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微血管通透性     
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  microvascular permeability
     Changes of Rat Brain Microvascular Permeability after Cerebral Ischemia-Reperfusion Injury
     大鼠脑缺血再灌注后微血管通透性变化
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     Myocardial contraction function, myocardial microvascular permeability, volume of regional myocardial blood flow, levels of myocardial myosin light chain 1(CMLC1), myocardial NF-κB activity, mygloperoxidase (MPO), TNFα mRNA expression and levels of myocardial TNFα were observed.
     检测心肌收缩功能、心肌微血管通透性分值、心肌局部血流量、血浆中心肌肌球蛋白轻链 1(CMLC1)、心肌组织核转录因子 (NF -κB)活性、髓过氧化物酶 (MPO)、TNFαmRNA表达和心肌组织TNFα含量。
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     Quantitative study oil organ microvascular permeability in rats
     正常大鼠不同脏器微血管通透性的定量研究
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     Methods The mRNA expressions of PLA 2 Ⅰ and PLA 2 Ⅱwere detected by blot hybridization,the activity of low molecular phospholipase A 2 in pulmonary homogenate was assayed with biochemistry, and simultaneously blood gases, lung water content and pulmonary microvascular permeability were measured.
     方法 利用斑点杂交技术检测肺内低分子磷脂酶A2 (PLA2 Ⅰ、PLA2 Ⅱ )mRNA表达量、肺含水量及肺微血管通透性
短句来源
     AIM and METHODS: The animal model of acute lung injury (ALI) caused by intratracheal instillation of lipopolysaccharides(LPS) in vivo and human peripheral blood polymorphonuclear neutrophil (PMN) in vitro were used to study the effects of sodium nitroprusside (SNP), nitric oxide (NO) donor, on LPS-induced PMN accumulation, microvascular permeability and PMN apoptosis.
     目的和方法 :应用气管内滴注脂多糖 (LPS)致SD大鼠急性肺损伤 (ALI)模型和体外培养人血中性粒细胞 (PMN) ,观察一氧化氮 (NO)供体硝普钠 (SNP)对LPS所致肺内PMN聚集、微血管通透性增高及PMN凋亡的影响。
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  capillary permeability
     The result indicate that external application of the Chinese herbs could enhance local capillary permeability of the wound surface.
     实验结果表明:外用中药可增强创面局部微血管通透性
短句来源
     The values of CBF,CBV,MTT,PS were different in different tumor and PS images could clearly display essential part of brain tumors and capillary permeability.
     不同类型肿瘤的CBF、CBV、MTT及PS值不尽相同,灌注伪彩图表现不同; PS图能清晰显示脑肿瘤的实质部分及微血管通透性
短句来源
     Conclusion: MSCTPI provides a new method in quantificational research of brain tumor flow perfusion and is valuable in realizing inner flow state of brain tumor and change of capillary permeability.
     结论:MSCT灌注成像对脑肿瘤血流灌注的定量研究提供了新的方法,对了解脑肿瘤内部血流状态及微血管通透性的改变有一定的价值。
短句来源
     PS images could clearly display essentral part of brain tumor and capillary permeability than other images.
     PS图较其他灌注图更能显示肿瘤的大小、轮廓及肿瘤的微血管通透性
短句来源
  microvessel permeability
     Effects of erythrocyte cardioplegia on myocardial protection and microvessel permeability
     红细胞停跳液的心肌保护作用及对心肌微血管通透性的影响
短句来源
     The changes of biological variables, including lung wet/dry weight ratio,count of polymorphonuclearneutrophil(PMN)and protein level in bronchoalveolar lavage fluid(BALF),and pulmonary microvessel permeability,were analyzed in bilateral lungs,and changed significantly the expression of TNF-αmRNA in the lung was determined with reverse transcription polymerase chain raction (RT-PCR)technique at 1,6 hours,and 24 hours after unilateral chest blunt trauma.
     在伤后1、6、24 h测定双肺生物学标志(肺湿/干重比、支气管肺泡灌洗液中性粒细胞、蛋白含量、肺微血管通透性)的变化,RT-PCR法测TNF-αmRNA在双侧肺组织中的表达。
短句来源
     The Changes of Microvessel Permeability in Different Organ Explosive Injury on the Chest-abdomen of Rabbits
     兔胸腹部爆炸伤后不同器官微血管通透性的变化
短句来源
     Methods The model of explosive blast wound on rabbits and the method of 125I_albumin dilution have been used for investigating microvessel permeability changes of heart, lung, kidney, liver and brain in rabbits.
     方法采用雷管对兔的爆炸性冲击损伤模型 ,以及 125I-白蛋白标记的方法 ,观察爆炸性冲击波对兔心、脑、肺、肾、肝组织中的微血管通透性的影响。
短句来源
     Conclusions The explosive blast can cause hematocrit increase,plasma loss, and can increase the microvessel permeability of heart, lung, kidnry,liver and brain in rabbits.
     结论爆炸冲击波能引起红细胞压积明显增加 ,血浆丢失 ,以及心、脑、肺、肾组织中微血管通透性的增加。
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  micrangium permeability
     Objective :To study the effect of micrangium permeability and microcirculation of Xuemaining granule on animal model of thrombotic disease.
     目的 :探讨血脉宁颗粒对血栓性疾病动物模型微血管通透性及微循环的影响。
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  microvascular permeability
The effect of selenium and vitamin E on microvascular permeability of rat organs
      
The effects of dietary sodium selenite and vitamin E on the microvascular permeability of rat organs such as heart, brain, kidney, liver and eye were investigated by using the Evans blue leakage method.
      
These results suggested that low or high sodium selenite and vitamin E contents in diet could alter the microvascular permeability of different organs in different manners.
      
This review addresses the idea that Hcy induces microvascular permeability by attenuating the GABA-A/B receptors and increasing redox stress, which activates a disintegrin and metalloproteinase that suppresses tissue inhibitors of metalloproteinase.
      
A Role for the Endothelial Glycocalyx in Regulating Microvascular Permeability in Diabetes Mellitus
      
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  capillary permeability
Release of inflammatory mediators in sepsis, pneumonia or after shock result in an increased pulmonary capillary permeability.
      
Actions mediated by 5-HT2 receptors which could be of relevance to migraine comprise cranial vasoconstriction, increased cranial capillary permeability and platelet aggregation, and some central nervous system effects and neuroendocrine functions.
      
The causes of hyponatremia were attributed to rickettsial vasculitis and increased capillary permeability in the first patient and to the syndrome of inappropriate anti-diuretic hormone (ADH) secretion in the second patient.
      
The vascular fluid leakage is thought to result from an increased capillary permeability of mesothelial surfaces under the action of one or several vasoactive ovarian factor(s) produced by the multiple corpora lutea.
      
Vascular endothelial growth factor (VEGF) induces endothelial cell proliferation and an increase in capillary permeability.
      
更多          
  microvessel permeability
As a result, the nature of the increased microvessel permeability in diabetes mellitus and indeed its causes are poorly understood.
      
Baseline measures of microvessel permeability and Ppa in the PAR1-null preparations were indistinguishable from those in normal lungs.
      
It induces the vascular stroma not only as a direct endothelial cell mitogen, but also as a potent mediator of microvessel permeability.
      
  其他


The early changes in microcirculation of burns are of significant pathophsi-ologic importance. The experimental use of cimetidine, an antagonist of histamine H2-receptor, demonstrated its better effectiveness against the changes in microva-scular permeability. No observations on blood flowing state were made. The post-burn adherence of White blood cells to venules were noted recently by Eriksson, which was considered as a pathophysiologic mechanism of postburn tissue edema.A 20% body surface area third degree...

The early changes in microcirculation of burns are of significant pathophsi-ologic importance. The experimental use of cimetidine, an antagonist of histamine H2-receptor, demonstrated its better effectiveness against the changes in microva-scular permeability. No observations on blood flowing state were made. The post-burn adherence of White blood cells to venules were noted recently by Eriksson, which was considered as a pathophysiologic mechanism of postburn tissue edema.A 20% body surface area third degree burn is produced in rats, which is designed an experimental model for observation of the changes in mesenteric microcirculation and a comparative relationship between the adherence of white blood cells and the tissue water content, a comparative therapeutic effect of early resuscitation with Ringer's lactate solution and the results of blood flow in microcirculation, adherence of white blood cells, and aggregation of red blood cells obtained by administration of cimtldine.The chief findings of mesentery microcirculation of burned rat are venule dilatation with progressive slowing and sludging of blood flow; preipheral rotation and adherence of white blood cells; progressive aggregation of red blood cells. No edema is found by measuring water content of remote tissue, better therapeutic effects are shown both by resuscitation of fluid and administration of cimetidine, with the former in predeminanee. The present study indicates:1.Hypovolemia caused by postburn fluid loss and venule dilatation is a major factor leading to worsening of microcircuration blood flow. In order to maintain normal flow state, to assure abetter tissue perfusion, the administration of cimetidine associated with fluid resuscitation may be beneficial.2.Low flow state of microcirculation is the underlying cause, but not the only one of adherence of white blood cells postburn.3.Adherence of white blood cells within venules does not lead to tissue edema, it is, therefore, not the cause of remote tissue edema. The point of view of Eriksson merites further discussion.4.The problems of aggregation of red blood cells and remote unburned tissue edema are also discussed in the present artical. The primary danger of aggregation of red blood cells is to make worsen of the microcirculation, which aggravates the deficiency of blood and oxygen; timely correction of hypovolumia is a preventive measure of primary importance. The experimental model revealed that if no fluid is administered, some, then shows slightly mild dehydration. This might probably be due to the compensatory responce of the remote area to hemoconcen-tration.

烧伤后早期微循环的变化是具有重要意义的病理生理过程。对于伤后微血管通透性的变化,目前对H_2受体拮抗剂甲氰咪胺的实验研究取得了较好的效果,但对血流状态的影响未予观察。伤后微循环中血细胞的变化,近年Eriksson特别注意到伤后微静脉内白细胞的粘附现象,认为是防后组织水肿的病理机制。 本实验用大鼠70只,体重在180~220g,性别不拘,实验前不禁食、不禁水,分为对照、烧伤、治疗三组。实验设计用20%三度烧伤大鼠进行肠系膜微循环变化的观察,并在作白细胞粘附观察的同时,测定组织实验含水量,比较两者的关系;并观察比较使用乳酸林格氏溶液复苏纠正低血容量和投用甲氰咪胺后对微循环血流状态、白细胞粘附和红细胞凝聚的影响。 大鼠20%三度烧伤后肠系膜微循环在活体观察下的主要发现是:微静脉扩张;血流缓慢,渐呈淤泥,钟摆样;白细胞发生贴壁翻滚、粘附,红细胞凝聚进行性加重。在观察白细胞粘附的同时测定相应各时期远隔组织的含水量,未发现组织水肿。液体复苏和甲氰咪胺对改善微循环的流态均具有较好的效果。尤其是液体复苏,效果更为满意。 本文认为: 1.伤后体液丢失和微静脉扩张所引起的低血容量是导致微循环流态恶化的因素。为维持流态...

烧伤后早期微循环的变化是具有重要意义的病理生理过程。对于伤后微血管通透性的变化,目前对H_2受体拮抗剂甲氰咪胺的实验研究取得了较好的效果,但对血流状态的影响未予观察。伤后微循环中血细胞的变化,近年Eriksson特别注意到伤后微静脉内白细胞的粘附现象,认为是防后组织水肿的病理机制。 本实验用大鼠70只,体重在180~220g,性别不拘,实验前不禁食、不禁水,分为对照、烧伤、治疗三组。实验设计用20%三度烧伤大鼠进行肠系膜微循环变化的观察,并在作白细胞粘附观察的同时,测定组织实验含水量,比较两者的关系;并观察比较使用乳酸林格氏溶液复苏纠正低血容量和投用甲氰咪胺后对微循环血流状态、白细胞粘附和红细胞凝聚的影响。 大鼠20%三度烧伤后肠系膜微循环在活体观察下的主要发现是:微静脉扩张;血流缓慢,渐呈淤泥,钟摆样;白细胞发生贴壁翻滚、粘附,红细胞凝聚进行性加重。在观察白细胞粘附的同时测定相应各时期远隔组织的含水量,未发现组织水肿。液体复苏和甲氰咪胺对改善微循环的流态均具有较好的效果。尤其是液体复苏,效果更为满意。 本文认为: 1.伤后体液丢失和微静脉扩张所引起的低血容量是导致微循环流态恶化的因素。为维持流态正常,确保组织良好灌流,在液体复苏的同时联合应用H_2受体拮抗剂甲?

The microcirculation of the bulbar conjunctiva and finger nail fold was observed among 108 cases of diabetes mellitus without fundoscopic retinopathy (group 1), 108 cases with fundoscopic retinopathy (group 2) and 100 cases of non-diabetics as control (group 3). It was found that there were more marked disturbances in microvessels, blood flow and microvascular permeability of the bulbar conjunctiva and nail fold in groups 1 and 2 than in group 3. Moreover, there were higher incidences of disturbances of the...

The microcirculation of the bulbar conjunctiva and finger nail fold was observed among 108 cases of diabetes mellitus without fundoscopic retinopathy (group 1), 108 cases with fundoscopic retinopathy (group 2) and 100 cases of non-diabetics as control (group 3). It was found that there were more marked disturbances in microvessels, blood flow and microvascular permeability of the bulbar conjunctiva and nail fold in groups 1 and 2 than in group 3. Moreover, there were higher incidences of disturbances of the microvessels and blood flow in the bulbar conjunctiva than in the nail fold. No significant difference in the walls of microvessels was found between groups 1 and 2 but the distubances of blood flow and microvascular permeability in group 2 were more severe than those in group 1. Significant abnormality of blood flow in the bulbar conjunctiva was found in the diabetics with proliferative retinopathy. The results suggest that the disturbances of microcirculation in diabetics might be the pathological basis in the development of diabetic retinopathy.

本文对100名健康人(对照组),108例糖尿病不伴有视网膜病变的患者(非糖网组),108例糖尿病伴有视网膜病变的患者(糖网组)进行眼球结膜和甲皱微循环的观察。结果表明:糖尿病患者眼球结膜和手指甲皱微循环,在微血管、微血流及微血管通透性三方面均有明显的障碍。但眼球结膜微循环的微血管和微血流的异常发生率高于手指甲皱。伴有视网膜病变的糖尿病患者微血流流态及微血管通透性的异常发生率明显地高于不伴有视网膜病变的糖尿病患者,但两者微血管形态的异常没有明显的差别。视网膜病变严重的糖尿病患者眼球结膜微血流流态的障碍更为严重。上述结果提示:糖尿病患者有明显的微循环障碍,而这是发生视网膜病变的病理生理学基础。

Smoke inhalation injury was inflicted to 13 goats with chrnoic lung lymph fistula. The contents of thromboxane A2(TXA2) and prostacyclin(PGI2) in plasma, lung lymph fluid, and lung tissues were measured in the 6-hour-periodafter injury with radioimmunoassay. Extravascular lung water(EVLW), thequantity of lymph fluid (QL), the ratio of protein contents in lung lymph and plasma(L/P), hemodynamic parameters, blood gases, platelet count and WBCcount were also determined.It was found that EVLW. QL and L/P increased...

Smoke inhalation injury was inflicted to 13 goats with chrnoic lung lymph fistula. The contents of thromboxane A2(TXA2) and prostacyclin(PGI2) in plasma, lung lymph fluid, and lung tissues were measured in the 6-hour-periodafter injury with radioimmunoassay. Extravascular lung water(EVLW), thequantity of lymph fluid (QL), the ratio of protein contents in lung lymph and plasma(L/P), hemodynamic parameters, blood gases, platelet count and WBCcount were also determined.It was found that EVLW. QL and L/P increased markedly in the 1st hour, reached the peak of elevation in the 2nd hour, and remained higher than the pre-injury level in the 6th hour after injury, which implies the existence of permea-bility pulmonary edema . Both TXA2 and PGI2 also increased markedly after, injury. The changes of TXA2 and TXA2/PGI2 ratio were in parallelto those of EVLW, QL and L/P in time course and in degree, which implies that the changes of TXA2 and PGI2 might play an important role in the genesis of permeability pulmonary edema. The mechanism might be the direct injury of the two substances and/or the precipitation of platelet and WBC aggregation which can release certain chemical mediators to increase the permeability of the capillaries. In addition, TXA2 might also result in the early elevation of pulmonary arterial pressure.

本实验制作慢性肺淋巴瘘模型13只,行烟雾吸入致伤。观测了伤后6小时内TXA_2和PGI_2含量,EVLW、Q_L和L/P、血流动力指标、血气、血小板及白细胞计数。EVLW、Q_L和L/P 1小时均明显增加,2小时达高峰,6小时仍在较高水平,提示为通透性肺水肿。TXA_2和PGI_2伤后均增加,TXA_2和TXA_2/PGI_2比值与EVLW和L/P在时间和程度上变化一致,提示它们可能与肺水肿形成有关。其机理可能通过直接作用和/或使血小板、白细胞聚集,释放化学介质,导致肺微血管通透性增加。此外,TXA_2在早期PAP增加中也可能起一定作用。

 
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