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gene
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  基因
    The establishment of the gene expression profile and cloning the genes associated with differentiation inducing of human glioma cells.*
    人脑胶质瘤细胞诱导分化基因谱的建立及相关基因的克隆
短句来源
    Adenovirus mediated angiostatin gene therapy for rat glioma
    重组腺病毒载体介导血管抑素基因治疗大鼠脑胶质瘤的实验研究
短句来源
    Observation on the Biological Charateristics of Glioma Cells Transfected with p16 Gene
    人胶质瘤细胞转染p16基因后生物学特性的观察
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    In Vitro and in Vivo Inhibitory Effect on Growth of Human Gliomas with P21~(WAF-1/CIP1) Gene Mediated by Targeted Non-viral Vector GE7 System
    靶向性非病毒载体GE7系统介导P21~(WAF-1/CIP1)基因治疗脑胶质瘤的实验研究
短句来源
    Effects of ABCA1, PON1 and ApoE Gene Polymorphisms on Plasma Lipid Levels and Cerebrovascular Disease
    ABCA1、PON1及ApoE基因多态性与血脂和脑血管疾病
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  “gene”译为未确定词的双语例句
    Experimental Study of Combination Gene Transfer FasL and P21~(WAF1/CIP1)Therapy for Human Gliomas
    联合基因转移FasL、P21~(WAF1/CIP1)治疗人脑胶质瘤的实验研究
短句来源
    Gene Therapy of Interleukin 12 Expressed in Mesenchymal Stem Cells for Glioma in Rats
    转染IL-12的骨髓间充质干细胞治疗胶质瘤的研究
短句来源
    Mapping the Disease Gene in SCA Families and the Frequency Distribution Analysis of Different Subtypes of SCA in Hans of China
    SCA致病基因定位及中国汉族人群SCA亚型频率分布
短句来源
    THE DISTRIBUTION OF TUMOR SUPPRESSOR GENE APC mRNA IN GUINEA PIG BRAIN
    肿瘤抑制基因APCmRNA在豚鼠脑中的分布
短句来源
    Changes of cNOS mRNA Gene Expression After Cerebral Ischemia: the Protective Effect of NO on Cerebral Ischemia in the Early Stage
    脑缺血后cNOS mRNA表达:NO在脑缺血早期的保护作用
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  gene
NEW TECHNOLOGY FOR DRUG DISCOVERY BASED UPON INSERTION OF LIGANDS INTO GENE SEQUENCES BY NUCLEAR RECEPTOR PROTEINS
      
A gene regulatory mechanism has been proposed in which steroid hormones and certain other drugs bind to nuclear receptor proteins followed by transfer to DNA where they are inserted between base pairs.
      
Polymerase chain reaction was used to amplify a 439-bp fragment of a 65,000-kDa (Mr) heat shock protein gene (hsp65) of Mycobacterium.
      
Cloning of an APETALA3 homologous gene (PtAP3) from Populus tomentosa and genetic transformation of its sense and anti-sense con
      
A pair of primers were designed according to published literature on Populus trichocarpa gene (PTD), and PtAP3, an AP3 homologous gene from Populus tomentosa was isolated by PCR using genomic DNA of the male clone of P.
      
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Gene dosages of β-amyloid protein (β-AP) in Alzheimer's disease patients and normal aged personswere detected with cloned β-AP cDNA probe. The results showed that dosages of the gene in patientswith Alzheimer's disease were incerased.

用编码老年性痴呆脑中老年斑核仁成分β-淀粉样蛋白(β-Amyloiol Protein,β-AP)的cDNA探针测定老年性痴呆及正常老人的该基因剂量,结果显示老年性痴呆患者的β-AP基因剂量较正常人高。

Detailed neurological examination were made in 27 patients (male 13, female 14) from 56 directly related offsprings of a large Chinese Huang's family with Joseph disease, which consisted of 76 persons in 5 generations. The family is the largest one of the Joseph disease family in Asia, and this is the first report in China. The Huang's family lives in the area of Beijing, Tianjin and Tangshan. And beginning from May 1988, we have made a systematic study in the Joseph disease in different respects, including...

Detailed neurological examination were made in 27 patients (male 13, female 14) from 56 directly related offsprings of a large Chinese Huang's family with Joseph disease, which consisted of 76 persons in 5 generations. The family is the largest one of the Joseph disease family in Asia, and this is the first report in China. The Huang's family lives in the area of Beijing, Tianjin and Tangshan. And beginning from May 1988, we have made a systematic study in the Joseph disease in different respects, including clinical classification, peripheral neuropathology, karyotype and haplotype analysis, electrophysiology, histochemical and submicroscopic obsearvations. The clinical features showed various combinations of the following symptoms: normal intellegence, bulging eyes, nystagsmus, curling hair, wide metatarsals, facial muscular spasm, tongue muscle fasiculation, ocular paralysis, cerebellar ataxia, pyramidal and extrapyramidal signs, marked. spasticity with pathololgical reflexes, clonus and peripheral neuropathy. Normal karyotype findings suggested that the disease of Huang' s family is a hereditary disease of genetic type. Haplotype analysis showed that the Joseph disease gene might loosely link with HLA with a lods score equals to 1. 27. HVA and dopamine metabolites were siginificanty low as compared with other non degeneration disease. Needle EMG disclosed a systematic neurogenic change with high amplitude potentials and reduced interference pattern. Sensory evoked potentials were adnormal. Atrophy of the cerebellum was remarkable in the cranial CT and MRI. Peripheral nerve biopsis were taken from 3 patients. 2 patients were found to be type Ⅰ and type Ⅱ. another one was type sub Ⅲ. All cases shared the same pathological features, which consisted of a reduction in density of myelinated and unmyelinated fibers, myelin degeneration and an increase in endoneurial collagen. It was also observed that some Schwann cells were not near to axons. whilst others showed numerous budding processes. These results might be valuable data for the research of the Joseph disease and would be of great importance for eurololgy eugenics.

本文报告了对Joseph病黄氏家系的综合性研究及流行病学调查的结果。该家系包括5代共76人,其直系亲属56人中27人发病,已死亡9人。本文报告18例病例简介,临床表现智力正常,卷发、突眼、面肌抽搐、小脑性共济失调、锥体系及锥体外系症状、下肢痉挛性瘫及周围神经受损,肌电图示有高而宽大的运动电位,周围神经运动传导速度低于正常。按其临床特性进行了分型。对部分患者还进行了血液中LPO、SOD值、脑脊液HVA值检查,氮基酸分析,小脑CT及MRI检查,染色体检查,皮肤、肌肉、神经活检,成纤维细胞培养及免疫组化研究。Joseph病的研究,对我国的优生优育有重要意义。

One family with BMD and two families with DMD were Studied with the probes DXS28, J-Bir, DXS164 and DXS84.All the J-Bir/BamH I and pE-RTS7-1/Msp I were informative in the family with DMD and the pERT87-1 deletion was identified in the families with BMD. The carriers of DMD pathogenic gene were detected in the three families by RELP linkage analysis and deletion analysis. The mechanism of the deletion was also discussed.

用DXS28、J-Bir、DXS164和DXS84探针对一个BMD和两个DMD家系进行RFLP连锁分析,在DMD家系中,JBir/BamH Ⅰ和pERT87-1Msp Ⅰ杂交组合均能提供信息,表明该家系中先证者之妹是DMD致病基因携带者。在一个BMD家系中发现有pERT87-1的缺失,并进行了缺失分析。

 
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