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神经元细胞     
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  neuronal cells
     RESULTS:Positive expression of HIF 1 αwas observed in neuronal cells and glial cells,mainly in the gray matter,0.5-6 hours after injury.
     结果:损伤后0.5~6h主要在灰质中的神经元细胞和胶质细胞有HIF-1α的阳性表达。
短句来源
     Influence of AVP_(4-8) on MAPK activity in astrocytic glial and fetal neuronal cells in primary culture
     精氨酸升压素(4-8)对原代培养星状胶质细胞及胚胎神经元细胞中MAPK活性的影响(英文)
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     At 24 hours after epilepsy, apoptosis of neuronal cells in hippocampus could be seen in rats of model group, western medicine group, routine acupuncture group and acupoint embedding therapy group.
     致痫后24h,模型组、西药组、常规针刺组和穴位埋药线组的大鼠海马神经元细胞均可见不同程度的凋亡表现。
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     ② Ectogenous nerve growth factor of proper concentration 80 μg/L can accelerate the repair of hypoxic-ischemic neuronal cells and protect mitochondrion.
     ②适宜浓度(80μg/L)的外源性神经生长因子能够促进缺新生大鼠脑缺氧缺血性神经元细胞修复,保护线粒体。
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     Conclusion In 24 h after treatment with ketamine, the expression of synaptophysin was decreased in hippocampl neural cells, the number of apoptotic neuronal cells were increased, which may be involved in neurotoxicity caused by ketamine.
     结论注射氯胺酮后24h,SD大鼠海马神经元细胞大量凋亡,突触素表达减少可能和氯胺酮神经毒性有关。
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  neuron cells
     After using naloxone the increase range of the Ca2+i under hypoxia and after giving oxygen condition significantly reduced compared with hypoxia group which suggests that naloxone has stable action on hypoxia neuron Ca2+i and can decrease the increase of neuron Ca2+I to play a protective action on neuron cells.
     应用纳洛酮后,在缺氧条件下和缺氧后复氧期间的犤Ca2+犦i升高幅度较缺氧组明显减小,说明纳洛酮对缺氧的神经元犤Ca2+犦i具有一定的稳定作用,可以减轻神经元犤Ca2+犦i的升高,进而对神经元细胞起到保护作用。
短句来源
     Neuron cells were treated with different dosages of PAF for 24 hours,and pretreated with 5 μ mol/L BN52021.Then cells were stained with iodide or calcein to detect apoptosis.
     不同剂量PAF处理神经元细胞24h,并经PAF拮抗剂5μmol/LBN52021预处理;
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     Objective To establish a neuron cells model for Hantaan virus (HTNV) infection by using the cultures of cortical neurons of mouse embryo.
     目的 利用体外培养的昆明小鼠胚胎脑皮质神经元 ,感染汉滩病毒 (Hantaanvirus .HT NV) ,建立病毒感染的神经元细胞模型 ,供探讨病毒感染神经元后细胞的变化及损伤机制之用。
短句来源
     Results The marked neuron cells were found at the anterior horn of spinal cord at 3 groups.
     结果 在兔脊髓前角 3组均发现被标记的神经元细胞
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     Protective Effect of Diphenyl Ethylene Glycoside on Hippocampal Neuron Cells Ischemic Injury Model Rats
     二苯乙烯苷对海马神经元细胞缺血性损伤模型大鼠的保护作用研究
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  neuron cell
     Results The Glu synthesizing of epilepsy hippocampus neuron cell increased relatively, the Glu secretion increased significantly( P <0 01).
     结果 致痫海马神经元细胞Glu合成相对增加 ,分泌显著性增加 (P <0 0 1) ;
短句来源
     Methods The primary cultured neuron cell of l8-day-pregnan embryoid Sprague-Dawley rat's hippocampus has been selected as the experimental material. The experimental concentration of magnesium sulfate will be 0 mmol/L,1 mmol/L,2 mmol/L,4 mmol/L;
     方法 体外原代培养孕 18d胎鼠海马神经元细胞 ,分别使用硫酸镁终浓度为 0、1、2、4mmol/L ,观察不同浓度硫酸镁对神经元的保护作用。
短句来源
     Establishment of a Hantaan virus infected mouse neuron cell model
     汉滩病毒感染神经元细胞模型的建立
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     Conclusion Mild hypothermia treatment can inhibit the release of serum βEP,NSE and protect the cortical neuron cell and neuroendocrine cell from the secondary damage.
     结论亚低温疗法可抑制β-EP、NSE释放,有保护重症病脑患儿神经元细胞和神经内分泌细胞继发性损害的作用。
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     In Gansu zokor, the vomeronasal epidermis thickness in adult males, the zones width of accessory olfactory bulb granular cells and the mitral cells are remarkably wider than that of the females (P<0.05). But the length of vomeronasalmucous, the density of vomeronasal epidermis neuron cells, the diameter of neuron cell nucleus, the zones length of accessory olfactory bulb granular cells and the mitral cells in females and males are no significant difference (P>0.05).
     成年雄性甘肃鼢鼠的犁鼻上皮厚度、副嗅球颗粒细胞带宽与僧帽细胞带宽雄性显著大于雌性(P<0.05),而犁鼻黏膜长度、犁鼻上皮神经元细胞密度、神经元细胞核直径、副嗅球颗粒细胞带长与僧帽细胞带长等在雌雄间差异不显著(P>0.05)。
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  neuronal cell
     Methods neurons were randomly divided into normal control(24 h,48 h) group,hydraulic impact injury(24 h,48 h) model group,taurine group(to hydraulic impact injury at 24 h,48 h),alteration in Ca2+ in SD rat's neuronal cell following hydraulic impact injury was measured by means of a confocal laser scanning microscope using Fluo-3/AM as a calcium indicator.
     方法将体外培养的大鼠神经元细胞随机分为正常对照(24 h4、8 h)组、液压冲击伤(24 h、48 h)模型组、牛磺酸(伤后24 h、48 h)治疗组,以Fluo-3/AM为细胞内钙离子荧光指示剂,用激光扫描共聚焦显微镜(CLSM)测定各组神经元细胞内Ca2+的变化。
短句来源
     The positive cells of the ET 1 mRNA expression were localized to neuronal cell bodies in spinal cord. The highly expressive regions were in the ventral horn (laminae IX).
     ET- 1 mRNA表达阳性细胞主要是神经元细胞,高表达区在脊髓前角( Ⅸ区)。
短句来源
     ②Hippocampus neuronal cell activity: The cell activity was obviously higher in control group and Rg1 pretreatment groups of 2 and 4 μmol/L than in model group (P < 0.05), and the highest in Rg1 pretreatment group of 4 μmol/L;
     ②海马神经元细胞活力:正常组和2,4μmol/L人参皂甙Rg1预处理组明显高于模型组(P<0.05),以4μmol/L人参皂甙Rg1预处理组最高;
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     The extracellular matrix molecules constitute the basement membrane underlying the vasculature and play a critical role in maintaining the integrity of the BBB. After stroke, there is a breakdown of the BBB by activated MMP -9 with an associated increase in vascular permeability, inflammatory cell influx, and neuronal cell death.
     ICH后,MMP-9被激活,通过对富含基膜的脑毛细血管床的过度降解作用,使得BBB遭到破坏,血管渗透性增加,炎性细胞浸润乃至神经元细胞死亡。
短句来源
     Neuron specific enolase (NSE) is a glycolytic enzyme, which is localized specifically to the neuronal cyloplasm and has been proposed as a biochemical marker of neuronal cell damage.
     神经元特异性烯醇化酶(NSE)是一种参与糖代谢功能酶,特异性位于神经元细胞
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  neuronal cells
The expression of β-III tubulin, a marker protein of early neuronal cells, was studied by molecular genetic and immunochemical techniques.
      
β-Amyloid peptide (Aβ), a normal constituent of neuronal and non-neuronal cells, has been proved to be the major component of extracellular plaque of Alzheimer's disease (AD).
      
Role of Lipids in the Assembly of Endoplasmic Reticulum and Dictyosomes in Neuronal Cells from the Cerebral Cortex of Yakutian G
      
Since leukocytes are much more available than hepatocytes or neuronal cells in humans, we assume that CE in peripheral blood leukocytes (neutrophils and monocytes) can be used as markers for indication of pending liver damage by CPZ.
      
When Na/K-ATPase was inhibited by high ouabain concentrations (10-5-5·10-4 M), an increase in stationary ROS level in neuronal cells was noted, this effect being attenuated by NMDA antagonists, MK-801 and D-AP5.
      
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  neuron cells
Unlike skeletal muscles that can survive for hours without oxygen, neuron cells in the brain are easily subjected to an irreversible damage within minutes from the onset of oxygen deficiency.
      
Dactylorhin B reduces toxic effects of β-amyloid fragment (25-35) on neuron cells and isolated rat brain mitochondria
      
Indeed, foot and mantle mucocytes exhibited muramic acid in a terminal position, linked to (subterminal) N-acetylgalactosamine, whereas in neuron cells muramic acid was present in an internal position and linked to N-acetylglucosamine.
      
In bursting excitable cells such as pancreaticβ-cells and molluscanAplysia neuron cells, intracellular Ca2+ ion plays a central role in various cellular functions.
      
As shown by MTT test, PrP106-126 induced significant death of neuron cells and marked proliferation of astrocytes after 10 days of treatment.
      
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  neuron cell
PPN are not rare disorders and correspond to a wide range of diseases of the PNS, involving either the neuron cell body, the axon, or the myline sheath.
      
3 and 5) and induces the capacity to sustain prolonged bursts triggered by brief current stimulation in the isolated neuron cell body (Fig.
      
Localization of stomatogastric IV neuron cell bodies in lobster brain
      
It was concluded that the position of the sensory neuron cell body does control its axonal morphology and synaptic connectivity, but that these characteristics are produced by independent mechanisms.
      
Normative data on the intermediolateral column (ILC) neuron cell bodies is lacking.
      
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  neuronal cell
Studies of mechanisms involved in neuronal cell death induced by chronic stress in rats
      
Mechanisms of peroxynitrite-induced [Ca2+]i increase in single neuronal cell
      
A proteomic analysis of the effect of mapk pathway activation on l-glutamate-induced neuronal cell death
      
In this study, we used proteomic analysis to investigate the role of the MAPK pathway in oxidative stress-induced neuronal cell death.
      
The results demonstrated that several proteins, including eukaryotic translation elongation factor 2 (eEF2) and enolase I, showed a differential expression pattern during the neuronal cell death process, and this was MAPK pathway dependent.
      
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