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treatment regimen
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  治疗方案
    Methods The clinical manifestations,treatment regimen and pathological characters of 11 cases(age:4-14 y) with pheochromocytoma in children were reviewed retrospectively.
    方法 :对 11例 4~ 14岁嗜铬细胞瘤患儿的临床表现、治疗方案及病理特征等进行回顾性分析。
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    To see the traits of HIV children and to find a treatment regimen for AIDS children.
    了解儿童艾滋病的特点,探讨适合我国儿童艾滋病的治疗方案
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  “treatment regimen”译为未确定词的双语例句
    The pre treatment regimen was “BU/CY+ATG” [busulfan (BU) with the dosage of 1 mg/kg, once every 6 hrs, and 8 times in all; cyclophosphamide (CY) with the dosage of 50 mg/kg daily for 4 days and antihuman thymocyte globulin (ATG) with the dosage of 100 mg daily for 4 days].
    环磷酰胺 (CY)每日 5 0mg/kg ,用 4d ; 抗胸腺球蛋白 (ATG) 1 0 0mg/d ,用 4d。
短句来源
    Conclusion:CsA Combined with IVIG and ligustrazine is a effective and safe treatment regimen of SAA.
    结论:CsA、IVIG、川芎嗪三者联用治疗SAA有效、安全,是治疗SAA的有效方案之一。
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    Objective To explore the effective treatment regimen to improve the function of children with cerebral palsy (CP).
    目的 探讨治疗脑性瘫痪 (CP)的有效方法。
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  相似匹配句对
    After treatment .
    采用IFN和TF联合治疗后取得了一定的疗效。
短句来源
    To see the traits of HIV children and to find a treatment regimen for AIDS children.
    了解儿童艾滋病的特点,探讨适合我国儿童艾滋病的治疗方案。
短句来源
    Conclusion:CsA Combined with IVIG and ligustrazine is a effective and safe treatment regimen of SAA.
    结论:CsA、IVIG、川芎嗪三者联用治疗SAA有效、安全,是治疗SAA的有效方案之一。
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    Treatment of Hirschsprang’s Disease
    赫什朋氏病的治疗
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  treatment regimen
The aim of the study is to explore the association between Immunophenotype and treatment regimen.
      
Accordingly, glycoprotein IIb/IIIa antagonists represent a major achievement for the treatment regimen of patients with unstable angina.
      
The importance of maintaining the most effective treatment regimen has been shown in another study.
      
A Major barrier to the management of hypertension is the extent to which patients comply with the treatment regimen.
      
While clinicians should be aware of the psychological aspect of patients with NCCP and cardiac syndrome X, further research is needed in order to establish a comprehensive physiological and psychological treatment regimen.
      
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Within the last two decades,the event free survival (EFS) rate of children with ALL has increased to over 70%.Strategies responsible for this accomplishment include:①The advances in molecular biology (MIC develops into MICM) made it possible to predict precisely the prognosis of the patients.Different treatment regimens had been designed according to different classification.②Properly combined chemotherapy protocols reduced therapy intensity and decreased drug toxicities to patients with standard risk...

Within the last two decades,the event free survival (EFS) rate of children with ALL has increased to over 70%.Strategies responsible for this accomplishment include:①The advances in molecular biology (MIC develops into MICM) made it possible to predict precisely the prognosis of the patients.Different treatment regimens had been designed according to different classification.②Properly combined chemotherapy protocols reduced therapy intensity and decreased drug toxicities to patients with standard risk ALL.High dose methotrexate (HD MTX),instead of craniospinal radiation,had become effective phylaxis,and decreased the incidence of extramedullary relapse and CNSL (if indispensably,radiation dose was reduced).This measurement reduced radiation chemotherapy long term side effects and improved survival quality.③Potent support therapy such as protection and treatment of infection during the marrow suppression by setting up clean laminar airflow room,using high valent gammaglobulin,effective antibiotics,antivirus agents,antifungi agents.Availability of G CSF,GM CSF and infusing condensed platelets shortened duration of marrow suppression and accelerated hematopoietic reconstitution so as to protect and treat hemorrhage.

近20余年儿童急性淋巴细胞白血病 (ALL)长期无病生存率 (EFS)达70%~80%。长期ESF率的提高主要归功以下三方面工作 :①由于分子生物学研究进展 ,更准确地判断预后 ,采用MIC或MICM分型 ,按型用化疗方案。②化疗方案合理组合在保证疗效的同时对ALL后期治疗降低强度 ,减少药物毒性 ,同时改进了庇护所预防措施———采用大剂量氨甲蝶呤 (HD MTX) ,大大减少了髓外复发 ,使相当一部分患儿放弃以往颅脑预防性放疗 ,即使需要放疗者也降低了剂量 ,从而减少放化疗远期毒副作用 ,提高了生存质量。③强有力的支持治疗如 :空气净化层流病房的建立 ,有效抗菌素、抗病毒、抗霉菌药物高效价丙种球蛋白等的应用 ,预防治疗了骨髓抑制期的感染。造血因子、GM、CSF、G CSF浓缩红细胞、血小板的输注缩短了强化疗所致骨髓抑制期 ,加速了造血的恢复。

Objective To investigate the clinical and neurodevelopmental profiles of patients with biotinidase deficiency and to determine the efficacy of current therapy with respect to outcome.Methods Six patients aged from 3 months to 14 years with biotinidase deficiency were confirmed by urinary organic acid analysis with gas chromatography/mass spectrometry (GC/MS) and biotinidase assay on dried blood spots. Biotin was supplemented individually (10 ~40 mg/d). Their clinical features, laboratory findings, and treatment...

Objective To investigate the clinical and neurodevelopmental profiles of patients with biotinidase deficiency and to determine the efficacy of current therapy with respect to outcome.Methods Six patients aged from 3 months to 14 years with biotinidase deficiency were confirmed by urinary organic acid analysis with gas chromatography/mass spectrometry (GC/MS) and biotinidase assay on dried blood spots. Biotin was supplemented individually (10 ~40 mg/d). Their clinical features, laboratory findings, and treatment regimen were reviewed.Results All the 6 patients presented with some extent of neurological abnormalities and dermatological lesions. Cases 1 3 had poor feeding, vomiting, seizures, mental retardation, and lethargy onset from their early infancy, with varied degree of anemia, ketosis, acidosis, and hypoglycemia. Case 2 exhibited eczema and dermatitis from his age of 7 months. Case 4 displayed motor deficit and ataxia after 6 months of age, and generalized pustular psoriasis when he was 8 months old. Cases 5 and 6 gradually showed muscle weakness and paraplegia at the age of 7 years and 5 years, respectively. Inflammatory demyelination changes of cervical cord were evident on magnetic resonance imaging in these two patients. Case 6 had progressive optic atrophy, eczema and alopecia. Remarkable elevations of urinary lactate, pyruvate, 3 OH propionate, methylcitrate, propionylglycine, 3 OH isovalerate, 3 methylcrontonylglycine were confirmed in cases 1, 2, 3 and 5. Slight increase of urinary lactate, pyruvate, and 3 methylcrontonylglycine was observed in cases 4 and 6. Biotinidase activities assayed on dried blood spots from all the patients were below 0.1 pmol/(min·3 mm) Biotin supplementation for all the patients, except for case 3 who was not treated, resulted in pronounced and rapid clinical and biochemical improvement. Cases 4 and 6 had residual neurological damage comprising ataxia and motor handicap of legs, due to prolonged disease course.Conclusions Biotinidase deficiency intensively impairs nervous system and skin in the affected patients. Urinary organic acid analysis and blood biotinidase assay are crucial to the diagnosis. Early diagnosis and biotin supplementation can contribute significantly to the improvement of prognosis.

目的 探讨生物素酶缺乏症的临床特征、诊断与治疗方法。方法 运用尿有机酸分析(气相色谱 质谱联用 )及干燥滤纸血片生物素酶测定进行筛查与诊断 ,对 6例生物素酶缺乏症患儿的临床经过进行分析。结果  (1 ) 6例干燥滤纸血片生物素酶活性均小于 0 .1pmol/ (min·3mm)。尿有机酸分析显示例 1、2、3、5乳酸、3 羟基丙酸、丙酮酸、丙酰甘氨酸、甲基巴豆酰甘氨酸、β 羟基异戊酸浓度明显增高 ,例 4、6仅显示乳酸、丙酮酸、甲基巴豆酰甘氨酸增高。 (2 ) 6例各有不同程度的神经系统损害和皮肤粘膜异常。例 1~ 3为婴儿期起病 ,因惊厥、智力运动倒退、呕吐、意识障碍来院 ,合并贫血、酮症酸中毒、低血糖。例 2于 7个月起出现顽固性湿疹 ,口角、肛门周围糜烂。例 4以扭转痉挛及全身性脓疱型牛皮癣为主。例 5、6分别于 7岁、5岁起病 ,进行性肢体运动障碍 ,MRI显示颈髓炎性脱髓鞘病变。例 6伴视神经萎缩、脱发。除例 3未接受治疗外 ,5例经生物素补充治疗后疗效显著 ,皮肤损害均已控制 ,神经系统情况逐渐好转。例 4、6仍存在明显的下肢运动障碍。结论 生物素酶缺乏症患儿临床表现复杂 ,生物素补充治疗疗效...

目的 探讨生物素酶缺乏症的临床特征、诊断与治疗方法。方法 运用尿有机酸分析(气相色谱 质谱联用 )及干燥滤纸血片生物素酶测定进行筛查与诊断 ,对 6例生物素酶缺乏症患儿的临床经过进行分析。结果  (1 ) 6例干燥滤纸血片生物素酶活性均小于 0 .1pmol/ (min·3mm)。尿有机酸分析显示例 1、2、3、5乳酸、3 羟基丙酸、丙酮酸、丙酰甘氨酸、甲基巴豆酰甘氨酸、β 羟基异戊酸浓度明显增高 ,例 4、6仅显示乳酸、丙酮酸、甲基巴豆酰甘氨酸增高。 (2 ) 6例各有不同程度的神经系统损害和皮肤粘膜异常。例 1~ 3为婴儿期起病 ,因惊厥、智力运动倒退、呕吐、意识障碍来院 ,合并贫血、酮症酸中毒、低血糖。例 2于 7个月起出现顽固性湿疹 ,口角、肛门周围糜烂。例 4以扭转痉挛及全身性脓疱型牛皮癣为主。例 5、6分别于 7岁、5岁起病 ,进行性肢体运动障碍 ,MRI显示颈髓炎性脱髓鞘病变。例 6伴视神经萎缩、脱发。除例 3未接受治疗外 ,5例经生物素补充治疗后疗效显著 ,皮肤损害均已控制 ,神经系统情况逐渐好转。例 4、6仍存在明显的下肢运动障碍。结论 生物素酶缺乏症患儿临床表现复杂 ,生物素补充治疗疗效显著 ,早期发现、合理治疗可有效地改善预后。对可疑患者应及早进行尿有机酸分析和血清生物素酶测定等有关?

Objective To study clinical characteristics of children with enthesitis related arthritis (ERA).Methods Twelve patients fulfilling the international league of associations for rheumatolgy (ILAR) criteria for classification of juvenile idiopathic arthritis (JIA) and ERA were referred to our department between August and November, 2002. Their gender, age, family history, clinical manifestations, imaging data, laboratory data and treatment regimens were analyzed retrospectively.Results Of the 12 patients,...

Objective To study clinical characteristics of children with enthesitis related arthritis (ERA).Methods Twelve patients fulfilling the international league of associations for rheumatolgy (ILAR) criteria for classification of juvenile idiopathic arthritis (JIA) and ERA were referred to our department between August and November, 2002. Their gender, age, family history, clinical manifestations, imaging data, laboratory data and treatment regimens were analyzed retrospectively.Results Of the 12 patients, 11 were male, only one was female; their age ranged from 4 to 16 years, and the median age was 10.5 years. Ten (83%) of the patients were older than eight years. Among their first degree relatives, one had definite ankylosing spondylitis (AS), and 3 presented with a history of inflammatory low back pain. Enthesitis occurred in 9. Synovitis occurred in 11, most of which were oligoarthritis, predominantly affecting large joints of the lower limbs in an asymmetric pattern. Seven patients underwent CT scan, and only one had erosions of the sacroiliac joints to achieve a diagnosis of juvenile AS. Ten had fever at the onset, and one had a history of diarrhea preceding the symptoms of arthritis. Urethritis and balanitis circinata occurred in 3 cases fulfilling the classification criteria of Reiter's syndrome, with conjunctivitis in 2 and corneitis in one. Elevated inflammatory indicators such as white blood cell, neutrophil, platelet, erythrocyte sedimentation rate, C-reactive protein,immunoglobulins and serum complement C3 were common during the acute illness. Mild anemia was found in 4, and reactive hemophagocytosis in 4 bone marrow specimens. DNA of human parvovirus B19 was detected in sera of 2 cases. One had positive IgM antibody to coxsackie virus B. All 12 cases were rheumatoid factor negative and HLA-B27 positive. Nonsteroidal anti-inflammatory drugs and sulfasalazine were the mainstay of treatment. Corticosteroids were added in 3 cases as a “bridge” therapy due to the severe systemic inflammation. Methotrexate was used in 4 cases with refractory disease or with the hip involved, and in 2 of them cyclophosphamide was added. Conclusion The clinical characteristics of ERA can facilitate an early diagnosis so as to avoid joint damage and disabilities.

目的 分析附着点炎相关关节炎 (ERA)的临床特征。方法  2 0 0 2年 8~ 11月共收治12例ERA ,全部符合ILAR关于幼年特发性关节炎 (JIA)和ERA的分类标准。对性别、年龄、家族史、临床表现、影像学改变、实验室资料及用药选择进行回顾性分析。结果  12例患儿中 ,男 11例 ,女 1例。年龄 4~ 16岁 ,中位数年龄 10 5岁 ,其中 8岁以上 10例 (83% )。一级亲属中强直性脊柱炎 (AS) 1例 ,炎性下腰痛 3例。 9例存在附着点炎。 11例出现滑膜炎 ,主要为下肢大关节的非对称性少关节炎。 7例对骶髂关节进行了CT扫描 ,1例发现侵蚀样改变而构成幼年AS诊断。 10例有发热 ,1例起病前有腹泻史。 3例因合并尿道炎和漩涡状龟头炎而同时满足赖特综合征的诊断 ,其中 2例出现结膜炎 ,1例合并角膜炎。急性期普遍有炎症指标如白细胞、中性粒细胞、血小板、血沉、C反应蛋白、免疫球蛋白及补体C3 的增高。 4例有轻度贫血。 4例骨髓中见到组织细胞吞噬血细胞现象。 2例血中检测到细小病毒B19DNA ,1例血柯萨奇病毒IgM(+)。本组类风湿因子均为阴性 ,HLA B2 7均为阳性。...

目的 分析附着点炎相关关节炎 (ERA)的临床特征。方法  2 0 0 2年 8~ 11月共收治12例ERA ,全部符合ILAR关于幼年特发性关节炎 (JIA)和ERA的分类标准。对性别、年龄、家族史、临床表现、影像学改变、实验室资料及用药选择进行回顾性分析。结果  12例患儿中 ,男 11例 ,女 1例。年龄 4~ 16岁 ,中位数年龄 10 5岁 ,其中 8岁以上 10例 (83% )。一级亲属中强直性脊柱炎 (AS) 1例 ,炎性下腰痛 3例。 9例存在附着点炎。 11例出现滑膜炎 ,主要为下肢大关节的非对称性少关节炎。 7例对骶髂关节进行了CT扫描 ,1例发现侵蚀样改变而构成幼年AS诊断。 10例有发热 ,1例起病前有腹泻史。 3例因合并尿道炎和漩涡状龟头炎而同时满足赖特综合征的诊断 ,其中 2例出现结膜炎 ,1例合并角膜炎。急性期普遍有炎症指标如白细胞、中性粒细胞、血小板、血沉、C反应蛋白、免疫球蛋白及补体C3 的增高。 4例有轻度贫血。 4例骨髓中见到组织细胞吞噬血细胞现象。 2例血中检测到细小病毒B19DNA ,1例血柯萨奇病毒IgM(+)。本组类风湿因子均为阴性 ,HLA B2 7均为阳性。非甾体抗炎药和柳氮磺吡啶是基本治疗药物。 3例因全身炎症反应剧烈而加用了肾上腺皮质激素作为“桥”治疗。 4例难治性或有髋关节受累者加用了氨甲喋呤 ,其中 2例还联

 
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