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intra-arterial
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  动脉内
     Results The mean intra-arterial systolic and diastolic pressures of the feeding arteries were (78.9±20.0)mmHg, (67.3±15.6)mmHg respectively before the embolization, and (97.1±20.9)mmHg, (84±18.4)mmHg (P<0.01) respectively after the embolization.
     结果 栓塞前供血动脉内平均收缩压、舒张压分别为(78.9±20.0)mmHg、(67.3±15.6)mmHg; 栓塞后分别为(97.1±20.9)mmHg、(84±18.4)mmHg,栓塞前后有显著性差异(P<0.01)。
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     Bombesin(intra-arterial, 2×10~(-10) mol/L, 0.3 ml/min)stimulated gastric acid secretion from basal 2.50±0.05×10~(-1) to 8.40±1.50×10~(-1) mEq/min (P<0.001). Exogenous addition of pentagastrin did not potentiate this effect of bombesin.
     2×10~(?) mol/L铃蟾肽以0.3ml/min速度作动脉内输注,可刺激胃酸的分泌,自2.50±0.05×10~(-1)增至5.50±1.50×10~(-1)mEq/min,但与外源性五肽胃泌素无协同作用。
短句来源
     (3)With the constant autoperfusion method,renal intra-arterial administration ofAP Ⅲ (0.8/μg/kg)decreased the mean renal perfusing pressure by 21.2±3.3mmHg(n=9,P<0.001). By injecting AP Ⅲ into the femoral artery,the meanperfusing pressure of hindlimb lowered by 7.8±1.1 mmHg(n=7,P<0.001).
     (3)在自体血恒量灌流条件下,局部动脉内注射 AP Ⅱ(0.8μg/kg)时,肾血管灌流压降低21.2±3.3mmHg(n=9,P<0.001),后肢血管灌流压降低7.8±1.1mmHg(n=7,P<0.001);
短句来源
     Urgent intra-arterial thrombolysis and low molecular weight heparin therapy for acute cerebral infarction: a report of 11 cases
     动脉内溶栓后低分子肝素治疗急性脑梗死11例
短句来源
     Bilateral local intra-arterial thrombolysis therapy of acute basilar artery occlusion:report of 12 cases
     双向局部动脉内溶栓治疗12例急性基底动脉梗塞的临床报告
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  动脉灌注
     Methods Twelve patients were received intra-arterial chemotherapy or received intra-arterial chemotherapy and embolism chemotherapy(ADM 50 mg or DDP 60 mg,5-Fu 0.5~0.75,MMC 10 mg,HCPT 10 mg). They were repeated for 4 weeks.
     方法对12例晚期胰腺癌患者予以ADM 50mg(或DDP 60 mg)5、-Fu 0.5~0.75g、MMC 10 mg、HCPT 10 mg进行动脉灌注或同时联合肝转移癌栓塞,4周重复。
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     While drugs in blood by intra-venous method was 1.92,2.03,1.77, 1.92,1.40 times as much as by intra-arterial method.
     血液中平均药物浓度静脉途径分别为动脉灌注方法的1、92、2.03、1.77、1.92、1.40倍。
短句来源
     Effects of intra-arterial infusion of IL-10 on IL-6 and TNFα after acute pancreatitis in rats
     IL-10区域动脉灌注对急性胰腺炎大鼠血浆IL-6和TNF-α的影响
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     groupⅥ animals of SAP treated with intra-arterial carthamus tinctorius L injection (0.5ml/kg·h-1, treatment started 3 hours after the induction of SAP);
     红花注射液(0.5ml/kg·h-1)经腹腔动脉灌注,按给药时间分二组:Ⅵ组,制模后3h给药;
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     Clinical research of intra-arterial radioembolization with ~(32)P-glass microspheres combined with chemoembolization for treatment of liver cancer
     经肝动脉灌注~(32)P-玻璃微球联合化疗栓塞治疗肝癌的临床研究
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  动脉系统
     Intra-arterial thrombolysis in 28 patients with acute internal carotid circulation infarction
     28例颈内动脉系统急性脑梗塞动脉溶栓治疗
短句来源
     Local intra-arterial thrombolysis in acute ischemic stroke of the anterior circulation
     急性颈内动脉系统脑梗死的局部动脉溶栓治疗
短句来源
     Objective To evaluate the safety and efficacy of local intra-arterial thrombolysis (LIT) using urokinase in patients with acute ischemic stroke of the anterior circulation.
     目的 检验局部注射尿激酶动脉溶栓治疗急性颈内动脉系统脑梗死的安全性和疗效。
短句来源
     Objective To evaluate the safety and efficacy of intra-arterial thrombolysis in patients with acute internal carotid circulation infarction.
     目的分析颈内动脉系统急性脑梗塞动脉溶栓治疗的有效性和安全性。
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  “intra-arterial”译为未确定词的双语例句
     Results Before intra-arterial chemotherapy the expression rate of P-gp was 30%,GST-p was 10%,Topo Ⅱ was 50%.
     结果:灌注前P-gp的表达率为30%,GST-p为10%,TopoⅡ为50%。
短句来源
     After intra-arterial chemotherapy,the expression rate of P-gp was 50%,GST-p was 20%,Topo Ⅱ was 40%,respectively.
     灌注后P-gp的表达率为50%,GST-p为10%,TopoⅡ为40%。
短句来源
     after Intra-arterial chemotherapy, P53 was 50%, bcl-2 was negative and VEGF was 50%.
     灌注后P53的表达率为50%(+~++),bcl-2为阴性,VEGF的表达率为50%(+)。
短句来源
     1. Apoptosis index (AI) in cervical cancer group before intra-arterial influsion chemotherapy(5.77(0.97)and after intra-arterial influsion chemotherapy(11.65(2.36)were higher than that in normal cervix group (2.54(0.87,). AI in cervical cancer group before intra-arterial influsion chemotherapy was significantly lower than that after intra-arterial influsion chemotherapy (p<0.05).
     1.宫颈癌介入化疗前的AI(5.77(0.97)、介入化疗后的AI(11.65(2.36)均明显高于正常组(2.54(0.87,p<0.05),介入化疗后组的AI明显高于介入化疗前组(p<0.05)。
短句来源
     Results Before Intra-arterial chemotherapy, the expression rate of P53, bcl-2 and VEGF were all 50%;
     结果灌注前P53的表达率为50%(+~++),bcl-2的表达率为50%(+~+++),VEGF的表达率为50%(+~++);
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  intra-arterial
Comparison of simultaneous measurements of intra-arterial pressure in the brachial artery was performed on 33 patients suspected of having hypertension.
      
Maximal vasodilatation was induced by intra-arterial infusion of adenosine.
      
We studied the diagnostic value of computed tomographic angiography (CTA) in the posterior circulation as a noninvasive substitute for intra-arterial digital subtraction angiography (DSA).
      
In the first part of the analysis we examined the correlation between CTA and intra-arterial DSA (n=22), and in the second part that between CTA and Doppler ultrasound (n=103).
      
Intra-arterial DSA identified 11 stenoses, 13 occlusions, and 3 hypoplasias.
      
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Nn-9 was a non-crystalline alkaloid isolated from the embryo of the seeds of the Asiatic lotus, Nelumbo nucifera. In anesthetized cats, iv.injections of 1—2 mg/kg lowered, after a delay of 1/2—1 minute, the blood pressure by about 50% from the original level, lasting 2—3 hours. In dogs, the blood pressure returned to normal in 1/2 hour. This dosage was ineffective in rabbits. Tachyphylaxis developed on repeated administrations. After the cats were pretreated with Diphenhydramine or Promethazine, the hypotensive...

Nn-9 was a non-crystalline alkaloid isolated from the embryo of the seeds of the Asiatic lotus, Nelumbo nucifera. In anesthetized cats, iv.injections of 1—2 mg/kg lowered, after a delay of 1/2—1 minute, the blood pressure by about 50% from the original level, lasting 2—3 hours. In dogs, the blood pressure returned to normal in 1/2 hour. This dosage was ineffective in rabbits. Tachyphylaxis developed on repeated administrations. After the cats were pretreated with Diphenhydramine or Promethazine, the hypotensive effect of Nn-9 was remarkably diminished. Pretreatment with Nn-9 also lessened the hypotensive action of histamine. When the cats were given 2 mg/kg of Nn-9 intravenously, the hypotension was accompanied by a reduction of cerebral vascular resistance. Intra-arterial injections yielded an increase of both cerebral and femoral blood flows, associated with a diminution of the vascular resistance. In spinal cats, i.v. injection of 2 mg/kg produced a significant drop of blood pressure. The pressor responses elicited by i.v. adrenalin, noradrenalin, pituitrin, or by spinal compression below C-7 were all inhibited for 1/2—1 hour. The retraction of nictitating membrane by preganglionic faradization was also inhibited. But it had no apparent influence on parasympathetic ganglion and M-cholinergic reactive system. Intracarotid injections produced a hypotension, together with a decrease of the carotid occlusion reflex pressor response. In conclusion, Nn-9 seems to act primarily through histamine release. Secondarily, a nervous factor is also involved.

蓮子心非結晶生物碱Nn-9对麻醉猫靜脉注射1-2毫克/公斤可降低原血压水平約50%,維持2-3小时。狗血压于1/2小时卽恢复,兔子不降压。有快速耐受性。經苯海拉明及异丙嗪处理猫再注射Nn-9碱2—3.5毫克/公斤,降压作用明显減弱。对靜脉注射組織胺引起的降压被Nn-9所減弱。猫靜脉注射2毫克/公斤的Nn-9后,血压下降伴随脑血管阻力減低,动脉注射0.2毫克/公斤使脑血流及后肢血流均增加,血管阻力減低。脊猫靜脉注射2毫克/公斤,有明显的降压出现。靜脉注射腎上腺素、去甲腎上腺素及脑垂体后叶素或在第七頸椎以下加高脊髓腔压所引起的升压反应均被抑制,历1/2—1小时恢复。电刺激頸交感神經节节前纤維所致的瞬膜收縮也受到抑制。对副交感神經节及M-胆碱反应系統无明显影响。頸动脉注射药液也出现降压,并抑制頸总动脉血流阻断所引起的升压反应。总結实驗結果,Nn-9的降压机制主要是释放組織胺,使外周血管扩张,其次神經因素也有关。

In anesthetized cats, an intravenous injection of lycoramine at 3—5 mg/kg produced a transient fall of systemic blood pressure, potentiated the hypotensive response elicited by acetylcholine or by the electrical stimulation of the peripheral end of vagus, and increased the activity of smooth muscle in the intestines. This drug also enhanced the blocking action of suceinylcholine on the myoneural junction. Under the same experimental conditions, galanthamine in doses of 0.25—2mg/kg produced similar effects. When...

In anesthetized cats, an intravenous injection of lycoramine at 3—5 mg/kg produced a transient fall of systemic blood pressure, potentiated the hypotensive response elicited by acetylcholine or by the electrical stimulation of the peripheral end of vagus, and increased the activity of smooth muscle in the intestines. This drug also enhanced the blocking action of suceinylcholine on the myoneural junction. Under the same experimental conditions, galanthamine in doses of 0.25—2mg/kg produced similar effects. When the solution of lycoramine was applied Iocally to the rabbit's eye, it caused pupillary constriction and abolished the mydriatic action of atropine. In vitro, lycoramine increased the reactivity of guinea-pig ileum and frog's rectus abdominis muscle to acetylcholine. In cats intravenous or intra-arterial (through lingual artery) injections of lycoramine or galanthamino produced no marked influence on the contractions of nictitating membranes elicited by electrical stimulation of preganglionic fibers, but theypotentiated the action of acetylcholine injected through lingual artery. In EEG recordings of normal rabbits lyooramine (15—20mg/kg) or galanthamine(3—5mg/kg)induced the arousal response and this action could be antagonized by some anticholinergic drugs such as atropine, scopolamine or benactyzine.

給麻醉猫靜脉注射力可拉敏3—5毫克/公斤后,能引起血压的短暫下降,加强注射乙酰胆碱或电刺激迷走神經外周端的降压反应,兴奋腸肌,和加强琥珀酰胆碱的阻断神經肌肉間的傳递作用。用加兰他敏0.25—2毫克/公斤試驗时,也能观察到上述作用。将力可拉敏溶液滴入兔眼內,能使瞳孔縮小,并有对抗阿托品的扩瞳作用。在离体試驗时,力可拉敏能提高豚鼠腸肌或蛙腹直肌对乙酰胆碱的敏感性。在麻醉猫的頸交感神經节試驗中,靜脉或动脉內注射力可拉敏和加兰他敏,对电刺激交感节前纤維引起的瞬膜收縮无明显影响,但能加强舌动脉注射乙酰胆碱的作用。力可拉敏(15—20毫克/公斤)和加兰他敏(3—5毫克/公斤)能使清醒家兔的皮层自发脑电活动,产生惊醒反应。阿托品,东茛菪碱或貝納替秦对上述惊醒反应有对抗作用。

The effects of Citrus aurantium L(Ⅰ) and one of its active ingredients N-methyl-tyramine (Ⅱ) on vascular resistance were studied with roller pump in anethetized dogs. Their actions on receptor were analysed by antagonists. Intra-arterial and intra-venous injection of Ⅰ and Ⅱ increased the renal and cerebral vascular resistance signi-ficantly, which could be antagonized by phentolamine. This suggests that α-receptor sti-mulation is involved. Ⅱ exerted a biphasic effect on femoral artery resistance. This...

The effects of Citrus aurantium L(Ⅰ) and one of its active ingredients N-methyl-tyramine (Ⅱ) on vascular resistance were studied with roller pump in anethetized dogs. Their actions on receptor were analysed by antagonists. Intra-arterial and intra-venous injection of Ⅰ and Ⅱ increased the renal and cerebral vascular resistance signi-ficantly, which could be antagonized by phentolamine. This suggests that α-receptor sti-mulation is involved. Ⅱ exerted a biphasic effect on femoral artery resistance. This is explained on the differences of distribution in a and β-adrenoceptors.Under constant renal infusion, the transient diuretic action observed after injection of Ⅰ or Ⅱ is independent of the change of renal blood flow.Receptor action of the positive inotropic effect of Ⅱ in guinea pig's auricle strip in vitro, and rabbit heart in situ was attributed mainly to the stimulation of β-adren-ergic receptor.In reserpinized rats and dogs the hypertensive effects of Ⅱ were significantly dee-reased. Pressor tachyphylaxis of Ⅱ similar to tyramine was observed in rats. Assay of adrenergic mediator content in rat myocardium showed significant decrease in nor-epinephrine content after repeated injections of Ⅱ and tyrarnine, while dopamine remained unchanged. It was concluded that the cardiovascular effects of Ⅱ similar to tyramine are resulted from liberation of intrinsic adrenergic mediator, while Ⅰ contains synephrine as well, which shares a direct agonist action on α-receptors.

用恒速灌流泵灌流狗肾、脑及下肢血管,记录血管阻力。动脉或静脉注射枳实或其有效成分N-甲基酪胺,均明显增加肾、脑的血管阻力,此作用可被酚妥拉明所阻滞,提示系通过兴奋血管α受体产生的。N-甲基酪胺对股动脉血管阻力呈先降低后增高的双相反应,其升高阻力的作用是兴奋α受体所致,而其降低下肢血管阻力的作用可能是兴奋骨骼肌血管β_2受体的表现。 在恒速灌流使肾血流量已固定的条件下,枳实和N-甲基酪胺在升高血压与肾血管阻力时,仍有短时间的利尿作用,说明此作用与肾血流量的变化无关。 N-甲基酪胺能加强离体和在体心脏收缩力,β-受体阻滞剂心得舒能拮抗此作用。 利血平化动物实验结果及升压快速耐受性形成,以及减少心肌去甲肾上腺素含量而不影响多巴胺含量等事实表明:N-甲基酪胺与酪胺相似,通过释放去甲肾上腺素兴奋心血管的α和β受体产生效应。

 
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