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   肾间质纤维化 的翻译结果: 查询用时:0.078秒
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肾间质纤维化     
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  renal interstitial fibrosis
     The expression of Smurf2mRNA and protein was positively correlated with that of Smad2(r=0.975,0.979,P<0.01),and the index of interstitial fibrosis(r=0.911,0.964,P<0.01)during the progressive phase of renal interstitial fibrosis(RIF).
     ⑵Smurf2 mRNA和蛋白质与肾间质纤维化指数呈正相关(r=0.911,0.964,P<0.01),与肾间质炎性细胞浸润指数无相关,与Sm ad2 mRNA和蛋白呈正相关(r=0.975,0.979,P<0.01)。
短句来源
     CONCLUSION: IL-18Rα mRNA and IL-18Rβ mRNA are expressed in RTECs, which provide the experimental basis for exploring IL-18R role in renal interstitial fibrosis.
     结论:在RTECs中有IL-18RαmRNA和IL-18RβmRNA的表达,为探讨IL-18R在肾间质纤维化中提供了实验依据的作用。
短句来源
     Conclusion: TGF-β_1、 TIMP-1 and MMP-9 play an important effection on renal interstitial fibrosis.
     结论:TGF-β_1、TIMP-1及MMP-9在肾间质纤维化过程中起着重要作用;
短句来源
     P21 expression in renal interstitial fibrosis and regulative effect of enalapril
     P21在单侧输尿管梗阻大鼠肾间质纤维化中的表达及依那普利对其表达的调节
短句来源
     Objective To investigate the expression of P21 in renal interstitial fibrosis rats and the effect of enalapril on it.
     目的:动态观察P21在单侧输尿管梗阻肾间质纤维化大鼠模型中的表达,以及依那普利对其表达的调节。
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  tubulointerstitial fibrosis
     Expression of p-Smad2/3 and smad7 in experimental renal tubulointerstitial fibrosis rat model
     p-Smad2/3、Smad7在大鼠实验性肾间质纤维化模型中的表达
短句来源
     Conclusions TGF-β1/Smad signaling is involved in the progression of renal tubulointerstitial fibrosis.
     结论 TGF-β1/Smad信号通路参与了肾间质纤维化进程。
短句来源
     Changes of extracellular matrix and metalloproteinases in TBM mediated tubulointerstitial fibrosis
     TBM介导的肾间质纤维化中细胞外基质和金属蛋白酶的动态变化
短句来源
     Objective To examine the expression of the TGF-β1/Smads signaling pathway in renal tubulointerstitial fibrosis.
     目的观察TGF-β1/Smads信号传导途径对大鼠肾间质纤维化表达的影响。
短句来源
     Conclusion: PGE1 could alleviate tubulointerstitial fibrosis through inhibiting the expressions of CTGF and TGF-β1 in tubulointerstitium.
     结论:PGE1可以抑制TGF-β1、CTGF的表达,减轻肾间质纤维化,从而防治梗阻性肾病。
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  interstitial fibrosis
     The expression of Smurf2mRNA and protein was positively correlated with that of Smad2(r=0.975,0.979,P<0.01),and the index of interstitial fibrosis(r=0.911,0.964,P<0.01)during the progressive phase of renal interstitial fibrosis(RIF).
     ⑵Smurf2 mRNA和蛋白质与肾间质纤维化指数呈正相关(r=0.911,0.964,P<0.01),与肾间质炎性细胞浸润指数无相关,与Sm ad2 mRNA和蛋白呈正相关(r=0.975,0.979,P<0.01)。
短句来源
     The degree of interstitial fibrosis was significantly correlated with TGF-beta(l) mRNAs (r=0.51, P = 0.001) and proteins (r=0.52, P=0.01) , and also significantly correlated with CTGF mRNAs (r=0.53, P = 0.005) and proteins (r=0.49, P=0.003) too.
     TGF-β 1mRNA和蛋白与肾间质纤维化的程度(r=0.51,p=0.001及r=0.52,P=0.01),CTGFmRNA和蛋白的表达与肾间质纤维化的程度(r=0.53,D=0.005及r=0.49,P=0.003)都呈明显正相关。
短句来源
     CONCLUSION: IL-18Rα mRNA and IL-18Rβ mRNA are expressed in RTECs, which provide the experimental basis for exploring IL-18R role in renal interstitial fibrosis.
     结论:在RTECs中有IL-18RαmRNA和IL-18RβmRNA的表达,为探讨IL-18R在肾间质纤维化中提供了实验依据的作用。
短句来源
     On day 7 of the UUO model, the percentage of tubular lesions and interstitial fibrosis score were significantly lower in the retinoic acid-treated group than those in control group [(15.9±2.0)% vs (27.3±2.2)% and (0.47±0.12) vs (1.65±0.18), P<0.01].
     在UUO模型第 7d ,维甲酸治疗组肾小管损害百分比和肾间质纤维化分值显著低于对照组 [(15 9± 2 0 ) %vs (2 7 3±2 2 ) %和 (0 4 7± 0 12 )vs (1 6 5± 0 18) ,P <0 0 1];
短句来源
     Localization of Smad 2, 3, 6, 7 and their changes of expression in experimental interstitial fibrosis model in rats
     Smad 2,3,6,7蛋白在实验性肾间质纤维化模型中的定位和表达变化
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  renal tubulointerstitial fibrosis
     Expression of p-Smad2/3 and smad7 in experimental renal tubulointerstitial fibrosis rat model
     p-Smad2/3、Smad7在大鼠实验性肾间质纤维化模型中的表达
短句来源
     Conclusions TGF-β1/Smad signaling is involved in the progression of renal tubulointerstitial fibrosis.
     结论 TGF-β1/Smad信号通路参与了肾间质纤维化进程。
短句来源
     Objective To examine the expression of the TGF-β1/Smads signaling pathway in renal tubulointerstitial fibrosis.
     目的观察TGF-β1/Smads信号传导途径对大鼠肾间质纤维化表达的影响。
短句来源
     The Role of Notch/Jagged1 Signaling Pathway and Inhibition of Jagged1 in the Development of Renal Tubulointerstitial Fibrosis in Vivo and in Vitro
     Notch1/Jagged1在肾间质纤维化发病机制中的作用及干预的研究
短句来源
     CONCLUSION: As the expression of TGF β 1 intensively increased in hypertensive course, TGF β 1 may be the important factor leaded to the renal tubulointerstitial fibrosis.
     结论 :TGF β1在自发性高血压大鼠肾小管的表达显著增加 ,它可能是致肾间质纤维化的重要因素。
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  renal interstitial fibrosis
Effects of mycophenolate mofetil on renal interstitial fibrosis after unilateral ureteral obstruction in rats
      
To investigate the effects of mycophenolate mofetil (MMF) on the process of renal interstitial fibrosis, unilateral ureteral obstruction (UUO) model was established in rats.
      
Role of protease activated receptor-2 expression in renal interstitial fibrosis model in mice
      
The rats in the model group developed glomerular sclerosis and renal interstitial fibrosis.
      
The progression of renal diseases: On the pathogenesis of renal interstitial fibrosis
      
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  tubulointerstitial fibrosis
Expression of connective tissue growth factor in renal tubulointerstitial fibrosis in rats and its pathogenic role
      
In order to explore the role of connective tissue growth factor (CTGF) in the pathogenesis of renal tubulointerstitial fibrosis, 48 Wistar rats were randomly divided into sham-operated and unilateral ureteral obstruction (UUO) group.
      
The increased expression of CTGF may play a crucial role in the development and progression of tubulointerstitial fibrosis.
      
It was concluded that CTGF might play a crucial role in the degradation of excessive ECM during tubulointerstitial fibrosis, and blocking the biological effect of CTGF may be a novel way in preventing renal fibrosis.
      
The PTC network is a main target of kidney transplant rejection and of progressive tubulointerstitial fibrosis, which typifies all progressive renal diseases.
      
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  interstitial fibrosis
Objective: To explore the effect and mechanism of matrine (Mt.) on myocardial interstitial fibrosis induced by pressure overload.
      
treatment can significantly increase survival rate (P>amp;lt;0.05), decrease LVW/BW (P>amp;lt;0.05) and MMP-2 activity (P>amp;lt;0.05), decrease size of cardiomyocytes and interstitial fibrosis compared with operation group.
      
Morphologically, hibernating myocardium displays features of dedifferentiation with loss of cardiomyocytes and myofibrils and of degeneration with increased interstitial fibrosis.
      
Mitochondrial volume and interstitial fibrosis were not different between groups.
      
Likewise, interstitial fibrosis in congestive heart failure may enhance the anisotropy.
      
更多          
  renal tubulointerstitial fibrosis
Expression of connective tissue growth factor in renal tubulointerstitial fibrosis in rats and its pathogenic role
      
In order to explore the role of connective tissue growth factor (CTGF) in the pathogenesis of renal tubulointerstitial fibrosis, 48 Wistar rats were randomly divided into sham-operated and unilateral ureteral obstruction (UUO) group.
      
As a result, the highly crosslinked collagens induce an irreversible progression of chronic renal tubulointerstitial fibrosis in the kidneys of ICGN mice.
      
The Institute of Cancer Research (ICR)-derived glomerulonephritis (ICGN) mouse is a hereditary model animal for nephrotic syndrome with chronic renal tubulointerstitial fibrosis.
      
Cellular and molecular events leading to renal tubulointerstitial fibrosis
      
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