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   22肿瘤 的翻译结果: 查询用时:0.032秒
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肿瘤
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  22 hepatocarcinoma
     Experimental Study on Mechanisms of Lyophilized Powder of Fresh Gekko Chinenis in Inhibiting H22 Hepatocarcinoma Angiogenesis
     鲜壁虎冻干粉抑制H22肿瘤血管生成机理的实验研究
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     Conclusion As_2O_3 and ATRA have marked inhibition effects on H22 hepatocarcinoma cells in mice both in vitro and in vivo. The enhanced activity might result from the combination of As_2O_3 and ATRA.
     结论As2O3和ATRA在体内和体外对H22肿瘤细胞的生长均有抑制作用,联合应用作用加强,有协同作用。
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  “22肿瘤”译为未确定词的双语例句
     Method ATPase test kits are used to determine the activities of Ca2+-ATPase? Na+K+-ATPase and Mg2+-ATPase in tumor cells of H22 type.
     方法按照测定无机磷含量的方法对H22肿瘤细胞的Ca2+-ATPase、Na+K+-ATPase和Mg2+-ATPase活性进行了测定。
短句来源
     RESULTS: Spleen lymphocyte proliferation rates were significantly higher in H22 HSP70-peptide complex group and B16 HSP70-peptide complex group than those in serum group,H22 tumor supernatant group and H22 treated tumor supernatant group,P<0.001;
     结果:B16肿瘤HSP70-肽复合物组和H22肿瘤HSP70-肽复合物组的脾淋巴细胞的增殖率均高于阴性对照组、H22肿瘤粗提物组和HIFU后H22肿瘤粗提物组,P<0.001;
短句来源
     Screening,cloning and structure analysis of aptamer binding to H22 tumor cells
     H22肿瘤细胞aptamer的筛选与结构分析
短句来源
     2. Activation of DCs with tumor antigensThe expression of CD80, CD86 and MHCII, and the level of IL-12 and INF-γ were increased in H22 tumor supernatant group, H22 treated tumor supernatant group, B16 HSP70-peptide complex group and H22 HSP70-peptide complex group respectively.
     H22肿瘤粗提物组、B16肿瘤HSP70-肽复合物组、HIFU后H22肿瘤粗提物组和H22肿瘤HSP70-肽复合物组CD80、CD86和MHCII分子的表达、IL-12和INF-γ水平均高于鼠血清组,P<0.001;
短句来源
     Relationship between Hsp70-H22 Tumor Antigen Peptide Complexes and Maturation of DC
     Hsp70-H22肿瘤细胞肽复合物与树突状细胞成熟的关系
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  相似匹配句对
     22.And the types of aberration include deletion,translocation.
     22;
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     Primary epididymal tumour 22 cases
     原发性附睾肿瘤22
短句来源
     An Analysis of Misdiagnosed Cases of Intracranial Tumour
     22例颅内肿瘤误诊分析
短句来源
     22 Key Words
     22个词组
短句来源
     Tumor lymphangiogenesis
     肿瘤淋巴管生成
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  22 hepatocarcinoma
Inhibitory effect of melatonin on the growth of H22 hepatocarcinoma cells by inducing apoptosis
      
Whether melatonin not only inhibits the growth of H22 hepatocarcinoma cells but also induces apoptosis in vitro was assessed.
      
It was found that melatonin could significantly inhibit the growth of H22 hepatocarcinoma cells.
      
It was suggested that melationin could directly inhibit the growth of H22 hepatocarcinoma cells by inducing apoptosis and extending the length of cell cycle of the tumor cells.
      


Ethyl cyanoacetate in ethyl alcohol (triethanolamine catalyst) was treated with hydrogen sulfide gas at 408—50° for 60 hours in order to get ethyl thiocarbamylacetate (Ⅰ). Treatment of the crude reaction mixture with 1, 3-dichloroacetone, however, gave 2-methyl-4-chloromethylthiazole hydrochloride (Ⅲ) instead of the expected ethyl 4-chloromethyl-2-thiazolylacetate (Ⅱ). It has been reported that ethyl thiocarbamylacetate (Ⅰ) reacted normally with α-halocarbonyl compounds to form thiazole derivatives. The method...

Ethyl cyanoacetate in ethyl alcohol (triethanolamine catalyst) was treated with hydrogen sulfide gas at 408—50° for 60 hours in order to get ethyl thiocarbamylacetate (Ⅰ). Treatment of the crude reaction mixture with 1, 3-dichloroacetone, however, gave 2-methyl-4-chloromethylthiazole hydrochloride (Ⅲ) instead of the expected ethyl 4-chloromethyl-2-thiazolylacetate (Ⅱ). It has been reported that ethyl thiocarbamylacetate (Ⅰ) reacted normally with α-halocarbonyl compounds to form thiazole derivatives. The method of preparation of Ⅰ in the literature cited consists of treatment of ethyl cyanoacetate in ether (triethylamine catalyst) with hydrogen sulfide at 65—70° for 2 hours. Probably our experimental condition was more strenuous so that Ⅰ suffered further decomposition giving thioacetamide as the result of hydrolysis and decarboxylation. The formation of Ⅲ from thioacetamide is straightforward. As part of a program on the study of physiologically active heterocycles, Ⅲ was treated with diethanolamine and then with thionyl chloride to give the nitrogen mustard, 2-methyl-4-[bis-(2'-chloroethyl)-aminomethyl]-thiazole hydrochloride (Ⅵ).

本文证实了氰乙酸乙酯与硫化氢作用所得的反应液与1,3-二氯丙酮在无水乙醇中反应后,进行昇华,所得的产物是2-甲基-4-氯甲基噻唑盐酸盐。并由它合成了氮芥型化合物2-甲基-4-[双-(2′-氯乙基)氨甲基]噻唑盐酸盐,供作抗肿瘤的试验。

The antitumor activity of Sb-derivatives of complexones has not yet been reported in the literature.In this work we found that Sb-26 (Sb Na ethylenediamine tetraacetate 15—20 mg/kg), Sb-57(Sb Na propylenediamine tetraacetate 30—50 mg/kg),Sb-66(Sb β-hydroxyethyl imino- diacetic acid 25—30 mg/kg)and Sb-71(Sb ammonia triacetic acid 20—40 mg/kg)could significantly inhibit the growth of Ehrlich ascites carcinoma of mice and prolonged the survival time for 4—22 days(36—147%,P<0.05)in comparison with the control group.These...

The antitumor activity of Sb-derivatives of complexones has not yet been reported in the literature.In this work we found that Sb-26 (Sb Na ethylenediamine tetraacetate 15—20 mg/kg), Sb-57(Sb Na propylenediamine tetraacetate 30—50 mg/kg),Sb-66(Sb β-hydroxyethyl imino- diacetic acid 25—30 mg/kg)and Sb-71(Sb ammonia triacetic acid 20—40 mg/kg)could significantly inhibit the growth of Ehrlich ascites carcinoma of mice and prolonged the survival time for 4—22 days(36—147%,P<0.05)in comparison with the control group.These com- pounds at the above mentioned dosages inhibited also the growth of Guerin’s carcinoma.The degree of inhibition was about 58—70%(P<0.05).The survival time of mice bearing spindle cell sarcoma(in ascitic form)was also prolonged for 5—16 days(50—160%,P<0.05)by treatment with the latter three drugs,among which Sb-71 was most active. Experiments were carried out on mice bearing sarcoma 180 or sarcoma AK treated with Sb-26,Sb-57,Sb-71 or Sb-66 and on mice bearing Ehrlich carcinoma or lymphatic leucemia (solid form)treated with the former three drugs.It was found that only Sb-71 possessed certain inhibitory action on sarcoma 180. In the present study,Sb-8,Sb-11,Sb-42,Sb-15,Sb-64 and Sb-85 were all lacking in inhibitory effect on Ehrlich ascites carcinoma of mice. In mice,the acute LD_(50)levels after intraperitoneal injections of Sb-57 and Sb-71 were found to be 131 and 62 mg/kg respectively;the subacute LD_(50)(10 injections)were equal to 75 and 58 mg/kg respectively.

1.锑胺羧螯合物是一种新型的抗肿瘤药物,应用 Sb-26(EDTA-SbNa)、Sb-57(PDTA-SbNa)、Sb-66(HEDT-Sb)和 Sb-71(ATA-Sb)分别为15—20、30—50、25—30和20—40毫克/公斤剂量时,均能显著地抑制小白鼠 Ehrlich 腹水瘤的生长,平均延长生存时间4—22天,延长率36—147%.上述剂量对大白鼠 Guerin 氏癌也有抑制作用,抑制率为58—70%.后3个药物,还能使小白鼠梭形细胞肉瘤腹水型的生存时间延长5—16天,延长率50—160%,其中以 Sb-71的疗效最著.Sb-26、Sb-57、Sb-71、Sb-66对肉瘤180和 AK 肉瘤及前3者对 Ehrlich 固体瘤和淋巴白血病固体型的实验结果,除 Sb-71对肉瘤180有抑制作用外,其他均无疗效.2.4 类锑化合物:3个(月弟)酸化合物(Sb-8,Sb-11和 Sb-42)及1个酒石酸锑(Sb-15),与螯合的间苯二酚锑(Sb-64)和8羟基喹啉锑(Sb-85)各1个,对 Ehrlich 腹水瘤的实验结果,均无明显疗效.3.小白鼠的急性半数致死量,Sb-57和 Sb-71分别为...

1.锑胺羧螯合物是一种新型的抗肿瘤药物,应用 Sb-26(EDTA-SbNa)、Sb-57(PDTA-SbNa)、Sb-66(HEDT-Sb)和 Sb-71(ATA-Sb)分别为15—20、30—50、25—30和20—40毫克/公斤剂量时,均能显著地抑制小白鼠 Ehrlich 腹水瘤的生长,平均延长生存时间4—22天,延长率36—147%.上述剂量对大白鼠 Guerin 氏癌也有抑制作用,抑制率为58—70%.后3个药物,还能使小白鼠梭形细胞肉瘤腹水型的生存时间延长5—16天,延长率50—160%,其中以 Sb-71的疗效最著.Sb-26、Sb-57、Sb-71、Sb-66对肉瘤180和 AK 肉瘤及前3者对 Ehrlich 固体瘤和淋巴白血病固体型的实验结果,除 Sb-71对肉瘤180有抑制作用外,其他均无疗效.2.4 类锑化合物:3个(月弟)酸化合物(Sb-8,Sb-11和 Sb-42)及1个酒石酸锑(Sb-15),与螯合的间苯二酚锑(Sb-64)和8羟基喹啉锑(Sb-85)各1个,对 Ehrlich 腹水瘤的实验结果,均无明显疗效.3.小白鼠的急性半数致死量,Sb-57和 Sb-71分别为131和62毫克/公斤,亚急性半数致死量分别为75和58毫克/公斤.

In view of some analogues of chloramphenicol show more or less in vitro activity towards quite a few species of tumour cells,the authours have examined the in vitro anti-Ehrlich ascites activity of 5 compounds prepared by one of us and Par before,namely,α-dichloroacetamido- (or acetamido)-β-hydroxy-p-nitro-o-hyrdroxy-(or methoxy)-propiophenone(Ⅰ,Ⅱ,Ⅲ),and α-dichloro- acetamido-β-hydroxy-p-amino-o-hydroxy-propiophenone(IV, V).The results(see Tab. 1)showed that the compounds carrying a nitro group in the benzene...

In view of some analogues of chloramphenicol show more or less in vitro activity towards quite a few species of tumour cells,the authours have examined the in vitro anti-Ehrlich ascites activity of 5 compounds prepared by one of us and Par before,namely,α-dichloroacetamido- (or acetamido)-β-hydroxy-p-nitro-o-hyrdroxy-(or methoxy)-propiophenone(Ⅰ,Ⅱ,Ⅲ),and α-dichloro- acetamido-β-hydroxy-p-amino-o-hydroxy-propiophenone(IV, V).The results(see Tab. 1)showed that the compounds carrying a nitro group in the benzene ring possessed invariably higher activity as compared with their corresponding amino analogues.Therefore,it appears that the nitro group attached to benzene ring plays somewhat important role in producing the in vitro anti-Ehrlich ascites activity. These facts led us to prepare some arylaliphatic nitroalcohols(Ⅶ,Ⅸ,Ⅹ,Ⅺ)and some derivatives of β-nitrostyrenes (Ⅻ, ⅩⅣ,ⅩⅤ,ⅩⅥ,ⅩⅦ,ⅩⅧ,ⅩⅨ,ⅩⅩ) for the purpose of examining their anti-Ehrlich ascites activity.The results(see Tab.2)showed that the nitro group attached to saturated aliphatic carbon atom has no obvious influence in enhancing the anti-tumour activity,in some cases even with counter effect,but that which attached to unsaturated aliphatic carbon atom,as in the case of a series of β-nitrostyrene derivatives,did show higher activity.Therefore, the phenomenon is in harmony with the result described for I,II in the foregoing paragraph,that is,nitro group attached to unsaturated carbon atom is equivalent to that attached to benzene ring. 1-Phenyl-4-nitrobutadiene(ⅩⅥ),in which the nitro group being in the vinylogous position of β-nitrostyrene,also possessed considerable activity.The groups,except that with greater molar volume,substituted in'the benzene nucleus of β-nitrostyrene appear to have no significant influ- ence. The m.ps.of the compounds prepared are recorded in Tab.2.

1.試驗了α-二氯乙酰胺(或乙酰胺基)-β-羥基-对-硝基-邻羥基(或甲氧基)苯丙酮及其相当的氨基化合物的体外抗艾氏腹水瘤細胞的作用,表示硝基对产生活力有影响。2.合成了若干芳香脂肪硝基化合物及β-硝基苯乙烯类化合物,并試驗了該項化合物的体外抗艾氏腹水瘤細胞的活力。3.从活力与化学結构的关系看,脂肪族飽和碳原子上的硝基对产生抗瘤細胞活力无影响,而硝基連在不飽和碳原子上时对活力的增強有很大关系。故一系列的β-硝基苯乙烯类化合物均有相当好的抗肿瘤細胞作用。至于苯乙烯环上的取代基則关系不大,除非取代的基团体积較大,活力也减小。

 
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