc.,characterised by acute renal failure with oliguria and azotemia, which pathologically may be shown as functional renal failure or acute tubular necrosis.This paper discussed the pathogenesis of functional renal failure and acute tubular necrosis, the relationship between renal failure and histologic changes of kidneys, as well as the prognosis of hepatorenal syndrome.

本文观察了71例有较完整临床病史的几种肝病尸检肾的组织学改变。在40例发生肝肾综合征的病例中,11例为功能性肾衰,29例为急性肾小管坏死,前者也可发展为后者。因此,我们认为肝肾综合征的定义应该是:各种严重肝病(如肝硬化、肝癌、暴发性肝炎等)在晚期并发急性肾功能衰竭,出现少尿及氮质血症等,在病理上可表现为功能性肾衰或急性肾小管坏死

The rats were given a single ip dose of cis-dichloro-diamminoplatinum (cis-DDP),cis-dichloro-trans-dihydroxobis (isopro-pylamin) platinum (CHIP),and malonato-1,2-diamminocyclohexane-platinum (PHM) 0.023m mol/kg.Two days after cis-DDP being given,theblood urea nitrogen level,urinary glucose concentration,and excretionof LDH in urine were found to be significantly increased.And therewas a tendency towards continual elevation in these observed indexes.The only excretion of LDH in urine was found significantly increasedin...

The rats were given a single ip dose of cis-dichloro-diamminoplatinum (cis-DDP),cis-dichloro-trans-dihydroxobis (isopro-pylamin) platinum (CHIP),and malonato-1,2-diamminocyclohexane-platinum (PHM) 0.023m mol/kg.Two days after cis-DDP being given,theblood urea nitrogen level,urinary glucose concentration,and excretionof LDH in urine were found to be significantly increased.And therewas a tendency towards continual elevation in these observed indexes.The only excretion of LDH in urine was found significantly increasedin the group of CHIP.The observed indexes in the rats given PHMdid not change markedly.These findings indicated that BUN,urinaryglucose concentration and LDH activity were sensitive indexesevaluating nephrotoxicity induced by platinum complexes;and that asingle ip cis-DDP (0.023m mol/kg) to rat resulted in a marked kidneydamage and the CHIP and PHM given to rats in the same doses didnot induce marked kidney damage.Microscopic examination of thekidneys in the group of cis-DDP showed not only acute tubularnecroses but also damage to the endangium cells of glomerule.Six days after ip cis-DDP and CHIP,the serum lipid peroxidationwas found greatly increased.The relation between nephrotoxicityinduced by platinum complexes and lipid peroxidation needs furtherinvestigation.

给大鼠腹注顺铂、CHIP和PHM,剂量均为0.023mmol/kg。给药后2天,顺铂引起尿糖、BUN含量和LDH活性明显增高,且呈持续增高趋势。给CHIP仅使LDH活性升高;而给PHM各项观察指标均未见显著变化。结果提示,BUN、尿糖含量和LDH活性是评价铂络合物致肾损伤的敏感指标;顺铂可引起明显肾损伤,而给予相等mmol的 CHIP和PHM则未发现明显肾毒性。顺铂致肾脏病理组织学改变,除急性肾小管坏死外,对肾小球内皮细胞也有损伤作用。给予顺铂与CHIP后第6天血脂质过氧化物增高,铂络合物引起的肾毒性与脂质过氧化的关系尚待研究。

 
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