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   共刺激 的翻译结果: 查询用时:1.337秒
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共刺激     
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  co-stimulation
     The role of B7:CD28 co-stimulation in the development of rheumatoid arthritis
     共刺激分子CD28和B7在关节炎发病中的作用
短句来源
     Conclusion: B7:CD28 co-stimulation plays a role in the autoimmune diseases mediated by T cell, and anti-B7-1 may have potential therapy effect.
     结论:B7:CD28共刺激信号在诱发 T细胞介导的自身免疫疾病中起作用,抗B7-1抗体具有潜在的治疗作用。
短句来源
     CONCLUSION: Co-stimulation of TEC by IL-4 and CD40mAb up-regulated the RANTES production, suggesting the RANTES may participate in the inflammation of TEC.
     结论 :IL - 4及CD4 0 -CD4 0L共刺激信号可调控RANTES合成及分泌 ,参与TEC炎症过程。
短句来源
     Results: The level of p65 in nuclear extracts of Hut-78 cells was much lower during the CD3/CD28 co-stimulation in 30 min, however arsenic trioxide seemed to inhibit the effect of CD3/CD28 co-stimulation.
     结果:Hut-78细胞在CD3/CD28双信号通路共刺激30min后,细胞核中p65的表达显著下降(P<0.05)。 三氧化二砷组p65表达要高于单纯CD3/CD28作用组。
短句来源
     Conclusion: NF-κB is suppressed during the early phase of CD3/CD28 co-stimulation in human T cells.
     结论:T细胞经过CD3/CD28共刺激,细胞内NF-κB不能得到迅速活化,反而有下降的趋势。 三氧化二砷能够促进NF-κB的早期活化。
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  co-stimulatory
     The Research on Interfering CD40-CD40L in Blocking Co-stimulatory Pathway by CD40 shRNA
     CD40 shRNA干扰CD40-CD40L在阻断共刺激通路作用的实验研究
短句来源
     Humoral immune response induced by co-stimulatory molecule B7-2 with HPV16L1 gene
     共刺激分子B7-2与HPV16L1联合基因免疫小鼠的免疫应答
短句来源
     The Anti-rejection Effect of Blocking B7/CD28 Co-stimulatory Pathway by RNAi in Mice Heart Transplantation
     RNAi阻断B7/CD28共刺激通路在小鼠心脏移植中的抗排斥反应作用
短句来源
     Expression of co-stimulatory molecules CD28/CTLA-4:B7 in peripheral blood lymphocytes of patients with ankylosing spondylitis
     共刺激分子CD_(28)/CTLA-4:B7在强直性脊柱炎患者外周血淋巴细胞中的表达
短句来源
     The Anti-rejection Effect of Blocking B7/CD28 Co-stimulatory Pathway by RNAi in Mice Small Bowel Transplantation
     RNAi阻断B7/CD28共刺激通路在小鼠小肠移植中的抗排斥反应作用
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  co-stimulating
     Objective:To construct a recombinant adeno-associated virus vector rAAV2-PD-L1 and to investigate the biological efficiency of rAAV2-PD-L1-transfected vascular endothelial cells in co-stimulating secretion of cytokines by T cells.
     目的:构建腺相关病毒rAAV2介导的PD-L1表达系统,检测感染rAAV2-PD-L1的血管内皮细胞共刺激T细胞分泌细胞因子的作用。
短句来源
     RESULTS: Compared with CT-26 lysates, pulsing with HSCT-26 lysate enhanced the expression of MHC-II molecule (66.3% vs 59.1 %) and co-stimulating molecule CD86 (39.4% vs 36.7%) on DC surface.
     结果:热休克处理肿瘤细胞可提高肿瘤细胞抗原负载的树突状细胞表面MHC-Ⅱ类分子(66.3% vs 59.1%)、共刺激分子CD86的表达(39.4% vs 36.7%);
短句来源
     Results The DCs were fewer (P<0.05) and the expressive level of CD83 and CD86 which were DCs surface co-stimulating factors were lower(P<0.05) in patients with chronic hepatitis B than the controls.
     结果与正常对照组相比较,慢性乙型肝炎患者组PBMCs来源的DCs表面共刺激分子CD83、CD86的表达水平明显降低(P<0.05)。
短句来源
     Results After treated with A23187 (180 ng/mL) or plus rhIFN-γ (1000 U/mL) for 20 hours, some of HL-60 cells were found to have the dendritic appearance on cell surface; and the CD83 as a characteristic marker of mature DCs, co-stimulating molecules CD80 and CD86 got an up-regulated expression.
     结果HL-60细胞加入适量A23187(180ng/mL)或联合rhIFN-γ(1000U/mL)诱导20h后,即可出现细胞表面树状突起,且树突状细胞表面特异性成熟标志抗原CD83、共刺激分子CD80、CD86表达升高;
短句来源
     CD 86 co-stimulating factor expression on monocy te declined (66.96±13.87)% and mean fluorescence intensity (MI F) decreased (31.31±12.91)% after rhG-CSF administration. Expression of CD 80 on B lymophocyte decreased (45.77±26.58)%.
     单核细胞表达共刺激信号CD86下降了 (66 .96± 1 3 .87) % ,相对平均荧光强度降低了 (31 .31± 1 2 .91 ) % ,B淋巴细胞中表达CD80 降低了 (45 .77± 2 6 .58) %。
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  co stimulatory
     Objective To compare humoral immune response by co inoculating mice with co stimulatory molecule B7 2 and HPV16L1 recombinant plasmids and observe the neutralizing antibody activity in vitro .
     目的 研究共刺激分子B7 2和HPV16L1重组质粒免疫小鼠的体液免疫反应。
短句来源
     Conclusion (1)In ABM, the remarkably increased expression of co stimulatory molecule CD86 in CD3 - MNC may enhance the anti tumor effect of antigen specific CTLs.
     结论  (1)ABM中CD3-细胞表面共刺激分子CD86表达明显增加 ,与ABM中特异性细胞毒性T淋巴细胞(CTLs)的生成有关。
短句来源
     Expression feature of CD 137 (human 4 1BB),a new co stimulatory signal on T cells and T cell subsets
     一种新的共刺激信号CD_(137)在人T细胞及其亚群中的表达特点
短句来源
     Conclusion The high expression of CD40L is related to the development of GVHD in HSCT, and this indicates that CD40 CD40L co stimulatory pathway plays an important role in GVHD.
     结论 CD40L高表达与GVHD的发生相关 ,提示CD40 CD40L共刺激途径在GVHD的发生中可能起着重要作用
短句来源
     Conclusion (1)The abnormal high expression of peripheral blood co stimulatory molecules CD28 suggested CD28 disorder may play an important role in immuno pathogenesis of CAA.
     结论 (1)CAA患者外周血共刺激分子CD2 8异常高表达 ,提示CD2 8失调可能在CAA免疫发病中起重要作用。
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  co-stimulation
(iv) Co-stimulation with IL-10 resulted in a ~55 % reduction in TNFα-stimulated MCP-1 levels in cardiomyocyte culture supernatants.
      
Currently, it is generally believed that the blockade of T cell co-stimulation offers considerable potential for achieving tolerance in the clinical setting.
      
Thus, vascular SMC-like endothelial cells appear to contribute to the maintenance of an inflammatory response in the atherosclerotic vessel wall upon CD40-CD154 co-stimulation.
      
Activator protein 1 activation in response to either high glucose or co-stimulation with high glucose and Ang II was inhibited completely by calphostin C (a PKC inhibitor) and partially by genistein (a protein tyrosine kinase inhibitor).
      
Nevertheless, the proliferation achieved in the indirect presentation model required co-stimulation by LFA-1, CD2 and CD28, engaged by co-stimulation molecules expressed in the cis-form by the human monocytes.
      
更多          
  co-stimulatory
Elevated pressure significantly increased expression of all co-stimulatory and MHC molecules on mature DC.
      
Upstream promotion of Treg cell differentiation by blockade of the co-stimulatory molecule CD154 may also be of benefit in preventing autoimmune disease, as illustrated by a promising exploratory Phase I study in multiple sclerosis.
      
According to the danger hypothesis, the immune response to a drug-derived antigen requires the presence of co-stimulatory signals and cytokines, which propagate and determine the type of immune response.
      
However, impaired expression of co-stimulatory molecules including CD80, CD86, and CD40L may contribute to low polyfunctional CD4+ T cell as well CD8+ T-cell activity.
      
Co-stimulatory blockade as therapy for rheumatoid arthritis
      
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  co-stimulating
The expression of co-stimulating factor CD86 (B7-2) on the surface of DC was detected by flow cytometry, and the stimulating capacity of DC was determined by mixed lymphocyte reaction (MLR).
      
Realtime PCR examination demonstrated that PIR-A and co-stimulating molecules such as CD80, CD86 and MHC-II were increased significantly after stimulation with LPS.
      
A chicken homologue of the co-stimulating molecule CD80 which binds to mammalian CTLA-4
      
However, the appropriate molecules involved in co-stimulating T-cells must be present on the ker atinocyte membrane if T-cells are to be activated.
      
  co stimulatory
Additional stimuli from the APC have been collectively referred to as costimula tory factors, although the definition of such factors remains vague.
      
Co stimulatory pathways between APC and the T-lymphocyte are required for full T-lymphocyte activation.
      
CD137 and CD28 are co stimulatory molecules of T cell activation.
      
Furthermore, these particles can be potent stimulators of antigen presenting cells in vitro as measured by several important co stimulatory molecules.
      
This strongly suggests that BSF-1 costimula tory effects are not mediated in a manner entirely analogous to those of the calcium ionophore.
      
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