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  neonatal rat
The effects of antagonist of α2-adrenoceptors yohimbine and their agonist clonidine on Bax and Bcl-XL mRNA levels in neonatal rat brain were studied.
      
Maximal Na+,K+-ATPase activity along with minimal passive K+ influx was observed within oxygen concentration range characteristic for neonatal rat cerebellum.
      
Analysis of α1-adrenoceptor subtypes in neonatal rat heart
      
The expression levels of 3 subtypes (αlA, αlB and α1D) of α1-adrenoceptor have been determined by radioligand binding assay and reverse-transcription polymerase chain reaction (RT-PCR) assay in neonatal rat heart.
      
When two spontaneously beating neonatal rat heart cells in tissue culture were allowed to grow together they synchronized their originally independent beats to a common rhythm, as measured with an opto-electronic technique.
      
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  neonatal rats
ATP Effect on Spontaneous GABAergic Activity in the Hippocampus of Neonatal Rats
      
This study focuses on the oxygen-dependence of active and passive K+ fluxes across membranes of cerebellar granule cells of neonatal rats.
      
Neonatal rats were injected with 6-hydroxydopamine (6-OHDA) for chemical sympathectomy.
      
NE activated Raf-1 kinase and MAP kinases and increased amino acid uptake in cardiomyocytes of neonatal rats.
      
Ouabain caused a positive inotropic effect in right ventricular strips of neonatal rats up to the age of about 30 days but a negative inotropic effect in the adult cardiac tissue.
      
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  newborn rat
ATP induces generation of slow depolarizing waves in the CA3 region of the newborn rat hippocampus
      
Purinergic P2X4 Receptors Are Involved in the Modulation of Giant Depolarizing Potentials in the Hippocampus of Newborn Rat
      
Antioxidant N-acetylcycteine modifies the purinergic modulation of spontaneous postsynaptic potentials in the newborn rat hippoc
      
SEM observations on the effect of anthracycline drugs on cultured newborn rat cardiomyocytes
      
To study the protective effects of radix astragali against anoxic damages to in vitro cultured neurons in rats, NaCN was used to develop a hypoxic model of in vitro cultured neurons from newborn rat cerebral cortex.
      
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  newborn rats
Responses of the skeletal muscle tissue and thymus to muscle injury (complete transection) and wound xenoplasty with the minced muscle tissue of newborn rats (tissue therapy) were studied in mdx mice aged 12-16 and 40-48 weeks.
      
Hyperactivation of succinate dehydrogenase in lymphocytes of newborn rats
      
SDH activity was studied in newborn rats characterized by natural hyperadrenergic status and also in adult animals injected with epinephrine.
      
The increase in succinate oxidation by catecholamines includes activation of the faster pathways of succinate generation than the complete Krebs cycle, in particular, the glyoxylate cycle that is shown in the newborn rats in the present study.
      
Exorphin C and rubiscolin-5, which exhibited relative δ-selectivity, were shown to exert similar effects on the behavior of white newborn rats.
      
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  其他


Sixty eight 7-day old wistar rats were operated by ligating their left common carotid artery and exposing them to 8% oxygen for two hours. Sixty surviving animals were observed. Results showed that the treat- ed rats grew much slower than the control animals. 52/60 rats developed signs of circling to left after their tails were pinched. All rats were killed after hypoxia 44-48 hours. Macroscopically, 34/60 brains showed swelling, turning white in color, and softening. Under microseopynecrosis of the neural cells...

Sixty eight 7-day old wistar rats were operated by ligating their left common carotid artery and exposing them to 8% oxygen for two hours. Sixty surviving animals were observed. Results showed that the treat- ed rats grew much slower than the control animals. 52/60 rats developed signs of circling to left after their tails were pinched. All rats were killed after hypoxia 44-48 hours. Macroscopically, 34/60 brains showed swelling, turning white in color, and softening. Under microseopynecrosis of the neural cells were found in 32/35 cases. Water content significan- tly increased at the ipsilatereal hemispheres in hypoxia- ischem is group. It showed that this model were good for research of newborn hypoxia-isc- hemic encephalopathy.

结扎68只7日龄新生大鼠左侧颈总动脉,然后吸入8%浓度氧2小时,死亡8只。存活的60只新生大鼠体重增长缓慢,52/60只有夹尾左旋;缺氧后44~48小时处死,大体检查可见显示一侧脑变大,颜色变白或转化;显微镜下检查见33/35只的脑组织有不同程度的神经细胞坏死;缺氧缺血组的结扎侧脑半球含水量显著增高。实验结果表明此模型可以较好地用于新生儿缺氧缺血体脑病的进一步研究。

Ninety-nine newborn rats were divided randomly into three groups in the research of hypoxic-ischemic encephalopathy. The preventive and treatment groups were given flunarizing orally before and after hypoxic-ischemic damages, respectively. The behavior, brain water content, morphologic and histologic changes were examined. Water content in preventive group was 88.34±1.12%,much lower than 90.38±2.24% of control group (P<0.005).Morphologic and histologic changes were much lesser in the preventive group than that...

Ninety-nine newborn rats were divided randomly into three groups in the research of hypoxic-ischemic encephalopathy. The preventive and treatment groups were given flunarizing orally before and after hypoxic-ischemic damages, respectively. The behavior, brain water content, morphologic and histologic changes were examined. Water content in preventive group was 88.34±1.12%,much lower than 90.38±2.24% of control group (P<0.005).Morphologic and histologic changes were much lesser in the preventive group than that of control groug (P<0.001 and P<0.01). All damages were lesser in treatment group than control group but without statistically significant difference (P>0.05). These observations confirmed that flunarizing has a neuroprotective effect against hypoxic-ischemic injury in the developing brain.

99只新生大鼠缺氧缺血性脑病模型随机分为三组。预防组和治疗组分别于缺氧缺血前、后给予氟桂嗪,观察各组模型的行为改变、脑含水量、脑大体和显微镜下改变。预防组脑含水量88.34±1.12%,明显低于对照组的90.38±2.24%(P<0.005),脑大体改变和显微镜下改变也比对照组明显减轻(P<0.001和P<0.01)。治疗组各项检查虽有减轻,却无显著性差异(P>0.05)。本实验表明氟桂嗪能减轻新生大鼠缺氧缺血性脑损伤。

he rat model of perinatal cerebral hypoxia-ischemia was prepared by permanent ligation of right common carotid artery combined with a temporary systemic hypoxic (inhaling 8% O2 + 92 % .N2) at 37℃ in 7-day-old SD rats. The relationship between beta-endorphin and encephaledema was observed in the established models. The encephaledema was deteriorated after the injection of betaendorphin into cerebellomedullary cistern, while it was alleviated by the injectionof beta-endorphin antiserum. It is suggested that beta--endorphin...

he rat model of perinatal cerebral hypoxia-ischemia was prepared by permanent ligation of right common carotid artery combined with a temporary systemic hypoxic (inhaling 8% O2 + 92 % .N2) at 37℃ in 7-day-old SD rats. The relationship between beta-endorphin and encephaledema was observed in the established models. The encephaledema was deteriorated after the injection of betaendorphin into cerebellomedullary cistern, while it was alleviated by the injectionof beta-endorphin antiserum. It is suggested that beta--endorphin might play a role in the formation of encephaledema induced by cerebral hypoxic-ischemic injuryin neonatal rats and plethoric beta-endorphin will aggravate encephaledema.

通过结扎7日龄大鼠右侧颈总动脉合并高温低氧环境制备新生鼠缺氧缺血性脑损伤动物模型,观察新生鼠缺氧缺血性脑损伤时β-内啡肽(β-EP)与脑水肿的关系及给予β-EP或β-EP抗血清对脑水肿的影响。结果表明,外源性β-EP可使脑水肿明显加重,而给予β-EP抗血清则使脑水肿明显减轻。提示β-EP可能是新生大鼠缺氧缺血性脑损伤时引起脑水肿的病理“介质”之一,过量的β-EP有加重脑水肿的作用。

 
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