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scar formation     
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  瘢痕
     Aim: To investigate the expressions of apoptosis-related Survivin, P33ING1, and Caspase-3 in pathologic scar formation.
     目的检测病理性瘢痕组织中Survivin、P33ING1、Caspase-3蛋白的表达情况。
短句来源
     This experiment implanted artificial vertebral lamina made from n-HA/PA66 into the defect of vertebral lamina, and evaluated its value in preventing postlaminectomy scar formation,the dura adherence in the spinal canal, the compression of cauda equina and nerve root as well as the reconstruction of vertebral integrality.
     本实验将纳米羟基磷灰石/聚酰胺(nano-hydroxyapatite/polyamide-66, n-HA/PA66)复合材料制成人工椎板植入脊柱椎板缺损处,探讨n-HA/PA66复合人工椎板在防止脊柱后路术后椎管内瘢痕粘连,马尾及神经根受压,重建脊柱后部结构的完整性,预防全椎板切除术后并发症方面的价值。
短句来源
     5 lesions (0.5%) appeared temporary pigmented disorder and 4 lesions(0.4%)appeared light scar formation.
     术后出现暂时性色素沉着5例(0.5%),轻度瘢痕4例(0.4%)。
短句来源
     CHARACTERISTICS OF P38 MITOGEN-ACTIVATED PROTEIN KINASE AND c-Jun EXPRESSION IN HYPERTROPHIC SCAR AND THEIR EFFECTS ON SCAR FORMATION
     增生性瘢痕中磷酸化P38丝裂原活化蛋白激酶和c-Jun蛋白的表达特征及其与瘢痕形成的关系
短句来源
     Effects of gene expression of fibroblast proto-oncogene N33 on the hypertrophic scar formation
     原癌基因N33在增生性瘢痕中表达的研究
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  瘢痕形成
     Role of p-ERK、IGF-1R、PTEN and PCNA in Pathologic Scar Formation
     p-ERK、IGF-1R、PTEN及PCNA在病理性瘢痕形成中的作用
短句来源
     Function of Apoptosis-Related SURVIVIN, P33ING1, CASPASE-3 in Pathologic Scar Formation
     凋亡相关因子SURVIVIN、P33/ING1、CASPASE-3在病理性瘢痕形成中的作用
短句来源
     These indicate that TGF-βand its downstream signal molecules sand 2, sand 3 and sand 4 might be involved in hyperplastic scar formation.
     TGF - β和它的下游信号分子Sand 2、Sand 3与Sand 4可能与增生性瘢痕形成密切相关
短句来源
     The experimental study on antisense TGF-β_1 in inhibiting scar formation in Ⅲ° burn wound
     反义TGF-β_1抑制Ⅲ度烫伤愈合中瘢痕形成的实验研究
短句来源
     RESULTS The average length of wound heal was 15.3 days, and the percentage of pigmentation was 10/51, and scar formation was 3/51, and infection of cut part was 2/51 for M group.
     结果M组平均创面愈合时间15.3d,色素改变10/51,瘢痕形成3/51,创面感染2/51。
短句来源
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  瘢痕粘连
     This experiment implanted artificial vertebral lamina made from n-HA/PA66 into the defect of vertebral lamina, and evaluated its value in preventing postlaminectomy scar formation,the dura adherence in the spinal canal, the compression of cauda equina and nerve root as well as the reconstruction of vertebral integrality.
     本实验将纳米羟基磷灰石/聚酰胺(nano-hydroxyapatite/polyamide-66, n-HA/PA66)复合材料制成人工椎板植入脊柱椎板缺损处,探讨n-HA/PA66复合人工椎板在防止脊柱后路术后椎管内瘢痕粘连,马尾及神经根受压,重建脊柱后部结构的完整性,预防全椎板切除术后并发症方面的价值。
短句来源
     Objective To study the clinical effects of artificial vertebral laminae of the biomimetic nano-hydroxyapatite/polyamide 66(n-HA/PA66)composites in prevention of the scar formation in lumbar spinal canal and the reconstruction of posterior vertebral laminae structure.
     目的探讨自行研制的纳米羟基磷灰石/聚酰胺66(nano-hydroxyapatite/polyam ide66,n-HA/PA66)复合生物活性人工椎板,在防止椎管内瘢痕粘连及重建椎管后部结构中的作用。
短句来源
     Objective:To evaluate the effect of artificial vertebral lamina made from nano-hydroxyapatite/polyamide-66(n-HA/PA66) on preventing the postlaminectomy scar formation and the dura adherence in the spinal canal,and constructing vertebral canal posterior elements integraty.
     目的:探讨纳米羟基磷灰石/聚酰胺(nano-hydroxyapatite/polyamide-66,n-HA/PA66)复合人工椎板在防止脊柱后路术后椎管内瘢痕粘连,重建脊柱后部结构完整性的价值。
短句来源
     [Result] Experimentally, chitin and beta-tricalcium phosphate had significant effect on prevention of scar formation, the effect of group B, C, D were obveriously better than group A P<0.01, but the effect of group D was superior to that of group B and C, P<0.05, no significant difference between group B and C.
     结果几丁糖和人工骨有明显防硬膜外瘢痕粘连的效果。 在实验中B、C、D组均优于A组,P<0.01,D组为最佳防粘连方法,而B组与C组比较差异无显著性,P>0.05。
短句来源
     Experimental Study on the Effect of Titanium Alloy Particles on Epidural Scar Formation
     钛合金微粒对硬膜外瘢痕粘连形成影响的实验研究
短句来源
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  疤痕形成
     The Role and Mechanisms of Cell Proliferation Negative Factor p16 and Op18 in Burn Wound Healing and Scar Formation
     细胞增殖负性因子p16和Op18在烧伤愈合及疤痕形成中作用及机理
短句来源
     There has been extensive study on TGF - β1, which is believed to play a pivotal role in liver cirrhosis, glomerulonephritis and skin scar formation.
     其中TGF-β1研究得最广泛,其在纤维化性疾病如肝纤维化、肾间质纤维化、皮肤疤痕形成等疾病中均发挥重要作用。
短句来源
     3. MEBO helps to prevent scar formation andstricture of the anus.
     3:防止术后疤痕形成及肛门狭窄之效。
短句来源
     Conclusion Endogenous changes of EGF and EGFr, as a physiological changes during the wound healing, both can promote repair and avoid scar formation.
     结论 内源性EGF、EGFr变化为机体的生理变化,可以促进修复,也可避免疤痕形成
短句来源
     Conclusions From 3 hours to 2 months after cerebral ischemia there was astrocytes produced IL 1 β protein in the ischemic hemisphere, and IL 1 β may play a role in the process of astroglial proliferation and scar formation.
     结论 脑缺血后3小时至 2个月 ,脑内有内源性的、蛋白水平的IL 1β产生 ,来源为星形胶质细胞。 IL 1β可能与星形胶质细胞增生及疤痕形成有密切的关系。
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  scar formation
Effect of the Heat Shock Protein HSP70 on the Glial Scar Formation in Neurotransplantation
      
Glial and neurite penetration into places without symphysis, predominantly in the grey matter of the spinal cord, and the prevention of scar formation in the regions of damage were demonstrated.
      
PRP-3 prevented scar formation and favored ingrowth of nerve fibers of the white matter in the damaged area and thus prevented degeneration of neuroglial elements.
      
It was demonstrated that the inflammation process and scar formation in the cell therapy group were progressing at a rate 25-30% faster than in the control group.
      
It is suggested thatGFAP plays an important role in glial scar formation.
      
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