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   慢性病毒 的翻译结果: 查询用时:0.408秒
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慢性病毒
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  chronic viral
     The PD-1/PD-L pathway in chronic viral infection
     PD-1/PD-L负性协同刺激途径与慢性病毒感染
短句来源
     Significance of apoptosis gene Fas,FasL and bax expression in chronic viral hepatitis
     凋亡相关基因Fas、Fas配体及bax在慢性病毒肝炎组织中的表达及意义
短句来源
     HIV infection can impair the immune system. There is close correlation between the expressions of CD81 、CCR5、CXCR3 and CD25 on CD4+ and CD8+ T lymphocytes and the immune responses to chronic viral infection. So far, the study on how the immune response alters when HCV/HIV co -infection is too limited.
     CD4~+及CD8~+T淋巴细胞表面CD81、CCR5、CXCR3、CD25的表达与机体对慢性病毒感染的免疫应答密切相关,那么,在HCV合并HIV感染时,CD4~+及CD8~+T淋巴细胞表面CD81、CCR5、CXCR3、CD25的表达如何变化目前国内外未见报道。
短句来源
     Results: 1.CA was chronic, viral, hyperplastic inflammation of shallow layer epidermal proliferation unit, the pathological changes were special hydropic degeneration (CA cell group) of epidermal cell column which contain HPV and papillose hyperplasia of stratum spinosum cell. The cooperation of epidermal infection, differentiation, apoptosis and hyperplasia was the pathogenesis basis 2.DNA infected by HPV was observed in nucli;
     结果:①CA主要是薄层表皮增殖单位(epidermal pro- liferation unit)的慢性病毒(HPV6、11)性增生性炎,病变特点是含HPV表皮细胞柱的特殊水变性(CA 细胞群),乳头状增殖、分化及凋亡,三者协同作用是本病的发病基础。
短句来源
     Thus, further investigation into the biological role of IL-2 in chronic viral infection has been made posible by this experiment.
     本实验为进一步研究白细胞间素-2在慢性病毒感染中的作用打下了基础.
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  “慢性病毒”译为未确定词的双语例句
     CD8+/ CD28- regulator lymphocyte in patients with chronic hepatitis B(79.53± 16.07)% and ASC(89.30± 5.12)% were significantly high than that of controls(63.93± 7.85)% ;
     慢性乙型肝炎患者和慢性病毒携带者外周血中CD8+CD28-调节性淋巴细胞分别为(79.53±16.07)和89.30±5.12)%,均明显高于正常健康对照(63.93±7.85)%(P<0.05);
短句来源
     Conclusion The condition was in virus carried of HBV asymptomatic carriers was related to the raising level of CD4+/CD25+ and CD8+/ CD28- regulator lymphocytes.
     结论慢性病毒携带者处于病毒携带状态可能与体内调节性淋巴胞CD4+/CD25+和CD8+/CD28-的升高有关。
短句来源
     Methods CD4~+/CD25~+ and CD8~+/CD28~- regulator lymphocyte were detected by flow cytometry with multicolor fluorescence technology in 280 cases of chronic hepatitis B, 28 cases of HBV asymptomatic carriers (ASC) and 22 healthy volunteers.
     方法采用流式细胞技术多色荧光分析法,对280例慢性乙型肝炎患者、28例慢性病毒携带者以及22例健康体检者外周血中CD4+/CD25+和CD8+/CD28-淋巴细胞测定。
短句来源
     Results The percent of CD4+/CD25+ lymphocytes of ASC(3.23± 1.37)% was significantly high than that of chronic hepatitis B(1.85± 1.38)% and controls(1.59± 0.54)% . There was no significantly difference between the chronic hepatitis B and that of controls;
     结果慢性病毒携带者外周血中CD4+/CD25+调节性淋巴细胞为(3.23±1.37)%,明显高于慢性乙型肝炎患者(1.85±1.38)%和正常健康对照(1.59±0.54)%(P<0.05),但慢性乙型肝炎患者与正常健康对照差别无统计学意义(P>0.05);
短句来源
     Firstly, lentivirus vectors (three plasmids pNL-EGFP/pNL-EGFP- mES2-sFrp2、 pVSVG, pHelper) were exploited to transfect 293T packaging cell lines to produce EGFP- IRES2-. sFrp2 Lentivirus.
     主要研究工作包括:利用慢性病毒载体三质粒系统pNL-EGFP/pNL-EGFP-sFrp2、pVSVG、pHelper共转染293T包装细胞,生产EGFP-IRES2-sFrp2、EGFP慢性病毒并进行病毒的收集、冻存;
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  相似匹配句对
     The correlation between virus infection and chronic idiopathic thrombocytopenic purpura
     病毒感染与慢性特发性血小板减少性紫癜
短句来源
     Viral infection in patients with chronic obstructive pulmonary disease
     慢性阻塞性肺病患者的病毒感染
短句来源
     sendai virus
     仙台病毒
短句来源
     Chronic Abdominal Pain
     慢性腹痛
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     Chronic low back pain
     慢性腰痛
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  chronic viral
Association of the Ile50Val Polymorphism of the Interleukin-4 Receptor Gene IL4RA with Chronic Viral Hepatitis
      
The Ile50Val polymorphism of the IL4RA gene was tested for association with chronic viral hepatitis and the character of its progression (the stage of hepatic fibrosis).
      
In all 5 acute viral hepatites (AVHs) and chronic viral hepatites (CVHs) there was the increase of erythrocyte activities of glutathione peroxidase (GPx) and glutathione reductase (GR), and the decrease in reduced glutathione (GSH) concentration.
      
A primary study of the subgroups of T lymphocytes in MHV-3 induced chronic viral hepatitis
      
Epidemiology of chronic viral hepatitis in the mediterranean area: Present status and trends
      
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In this article are discussed the etiology,pathogenesis, Clinical and ECG manifestation as well as treatment of the familial long Q—T interval syndrome through census of familial systems.The resulst showed that the disease is mostly considered as a hereditary disease, though some reports pointed out that the disease resulted from chronical viral infection or noninfectional degeneration.The pathogenesis may be associated with autonomic nervous disfunction.The disease originates from early age and may,or may not,be...

In this article are discussed the etiology,pathogenesis, Clinical and ECG manifestation as well as treatment of the familial long Q—T interval syndrome through census of familial systems.The resulst showed that the disease is mostly considered as a hereditary disease, though some reports pointed out that the disease resulted from chronical viral infection or noninfectional degeneration.The pathogenesis may be associated with autonomic nervous disfunction.The disease originates from early age and may,or may not,be accompanied by congenital deafness.There are commonly sudden death cases in the familial system. The clinical features include repeated episodes of syncope and twitch. There appear polymorphous ventricular tachycardia,sometimes consistent with Torsede de point and occasionally ventricular flutter,ventricular fiborillation on ECG in the episode.In the course of episode,drugs of choice are atropine,isoprenaline and other such heart stimulants and installation of pacemaker showed be used for treatment.Contraindication includes,qunidine and similar drugs because of their negative inotropic effect.In order to prevent from episodes of polymorphous Ventricular tachycardia patients have to take propranolol or atropine between the episodes.

本文通过二例及家系发病调查,对家族性 Q—T 间期延长综合征的病因、发病机理、临床、心电图表现及治疗进行了讨论。指出:本病虽多认为是遗传性疾病,但亦有报告认为有的病例为慢性病毒感染或某种非感染性变性所致。病机可能与植物神经系统功能障碍有关。本病多自幼起病,可伴有或不伴有先天性耳聋,家族中常有猝死的病例。临床特点为反复出现晕厥与抽搐,发作时心电图常表现为多形性室速,部分符合尖端扭转型,间有室扑、室颤。发作时宜选用阿托品、异丙肾等心脏兴奋剂及人工心脏起搏治疗,奎尼丁一类心肌抑制剂为禁忌,间歇期可口服心得安、阿托品等预防多形性室速发作。

Human Interleukin2 (IL-2)has been purified for more than 1300 folds from lymphocyte-conditioned media by utilizing a sequence of ammonium sulfate precipitation,ion exchange chromatography with DEAE-Sephadex as well as gel filtration with Sephadex G.This mitog- enie protein released by T cells into the incubating media after exposure to a Lactin(PHA)and TPA,has a Mw fo 15000 as determined by SDS-PAGE.Partial purified IL-2 is unstable and requires the addi- tion of polyothylene glycol to maintain its biological...

Human Interleukin2 (IL-2)has been purified for more than 1300 folds from lymphocyte-conditioned media by utilizing a sequence of ammonium sulfate precipitation,ion exchange chromatography with DEAE-Sephadex as well as gel filtration with Sephadex G.This mitog- enie protein released by T cells into the incubating media after exposure to a Lactin(PHA)and TPA,has a Mw fo 15000 as determined by SDS-PAGE.Partial purified IL-2 is unstable and requires the addi- tion of polyothylene glycol to maintain its biological activity.Thus, further investigation into the biological role of IL-2 in chronic viral infection has been made posible by this experiment.

采用饱和硫酸铵分级沉淀法,DEAE 阴离子交换层析和葡聚糖凝胶过滤技术,将人淋巴细胞产生的白细胞间素-2提纯约1300倍.T 淋巴细胞经 PHA-TPA刺激后分泌的白细胞间素-2.用 SDS-DAGE 检测其分子量为15000.部分纯化的白细胞间素-2稳定性差,加入聚乙二醇可以维持其生物活性.本实验为进一步研究白细胞间素-2在慢性病毒感染中的作用打下了基础.

In this paper chronic hepatitis B and its virus carriers were treated with HB Vaccine and small doses of interferon and thymosin. After one course of interferon group, placenta peptide group and HB-Vaccine group, the negative percentage of HBsAg were 13.7%, 5.9% and 0%, respectively. The changes of HBeAg were 68.8%, 72.7% and 20%, respectively. The results showed that combination of interferon and immunologic stimulant can control the replication of Hepatitis B Virus, and impel HBeAg to change, but can not clear...

In this paper chronic hepatitis B and its virus carriers were treated with HB Vaccine and small doses of interferon and thymosin. After one course of interferon group, placenta peptide group and HB-Vaccine group, the negative percentage of HBsAg were 13.7%, 5.9% and 0%, respectively. The changes of HBeAg were 68.8%, 72.7% and 20%, respectively. The results showed that combination of interferon and immunologic stimulant can control the replication of Hepatitis B Virus, and impel HBeAg to change, but can not clear away HBsAg.

本文用乙肝疫苗与小剂量干扰素等药物配伍治疗慢性乙肝及慢性病毒携带者。经一个疗程后,干扰素组、胎盘肽组和乙肝疫苗组HBsAg转阴率分别为13.7%,5.9%和0%;HBeAg转换率分别为68.8%,72.7%和20%。表明干扰素和免疫增强剂配伍对乙肝病毒复制有一定抑制作用,促使HBeAg转换,但对清除HBsAg无效。

 
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