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  deafness
     Pedigree analysis and sequence analysis of mtDNA 12SrRNA,tRNA~(Leu(UUR)),tRNA~(Ser(UCN)) gene in nonsyndromicinherited deafness pedigrees
     非综合征型遗传性家系系谱分析及mtDNA 12SrRNA tRNA~(Leu(UUR)) tRNA~(Ser(UCN))基因突变分析
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     maximum discrimination scor (PBmax) in normal hearing (181 ears) was 90. 8±3. 7%, in conductive deafness (85 ears) was 87. 0±5. 6%, in sensorineural deafness (86 ears) was 80. 2±11. 5%;
     最大识别率(PBmax):正常听力(181耳)90.8±3.7%,传导性(85耳)87.0±5.6%,感音神经性(86耳)80.2±11.5%;
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     It was observed that TXB 2 and the ratio of TXB 2 to 6 keto PGF 1a (T/P) significantly increased,while 6 keto PGF 1a significantly decreased in the sudden deafness patients.
     结果:突患者TXB2、TXB2/6KPGF1a(T/P)比值明显增高,6KPGF1a显著降低;
短句来源
     Detection of mutation in mitochondrial 12S and 16S ribosomal RNA genes in aminoglycoside-induced deafness
     氨基糖苷类抗生素致家系线粒体DNA 12S和16S rRNA基因突变分析
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     Results The effective rate of treatment was 59. 0% in 78 cases of sudden deafness with vertigo and the effective rate was 87.77%, 139 cases of sudden deafness without vertigo (P <0.05).
     结果 突发性伴眩晕78例,治疗有效率为59.0%,不伴眩晕患者139例,治疗有效率为87.77%(P<0.05);
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  deaf
     Results: At the pretreatment of deaf guinea pigs, the threshold of ABR was 55 .30±6.73 in control group,50.20±7.32 in treatment group 1 and 47.60±8.10 in treatment group 2. After two months, the threshold of ABR was 64.60±6.63,38.10± 9.70 ,and 46.50±7.40 respectively.
     结果 :致豚鼠治疗前ABR阈 :对照组 5 5 .30± 6 .73,治疗组 1:5 0 .2 0± 7.32 ,治疗组 2 :47.6 0± 8.10 ; 2个月后ABR阈分别为 :6 4.10± 6 .6 3,38.10± 9.70和 46 .5 0± 7.40。
短句来源
     groupof profound hearing loss:7.7±5.7、7.1±5.5、5.6±5.6and5.1±5.5dB.Conclusion Using MFSSR data topredict the deaf children’s, hearing threshold and can help us to fit hearing aids for the deaf children whofailed in behavior audiometry.
     极重度组:7.7±5.7、7.1±5.5、5.6±5.6和5.1±5.5dB。 结论 MFSSR反应阈值可以预测行为听力图,为低龄儿童及难以检测行为听力的患儿验配助听器提供依据。
短句来源
     Detection and Comparison of ABR and 40 Hz AERP in 401 Deaf Children
     401例儿的ABR及40Hz AERP测试结果分析
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     the Chinese deaf learn English in trilingual and bicultural environment;
     中国人英语学习是三语的语言环境—汉语书面语、中国手语以及英语;
短句来源
     Application of Multiple Intelligences Theory in Deaf Education
     试谈多元智能理论在教育中的运用
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  hearing loss
     group of severe hearing loss:14.3±8.8、10.3±8.4、10.8±9.0 and 9.1±10.0dB;
     重度组:14.3±8.8、10.3±8.4、10.8±9.0和9.1±10.0dB;
短句来源
     Their results in different hearinggroups at the four frequencies were: group of mild-severe hearing loss:16.5±11.8、15.0±13.3、12.7±17.4and 11.5±12.7dB;
     在中重度、重度和极重度三组儿0.5k、1k、2k和4kHz处两种测试的阈值差分别为中重度组:16.5±11.8、15.0±13.3、12.7±17.4和11.5±12.7dB;
短句来源
     Screening of GJB2 235delC mutation,SLC26A4 IVS7-2A>G mutation and mtDNA 12SrRNA A1555G mutation in severe-profound hearing loss patients in Zhuozhou and Gaobeidian,Hebei province
     河北涿州、高碑店市特教学校非综合征性分子病因学分析——GJB2 235delC突变、SLC26A4 IVS7-2A>G突变和线粒体DNA 12SrRNA A1555G突变筛查报告
短句来源
     Connexins are translated by the gap junction protein genes that belong to one gene family. GJB2 (Cx26), GJB3 (Cx31), GJB6(Cx30), GJA1 (Cx43), and GJB1 (Cx32) are responsible for hereditary hearing loss.
     连接蛋白是同一基因家族编码,其中GJB2(Cx26)、GJB3(Cx31)、GJB6(Cx30)、GJA1(Cx43)、GJB1(Cx32)都与遗传性有关。
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     Long term exposure to excessive noise will cause noise-induced hearing loss (NIHL).
     众所周知,过多过强的噪声暴露常引起噪声性听力损失,继而导致噪声性(Noise-induced hearing loss,NIHL)。
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  anacusis
     Objective:To investigate the risk factors and clinical features of anacusis or severe deafness associated with chronic suppurative otitis media (CSOM ).
     目的 :研究慢性化脓性中耳炎 (CSOM )引起全或极度的相关危险因素。
短句来源
     The surgical procedure should be performed earlier to eradicate the chronic or acute infection and to avoid anacusis or severe deafness.
     胆脂瘤和中耳感染如不及时控制可能导致全或极度
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  deafness
Inherited LQTS is represented by the autosomal dominant Romano-ward syndrome (RWS), which is not accompanied by congenital deafness, and the autosomal recessive Jervell and Lange-Nielsen syndrome (JLNS), which is accompanied by congenital deafness.
      
Frequency of the 35delG Mutation of the Connexin 26 Gene (GJB2) in Patients with Non-Syndromic Recessive Deafness from Bashkorto
      
Prevalence and Molecular Genetic Typing of Nonsyndromic Sensorineural Deafness in Chuvash Republic
      
Summarized genetic epidemiological characteristics of nonsyndromic sensorineural deafness in six raions of Chuvash Republic (Cheboksary, Kanash, Morgaushi, Tsivil'sk, Mariinski Posad, and Alatyr') are presented.
      
Forty-five families (60 affected individuals) with autosomal recessive (AR) and 8 families (18 affected individuals) with autosomal dominant (AD) nonsyndromic sensorineural deafness (NSSD) were identified.
      
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  deaf
Background: Prelingually deaf persons usually gain only a rudimentary command of speech and prefer sign language to communicate within the deaf community without the handicap they experience in the hearing world.
      
Method: Data were collected with the help of semi-structured interviews; with the deaf patients these were conducted in German sign language.
      
Comparison of the verbal and visual language skills of the two deaf groups revealed a substantial deficit among the deaf schizophrenics.
      
An innovative and reliable way of measuring health-related quality of life and mental distress in the deaf community
      
Mental distress and quality of life in a deaf population
      
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  hearing loss
In addition to congenital ichthyosis he had also strabismus, horizantal nystagmus, bilateral neurosensory hearing loss, hepatomegaly and splenomegaly.
      
Hearing loss up to 30 dB (preserved socially adequate hearing) was found in 25 workers.
      
Some measures for preventing occupational noise-induced hearing loss were suggested.
      
Conclusion: The primary symptom of the patient was unilateral hearing loss.
      
In 73% of the cases the EAEP indicated the retrocochlear site of the lesion; in 27%, however, the results did not localize the exact site of the lesion owing to a lack of waves I, II and III due to a pronounced hearing loss.
      
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