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   心脏纤维化 在 内分泌腺及全身性疾病 分类中 的翻译结果: 查询用时:0.131秒
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内分泌腺及全身性疾病
心血管系统疾病
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心脏纤维化    
相关语句
  cardiac fibrosis
    Expression of Short Isoform of Leptin Receptor mRNA and Observation of Cardiac Fibrosis in Hearts of Streptozotocin-Diabetic Rats
    STZ糖尿病大鼠心肌短型瘦素受体基因的转录表达及心脏纤维化观察
短句来源
    Results Ob-Ra mRNA was expressed in myocardium of streptozotocin - diabetic and control rats with no significant difference between them (P>0.05),but there was higher expression of leptin receptor mRNA in cultured myocardial fibroblasts of diabetic rat.Distinct cardiac fibrosis was found and compained by the decreased heart function in diabetic rats.
    结果 STZ糖尿病大鼠和对照鼠心肌均可转录表达短型瘦素受体mRNA ,两组之间无显著差别 ,但糖尿病心肌成纤维细胞短型瘦素受体mRNA表达较高 ,伴心功能减退和显著的心脏纤维化病理改变。
短句来源
    Conclusions The coexistence of cardiac fibrosis and increased expression of Ob-Ra mRNA in myocardial fibroblasts were found in diabetic rats.
    结论 糖尿病大鼠心脏成纤维细胞短型瘦素受体表达增加与心脏纤维化并存。
短句来源
  cardiac fibrosis
    Expression of Short Isoform of Leptin Receptor mRNA and Observation of Cardiac Fibrosis in Hearts of Streptozotocin-Diabetic Rats
    STZ糖尿病大鼠心肌短型瘦素受体基因的转录表达及心脏纤维化观察
短句来源
    Results Ob-Ra mRNA was expressed in myocardium of streptozotocin - diabetic and control rats with no significant difference between them (P>0.05),but there was higher expression of leptin receptor mRNA in cultured myocardial fibroblasts of diabetic rat.Distinct cardiac fibrosis was found and compained by the decreased heart function in diabetic rats.
    结果 STZ糖尿病大鼠和对照鼠心肌均可转录表达短型瘦素受体mRNA ,两组之间无显著差别 ,但糖尿病心肌成纤维细胞短型瘦素受体mRNA表达较高 ,伴心功能减退和显著的心脏纤维化病理改变。
短句来源
    Conclusions The coexistence of cardiac fibrosis and increased expression of Ob-Ra mRNA in myocardial fibroblasts were found in diabetic rats.
    结论 糖尿病大鼠心脏成纤维细胞短型瘦素受体表达增加与心脏纤维化并存。
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  cardiac fibrosis
We found that noise stress significantly increased the microvessel wall area, the number of microvessels with an outer diameter >amp;gt;19 μm, the degree of cardiac fibrosis, and the extent of ischemic myocardial lesions in SHR, but not in NWR.
      
TNFα-antagonism attenuates the development of experimental diabetic cardiomyopathy associated with a reduction of intramyocardial inflammation and cardiac fibrosis.
      
Cardiac Fibrosis as a Cause of Diastolic Dysfunction
      
The interaction of coronary tone and cardiac fibrosis
      
Aldosterone has direct effects on the vasculature and has been associated with vascular smooth muscle cell hypertrophy, endothelial dysfunction, cardiac fibrosis, proteinuria, and renal vascular injury.
      
更多          
  cardiac fibrosis
We found that noise stress significantly increased the microvessel wall area, the number of microvessels with an outer diameter >amp;gt;19 μm, the degree of cardiac fibrosis, and the extent of ischemic myocardial lesions in SHR, but not in NWR.
      
TNFα-antagonism attenuates the development of experimental diabetic cardiomyopathy associated with a reduction of intramyocardial inflammation and cardiac fibrosis.
      
Cardiac Fibrosis as a Cause of Diastolic Dysfunction
      
The interaction of coronary tone and cardiac fibrosis
      
Aldosterone has direct effects on the vasculature and has been associated with vascular smooth muscle cell hypertrophy, endothelial dysfunction, cardiac fibrosis, proteinuria, and renal vascular injury.
      
更多          
  myocardial fibrosis
Midwall myocardial fibrosis in Becker-Kiener muscular dystrophy
      
In chronic myocarditis, viremia is often absent and myocardial fibrosis and dilated cardiomyopathy (DCM) resulting in heart failure can occur.
      
MRI is a sensitive tool detecting myocardial fibrosis on late images after application of paramagnetic contrast agents.
      
Patchy or homogenous myocardial fibrosis in Becker muscular dystrophy
      
Transforming growth factor-β receptor antagonism attenuates myocardial fibrosis in mice with cardiac-restricted overexpression o
      
更多          
  myocardial fibrosis
Midwall myocardial fibrosis in Becker-Kiener muscular dystrophy
      
In chronic myocarditis, viremia is often absent and myocardial fibrosis and dilated cardiomyopathy (DCM) resulting in heart failure can occur.
      
MRI is a sensitive tool detecting myocardial fibrosis on late images after application of paramagnetic contrast agents.
      
Patchy or homogenous myocardial fibrosis in Becker muscular dystrophy
      
Transforming growth factor-β receptor antagonism attenuates myocardial fibrosis in mice with cardiac-restricted overexpression o
      
更多          


Purpose To observe whether the leptin receptor mRNA is expressed and cardiac fibrosis is present in hearts of Streptozotocin - diabetic rats. Methods The experiment was done at twelfth week after the onset of diabetes.After blood pressure and cardiac function were mearsured,the rats were sacrificed then cardiac tissue and cultured myocardial fibroblasts were used for RT?PCR to show the expression of leptin receptor mRNA.The ultrastructure of myocardium were examed by electron microscopy.Serum leptin,insulin...

Purpose To observe whether the leptin receptor mRNA is expressed and cardiac fibrosis is present in hearts of Streptozotocin - diabetic rats. Methods The experiment was done at twelfth week after the onset of diabetes.After blood pressure and cardiac function were mearsured,the rats were sacrificed then cardiac tissue and cultured myocardial fibroblasts were used for RT?PCR to show the expression of leptin receptor mRNA.The ultrastructure of myocardium were examed by electron microscopy.Serum leptin,insulin were assessed by immunoassay. Results Ob-Ra mRNA was expressed in myocardium of streptozotocin - diabetic and control rats with no significant difference between them (P>0.05),but there was higher expression of leptin receptor mRNA in cultured myocardial fibroblasts of diabetic rat.Distinct cardiac fibrosis was found and compained by the decreased heart function in diabetic rats. Conclusions The coexistence of cardiac fibrosis and increased expression of Ob-Ra mRNA in myocardial fibroblasts were found in diabetic rats.

目的 研究瘦素受体在STZ糖尿病大鼠心肌中转录表达及心脏的纤维化情况。方法 用STZ制备糖尿病大鼠模型 ,在病程 12周时测定大鼠血压和心功能后处死 ,取心肌组织和分离、培养心脏成纤维细胞 ,用RT PCR检测长、短型瘦素受体mRNA转录表达 ,PCR产物测序 ,电镜观察心肌的超微结构。血清瘦素、胰岛素放射免疫测定。结果 STZ糖尿病大鼠和对照鼠心肌均可转录表达短型瘦素受体mRNA ,两组之间无显著差别 ,但糖尿病心肌成纤维细胞短型瘦素受体mRNA表达较高 ,伴心功能减退和显著的心脏纤维化病理改变。结论 糖尿病大鼠心脏成纤维细胞短型瘦素受体表达增加与心脏纤维化并存。

 
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