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血瘀
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  “血瘀”译为未确定词的双语例句
    Conclusions:The curative effects of ATM on ACI of Qixu Xueyu(气虚血瘀) and Fengtan Zuluo(风痰阻络) type are confirmed but on Ganyang Baokang(肝阳暴亢) and Tanre Fushi(痰热腑实) type are ordinary and it is possibly related to property of its supporting Yang of warm heat.
    结论:抗血栓合剂对气虚血瘀型、风痰阻络型急性脑梗死疗效肯定,对肝阳暴亢、痰热腑实型等疗效一般,可能与抗血栓合剂温热助阳之性有关。
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    Results:In pa- tients with BSS the index of ED ( 0 .4 67± 0 .14 5 ) was significantly lower than that ( 0 .5 0 8± 0 .14 1) in control group ( P <0 .0 5 ) ,furthermore its decrease in patients with CI( 0 .4 43± 0 .15 6) was much m ore significant ( P<0 .0 1) .
    结果 :血瘀证 TIA患者红细胞变形指数 ( 0 .4 67± 0 .14 5 )显著低于正常对照组 ( 0 .5 0 8± 0 .14 1) ,P<0 .0 5 ,脑梗死患者红细胞变形指数 ( 0 .4 43± 0 .15 6)降低更为明显( P<0 .0 1) ;
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    Results: Compared with the control group, the level of ET and ratio of ET/NO raised significantly in the ACI group and the BSS group (P < 0. 01 ).
    结果:血瘀证组、ACI组ET、ET/NO均显著高于健康对照组(P<0.01);
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    The levels of ET and ratio of ET/NO of mild, moderate and serious BSS increased in order, but only the serious BSS in comparing with mild, moderate group of BSS had significant difference (P < 0. 01).
    血瘀证组轻、中、重度组ET、ET/NO依次增高,重度组与轻、中度组比较有显著性差异(P<0.0),中度组与轻度组比较仅ET有显著性差异(P<0.01);
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    The NO levels of ACI group, non-BSS group in comparing with healthy Control, there was significant decrease (P < 0. 05, P < 0. 01 ).
    ACI组、血瘀证组、非血瘀证组NO含量与健康对照组比较均显著降低(P<0.05,P<0.01);
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37 cases with hemorrahage of upper digestive tract wereanalyzed with blood rheology to expound the relationship between the bleeding and stagnation of blood, The results were as follows:①Abnormal blood viscometer, ②The hyper function of the blood coagulation and fibrinolysis. These suggested that the breeding could cause the stagnation 0f blood, the later could simultaneously cause bleeding again.

本文报道通过对上消化道出血患者血液流变学指标的测定,探讨出血与血瘀的关系。结果提示:出血患者的血液流变性异常,凝血和纤溶功能活跃,表明“出血可致血瘀,血瘀可致再出血”的理论是有客观依据的。

Objective:To explore the relationship of blood stasis syndrome of cerebral embolism to fibrinolytic function of vascular endotheliocyte and platelet activating level.Methods:The plasma tissue plasminogen activator(tPA),plasminogen activating inhibitor(PAI) and granule membrane protein140(GMP140) contents were detected in 30 cases of blood stasis syndrome of cerebral embolism.Results:It was obvious difference of fibrinolytic function and platelet activation between cerebral embolism patients and health subjects.Plasma...

Objective:To explore the relationship of blood stasis syndrome of cerebral embolism to fibrinolytic function of vascular endotheliocyte and platelet activating level.Methods:The plasma tissue plasminogen activator(tPA),plasminogen activating inhibitor(PAI) and granule membrane protein140(GMP140) contents were detected in 30 cases of blood stasis syndrome of cerebral embolism.Results:It was obvious difference of fibrinolytic function and platelet activation between cerebral embolism patients and health subjects.Plasma tPA〔(131±047)kU/L〕 level in patients was lower obviously than that in control group〔(189±050)kU/L〕,but PAI〔(875±232)×103 AU/L〕 and GMP140 level〔(2785±860)μg/L〕 were higher obviously than control group〔(661±167)×103AU/L and (1331±342)μg/L,respectively〕.Conclusions:The pathogenesis of blood stasis syndrome of cerebral embolism was related closely to decrease of fibrinolytic function and change in platelet activation.

目的:探讨脑血栓血瘀证与血管内皮细胞纤溶功能及血小板活化程度的关系。方法:检测30例脑血栓血瘀证患者急性期血浆组织型纤溶酶原激活剂(tPA)、纤溶酶原激活剂抑制物(PAI)和α颗粒膜蛋白(GMP140)的含量。结果:脑血栓血瘀证患者与健康对照组的纤溶功能和血小板活化指标有明显差异。患者血浆tPA〔(1.31±0.47)kU/L〕明显低于对照组〔(1.89±0.50)kU/L〕,而PAI和GMP140〔分别为(8.75±2.32)×103AU/L和(27.85±8.60)μg/L〕则明显高于对照组〔分别为(6.61±1.67)×103AU/L和(13.31±3.42)μg/L〕。结论:脑血栓血瘀证的发病与纤溶功能减退及血小板活化程度的变化密切相关。

Recently, in hematological examination of ischemic stroke, we find Granular Membrane Protein-140(GMP-140)plays a important role in platelet activating and agglutinating.Now generally acknowledged pathogenesis of ischemic stroke is atherosclerosis and platelet abnormal agglutinating, the consequence is throwbosis.Chinese traditional medicine considers that the stagnation of vital energy and blood stasis is the carse of ischemic stroke.Then we can see,two kinds of medicine have some similarity in pathogensis of...

Recently, in hematological examination of ischemic stroke, we find Granular Membrane Protein-140(GMP-140)plays a important role in platelet activating and agglutinating.Now generally acknowledged pathogenesis of ischemic stroke is atherosclerosis and platelet abnormal agglutinating, the consequence is throwbosis.Chinese traditional medicine considers that the stagnation of vital energy and blood stasis is the carse of ischemic stroke.Then we can see,two kinds of medicine have some similarity in pathogensis of ischemic stroke.In this thesis, we observde 30 ischemic stroke patients and 30 healthy people.From examining,we found in the patients' blood serum, the GMP-140 content increased distinctly.So we conclude that GMP-140 can be regard as one of objective hematological examinations of ischemic stroke.

近年来,在缺血性中风的血液学检测方面,发现了血小板颗粒膜蛋白(GMP—140)在血小板活化、凝集过程中起着重要作用.缺血性中风的主要病机是与动脉粥样硬化和血小板异常凝集形成血栓有关.祖国医学认为缺血性中风是由于各种原因最终导致气滞血瘀而发病.两者在发病机理的认识上有相似之处.本文通过对30例缺血性中风患者及30例健康者的GMP—140含量的测定,发现缺血性中风者的含量明显高于健康者.认为缺血性中风病中血小板CMP—140含量增高可作为该病的客现血液学检测指标之一。

 
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