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   铅中毒 的翻译结果: 查询用时:0.058秒
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铅中毒     
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  lead poisoning
     Results: Among the 588 cases, maternal blood lead level (Bpb) were 0. 424±0.170 μmol/L, Bpb≥0.483 μmol/L ones were 19.05%, umbilical cord Bpb were 0.417±0.164 μmol/L, lead poisoning rate was 18.71%.
     结果:产妇血铅水平0.424±0.170μmol/L,Bpb≥0.483μmol/L者占19.05%,新生儿脐血铅水平0.417±0.164μmol/L,铅中毒率为18.71%;
短句来源
     Results The students mean blood lead level was(97.72±26.92)μg/L,lead poisoning rate accounting for 39.16%;
     结果学生血铅浓度为(97.72±26.91)μg/L,铅中毒率为39.16%;
短句来源
     In XRCC1 A399Q polymorphism,there were 0.37-fold and 0.52-fold decreased risk of lead poisoning in heterozygous mutation genotype(GA) and mutation genotype(GA+AA) compared with wild genotype(GG),respectively.
     XRCC1 A399Q的杂合突变型(GA)和突变型(GA+AA)分别减少铅中毒易感性达0.37和0.52倍;
短句来源
     Results The lead poisoning rate of children in Hefei was 9.1%.
     结果合肥市儿童铅中毒率为9.1%;
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     In XRCC1 A194W polymorphism,heterozygous mutation genotype (CT),homozygous mutation genotype(TT) and mutation genotype(CT+TT) could increase 2.03-fold,2.59-fold and 2.12-fold risk of lead poisoning compared with mild genotype(CC),respectively.
     XRCC1 A194W的杂合突变型(CT)、纯合突变型(TT)和突变型(CT+TT)分别增加铅中毒易感性达2.03,2.59和2.12倍。
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  plumbism
     Results The average level of blood lead among 440 children was 0.625±0.319 μmol/L, the prevailing rate of plumbism was 56.4% according to the level of plumbism ( 0. 483 μmol/L).
     结果440名儿童血铅为0.625±0.319μmol/L,其中铅中毒(血铅值≥0.483μmol/L)流行率为56.4%。
短句来源
     The total misdiagnosis rate was 9.88% (16/42). The misdiagnosis rates of occupational and nonoccupational plumbism were 2.84%(4/41) and 57.14 (12/21) respectively(P<0.01).
     ⑤误诊率为9.88% ( 16/ 162 ) ,职业性铅中毒误诊率为 2 .84 % ( 4 / 141) ,非职业性铅中毒误诊率为 57.14%( 12 / 2 1) ,两者有非常显著性差异 (P <0 .0 1) ;
短句来源
     CLINICAL ANALYSIS OF 148 CASES IN PROFESSIONAL PLUMBISM (ABSORPTION STATE OF PLUMBUM)
     职业性铅中毒(铅吸收)148例临床分析
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     CLINICAL ANALYSIS OF 162 CASES OF PLUMBISM
     铅中毒162例临床分析
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     141 cases (87.04%) were confirmed to be of occupational plumbism and 21 cases (12.96%) belonged to nonoccupational plumbism.
     ②职业性铅中毒141例 ,占 87.0 4 % ,非职业性铅中毒 2 1例 ,占 12 .96% ;
短句来源
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  saturnism
     Results:Average value of the blood lead level in 883 children is 84.24 μg/l(1 μg/l=0.00483 μg mol/l),32.84% of them surpasses international diagnosis standard (100 μg/L)of children saturnism.
     结果 :883名儿童血铅水平均数为 84.2 4 μg/L( 1 μg/L=0 .0 0 4 83μmol/L) ,其中高于目前国际公认的儿童铅中毒诊断标准 1 0 0μg/L的比例是 32 .84%。
短句来源
     STUDY ON BRAINSTEM AUDITORY EVOKED POTENTIALS OF PATIENTS WITH CHRONIC SLIGHT SATURNISM
     慢性轻度铅中毒患者的脑干听觉诱发电位研究
短句来源
     Conclusion:Currently,blood lead level and saturnism are high in children of Nanchang city,Which should bring to attention of the parent and the department concerned
     结论 :南昌市城区儿童血铅水平及铅中毒率已较高 ,应引起家长及有关方面的关注。
短句来源
     It could not be excluded that the decrease of neuro-conductive velocity in saturnism patients seemed to be related to the lowering of magnesium and calcium induced by the lead interference.
     铅中毒引起神经传导速度减慢与铅干扰镁、钙低下的因素不能排除。
短句来源
     Saturnism is a very common toxicosis disease in animals.
     铅中毒是动物较为常见的一种重金属中毒病。
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  pb poisoning
     The content of Zn,Cu,Fe,Ca and Mn in blood of 68 children with Pb poisoning was determined before and after the treatment by traditional Chinese medicine.
     为探讨中药驱铅对儿童血Pb及微量元素Zn、Cu、Fe、Ca、Mn水平的影响 ,检测了 68例儿童铅中毒患儿服用中药驱铅前后的血Pb及微量元素Zn、Cu、Fe、Ca、Mn水平。
短句来源
     The results showed that the content of Pb in blood of ADHD was 157 94±41 13 μg/L,that of contrast group was 94 73±29 93 μg/L. Incidence rate of children Pb poisoning in ADHD group was 88 75%.
     结果表明ADHD组血铅水平为 1 5 7 94± 4 1 1 3μg/L ,对照组为 94 73± 2 9 93μg/L ,其中儿童铅中毒发生率 ,ADHD组为 88 75 % ( 71 /80 ) ,对照组为 4 0 0 0 % ( 2 4 /60 ) ,经统计学处理 ,两组资料均具显著性差异 ;
短句来源
     Pb Metabolism and Perniciousness of Pb Poisoning to Children
     铅代谢及铅中毒对儿童的危害
短句来源
     Results At 17days, the blood pb of above three groups were 3.6±6.5μg/L, 410.8±46.63μg/L and >650μg/L,respectively,and belong to normal, middle pb poisoning, heavy pb poisoning.
     结果 第 17d断头处死各鼠 ,检测血铅值分别为 :对照组 3 5± 6 6 μg L、低铅组 4 10 8± 4 6 6 3μg L、高铅组均 >6 5 0 μg L ,正常组在正常范围内 ,低铅组已达中度铅中毒 ,而高铅组已达重度铅中毒
短句来源
     Methods Pb poisoning rat model was made by 0.02% lead acetate for four weeks, then fed by Vitamin C 100mg/kg.
     方法:大鼠自由饮用0.02%的醋酸铅四周造成铅中毒模型。
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  lead poisoning
Many ocular manifestations of lead poisoning have been reported so far.
      
Stable isotope mass spectrometry in childhood lead poisoning
      
However, coexposure to lead and ethanol could result in more serious depletion of calcium and magnesium, and this could be the cause of suspected synergism between alcohol consumption and lead poisoning.
      
Alterations in the hepatic enzymes following experimental lead poisoning
      
Whereas significant inhibition of succinic dehydrogenase was seen following lead poisoning, the activity acid and alkaline phosphatase increased with lead intoxication.
      
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  plumbism
Lead-exposed neonatal rats are frequently used as a model for plumbism in children.
      
Intravenous administration of 6 mg per kg lead acetate to rabbits resulted in plumbism with elevated erythrocyte lead levels and marked depression of activity of erythrocyte δ-aminolevulmic acid dehydratase.
      
The insensitivity of these erythrocyte enzymes to inhibition by lead excludes their usefulness for detection or diagnosis of plumbism.
      
In the in vitro inhibition test the same degree of inhibition of red cell P5N activity seen in hereditary red cell P5N deficiency was obtained by using a lead concentration 200-400 times higher than the lead levels detected in human plumbism.
      
Experimental plumbism in pigeons from the administration of metallic lead
      
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  saturnism
In spite of the considerable achievements of the investigations of the different aspects of the saturnism problem, the question of an early diagnosis of the widely spread professional poisoning is not yet concluded.
      
It was established in chronic experiments on dogs that the picture of experimental lead poisoning was to a considerable extent similar to that seen during clinical manifestations of saturnism in human beings.
      
The autoxidation of d-aminolevulinic acid, in the presence or absence of oxyhemoglobin has been proposed as a source of oxy and carbon-centred radicals in the cells of intermittent acute porphyria and saturnism carriers.
      
  pb poisoning
The authors propose that altered fatty acid metabolism may be responsible for some indications of Pb poisoning.
      
These results suggest that Pb exacerbates liver lipid peroxidation in PU rats and suggests the involvement of free radicals in the pathogenesis of Pb poisoning.
      
These negative results raise questions about the usefulness of chelation therapy in cases of acute Pb poisoning.
      
Screening programs for pediatric lead (Pb) poisoning routinely collect blood by finger stick onto specially designed filter paper (FP) for subsequent laboratory testing.
      
Most poisoned birds of the other species were emaciated but lacked other gross lesions caused by Pb poisoning.
      
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