助手标题  
全文文献 工具书 数字 学术定义 翻译助手 学术趋势 更多
查询帮助
意见反馈
   黏膜血 的翻译结果: 查询用时:0.445秒
图标索引 在分类学科中查询
所有学科
外科学
消化系统疾病
中药学
更多类别查询

图标索引 历史查询
 

黏膜血
相关语句
  mucosal blood
     Results Gastric mucosal blood flow(GMBF,mV) in infected group was significantly lower than in control group(43.7±2.8 vs. 57.9±2.7,P <0.05) at 3 hours after CLP,and rapidly dropped to the lowest(31.9±2.6,P <0.01) at 12 hours post-perforation.
     结果胃黏膜血流量 (mV)在盲肠穿孔后 3h (43 7± 2 8)与对照组 (5 7 9± 2 7)相比明显降低 (P <0 0 5 ) ,12h(31 9± 2 6 )降至最低(P <0 0 1) ,2 4h(44 7± 2 7)开始回升 ,4 8h(5 2 9± 2 8)仍低于对照组。
短句来源
     RESULTS: Compared with control mice in normal cage, on big platform, or the mice on the 1st day of sleep deprivation, the gastric mucosal blood flow of mice on the 3rd, 5th and 7th day of sleep deprivation (54.2±2.5, 53.7 ± 3.0,48.3±2.5 mv, respectively) were significantly lower.
     结果:在SD3 d、5 d、7 d大鼠胃黏膜血流量与CC组、TC组和sD1d组比较明显下降,血流量分别为(54.2±2.5)、(53.7±3.0)、(48.3±2.5)mv;
短句来源
     On the other hand,the intestine mucosal blood flow(IMBF),(mucosa) thickness,villous height,crypt depth and intestinal epithelial proliferation index were significantly decreased(P<0.05 or P<0.01).
     结果烧伤后肠黏膜损伤指数、通透性及血浆二胺氧化酶(DAO)活性均明显高于C组(P均<0.01),而肠黏膜血流量、肠黏膜厚度、绒毛高度、隐窝深度及肠上皮细胞增殖指数则明显降低(P<0.05或P<0.01)。
短句来源
     Results (1) When IAP was 10 mmHg, the intestinal mucosal blood flow in IAH1 group was similar to that of normal group( P >0. 05).
     结果 (1)IAH模型组兔IAP为10 mm Hg时,其肠黏膜血流量接近正常对照组(P>0.05);
短句来源
     Objective To investigate the regulation of electroacupuncture(EA)on gastric mucosal blood flow(GMBF) and its relation to the contents of plasma and gastric mucosal calcitonin gene-related peptide(CGRP) in dogs.
     目的 观察电针对狗胃黏膜血流量 (GMBF)的调控作用及其与血浆、胃黏膜组织中降钙素基因相关肽 (CGRP)水平变化的关系 ,以探讨电针对胃黏膜保护作用机制。
短句来源
更多       
  “黏膜血”译为未确定词的双语例句
     ②GMBF: The model and saline groups were significantly lower than the normal control group and AG group [(158.2±39.4), (161.7±38.6), (312.6±34.5), (251.3±39.1) mV, P < 0.01];
     ②胃黏膜血流量:模型组和生理盐水组明显低于正常对照组和氨基胍组犤(158.2±39.4),(161.7±38.6),(312.6±34.5),(251.3±39.1)mV,P<0.01犦;
短句来源
     Results The P + E group had the highest gastric blood flow of (103±14) U compared with the other 3 groups (P<0.05).
     结果P+E组胃黏膜血流量(103±14)U显著高于其他3组(P<0.05)。
短句来源
     Results The PVP (19 89±2 29 mmHg) and GMBF (116 21±13 40 BPU)in fundus were significantly higher in the portal hypertensive group than in the sham operated group (8 53±0 84 mmHg, 79 01±11 66 BPU, respectively)( P <0 01 or P <0 05 respectively).
     结果  PHT组大鼠门静脉压力 [(19.89± 2 .2 9) mm Hg]和胃底黏膜血流量 [(116 .2 1± 13.4 0 ) BPU]显著高于 SO组 [(8.5 3± 0 .84 ) mm Hg,(79.0 1± 11.6 6 ) BPU](P<0 .0 1,P<0 .0 5 )。
短句来源
     Results The blood flow in intestinal mucosa of rabbits without SMA occlusion was (102±5)PU,reduced to (48±6) PU after SMA occlusion, and increased to (77±3) PU after injection of carbachol.
     结果SMA阻断后肠部分I/R损伤组肠黏膜血流量为(48±6)PU,较正常对照组[(102±5)PU]明显减少,而肠道内注入卡巴胆碱后1h血流量增至(77±3)PU。
短句来源
     However, it was 44% and 80% lower than that of normal group when IAP was 20 and 30 mmHg,respectively( P <0.01).
     IAP为20、30 mm Hg时,肠黏膜血流量分别比正常对照组减少了44%、80%(P<0.01)。
短句来源
更多       
  相似匹配句对
     Bloody Mary
     玛丽
短句来源
     Effect of Extract A of Dried Radix Rehmanniae on Gastric Mucosal Blood Flow
     干地黄提取物A对胃黏膜流量的影响
短句来源
     TNF IL-8 concentration in blood and in gastric mucosa after liver transplantation in rats
     肝移植大鼠及胃黏膜中TNF和IL-8浓度测定
短句来源
     Blood Rain
     [与泪]
短句来源
     H. pylori infection was more frequent in the mucosal injury group than that in the non;
     黏膜损害组的H.
短句来源
查询“黏膜血”译词为用户自定义的双语例句

    我想查看译文中含有:的双语例句
例句
为了更好的帮助您理解掌握查询词或其译词在地道英语中的实际用法,我们为您准备了出自英文原文的大量英语例句,供您参考。
  mucosal blood
Oral mucosal blood flow following dry ice stimulation in humans
      
Mucosal blood flow (mBF) was obtained at the hard palate, at the tip of the tongue, on the midline of the oral vestibule, and at the lip.
      
Mucosal blood flow increased at all sites in response to application of dry ice (p>amp;lt;0.001), with peak flow at 0.5 minute to 1.5 minutes after onset of stimulation.
      
Stimulation by dry ice appears to be an effective, noninvasive, and tolerable means to investigate mucosal blood flow at different mucosal sites.
      
Preliminary data indicate different levels of responsiveness to painful cold stimulation at different sites on the oral and perioral mucosa; particularly, mucosal blood flow response at the tongue was least pronounced.
      
更多          


Objective To determine the role of endogenous NO in gastric mucosal tolerant cytoprotection under stress and its possible mechanism. Methods SD rats were exposed to WRS repeatedly during which L NAME, a non selective NOs inhibitor, and L Arg, a substrate for NO synthesis, were administered to inhibit or promote the synthesis of NO, GMBF was measured using LDF 3 flowmeter, NO levels in gastric mucosa were tested by Griess reaction and gastric mucosal lesions were evaluated by ulcer index (UI). Results...

Objective To determine the role of endogenous NO in gastric mucosal tolerant cytoprotection under stress and its possible mechanism. Methods SD rats were exposed to WRS repeatedly during which L NAME, a non selective NOs inhibitor, and L Arg, a substrate for NO synthesis, were administered to inhibit or promote the synthesis of NO, GMBF was measured using LDF 3 flowmeter, NO levels in gastric mucosa were tested by Griess reaction and gastric mucosal lesions were evaluated by ulcer index (UI). Results Gastric tolerant cytoprotection was accompanied by increased GMBF and NO levels in gastric mucosa. Inhibition of endogenous NO synthesis by L NAME worsened mucosal lesions induced by WRS. After repeated WRS, adaptive increase of GMBF was abolished and NO content in gastric mucosa significantly reduced. In contrast, enhancement of endogenous NO synthesis by L Arg attenuated mucosal erosions caused by WRS. GMBF and NO content in mucosa increased. After 4th WRS, mucosal lesions could be negligible. Conclusion By regulating GMBF, endogenous NO might play an important role in the gastric mucosal tolerant cytoprotection under stress. Inhibition of NO synthesis delayed the induction of tolerant cytoprotection, while increase NO synthesis will ptomote the induction of tolerant cytoprotection.

目的 探讨内源性一氧化氮 (NO)在应激状态下胃黏膜耐受性细胞保护中的作用及其可能的机制。方法 以重复浸水束缚应激 (WRS)制作动物模型 ,以左旋精氨酸甲酯 (L NAME)或左旋精氨酸 (L Arg)抑制或促进内源性NO的合成 ,动态检测胃黏膜血流量 (GMBF)、溃疡指数 (UI)、黏膜一氧化氮含量的变化。结果 重复应激后 ,实验对照组大鼠UI明显下降 ,同时GMBF上升 ,黏膜内NO含量增高 ;L NAME使WRS引起的胃黏膜损伤加重 ,消除了GMBF的递增趋势 ,黏膜NO含量明显下降 ;而L Arg可减轻WRS造成的黏膜损伤 ,GMBF、黏膜NO含量均相应增加 ;GMBF、UI、黏膜NO含量变化之间有相关关系。结论 内源性NO通过调节GMBF而介导耐受性细胞保护作用 ,L NAME抑制其合成 ,延缓这一作用 ,L Arg增加其合成 ,促进该作用

Objective Effects of melatonin and serotonin on ethanol induced ulceration in the rat stomach were investigated. Methods Serotonin was injected subcutaneously, then melatonin solution and finally ethanol was put into the ex vivo gastric lumen to prepare the experimental models. Glandular mucosal blood flow (GMBF) and the gastric mucosal injury were observed. Results Melatonin and serotonin administration did not induce observable gastric mucosal of damage in the ex vivo stomach, but the serotonin...

Objective Effects of melatonin and serotonin on ethanol induced ulceration in the rat stomach were investigated. Methods Serotonin was injected subcutaneously, then melatonin solution and finally ethanol was put into the ex vivo gastric lumen to prepare the experimental models. Glandular mucosal blood flow (GMBF) and the gastric mucosal injury were observed. Results Melatonin and serotonin administration did not induce observable gastric mucosal of damage in the ex vivo stomach, but the serotonin reduced glandular mucosal blood flow (GMBF) with dose dependence in this organ. Ethanol reduced GMBF and induced visible glandular mucosal damage. The latter effect was prevented by melatonin pretreatment. Serotonin pretreatment aggravated the gastric mucosal injury and GMBF changes induced by ethanol, but these effects were partially reversed by melatonin. Conclusion The findings indicate that the GMBF and gastric injury are related, the reduction in GMBF, however, may not be the sole factor responsible for ulceration. The antagonistic effects of melatonin on serotonin action on the stomach suggest that melatonin may play a modulator role for serotonin action on the gastrointestinal tract.

目的 探讨褪黑激素和血清素对鼠胃酒精性溃疡形成及黏膜血流的影响。方法 制备胃在体动物模型。分别皮下注入血清素及同容积生理盐水。用褪黑激素及同容积蒸馏水作为浸育液分别放入各组鼠的胃腔中。 30min末测毕胃黏膜血流后 ,采用 40 %的酒精作为浸育液分别放入各组鼠胃腔中 ,继续检测胃黏膜血流并测量胃黏膜损伤指数。结果 褪黑激素和血清素两者均不能损伤鼠胃黏膜 ,但血清素可降低胃黏膜血流 ,且与其剂量相关。褪黑激素可减轻由酒精引起的胃黏膜损伤和胃黏膜血流减少 ,而血清素可加重酒精引起的胃黏膜损伤和胃黏膜血流减少 ,但可被褪黑激素部分逆转。结论 胃黏膜的损伤与胃黏膜血流有关 ,但不是溃疡形成的唯一因素。因此 ,能拮抗血清素的褪黑激素可作为在胃肠道作用的调节剂

Objective:To investigate the changes of gastric mucosal blood flow(GMBF)and regulative effect of electro-acupuncture on GMBF in rats with micro-injection of vasoactive intestinal peptide(VIP) into dorsal motor nucleus of vagus complex(DVC).Method:The neurocentre of rat was located and injected by the stereotaxic apparatus of the rat brain,electro-acupuncture (EA)of Zusanli(ST-36) points was applied.GMBF was measured by the method of hydric gas clearance;The contents of VIP in the serum was assayed by radioimmunoassay.Result:The...

Objective:To investigate the changes of gastric mucosal blood flow(GMBF)and regulative effect of electro-acupuncture on GMBF in rats with micro-injection of vasoactive intestinal peptide(VIP) into dorsal motor nucleus of vagus complex(DVC).Method:The neurocentre of rat was located and injected by the stereotaxic apparatus of the rat brain,electro-acupuncture (EA)of Zusanli(ST-36) points was applied.GMBF was measured by the method of hydric gas clearance;The contents of VIP in the serum was assayed by radioimmunoassay.Result:The contents of VIP in the serum,transmucosal potential difference(PD) and GMBF were all increased(P<0.01) after micro-injection of VIP into DVC.These effects were enhanced by EA at Zusanli(ST-36) points.Conclusion:The magnifying effect on GMBF is induced by micro-injection of VIP into DVC,VIP is a strong neuropeptide in nerve endocrine immunity system(NEIS),DVC is a special part of neurocentre influenced by VIP,EA has the synergistic effects on micro-injection of VIP into DVC.

目的 :采用中枢迷走神经背核复合体 (DVC)微量注射血管活性肠肽 (VIP) ,观察胃黏膜血流量 (GMBF)变化和电针对GMBF的调整作用。方法 :采用脑立体定位仪对大鼠中枢进行定位和注射 ,电针足三里穴 ,用氢气清除法测定GMBF ;放射免疫法测量外周血中VIP含量。结果 :DVC微量注射VIP后 ,血中VIP含量增加 (P <0 .0 1) ,GMBF明显增加 (P <0 .0 1)。电针 (EA)足三里穴加强了GMBF放大效应。结论 :DVC注射VIP对GMBF有放大效应 ,证实在神经 内分泌 免疫网络系统 (NEIS)中VIP是重要的神经肽 ,DVC是VIP作用的神经中枢特异性部位之一 ,电针具有协同作用。

 
<< 更多相关文摘    
图标索引 相关查询

 


 
CNKI小工具
在英文学术搜索中查有关黏膜血的内容
在知识搜索中查有关黏膜血的内容
在数字搜索中查有关黏膜血的内容
在概念知识元中查有关黏膜血的内容
在学术趋势中查有关黏膜血的内容
 
 

CNKI主页设CNKI翻译助手为主页 | 收藏CNKI翻译助手 | 广告服务 | 英文学术搜索
版权图标  2008 CNKI-中国知网
京ICP证040431号 互联网出版许可证 新出网证(京)字008号
北京市公安局海淀分局 备案号:110 1081725
版权图标 2008中国知网(cnki) 中国学术期刊(光盘版)电子杂志社