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   慢性炎症反应 的翻译结果: 查询用时:0.028秒
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慢性炎症反应
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  chronic inflammation
     Conclusion:The production of the recombinant adenovirus vector encoding NF κB RNA aptamer provides a foundation for investigation of its activity and gene therapy for treatment of acute and chronic inflammation caused by NF κB over activity in clinic.
     结论 :产生具有生物学活性的重组腺病毒 Ad-aptamer,为下一步基因治疗因 NF- κB过度激活引起的急慢性炎症反应奠定基础
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     Conclusion Aspirin can change chronic inflammation in elderly patients with insulin resistance,and lower CRP,and improve insulin sensitivity.
     结论 阿司匹林可改变老年胰岛素抵抗患者慢性炎症反应 ,从而降低CRP水平 ,改善胰岛素抵抗
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     Conclusion:A low-grade chronic inflammation may be involved in the pathogenesis of ACIN.
     结论 :持续低程度的慢性炎症反应可能与ACIN的发病有关
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     Role of chronic inflammation in type 2 diabetes and obesity has been recognized in the past few years.
     慢性炎症反应在2型糖尿病和肥胖发生发展中的作用越来越受到人们的重视。
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     Results:Chronic inflammation occurred in liver of autoimmune mice and the serum titers of autoantibodies including anti dsDNA and antinuclear antibodies were significantly elevated.
     结果 :CJ致敏小鼠的肝脏组织发生了慢性炎症反应 ,血清自身抗体如抗核抗体和抗双链DNA抗体的水平明显升高 ;
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  chronic inflammatory reaction
     Conclusion: Manzhi Kechuanning Capsules can control chronic inflammatory reaction by improving the content of IL-2 and reducing the content of IL-8.
     结论:慢支咳喘宁胶囊可提高IL-2含量,降低IL-8含量,控制慢性炎症反应
短句来源
     Conclusion: Manzhi Kechuanning Capsules can control chronic inflammatory reaction by reducing the contents of TNF-α and ET-1.
     结论:慢支咳喘宁胶囊可降低血清中TNF-α及ET-1含量,控制慢性炎症反应
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     Psoriasis is a common skin disease characterized by over hyperplasia of epidermis and chronic inflammatory reaction of corium.
     银屑病(Psoriasis)是一种以表皮过度增生和真皮慢性炎症反应为特征的常见皮肤病。
短句来源
     Conclusion: Manzhi Kechuanning Capsules can control chronic inflammatory reaction by reducing the contents of TNF and ET.
     结论:慢支咳喘宁胶囊可降低TNF、ET含量,控制慢性炎症反应
短句来源
     Conclusion: Manzhi Kechuanning Capsules can control chronic inflammatory reaction by reducing the content of IL-4.
     结论:慢支咳喘宁胶囊可降低IL-4含量,控制慢性炎症反应
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  “慢性炎症反应”译为未确定词的双语例句
     (3) HE staining showed that there was severe chronic pulmonary inflammatory response in smoking control group, which was slight in the smoking plus exercise group;
     (3)HE染色显示 ,吸烟对照组肺组织出现明显的慢性炎症反应 ,吸烟加运动应激组炎症反应轻于吸烟对照组 ;
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     The pathogenesis of AR and BA has not been quite clearly, it may be involved in the interaction of many inflammatory mediators, cytokines and transcriptional factors.
     AR与BA的发病机制至今仍不十分清楚,其是涉及多种炎性细胞的相互作用,诸多炎性介质、细胞因子、转录因子等共同参与的慢性炎症反应
短句来源
     NF-κB, a transcription factor, contributes to the links of AS and shear stress.
     同时AS又是一个慢性炎症反应过程,而核因子κB(nuclear factor kappa B,NF-κB)作为调节炎症和免疫反应的核转录因子之一,在AS的形成过程中意义重大。
短句来源
     Conclusion: Manzhikechuanning capsules could control inflammatory reaction by increasing the contents of IL-2 and reducing the contents of IL-8.
     结论:慢支咳喘宁胶囊可提高IL-2含量,降低IL-8含量,控制慢性炎症反应
短句来源
     In this study, we chose two CAD candidate genes, MEF2A and ALOX5, which relate with endothelium development and inflammation respectively, to identify SNPs exist in this two genes, and investigate their association with CAD in Chinese Han polulation.
     本研究,我们选择了MEF2A和ALOX5这两个分别与血管内皮发育和慢性炎症反应机制有关的基因,进行中国汉族人群基因多态性分析,并探讨其与冠状动脉粥样硬化性心脏病的相关性。
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  chronic inflammation
In addition to having a key role in thrombogenesis, platelets are actively involved in acute and chronic inflammation: they induce the release of proinflammatory mediators, expose adhesion molecules, and recruit leukocytes.
      
Protease-antiprotease imbalance, generation of oxidative stress, and chronic inflammation are recognized as the principal mechanisms leading to irreversible airflow obstruction and parenchymal destruction in the lung.
      
Further characterizations of DN Treg cells are that infection indicates that MHV-3 could induce the chronic inflammation in livers of C3H/Hej mice.
      
When the endothelium is physically disrupted or functionally perturbed by postischemic reperfusion, acute and chronic inflammation, atherosclerosis, diabetes and chronic arterial hypertension, then completely opposing actions pertain.
      
Chronic inflammation or myocarditis and chronic alcohol abuse are considered two main etiologic factors in dilated cardiomyopathy.
      
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  chronic inflammatory reaction
The occurrence of a chronic inflammatory reaction in one of the two patients supports the idea that an autoimmune disease against GABA-ergic neurons may be involved in SMS.
      
The following chronic inflammatory reaction causes fistulas or sinuses and cysts.
      
This chronic inflammatory reaction could be the cause of cartilage degeneration seen in patients after ACL reconstruction.
      
They may cause a chronic inflammatory reaction that can result in abnormal findings on radiographs, including lytic or blastic osseous changes.
      
The mononuclear cell infiltration which characterizes the chronic inflammatory reaction results from the migration of lymphocytes and monocytes through the endothelium of the postcapillary venule.
      
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Precancerous lesions of the nasal cavities were induced by a large dose of DNP subcutaneous injection 12 times in 45 days in 12 rats. Cancerous lesions developed in all the animals when the observation period prolonged to 160 days. It suggests that the abnormal differentiated proliferation of the basic cells, which is reconized in the process transforming from the normel epithelium to well-differendiated carcinoma, should be always observed.Carcinogenesis of the nasocavity was inhibited by N-(4-Ethoxycarbophenyl)...

Precancerous lesions of the nasal cavities were induced by a large dose of DNP subcutaneous injection 12 times in 45 days in 12 rats. Cancerous lesions developed in all the animals when the observation period prolonged to 160 days. It suggests that the abnormal differentiated proliferation of the basic cells, which is reconized in the process transforming from the normel epithelium to well-differendiated carcinoma, should be always observed.Carcinogenesis of the nasocavity was inhibited by N-(4-Ethoxycarbophenyl) Retin-amide (RI). The mechanism Probably is the results of inhibitiing or reversing effect on the squamogenesis in the process of carcinogenesis.

1.短期大剂量DNP皮下注射12次经45天后12只大鼠全部出现鼻腔癌前期病变,动物延至160天全部出现癌,作者认为可用作鼻腔、鼻咽癌前期的动物实验模型;2.维甲酰胺对癌的发生有抑制作用,其机制可能是抑制或逆转癌变过程中的鳞化过程。维甲酰胺还表现出对急、慢性炎症反应有抑制作用;3.DNP诱发的鼻腔上皮病变中出现的急、慢性炎症可能是上皮病变的伴发反应和机体免疫状态的反映。

The distribution and pathological lesions induced by migrating larva of Toxocara canis were investigated by detecting larva and observing histologi-cal changes from various organs of the infected animals on 1,3,5,7,14,28, 42, 8lday after administration of 1000 eggs/mice groups of ten NIH male mice. The worm burden in liver and lung was more on day 1 to 7 after infection. During this period,degeneration and necropsies of hepatic cells and haemorrhages and oedema in the lungs occured. Inflammatory reaction was...

The distribution and pathological lesions induced by migrating larva of Toxocara canis were investigated by detecting larva and observing histologi-cal changes from various organs of the infected animals on 1,3,5,7,14,28, 42, 8lday after administration of 1000 eggs/mice groups of ten NIH male mice. The worm burden in liver and lung was more on day 1 to 7 after infection. During this period,degeneration and necropsies of hepatic cells and haemorrhages and oedema in the lungs occured. Inflammatory reaction was serious in these two organs.7 days after infection, the larvae migrated mostly to muscles and brain,with scattered inflammatory foci in muscles and slight inflammation in brain.Sometime focal granuloma appeared later.The studies showed that the pathological manifestation in visceral larva migrans resulted from mechanical lesions by the presence of the migrating larvae.

用犬弓首线虫感染性虫卵感染NIH小鼠,1000个虫卵/鼠,于感染后1、3、5、7、14、28、42、81天剖杀,观察幼虫在其肝、肺、脑、横纹肌等器官的分布及病理反应。感染后的前7天,幼虫主要分布在肝、肺,7天后主要在脑和肌肉。感染后的前28天为急性炎症反应,受累器官有出血及严重的炎症反应,其实质细胞有变性和坏死。晚期为慢性炎症反应过程,有典型的寄生虫性肉芽肿形成。

ight microscopy and scanning electron

应用光镜及扫描电镜对30只兔眼后房型人工晶体表面细胞反应进行了观察,结果发现有巨噬细胞、类上皮细胞、成纤维细胞样细胞、异物巨细胞、超大巨细胞及淋巴细胞粘附在人工晶体表面。扫描电镜下,这些细胞共同形成一层半透明薄膜,复盖在人工晶体表面。提示后房型人工晶体植入术后晶体表面细胞反应是由于人工晶体刺激引起的一种以异物反应为主,免疫细胞参与的慢性炎症反应,该反应由于术后时间推移而渐减弱,最后由眼内环境接受。

 
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