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activated platelet     
相关语句
  活化血小板
     Platelet can be obviously activated by 0.1 μmol/L ADP, and 10.0 μmol/L ADP leaded to the peak expression of FIB R and CD62P on the activated platelet membrane surface, FIB R and CD62P positive percent of 87.8% and 81.34% respectively.
     0 .1μmol/LADP可使血小板明显活化 ,10 μmol/LADP使活化血小板膜表面FIB R和CD6 2P表达至峰值 ,阳性百分率分别为 87.8%和 81.3%。
短句来源
     Results API of all concentrations (25 mg/L, 50 mg/L and 100 mg/L) could significantly decrease the MFI of GP Ⅱ b/Ⅲ a in a positive dose-dependent manner, as compared with that in activated platelet untreated with API;
     结果 与活化血小板组比较,不同浓度API(25mg/L、50mg/L、100mg/L)均能够显著抑制GPⅡb/Ⅲa的平均荧光强度,抑制作用强度与用药剂量成正比;
短句来源
     RGDPe(Ac Arg Gly Asp NHCH 2CH 2C 6H 5) is a mimetic of the RGD containing peptide sequence that is recognized by the platelet Ⅱb/Ⅲa receptor. RGDPe inhibit fgn binding to activated platelet(IC 50 =3.64±0 55×10 -5 M) and prevent platelet aggregation in response to ADP in platelet rich plasma(IC 50 =76.42±55 42×10 -5 μM).
     结果RGDPe抑制125I-Fgn与活化血小板GPⅡb/Ⅲa结合的IC50=3.64±0.55×10-5M(RGDS:6.03±1.24×10-5M),抗ADP诱导的血小板聚集IC50=76.42±55.42μM(RGDS:114.67±40.55μM)。
短句来源
     CD62P-FITC monoclonal antibody was used to label activated platelet and CD42a-PE monoclonal antibody was used to label PDMP.
     收集一般性临床资料,用CD62P-FITC单克隆抗体标记活化血小板(CD62P),CD42a-PE单克隆抗体标记PDMP(CD42a),用流式细胞仪检测CD62P、CD42a以测定血浆中活化血小板和PDMP表达率。
短句来源
     Methods: Using125I antihuman activated platelet GMP140 monoclonal antibody, the quantitative measurement of platelet GMP140 in 43 patients with CGN and 20 healthy individuals, by direct unit radioimmunoassay, and GFR by~(99m) TcDTPA were performed.
     方法:采用抗人活化血小板α-颗粒膜蛋白125I标记的单克隆抗体,以单位点放射免疫法直接定量测定43例CGN患者和20例正常人血小板GMP140,并用99mTcDTPA检测GFR。
短句来源
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  血小板活化
     Results:After treatment activated platelet markers(PAC-1 and CD62P)were reduced significantly in three groups(P<0.05 all);
     结果:治疗3d后,三组血小板活化指标PAC-1和CD62P较治疗前均明显降低,P均<0.05;
短句来源
     The purpose of the present study was to find out the relationship between activated platelet, microthrombus formation and postprocedural myocardial injury by measuring platelet membrane protein CD62P and membrane glycoprotein GPⅡb/Ⅲa、D-dimer、CK-MB and cTnI.
     本研究通过检测PCI手术前后CD62P、GPⅡb/Ⅲa、D-dimer、CK-MB及cTnI的变化了解血小板活化、微血栓形成与术后心肌损伤标志物增高之间的关系;
短句来源
     The activated platelet was obviously increased during acute phase of cerebral infarction,and still maintained a higher level in a long time even in the chronic phase compared with control group (CD 41 ,CD 62 P<0.05,CD 63 P<0.01).
     脑梗塞亚急性期和恢复期检测者,其血小板活化程度较急性期检测者为低(CD41、CD62PP<0.05,CD63P<0.01),但与对照组比仍然保持在一个相当高的活化水平上(P<0.05~0.01)。
短句来源
     There is a relation between activated platelet and the degree of seriousness,size of infarction in patients with cerebral infarction. PAC-1 and CD_(62P) can be served as parameter to evaluate the state of cerebral infarction.
     脑梗死病人血小板活化与病情及梗死灶大小有关,PAC-1、CD62P可作为判断病情的指标。
短句来源
     Methods:Salvia miltiorrhiza,which inhibits the expression of fibrinogen receptor(FIB-R) and P-selection(CD 62P) on activated platelet surface in vitro,were analyzed by tri-color flow cytomety.
     方法 :用流式细胞仪三色分析测定丹参在体外对血小板活化膜表面纤维蛋白原受体 (FIB R)和P 选择素 (CD62 P)的表达。
短句来源
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  血小板激活
     EFFECTS OF POTASSIUM QUERCETIN PHOSPHATE ON CALCIUM MOVEMENT OF ACTIVATED PLATELET
     槲皮素磷酸酯钾对血小板激活时钙流动的影响(英文)
短句来源
     Activated platelet induce the inflammatory signals to aggravate the atherogenesis. In the processes of atherothrombosis, platelet become activated and expose fibrinogen-binding sites on the membrane glycoprotein complex GP Ⅱb/Ⅲa.
     血小板的激活在动脉粥样硬化过程中起着关键作用,活化的血小板介导炎症信息的传递而加剧动脉粥样硬化的形成,在此过程中,血小板激活并且暴露其膜上纤维蛋白原结合位点—糖蛋白GPⅡb/Ⅲa复合物,这一过程同时加剧血小板的聚集。
短句来源
     Glycoprotein Ⅱb/Ⅲa(GPⅡb/Ⅲa) also play a key role in the adhesion and aggregation of platelet during coagulation. The activated platelet can result in the changes of GPⅡb/Ⅲa in its conformation,which then intensifies the binding between GPⅡb/Ⅲa and its ligand fibrinogen. Fibrinogen can make bridge between platelets close together,which then accelarates the aggregation of platelets.
     GPⅡb/Ⅲa在凝血过程中的血小板黏附和聚集中也起关键作用,血小板激活导致GPⅡb/Ⅲa构型发生变化,继而使GPⅡb/Ⅲa受体与配体纤维蛋白原的结合加强,纤维蛋白原在相邻血小板分子间形成桥梁,导致血小板聚集的加速。
短句来源
     AIM: To study the effects of PQP on calcium movement of activated platelet METHODS: Using Fura-2 fluorescence technique. RESULTS: PQP inhibited the rise of [Ca2+]i induced by thrombin in platelets.
     目的:研究槲皮素磷酸酯钾对血小板激活时钙流动的影响 方法:用荧光钙离子指示剂Fura-2观察槲皮素磷酸酯钾对血小板胞浆游离钙的影响。
短句来源
  活化的血小板
     Activated platelet induce the inflammatory signals to aggravate the atherogenesis. In the processes of atherothrombosis, platelet become activated and expose fibrinogen-binding sites on the membrane glycoprotein complex GP Ⅱb/Ⅲa.
     血小板的激活在动脉粥样硬化过程中起着关键作用,活化的血小板介导炎症信息的传递而加剧动脉粥样硬化的形成,在此过程中,血小板激活并且暴露其膜上纤维蛋白原结合位点—糖蛋白GPⅡb/Ⅲa复合物,这一过程同时加剧血小板的聚集。
短句来源
     Conclusions The high level Lp(a) and activated platelet PKC may play an important role in onset of acute cerebral infarction. Lp(a) level was closely associated with platelet PKC activation.
     结论 高水平的 Lp( a)与异常活化的血小板 PKC是老年人急性脑梗死的重要致病因素 ,而 Lp( a)与血小板 PKC的活化有密切的关系。
短句来源
     ③The enhancement effect of activated platelet on the procoagulant activity of M was blocked by P selectin McAb.
     ③抗血小板P 选择素单抗能阻断活化的血小板对单核细胞促凝活性的增强效应。
短句来源
     Many studys showed that its formation was associated with many aspects including blood components(lysed red blood cells,exudation of plasma protein,activated platelet),decreased regional cerebral blood flow around hematoma,inflammatory reaction after ICH and thrombin.
     大量的研究表明,血液成分(溶解的红细胞、血浆蛋白的渗出、活化的血小板)血肿周围局部血流量的减少、脑出血后的炎性反应和凝血酶均和脑出血血肿周围水肿的形成有关。
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  activated platelet
Effect of thoracotomy and cardiopulmonary bypass on activated platelet and neutrophil dynamics and platelet emboli in a pig mode
      
AGEPC and IgG-Agg in combination activated platelet aggregation synergistically, whereas release of the granular constituent serotonin was not increased.
      
Modulation of neutrophil functions by activated platelet release factors
      
The model requires estimation of four parameters to fit it to experimental data: shear-dependent platelet diffusivity and resting and activated platelet-surface and platelet-platelet reaction rate constants.
      
Co-localization of CD9 and GPIIb-IIIa (·IIb ·3 integrin) on activated platelet pseudopods and ·-granule membranes
      
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