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保护性基因
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  protective gene
     Conclusion The results suggest that HLA-B*51 and HLA-DRB1*14 might be the susceptible genes of BD patients, while HLA-DRB1*15 might be the protective gene of BD patients.
     结论本研究结果提示HLA-B*51、HLA-DRB1*14可能是BD的易感基因,HLA-DRB1*15可能是BD的保护性基因
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     Conclusion HLA DQA1*0301 allele might be a correlative gene with hereditary susceptibility of LS and EH, whereas HLA DQA1*0103 allele might be a protective gene in the patients with LS and EH.
     结论 HLA DQA1 0 3 0 1等位基因与LS及EH的发病有一定的关联 ,而HLA DQA1 0 1 0 3等位基因可能是LS及EH的保护性基因
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     Conclusion HLA DQA1*0301 allele might be a correlative gene with hereditary susceptibility of ABI and HLA DQA1*0301 allele might be a correlative gene in ABI patients with the family history of essential hypertension, whereas HLA DQA1*0103 allele might be a protective gene in the patients with ABI.
     结论  HL A- DQA1* 0 30 1等位基因与 ABI的发病有一定的关系 ,与有原发性高血压阳性家族史的 ABI的发病可能有关 ,而 HL A- DQA1* 0 10 3等位基因可能是 ABI的保护性基因
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     Conclusion DRB1*0301 allele and DR3/4 heterozygosity were the important susceptibility and heterozygosity genes while DR2 was the most important protective gene in Harbin.
     结论 DR位点的DRB1 0 30 1等位基因为哈尔滨市儿童Ⅰ型糖尿病的强易感基因 ,而DR2为保护性基因
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     Large-scale DNA microarray analysis of atopic skin lesions shows overexpression of an epidermal differentiation gene cluster in the alternative pathway and lack of protective gene expression in the cornified envelope
     异位性皮炎皮损的大片段DNA微点阵分析显示旁路途径中表皮分化基因簇过表达以及角质层保护性基因表达缺乏
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  “保护性基因”译为未确定词的双语例句
     DRB1*0301,DRB1*0405,DRB1*0601,DQB1*0203,DQB1*0306 are the susceptible alleles of GDM. DQB1*0402,DRB1*0317 may be the protective alleles of GDM.
     DRB1*0301、DRB1*0405、DRB1*0601、DQB1*0203、DQB1*0306可能是GDM的易感基因,DQB1*0402、DRB1*0317可能是GDM的保护性基因
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     HLA-B*35, DRB1*13, B*56 and A*69 are associ-ated with the protection against HBV infection.
     HLA-DRB1*13可能是抗HBV感染的保护性基因; HLA-B*35,B*56和A*69在中国北方汉族人可能也为抗HBV感染的保护性基因.
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     The DRB1*0404 might be the susceptibility gene of macrovascular diseases in type 2 DM,and the DRB1*0403 might be the susceptibility gene preventing type 2 DM from the macrovascular diseases.
     DRB1*0404单倍型可能是2型DM患者发生大血管病变的易患基因,DRB1*0403单倍型可能是2型DM患者发生大血管病变的保护性基因
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     Conclusions (1) HLA DRB1*08 might serve a genetic risk factor for Hp infection while DRB1*12 might play a role of protecting effect against Hp infection.
     结论  (1)HLA DRB1 0 8基因阳性可能增加对Hp的易感性 ,DRB1 12基因可能是抵御Hp感染的保护性基因
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     Ourresultsindicated thatthefrequenciesof DRB1* 0301,DQA1*0501,DQA1*0301andDQB1*0201,the susceptible alleles,weresignificantly increased inIDDM patients,and the frequencies of DQA1*0103and DQB1*0601,the protective alleles,significantly increased in controls。
     结果表明:DRB1*0301、DQA1*0501、0301和DQB1*0201为IDDM易感性基因,DQA1*0103、DQB1*0601为IDDM保护性基因。 DRB1*0301-DQA1*0501-DQB1*0201为IDDM易感性单倍型。
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     4 new genes were obtained.
     U基因
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     coli lac Z gene.
     colilacZ基因
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     Conclusion I/I genotype is a protecting factor for NIDDM nephropathy.
     结论I/I基因型是NIDDM肾病患者的保护性基因 ;
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     Hemagglutinin gene is an important gene of protective antigen of Avian Influenza Virus (AIV).
     HA基因是禽流感病毒重要的保护性抗原基因
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  protective gene
Liver homogenates of testicular feminized (Tfm) mice carrying the protective (ohv) gene were found to be less capable of converting testosterone to androstenedione than Tfm without the protective gene.
      
We postulated that pulsatile non-reversing shear stress (typical of the common carotid artery), would produce a more "athero-protective" gene expression pattern compared with steady shear stress of the same mean value.
      
Expression of a protective gene prolongs survival of T cells in human immunodeficiency virus-infected patients.
      
Heme oxygenase-1, a protective gene that prevents the rejection of transplanted organs.
      
However in order to be widely scattered among the population, a protective gene should offer its benefits prior to or during the childbearing age.
      
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HLA-DQ gene was analysed by PCR in 42 insulin treated IDDM patients and 40 healthy controls.The results showed in IDDM patients the frequencies of DQA1 52-arginine alleles 0301 and 0501 were significantly higher DQB1 57-nonaspartic acid allele 0302 was significantly increased and DQB1 57 aspartic acid allele 0503 was significantly decreased(P<0.01),In IDDM patients DQA1 52-arginine positive homozygote geneotype was much more(P<0.001).52-arginine negative homozygote genotype was much less(P<0.001).DQB1 57-nonaspartic...

HLA-DQ gene was analysed by PCR in 42 insulin treated IDDM patients and 40 healthy controls.The results showed in IDDM patients the frequencies of DQA1 52-arginine alleles 0301 and 0501 were significantly higher DQB1 57-nonaspartic acid allele 0302 was significantly increased and DQB1 57 aspartic acid allele 0503 was significantly decreased(P<0.01),In IDDM patients DQA1 52-arginine positive homozygote geneotype was much more(P<0.001).52-arginine negative homozygote genotype was much less(P<0.001).DQB1 57-nonaspartic acid homozygote was significantly more than in healthy controls.The results showed HLA DQA152-arginine and DQB1,57-no-naspartic acid contribute the suscebtilitity to IDDM.The frequencies of DQB-57 aspartic acid alleles in Chinese diabetic and controls were 79% and 87.5% respectively,as 57-aspartic acid is supposed to provide the resistance to IDDM in Chinese people,it may explain the low incidence of IDDM in Chinese population.

对42例无亲属关系的IDDM患者和40例健康对照者采用PCR方法对HLA-DQ基因进行了分析,42例皆为曾在本院诊断和住院患者,并一直用胰岛素注射治疗。结果:DQA_152位精氨酸0201和0301等位基因在患者组显著性增高,DQB1-57非门冬氨酸等位基因0302在患者中显著增多,DQB1*050357-门冬氨酸等位基因在患者中显著减少,P<0.01。DQA1基因型52-精氨酸同合子患者显著增加,p<0.001。52-精氨酸阴性同合子在患者中显著减少,P<0.001。说明HLA-DQA152-精氨酸为易感性基因。HLA-DQ57-非门冬氨酸同合子在患者中显著增多,说明DQB157-非门冬氨酸为易感性基因,门冬氨酸为保护性基因。中国患者和健康对照者携带一个门冬氨酸的基因频率分别为79%和87.5%,由于门冬氨酸基因频率高,可解释中国人IDDM患病率低的原因之一。

To study the dose effect rules in IDDM genetic susceptibility contributed by human leukocyte antigen DQ (HLA DQ) alleles, the authors applied polymerase chain reaction and sequence specific oligonucleotide probe dot blot hybridization techniques to classify the HLA DQ alleles of 54 children with IDDM and 40 normal controls. The results indicated that the individuals with 4 susceptive alleles could be found only in the patients, the number of individuals with 3 susceptive alleles was significantly...

To study the dose effect rules in IDDM genetic susceptibility contributed by human leukocyte antigen DQ (HLA DQ) alleles, the authors applied polymerase chain reaction and sequence specific oligonucleotide probe dot blot hybridization techniques to classify the HLA DQ alleles of 54 children with IDDM and 40 normal controls. The results indicated that the individuals with 4 susceptive alleles could be found only in the patients, the number of individuals with 3 susceptive alleles was significantly increased in the patients than in controls (33.3% vs 10%), while the frequency of the individual with 1 or 2 susceptive or protective alleles was a little higher in controls than in the patients, and the individuals carrying 3 protective alleles could only be seen in controls. These results suggest that the effect of susceptive alleles is somewhat recessive and could be accumulated. The individual with one or two susceptive alleles might not offer significant predisposition to IDDM, while the individuals with three or more susceptive ones could be associated with significant susceptibility. The effect of protective alleles is parfially predominant. The individual carrying one or two protective alleles could contribute certain protection to IDDM, while those with three protective ones might have complete resistance to IDDM.

为研究中国北方汉族人中组织相容性复合物DQ(HLA-DQ)基因与胰岛素依赖型糖尿病(IDDM)遗传易感性相关的剂量效应规律,采用聚合酶链反应和序列特异性寡核苷酸探针杂交技术,对54例胰岛素依赖型糖尿病患儿和40例正常成年供血员HLA-DQ基因进行了研究。结果:携带4个易感性基因的个体只见于患者,携带3个易感性基因的个体在患者中为33.3%,正常对照中为10%;携带2个或1个易感性或保护性基因的个体在正常对照中频率较患者为高,但差异无显著意义;携带3个保护性基因的个体只见于正常对照。提示:IDDM易感性基因具有部分隐性遗传的特点且具有累加效应。个体中1个或2个易感性基因的存在不能对IDDM构成显著的易感性,3个或3个以上易感性基因的存在方可对IDDM构成显著易感性。DQ保护性基因具有部分显性遗传的特点并且也具有累加效应。携带1个或2个保护性基因的个体患IDDM的机会将大大减少,而携带3个保护性基因的个体则可以不发生IDDM。

Objective To establish a method for sensitive, specific and rapid detection of the ACE genotypes and to study the relationship between the ACE gene polymophism and renal disorders of NIDDM. Methods A pair of primers for the exon 16 of ACE gene was designed. A sensitive and specific method for detection of the region of the insertion/deletion was determined by polymerase chain reaction (PCR). Results Subjects were classified, according to presence or absence of a 287 bp insertion in intron 16 of the...

Objective To establish a method for sensitive, specific and rapid detection of the ACE genotypes and to study the relationship between the ACE gene polymophism and renal disorders of NIDDM. Methods A pair of primers for the exon 16 of ACE gene was designed. A sensitive and specific method for detection of the region of the insertion/deletion was determined by polymerase chain reaction (PCR). Results Subjects were classified, according to presence or absence of a 287 bp insertion in intron 16 of the ACE gene, as Ⅱ, DD, or heterozygotes for deletion/insertion(DI). Conclusion Ⅱ genotype is a marker for reduced risk for diabetic nephropathy, and DD is a risk factor.

目的探讨血管紧张素Ⅰ转换酶(ACE)基因第16内含子插入/缺失多态性与糖尿病肾病的关系。方法聚合酶链反应。结果40例糖尿病肾病患者中ACE基因Ⅱ型和DD型频率分别为10%(4/40)和38%(15/40),而38例无肾病的糖尿病患者中则分别为42%(16/38)、16%(6/38)。结论提示Ⅱ型基因是糖尿病的保护性基因,DD型基因是糖尿病肾病易感基因。

 
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