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plasma glucose
相关语句
  血糖
     CORRELATION OF FASTING PLASMA GLUCOSE AND GLYCOSYLATED HEMOGLOBIN (HbA_1) TO THE NERVE CONDUCTION VELOCITY IN NIDDM
     Ⅱ型糖尿病周围神经病变患者的神经传导速度与空腹血糖和糖基化血红蛋白(HbA_1)的关系
短句来源
     Assessment of fasting plasma glucose in patients with oral lichen planus
     口腔扁平苔藓患者空腹血糖测定及评价
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     The correlation coefficient between U-mAlb/Cr and 24h-U-Alb,free plasma glucose and glycation hemoglobin A1c was 0.903,0.025 and 0.038.Conclusion There is a significant correlation between U-mAlb/Cr and 24h-U-Alb.
     尿微量白蛋白/肌酐比值与24h尿白蛋白定量,空腹血糖,糖化血红蛋白A1c的相关系数分别为0.903,0.025,0.038。
短句来源
     Method:198 inpatients with schizophrenia were divided randomly into chlorpromazine group and risperidone group. HbA1C and fasting plasma glucose were tested before treatment and at 1,2,3,6,12 month after treatment, and compared with clinical routine fasting plasma glucose test.
     方法:对198例单用氯丙嗪或利培酮治疗的精神分裂症住院患者于治疗前、治疗1、2、3、6、12个月测定HbA1C及血糖浓度并与常规血糖检测指标进行比较。
短句来源
     Results:The level of HbA1C trended to increase with the time of hospitalization and associated with the level of fasting plasma glucose and the incidence of imparied glucose regulation (IGR),whose abnormality rate was 11.6% one year after treatment.
     结果:HbA1C浓度随治疗时间延长而增高,与血糖浓度变化呈显著正相关,与血葡萄糖调节受损(IGR)的发生呈平行发展,治疗1年后异常率为11.6%。
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  血浆葡萄糖
     O_2 saturation (%) 96.664±2.040 and 56.661±10.049 plasma glucose concentrations (mg/100ml)66.20±7.87 and 12.36±2.22;
     O_2饱和度(%):母羊96.664±2.040,胎儿56.661±10.049; 血浆葡萄糖含量(mg/100ml):母体66.20±7.87,胎儿12.36±2.22;
短句来源
     RESULTS: The reverse of fasting plasma glucose-insulin product [ unit: L 2/(mmol·mU)×10 -3 ] was increased from 10.8±2.2 to 13±4 (P<0.05). The fasting plasma glucose(unit: mmol/L)was decreased from 6.2±0.7 to 6.0±0.5(P<0.05).
     结果:空腹血浆葡萄糖胰岛素乘积的倒数[单位:L2/(mmol·mU)×10-3]由10.8±2.2上升至13±4(P<0.05),空腹血糖(单位:mmol/L)由6.2±0.7降至6.0±0.5(P<0.05)。
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     The sensitivity、specificity and diagnostic efficiency of ADA、blood plasma glucose、ascites/blood plasma glucose for the diagnosis of tuberculous ascites were: 81.8%、90.9%、72.7%;
     ADA、GLU、腹水/血浆葡萄糖诊断结核性腹水的敏感性、特异性、诊断准确性分别为:81.8%、90.9%、72.7%;
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     For every 1 kg increase in birthweight,systolic blood pressure decreased by 2 9 mm Hg (95%CI,0 3~5 4 mm Hg) and the 2 hour plasma glucose level decreased by 5 1% ( 95%CI,0 7%~9 3%).
     出生体重每升高 1kg ,收缩压降低2 9mmHg (95 %CI:0 3~ 5 4mmHg) ,2h血浆葡萄糖水平降低 5 1 % (95 %CI:0 7%~ 9 3 % )。
短句来源
     The CGMS values in this study were significantly correlated with the values of plasma glucose and capillary glucose of finger tip(r=0.93,r=0.95,both P< 0.001).
     CGMS观测值与血浆葡萄糖值及指端毛细血管血糖值均呈显著正相关(r=0.93,r=0.95,P均<0.001)。
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  “plasma glucose”译为未确定词的双语例句
     (3) Correlation analysis showed that the serum IL-18 concentrations were positively correlated with fasting plasma glucose (FPG), 2 h postprandial glucose (2 hPG), glycosylated hemoglobin (HbA1c)( r =0.52, 0.71, 0.50; P <0.01, 0.01, 0.01).
     直线相关分析表明IL-18与FPG、2hPG、HbA1c水平成正相关(分别为r=0.52,P<0.01;r=0.71,P<0.01;r=0.50,P<0.01)。
短句来源
     the plasma glucose concentration were detected and recorded as G 2. The BV was calculated on the basis of: BV(ml)=3 082×HET(ml)/(G 2-G 1)/(1-Hct).
     根据公式 :BV(ml) =3 0 82×HET(ml) /(G2 -G1) /( 1-Hct)计算血容量。
短句来源
     Methods Divided 144acute myocardial infarction patients into 2 groups according plasma glucose level separately at the 6.1mmol/L,7.0mmol/Land 8.0mmol/L,compared the prognosis.
     方法分别以6.1mmol/L、7.0mmol/L和8.0mmol/L作为入院糖水平的切入点,对144例急性心肌梗死患者的预后进行研究,比较2组预后的差异。
短句来源
     Methods One hundred and eighteen subjects were divided into six groups by fasting plasma glucose (FPG),including FPG1:3.7~6.09 mmol/l;
     方法118例受试者根据FPG水平将研究对象分成6个组:FPG1:3.7~6.09mmol/L;
短句来源
     The variant of mtDNA 16189 T to C might be associated with insulin resistance, higher level of plasma glucose and earlier occurance of AS in type 2 DM.
     因此,mtDNA 16189 T→C变异可能与胰岛素抵抗、较高血糖水平和2型DM早期AS相关。
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  plasma glucose
In the diabetic group, serum VEGF appeared to be positively correlated with fasting plasma glucose, HbA1c, LDL, creatinin and microalbuminuria.
      
Measurements including anthropometry, blood pressure, fasting plasma glucose and insulin, serum lipid profile were done.
      
5.2% of the patients without a diagnosis of diabetes mellitus and a fasting plasma glucose level >amp;gt;125 mg/dl were defined as undiagnosed diabetics.
      
Mean basal plasma glucose level was slightly higher before S milk (11.4 vs.
      
The peak increment in plasma glucose was higher in S milk (9.4 vs.
      
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14 cases of hyperosmolar nonketotic diabetic coma were reported. These accounted for 15.5% of diabetic coma patients seen during the past 20 years. There were 9 female and 5 male patients. 8 were over 60 years old. Half or them gave no history of diabetes. The most important precipitating factor was infection (8 cases). Other factors included infusion of large dose of glucose or hydrocortisone, intake of large amount of sweet beverage, withdrawal of insulin, psychic stress and gastrointestinal disorders. The...

14 cases of hyperosmolar nonketotic diabetic coma were reported. These accounted for 15.5% of diabetic coma patients seen during the past 20 years. There were 9 female and 5 male patients. 8 were over 60 years old. Half or them gave no history of diabetes. The most important precipitating factor was infection (8 cases). Other factors included infusion of large dose of glucose or hydrocortisone, intake of large amount of sweet beverage, withdrawal of insulin, psychic stress and gastrointestinal disorders. The clinical pictures consisted mainly of neurological syndrome and profound dehydration. About half of the patients were in frank coma, the others were unconscious or mentally confused. 2 had paralysis, 1 had seizures and 3 had circulatory collapse. Laboratory findings were: mean plasma glucose 656.7 mg/dl, mean serum sodium 147.7 mEq/l and mean serum osmolarity 356.3 mOsm/kg. The mortality rate was 64.3% in this series. The main causes of death were infections, shock, electrolyte disturbances and cerebral edema. With adequate hydration, use of hypotonic saline, correction of electrolyte disturbances, small dose of insulin and vigorous treatment of infections, our last 4 patients survived.

本文报道14例高渗性非酮症性糖尿病昏迷。本病多见于老年,其主要诱因为感染,其他为胃肠道功能紊乱,停用胰岛素,进食过多甜食,精神刺激和用药不当。主要临床特征为神经系症状昏迷,严重脱水,重度高血糖,血浆渗透压增高。本综台征易于误诊,本组死亡率高达64.3%。治疗上补液要足量,一部分补液须用0.45%低渗盐水,胰岛素剂量宜较小,补钾要充分,需密切观察,随时调整,以期提高治愈率,降低死亡率。

Insulin secretion and carbohydrate andlipid metabolic changes were studied in31 normal subjects, 11 patients withmaturaty onset diabetes mellitus (MO-DM) and 59 persons of various degreeglucose intolerance. The plasma glucosetriglycerides, cholesterol, HDL-chole-sterol, free fatty acid (FFA), cAMP,lecithin cholesterol acyl transferase(LCAT) and immuno-reactive insulin (IRI) response to an oral load of100 gm of glucose were evaluated. The results of this study showed thatthe plasma glucose area in...

Insulin secretion and carbohydrate andlipid metabolic changes were studied in31 normal subjects, 11 patients withmaturaty onset diabetes mellitus (MO-DM) and 59 persons of various degreeglucose intolerance. The plasma glucosetriglycerides, cholesterol, HDL-chole-sterol, free fatty acid (FFA), cAMP,lecithin cholesterol acyl transferase(LCAT) and immuno-reactive insulin (IRI) response to an oral load of100 gm of glucose were evaluated. The results of this study showed thatthe plasma glucose area in the subjectsof slight, moderate and severe degree ofglucose intolerance and MODM wereincreased than that of the normal groupby 33.8%, 52.9%, 82.2% and 154.7%respectively after oral glucose. Whilethe plasma IRI area were increasedthan that of normal group by 67.9%92. 1 %, 90. 2% and 21. 4% respectiyely.The plasma IRI area/glucose area ratioin various glucose intolerance groupwere larger than that of normal groupby 25. 5%, 25. 5% and 4% respectively,but, the ratio in diabetic group weresmaller than that of normal subjects by52. 3%. Significant hyperglucemia andhyperinsulinemia were observed in thepatients with MODM and the subjectswith glucose intolerance after oral glu-cose. The plasma triglycerides, cholesterol,and FFA levels in the diabetic groupand glucose intolerance group were sig-nificantly higher than those in the nor-mal group (P<0. 001), and the levelsof triglycerides increased to the greatestextent, amounting to 189%, 264%,265% and 269% respectively. The pla-sma HDL-cholesterol levels and theHDL-cholesterol/cholesterol ratio inpatients with MODM and glucose in-tolerance were lower than those in thenormal group (P<0. 05 and P<0. 001). The plasma triglycerides levels of thesubjects with glucose intolerance andpatients with diabetes were higher thanthe fasting levels 30 minutes after theintake of glucose meal by 19.0%,17. 9%, 21. 7%and 10. 0% respectively. FFA levels were significantly decreased(P<0.01 and P<0.001) after oralglucose load. While the plasma LCATtended to reduce 2 hours after the in-take of 100 gm of glucose. The average levels of fasting plasmacAMP in the moderate and severe glu-cose intolerance groups were loweredby 30. 3% and 38.9% respectively ascompared with the fasting level in thenormal group (P<0.05), while thefasting plasma cAMP level in MODMgroup was the same as that in the nor-mal group (20. 1±1. 8 pmol/ml).The plasma cAMP levels in the subjectsof various glucose intolerance werelowered 60-120 minutes after the intakeof glucose, while that of the diabeticgroup was insignificantly decreased butstill significantly increased by 63. 4%as compared with the normal group(P<0. 05) 3 hours after oral glucose.Significant insulin resistance wasobserved in the subjects with variousdegree of glucose intolerance and pa-tients with MODM.The results are dis-cussed.

对31例正常人,11例成年型糖尿病患者及59例不同程度(轻、中、重度)耐糖量减低者负荷糖后胰岛素的分泌及血糖、血脂以及血浆cAMP的改变进行了分析。结果发现,耐糖量降低者,胰岛素的分泌有不同程度增加,峰值有不同程度之延后,胰岛素面积/血糖面积比值有不同程度之增加,而糖尿病组则未见明显之峰值,比值反较正常降低52.3%。血脂除HDL-胆固醇下降外,均较正常明显升高(P<0.001)。中度及重度耐糖降低组空腹血浆cAMP含量分别较正常降低30.3%及38.9%(P<0.05),而糖尿病组与正常组比较无差异。负荷糖后,糖尿病组cAMP含量不仅不下降,3小时后反较正常组升高63.4%(P<0.05)。对所得结果进行了讨论。

It was found in our earlier studiesthat in half of CHD cases and almosthalf of the hyperlipidemic (HL) casesthe mechanism to handle exogenous glu-cose was defective, and consequently,the time for the clearance of glucosewas prolonged, and glucose intolerancewas presented. Since insulin plays animportant role in the regulation of thecorrelation between carbohydrate andlipid metabolism. High blood sugarlevel is an important stimulator for thesecretion of insulin. Therefore, a studyon the effects of glucose meal...

It was found in our earlier studiesthat in half of CHD cases and almosthalf of the hyperlipidemic (HL) casesthe mechanism to handle exogenous glu-cose was defective, and consequently,the time for the clearance of glucosewas prolonged, and glucose intolerancewas presented. Since insulin plays animportant role in the regulation of thecorrelation between carbohydrate andlipid metabolism. High blood sugarlevel is an important stimulator for thesecretion of insulin. Therefore, a studyon the effects of glucose meal on theplasma immuno-reactive-insulin (IRI)levels was made in 31 normal individuals,75 cases of hyperlipidemia and 53 casesof coronary heart disease (CHD). Results of this study showed thatsignificant impairment of carbohydratemetabolism was observed in patientswith HL and CHD. 30 minutes aftertaking 100gm of glucose the plasma glu-cose level reached peak value(171.3±7.32~215.7±13.5mg%) in types Ⅱ_b andⅣ_2 of hyperlipidemia of HL and CHDgroups, and the plasma glucose peakvalue was significantly higher thanthat of the normal group (131.8±4.2mg%)and the fasting level of the same grouprespectively (P<0.001). The plasmaglucose level 3 hours after taking glucosein types Ⅱ_b and Ⅳ_2 hyperlipidemiaof HL and CHD groups did not returnto the fasting level, and was significantlyhigher than the fasting level(P<0.05and P<0.001). The insulin secretion in responseto an oral glucose loading in types Ⅱ_band Ⅳ_2 hyperlipidemia of the HL andCHD group was significantly excessiveas compared with that of the normalgroup. The plasma IRI mean peak value(97.2±13.8~151.1 ±29.4μU/ml) inpatients with types Ⅱ_b and Ⅳ_2 of HLand CHD was higher than that of thenormal group (84.5±8.6μU/ml, P<0.01and P<0.001) b The plasma IRI meanpeak values in the HL und CHD groupwere reached by 60 and 120 minutesafter glucose loading, and did not returnto the fasting level by 180 minutes The plasma IRI levels at 180 minutes afterglucose loading in the HL and CHDgroup were higher than that of thenormal group and the fasting level ofthe same group by 2~5.2 times (P<0.05and P<0.001). But the plasma IRI meanpeak value in the normal group wasachieved by 30 minutes after glucoseloading and subsequnetly rapidly return-ed to the fasting level by 180 minutes. The plasma glucose area and IRIarea after taking glucose within 3 hoursin types Ⅱ_b and Ⅳ_2 of hyperlipidemiaof the HL and CHD groups were higherthan that of the normal group by 36. 5~73.6% and 31.1~111.2% respectively. These results indicate that hyperin-sulinemia and glucose intolerance areusually seen in types Ⅱ_b and Ⅳ_2 ofhyperlipidemia. The role of hyperinsu-linemia in the pathogenesis of hyperli-pidemia, glucose intolerance and athero-sclerosis is discussed.

本文对31例正常人,75例高脂血症及53例冠心病患者口服100克葡萄糖后血糖及血浆胰岛素水平进行了分析。结果发现,高脂血症及冠心病患者(Ⅱb及Ⅳ2型)耐糖降低及血浆甘油三酯增高并非胰岛素分泌不足所致。相反,患者负荷糖后胰岛素的分泌较正常显著增加,峰值延后(60及120分钟);3小时后仍未恢复至空腹水平;胰岛素分泌面积增加;呈现高胰岛素血症。本文对高胰岛素血症的产生及其与血脂升高,耐糖降低及动脉硬化的关系进行了设想及讨论。

 
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