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   阿尔茨海默病 在 中药学 分类中 的翻译结果: 查询用时:0.511秒
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阿尔茨海默病     
相关语句
  alzheimer ' s disease
    Methods Treated human neuroblastoma SK-N-SH cells with Aβ_(25-35),simulating the pathologic damage in brain of patients with Alzheimer's disease,and antagonized the effect by adding KXS.
    方法用Aβ25-35处理人神经母细胞瘤细胞(SK-N-SH细胞),模拟阿尔茨海默病(AD)病人脑内神经元病理损伤,并以含开心散(KXS)药物血清拮抗其作用。
短句来源
    Study on relationship between effects of quercetin on learning and memory in Alzheimer's disease rats and apoptosis of hippocampus cells
    槲皮素对阿尔茨海默病大鼠学习记忆影响及与海马细胞凋亡关系的研究
短句来源
    Effect of Heart-benefitting Recipe on Alzheimer's disease rats after unilateral Aβ_(25-35) injection into amygdala
    调心方对杏仁核注射Aβ_(25-35)诱导的阿尔茨海默病模型大鼠的影响
短句来源
    Effects of lingcao liquid on energy metabolism in the brain of model rats of Alzheimer's disease
    灵草液对阿尔茨海默病模型大鼠脑组织能量代谢的影响
短句来源
    Effect of Jiannao Yizhi decoction on learning memory and oxidative stress in rats with Alzheimer's disease
    健脑益智汤对阿尔茨海默病大鼠学习记忆和氧化应激的影响
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  alzheimer ′ s disease
    Study on Establishment of Alzheimer′s Disease Animal Model and Intervening Effect of Zhinao Capsule on It
    阿尔茨海默病动物模型的建立及智脑胶囊干预作用的研究
短句来源
    Objective:To establish a convenient, economical and practical analogous oxidative damaged Alzheimer′s disease rat model (AD model) for exploring the effect of Tiaoxin Recipe (TXR) on the spatial memory capacity and β-amyloid protein (Aβ) deposition in the model.
    目的 :建立一种简便、经济、实用的氧化损伤类阿尔茨海默病 (Alzheimer′sdisease ,AD)大鼠模型 ,探讨调心方对该模型大鼠空间学习记忆能力和 β 淀粉样蛋白质 (β amyloidprotein ,Aβ)沉积的影响。
短句来源
    AIM The study was designed to investigate the ef fe ct of ginsenoside Rg1 on the neprilysin (NEP) expression in U251 cell line treat ed by lipopolysaccharide(LPS), in order to find out the drugs and methods f or treating Alzheimer ′s disease.
    目的 为寻找治疗阿尔茨海默病的药物及方法 ,探讨人参皂苷Rg1对脂多糖 (LPS)诱导的U2 5 1细胞株脑啡肽酶 (NEP)表达的影响。
短句来源
    Establishment of Analogous Oxidative Damaged Alzheimer′s Disease Rat Model and Effect of Tiaoxin Recipe on It
    氧化损伤类阿尔茨海默病大鼠模型的建立及调心方的作用
短句来源
    PROTECTIVE EFFECT OF GANODERMA LUCIDUM POLYSACCHARIDE ON THE BRAIN TISSUE OF RAT MODEL OF ALZHEIMER′S DISEASE INDUCED BY β-AMYLOID PROTEIN FRAGMENT25-35
    灵芝多糖对Aβ25-35诱导阿尔茨海默病模型大鼠脑组织的保护作用
短句来源
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  alzheimer ’ s disease
    Effect of mistura ginsenoside and choline on rat models of alzheimer’s disease
    参胆合剂对阿尔茨海默病模型大鼠学习记忆的影响
短句来源
    Effects of Extracts of Ginkgo Biloba Leaves on Learning and Memory in Alzheimer’s Disease Rats
    银杏叶提取物对雌性阿尔茨海默病大鼠学习记忆能力影响
短句来源
    Objective: According to beta-amyloid (Aβ) cascade hypothesis in Alzheimer’s disease(AD), this study is to observe effects of GETO (herbal extract) on generation and degradation of beta-amyloid protein and neuron plasticity, to look for effective target of GETO in the treating AD. Methods: APPV717I transgenic mouse and model rats with hippocampus injected by Aβ1-42, as AD models were used in this study.
    目的:基于阿尔茨海默病(AD)发病机理中的“Aβ级联假说”,研究金思维提取物对AD动物模型脑内Aβ的生成、聚集和降解途径及神经元可塑性的影响,探讨金思维提取物治疗AD的作用靶点。
短句来源
    Synapse loss is one of the most important pathological changes of Alzheimer’s disease (AD). Synaptic dysfunction and failure are processes that occur early in AD, it is the best current pathologic correlate of cognitive decline.
    突触功能障碍及缺失是发生于阿尔茨海默病(Alzheimer’s disease, AD)早期的病理过程,是AD疾病过程中的重要神经病理改变之一,并且是与认知功能下降最为密切的病理表现。
短句来源
    Objective: Alzheimer’s disease (AD) is a neurodegenerative disorder characterized by irreversible, progressive loss of memory followed by complete dementia, is the fourth largest cause of death for people over the age of 65 years.
    目的:阿尔茨海默病(Alzheimer’s disease,AD)是一种以进行性痴呆为主要特征的神经退行性疾病,是65岁以上人群的第四大死因。
短句来源
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  alzheimer ' s dementia
    CLINICAL STUDY ON HEART AND BRAIN BENEFITING FORMULA IN TREATING ALZHEIMER' S DEMENTIA
    养心健脑方治疗阿尔茨海默病临床研究
短句来源
    Thirty-three cases of Alzheimer's dementia(AD)were treated with Heart and Brain Benefiting Formula (HBBF).
    目的:观察具有益心气、通心窍作用的复方──养心健脑方治疗阿尔茨海默病(AD)的临床疗效,并与西药脑复康对照组比较。
短句来源
    Objective Though study the effects of Shouerzhi capsule upon dissociated Ca 2+ in hippocampus neuron cell, to explore preventive and therapeutic mechanism of TCM in treatment of Alzheimer's dementia(AD) .
    目的 研究寿尔智胶囊对海马神经元细胞内游离钙水平的影响 ,从而探讨中药防治阿尔茨海默病 (Alzheimer’sdementia ,AD)的机制。
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      alzheimer disease
    Effect of homocysteine and nitric oxide levels on specific Computed Axial Tomography measurements in Alzheimer disease
          
    We describe a patient who was clinically diagnosed with familial early-onset Alzheimer disease (AD) carrying both the E318G substitution in presenilin 1 (PSEN1) and an insertion of 7 octapeptide coding repeats in the prion protein gene (PRNP).
          
    It is far from clear that DLBD represents a specific disease entity rather an intermediate variant between Alzheimer disease and idiopathic parkinsonian syndromes.
          
    Pre-clinical diagnosis of Alzheimer disease combining platelet amyloid precursor protein ratio and rCBF spect analysis
          
    Working memory and FDG-PET dissociate early and late onset Alzheimer disease patients
          
    更多          
      alzheimer ' s disease
    Furthermore, acetylcholinesterase and butyrylcholinesterase inhibitors such as tacrine, donepezil, rivastigmine, and galantamine are currently used to manage Alzheimer's disease.
          
    Since amlodipine besylate is a very potent inhibitor of both cholinesterases, amlodipine besylate may, like donepezil, be useful in Alzheimer's disease treatment.
          
    Mutations in presenilin 1 (PS1) gene are closely associated with the early onset of familial Alzheimer's disease (EOFAD).
          
    Prospects of Non-drug Approaches to Alzheimer's Disease
          
    According to the immune status indices and our previous data on behavioral, biochemical, and morphological changes induced in bulbectomized mice, they have common symptoms with the Alzheimer's disease.
          
    更多          
      alzheimer ′ s disease
    The apoE gene has been identified as a major susceptibility locus for late-onset Alzheimer′s disease (LOAD).
          
      其他


    Morris water maze was used to observe preventive effect of Bushen Yizhi Decoction (BYD)on learning and memory in rat model with Alzheimer's disease(AD),and AD rat model was established by intraperitoneral injection of D_galactose combined with ibotenic acid injection into bilateral nucleus basalis of Meynert.The results showed that BYD improved learning and memory in AD rat model.Escape latency of AD rat model in Morris water maze was shortened (P<0.01) and distance of swimming in the platform quadrant was...

    Morris water maze was used to observe preventive effect of Bushen Yizhi Decoction (BYD)on learning and memory in rat model with Alzheimer's disease(AD),and AD rat model was established by intraperitoneral injection of D_galactose combined with ibotenic acid injection into bilateral nucleus basalis of Meynert.The results showed that BYD improved learning and memory in AD rat model.Escape latency of AD rat model in Morris water maze was shortened (P<0.01) and distance of swimming in the platform quadrant was prolonged (P<0.05) .It is suggested that BYD possesses a certain preventive effect on learning and memory in AD rat model.

    为寻求防治老年性痴呆的有效措施 ,运用Morris水迷宫评价了中药补肾益智方 (由蛇床子、枸杞子、女贞子、人参等组成 )对由D_半乳糖致亚急性衰老加上Meynert核损毁所致阿尔茨海默病模型大鼠的学习记忆能力的保护作用。结果表明 :补肾益智方能够改善模型大鼠的学习记忆成绩 ,包括缩短模型大鼠在Morris水迷宫中寻找到平台的逃避潜伏期 (P <0 .0 1) ,延长其在原平台象限游泳的距离 (P <0 .0 5)。提示补肾益智方对阿尔茨海默病模型大鼠的学习记忆能力有一定的保护作用

    To explore the effects of Bushen Yizhi Decoction(BYD)on the hippocampal long_term potentiation (LTP) in rat model with Alzheimer's disease(AD),AD rat model was established by intraperitoneral injection of D_galactose and ibotenic acid injection into nucleus basalis of Meynert.Electrophysiological method was used to assess the changes of long_term potentiation of dentate gyrus.BYD groups were treated with gastric infusion of BYD for 4 weeks.The results showed that the differences of potentiation of excited postsynaptic...

    To explore the effects of Bushen Yizhi Decoction(BYD)on the hippocampal long_term potentiation (LTP) in rat model with Alzheimer's disease(AD),AD rat model was established by intraperitoneral injection of D_galactose and ibotenic acid injection into nucleus basalis of Meynert.Electrophysiological method was used to assess the changes of long_term potentiation of dentate gyrus.BYD groups were treated with gastric infusion of BYD for 4 weeks.The results showed that the differences of potentiation of excited postsynaptic potentials(EPSPs)were not significant among the five groups (P> 0.05 ),and potentiation of population spike(PS)amplitude in BYD groups were significantly different from AD model group.It is suggested that BYD exerts an effect in improving hippocampal LTP in rat model of AD.

    观察补肾益智方 (蛇床子、枸杞子、女贞子、人参等组成 )对老年性痴呆 (又称阿尔茨海默病 ,Alzheimer’sdisease ,AD)模型大鼠突触传递长时程增强现象 (long_termpotentiation ,LTP)的影响 ,旨在探讨该方治疗AD作用的神经生物学机制。应用D -半乳糖致亚急性衰老合并Meynert基底核损毁建立AD模型大鼠 ,用补肾益智方对AD大鼠灌胃治疗 4周 ,采用电生理学方法 ,检测大鼠海马齿状回强直刺激诱发的LTP的变化。结果表明 :强直刺激后 ,各组间兴奋性突触后电位增强差异不明显 (P >0 .0 5) ,补肾益智方高、低剂量组群体峰电位幅度增强较模型组明显提高 (P <0 .0 1或P <0 .0 0 1)。提示补肾益智方对AD大鼠齿状回LTP具有改善作用

    To study the effects of Bushen Yizhi Decoction (BYD) on the pathological reconstruction of synapse, morphometry was applied for the quantitative analysis of synapse in molecular layer of hippocampal dentate gyrus in rats with Alzheimer's disease (AD). AD rat model was established by intraperitoneral injection of D-galactose and ibotenic acid injection into nucleus basalis of Meynert. The results showed that the numerical density (ND) of the synapse was significantly increased while the surface density (SD) and...

    To study the effects of Bushen Yizhi Decoction (BYD) on the pathological reconstruction of synapse, morphometry was applied for the quantitative analysis of synapse in molecular layer of hippocampal dentate gyrus in rats with Alzheimer's disease (AD). AD rat model was established by intraperitoneral injection of D-galactose and ibotenic acid injection into nucleus basalis of Meynert. The results showed that the numerical density (ND) of the synapse was significantly increased while the surface density (SD) and average area were decreased in the synaptic contact zones in BYD group as compared with the AD model group (P< 0.01 ). Meanwhile, there was no significant difference in ND, SD and average area between BYD group and normal control group (P> 0.05 ).It is suggested that BYD could inhibit the pathological reconstruction of hippocampal synapse in AD rats, and this may be one of the neurobiological mechanisms of BYD for the improvement of learning and memory.

    应用体视学定量分析方法 ,观察补肾益智方 (由蛇床子、枸杞子、女贞子、人参、首乌等组成 )对D -半乳糖致亚急性衰老合并Meynert核损毁的老年性痴呆 (又称阿尔茨海默病 ,Alzheimer’sdisease ,AD)模型大鼠海马齿状回分子层突触病理性重构的影响。结果显示 ,补肾益智方灌胃组大鼠突触数密度较AD模型组明显增加 (P <0 .0 1)、面密度和平均面积显著减少 (P <0 .0 1) ,而与正常对照组比较无明显差异 (P >0 .0 5) ,表明该方具有阻止AD模型大鼠海马突触病理性重构的作用 ,提示对突触可塑性的影响可能是该方改善学习记忆的神经生物学机制之一

     
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