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   粘膜血 的翻译结果: 查询用时:0.124秒
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粘膜血
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  mucosal blood
     Results Mucosal blood flow of fundus and antrum in rabbits with PHT group was (73.44 ± 25.47) BPU and (97. 85 ± 15.05) BPU respectively;
     结果 PHT组胃底、胃窦的粘膜血流量分别为(73.44±25.47)BPU. (97.85±15.05)BPU;
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     Results The portal vein pressure(2 65±0 30kPa) and the gastric mucosal blood flow(116 21±13 40BPU) in fundus were significantly higher in the portal hypertensive rats than that in the sham operated rats(1 13±0 11kPa?79 01±11 66BPU,respectively)( P <0 01 or P <0 05 respectively).
     结果 PHT组门静脉压力 (2 6 5± 0 30kPa)和胃底粘膜血流量 (116 2 1± 13 4 0BPU)显著高于SO组 [(1 13± 0 11kPa) ,(79 0 1± 11 6 6BPU) ](P <0 0 1,P <0 0 5 )。
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     Objective To observe the effects of sandostatin on gastric mucosal blood flow(GMBF) in rats with (portal hypertensive gastropathy, PHG) and its mechanisms.
     目的观察善得定对门静脉高压性胃病(portal hypertensive gnstropathy, PHG)大鼠胃粘膜血流量(gastric  mucosal blood flow, GMBF)的影响,并对其作用机制作初步探讨。
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     In the same time. the gastric mucosal blood flow(GMBF) was obviouslyimproved by De~Nol therapy(from 2.33±0.10 to 3.21±0.10V).
     对胃粘膜血流量的改善也非常明显,由2.33±0.10V增加到3.21±0.10V。
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     Using laser dopple flowmetry,we measured the gastric mucosal blood flow(GMBF)in 28 cases with gastritis[15 chronic atrophic gastritis(CAG),13 non chronic atrophic gastritis(NCAG)].
     为探明慢性萎缩性胃炎者胃粘膜血供情况,应用激光多普勒血流仪测定28例胃炎患者的胃粘膜血流量(GMBF),其中慢性萎缩性胃炎组15例,非萎缩性胃炎组13例。
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  “粘膜血”译为未确定词的双语例句
     Results Tetrandrine (15mg/kg , 30mg/kg ) can degrade the content of seru-mal ALT,PCⅢ,NO , 6-keto-PGF_(1α),and can degrade the blood flow of mucosaunderlayer ,and can increase the blood flow of mucosa ,and tetrandrine 30mg/kgis best.
     结果: Tet(15mg/kg、30mg/kg)可降低血中 ALT、Ⅲ型前胶原(PCⅢ)、NO、前列环素(6-keto-PGF_(1α))含量,使粘膜下血流量减少、粘膜血流量相对增加,尤以 Tet 30mg/kg 作用显著(P<0.05);
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     In PG,the blood flow of gastric mucosa was higher at 2h than 1/2h( P <0 001),then was low again at 6h and reached its minimum at 12h (contrast with 2h, P <0 001).
     P组组内胃粘膜血流量比较 ,2h较 1/ 2h有所回升 (P <0 0 0 1) ,6h又复下降 ,12h降至最低 (与 2h比较 ,P <0 0 0 1)。
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     Content of histamine and blood flow in nasal mucosa correlated each other ( P < 0.001).
     致敏组动物鼻粘膜中组胺含量和鼻粘膜血流量呈正相关 ,相关性非常显著 (P<0 .0 0 1,r=0 .96 7)。
短句来源
     2. It was confirmed by the method of hydrogen clearence that the gastric mucosal flow reduced significantly. After 15 minutes of the injection, it decreased from original 1.230±0.093 to 0.521±0.079 ((?) +SE) ml, min~(-1).
     2.氢气清除法证明,胃粘膜血流量明显减少,注后第15分钟时由注前1.230±0.093(M±SE)降至0.521±0.079ml·min~(-1)·100g~(-1)(P<0.01)。
短句来源
     TMP may also increase GMBF when gastric mucosal microcirculatory obstruction was induced by LTC_4; The GMBF was changed from 0.52±0.079 to 0.979±0.174ml.
     ③川芎嗪能提高LTC_4诱导的胃粘膜微循环障碍时的胃粘膜血流量,注后10分钟时由注前0.521±0.079增至0.979±0.174ml.
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  相似匹配句对
     Gastric Mucosa Blood Supply Analyzer
     胃粘膜供分析仪
短句来源
     Laser Doppler Flowmetry for Evaluation of Nasal Mucosa Microcirculation
     鼻粘膜微区流量测定
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     Bloody Mary
     玛丽
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     In the superficial layer of the mucosa .
     在胃粘膜表层.
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     Conclusion:It is suggested that the blood ?
     结论:认为!
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  mucosal blood
Oral mucosal blood flow following dry ice stimulation in humans
      
Mucosal blood flow (mBF) was obtained at the hard palate, at the tip of the tongue, on the midline of the oral vestibule, and at the lip.
      
Mucosal blood flow increased at all sites in response to application of dry ice (p>amp;lt;0.001), with peak flow at 0.5 minute to 1.5 minutes after onset of stimulation.
      
Stimulation by dry ice appears to be an effective, noninvasive, and tolerable means to investigate mucosal blood flow at different mucosal sites.
      
Preliminary data indicate different levels of responsiveness to painful cold stimulation at different sites on the oral and perioral mucosa; particularly, mucosal blood flow response at the tongue was least pronounced.
      
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Changes of gastric-duodenal mucosal blood flow (GDMBF) in the healing process of 81 patients with gastric ulcer and 130 with duodenal ulcer and 9 with active ulcer induced by gastric polypectomy were studied by endoscopic laser Dopple flowmetey, 108 cases of no gastric-duodenal lesions were used as controls.In normal sut mucosal blood flow at the angle and antrum of the lessor curvature and at the anterior wall of the duodenal bulb was the smallest (P<0.01).In patients with gastric ulcer, GMBF was decreased...

Changes of gastric-duodenal mucosal blood flow (GDMBF) in the healing process of 81 patients with gastric ulcer and 130 with duodenal ulcer and 9 with active ulcer induced by gastric polypectomy were studied by endoscopic laser Dopple flowmetey, 108 cases of no gastric-duodenal lesions were used as controls.In normal sut mucosal blood flow at the angle and antrum of the lessor curvature and at the anterior wall of the duodenal bulb was the smallest (P<0.01).In patients with gastric ulcer, GMBF was decreased significantly (P<0.01) at active stage and increased at healing stage.GDMBF was increased significantly (P<0.01) at active stage and returned to the normal at the healing stage of duodenal ulcer.GDMBF was unchanged in the healing process of the active ulcer induced by gastric polypectomy.The mucosal blood flow at ulcer centre and margin was decreased significantly at active stage and increased markedly at healing stage as compared with that of surrounding mucosa.We conclude that GMBF plays an important role in the developing and healing process of peptic ulcers

用激光多普勒血流计经胃镜测定了108例正常对照者、81例胃溃疡,180例溃疡和9例外胃息肉摘除后溃疡患者GDMBF。结果表明:正常人粘膜血流量胃大弯高于胃小弯;胃角、胃窦小弯和球部前壁最低(P<0.01)。活动期胃溃疡GMBF明显降低,愈合期增加(P<0.01)。球溃疡在活动期GDMBF明显增加,随着溃疡愈合而降至正常。胃息肉摘除后溃疡则无变化。三种溃疡其中央和边缘血流量在活动期显著降低(P<0.01),愈合期增加。本研究表明GMBF在消化性溃疡的发生和愈合中起着重要作用。

It has been known that portal hypertension ( PHT ) is often complicated by gastric mucosal lesions, but the mechanism of it remains unclear. The purpose of this study is to evaluate the changes of damaging factors and protective factors of gastric mucosa in PHT rats. Gastric acid, pepsin in gastric juice, gastric gel mucus (GGM) , gastric mucosal blood flow (G-MBF ) and gastric submucosal width(GSMW), were measured and compared with the normal. The results revealed that the output of fasting gastric acid and...

It has been known that portal hypertension ( PHT ) is often complicated by gastric mucosal lesions, but the mechanism of it remains unclear. The purpose of this study is to evaluate the changes of damaging factors and protective factors of gastric mucosa in PHT rats. Gastric acid, pepsin in gastric juice, gastric gel mucus (GGM) , gastric mucosal blood flow (G-MBF ) and gastric submucosal width(GSMW), were measured and compared with the normal. The results revealed that the output of fasting gastric acid and the activity of pepsin in fasting gastric juice did not differ significantly between PHT rats and normal rats(156 +48 vs 126+54 mEq/4h, p>0.05), ( 0.564+0.112 vs 0.514 +0,171 mg/h ml, p>0.10).The quantity of GGM in PHT rats decreased signifieantly(1.60+0.42 vs 2.30+0.30mg, p<0.001);GMBF decreased markedly(2.22+ 0,90 vs 18.43 +3.88ml/h kg,p< 0,001 ), GSMW in PHT rats was much bigger (30.72+3.49 vs 14.70 + 3.77um, p<0.001 ) . Comparison between post-devascularizative portal hy-pertensive(PDPH)and non-operative portal hypertensive(NOPH)rats showed the gastric acid,pepsin and GGM had no signficant changes; but the GMBF and GSMW decreased after operation ( 1.08+1.10 vs 2.22 +0.90 ml/h kg, p<0.05 ) , (25.43+4.08 vs 30.37+3.49um, p<0.05),although the GSMW in PDPH rats was still much bigger than the normal ( 25.43+4.08 vs 14.70 +3.77um, p<0.001 ) .

对门脉高压大鼠胃粘膜的损伤因素和防御因素进行了观察研究,包括测定空腹胃酸分泌量,空腹胃液中胃蛋白酶的活性,胃壁结合粘液量及胃粘膜下层厚度等,并与正常大鼠作了对比。结果发现门脉高压大鼠与正常大鼠的胃酸排出量和胃液中胃蛋白酶活性无显著性差异[(156±48VS 126±54m—Eq/4h,P>0.05)(0.564±0.112VS 0.514±0.717mg/h·ml,P>0.05)]门脉高压大鼠胃壁结合粘液量却明显低于正常对照鼠(1.60±0.42VS 2.30±0.30mg,P<0.001);门脉高压大鼠的胃粘膜血流量也明显低于正常对照鼠(2.22±0.90VS 18.43± 3.88ml/h·kg P<0.001):而门脉高压大鼠胃粘膜下层厚度却远大于正常对照鼠(30.72±3.42VS 14.70±3.77um,P<0.001)断流术半月后门脉高压大鼠的胃酸排出量和胃蛋白酶活性与非手术门脉高压鼠和正常对照鼠比较无显著性差异;其胃壁结合粘液量较非手术门脉高压鼠为低(1.40±0.23VS 1.60±0.42mg,P>0.10)但更低于正常对照鼠(1.40±0.23VS 2.30±0.30mg,P...

对门脉高压大鼠胃粘膜的损伤因素和防御因素进行了观察研究,包括测定空腹胃酸分泌量,空腹胃液中胃蛋白酶的活性,胃壁结合粘液量及胃粘膜下层厚度等,并与正常大鼠作了对比。结果发现门脉高压大鼠与正常大鼠的胃酸排出量和胃液中胃蛋白酶活性无显著性差异[(156±48VS 126±54m—Eq/4h,P>0.05)(0.564±0.112VS 0.514±0.717mg/h·ml,P>0.05)]门脉高压大鼠胃壁结合粘液量却明显低于正常对照鼠(1.60±0.42VS 2.30±0.30mg,P<0.001);门脉高压大鼠的胃粘膜血流量也明显低于正常对照鼠(2.22±0.90VS 18.43± 3.88ml/h·kg P<0.001):而门脉高压大鼠胃粘膜下层厚度却远大于正常对照鼠(30.72±3.42VS 14.70±3.77um,P<0.001)断流术半月后门脉高压大鼠的胃酸排出量和胃蛋白酶活性与非手术门脉高压鼠和正常对照鼠比较无显著性差异;其胃壁结合粘液量较非手术门脉高压鼠为低(1.40±0.23VS 1.60±0.42mg,P>0.10)但更低于正常对照鼠(1.40±0.23VS 2.30±0.30mg,P<0.001)断流术后大鼠的胃粘膜血流量也较非手术门脉高压大鼠为低(1.078±1.009VS 2.220±0.901ml/h·kg,P<0.05),且更低于正常对照鼠(1.078±1.009VS 18.43±3.880m/h·kg,P<0.001)断流术后大鼠的胃粘膜下层厚度比非手术门脉高压鼠虽明显变小(25.4±4.08VS 30.72

Gastric mucosal blood flow (GMBF) and acid secretion were studied before and after highly selective vagotomy (HSV) on 12 health dogs for two months. Before HSV the blood flow increased about 45% in the pentagastrin stimulated corpus, whereas there was no significat change in antrum.Though HSV had no significant influence on resting GMBF, it did influence the mucosal blood flow of corpus during pentagastrin infusion. After HSV the increase in pentagastrin-stimulated corpus mucosal blood flow was around 20%. Which...

Gastric mucosal blood flow (GMBF) and acid secretion were studied before and after highly selective vagotomy (HSV) on 12 health dogs for two months. Before HSV the blood flow increased about 45% in the pentagastrin stimulated corpus, whereas there was no significat change in antrum.Though HSV had no significant influence on resting GMBF, it did influence the mucosal blood flow of corpus during pentagastrin infusion. After HSV the increase in pentagastrin-stimulated corpus mucosal blood flow was around 20%. Which was significantly diminished compared with the increase observed before operation. The changes of acid secretion were similar to those of GMBF.Through the endoscope, modified hydrogen gas clearance method was used in the study. This method is noninvasive, nontoxic, and can be repeated on the same animal at specific intervals without being influenced by changes in secretory activity of the gastric mucosa. It seems to be a good method for measuring GMBF.

对12只健康狗在高选性迷走神经切断术前后的胃粘膜血流量及酸分泌进行了2个月的研究。术前,经胃泌素刺激,胃体粘膜血流量增加45%左右;术后,血流升高幅度明显低于术前,仅20%左右。对胃酸分泌的影响与此相似。本实验通过内窥镜,应用了改良氢气排出法。此法无毒无损伤,可反复在同一动物测定,是一种较好的胃粘膜血流量测定方法。

 
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