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antitumor drugs
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  抗肿瘤药物
     STUDIES ON ANTITUMOR DRUGS:SYNTHESIS OF 2,3-DIACETOXY-1,4-BIS,(3′,5′-DIOXO-N~4′-SUBSTITUTED PHENYL PIPERAZINYL METHYL)BENZENE
     抗肿瘤药物2,3-二乙酰氧基-1,4-双-(3′,5′-二酮-N_(4′)-取代苯基哌嗪甲基)苯的合成研究
短句来源
     STUDIES ON ANTITUMOR DRUGS(Ⅲ):SYNTHESIS OF 2,3-DIACETOXY-5-CARBOALKYLOXY-1-(3',5'-DIOXO-N(4')-SUBSTITUTED PIPERAZINYL METHYL) BENZENE
     抗肿瘤药物2,3-二乙酰氧基-5-烷氧羰基-1-(3′,5′-二酮-N(4′)-取代哌嗪甲基)苯的合成研究(Ⅲ)
短句来源
     STUDIES ON ANTITUMOR DRUGS :SYNTHESIS OF 2,5-DIACETOXY-1-(3', 5'-DIOXO-N~(4')-SUBSTITUTED PIPERAZINYL METHYL)BENZENE
     抗肿瘤药物2,5-二乙酰氧基-1-(3′,5′-二酮-N~(4′)-取代哌嗪甲基)苯的合成研究(Ⅱ)
短句来源
     Changes of the Usage of Antitumor Drugs and Analysis of the Tendency in the Hospitals of 6 Cities During 1999-2002
     1999年~2002年6城市医院抗肿瘤药物用药变化及趋势分析
短句来源
     STUDIES ON ANTITUMOR DRUGS: THE SYNTHESIS OF N′, N″-DISPIROTRIPIPERAZINIUMS
     抗肿瘤药物的研究:N′,N″-二螺三哌嗪类化合物的合成
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  抗癌药物
     BCL-2 Protein:A New Reacting Target of Antitumor Drugs
     BCL-2蛋白 :抗癌药物作用的新靶点
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     Results:(1)The HAP nanoparticles and antitumor drugs have suppressive effect on K562 cell line.
     结果:(1)HAP和抗癌药物对K562细胞增殖均有抑制作用;
短句来源
     Purification and 99m Technetium labeling of low density lipoprotein as a targeting carrier of antitumor drugs
     抗癌药物靶向载体低密度脂蛋白的提取和~(99m)锝标记
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     Effect of Antitumor Drugs on Cell Cycle of Human Gastric Cancer Cells
     抗癌药物对人胃癌细胞周期影响的研究
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     EFFECT OF SOME NONINTERCALATIVE ANTITUMOR DRUGS ON THE ACTIVITY OF CALF THYMUS DNA TOPOISOMERASE Ⅰ
     非嵌合性抗癌药物对小牛胸腺DNA拓扑异构酶Ⅰ活力的影响
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  抗癌药物的
     Chernosensitivity of K562 cell and HL60 cell to the common antitumor drugs was tested with MTT assay.
     本文采用MTT法,观察白血病细胞林K562、HL60细胞对临床常用的抗癌药物的敏感性反应。
短句来源
     Electrochemical Studies of Antitumor DrugsⅠ. Fundamental Electrochemical Characteristics of an Iron(Ⅲ) Schiff Base Complex and Its Interaction with DNA
     抗癌药物的电化学研究——Ⅰ.一种Fe(Ⅲ)Schiff碱配合物的基本电化学性质及其与DNA的相互作用
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     In the course of screening new antitumor drugs, a primary high throughput screening method has been established and the inhibitory activity of tested samples against GST π were spectro photometrically determined in 96 well plate through the variance of absorbance at 340nm by using CDNB and GSH as substrate.
     为了筛选新的抗癌药物的增敏剂 ,我们建立了一个 GST-π酶抑制剂的高通量筛选模型 ,该模型以 CDNB和 GSH为底物 ,在 96 -孔板上通过对 340 nm处吸光度的变化来检测样品对 GST-π酶的抑制活性。
短句来源
     This study was to detect drug concentration in axillary lymph nodes of patients with breast cancer after lymphatic chemotherapy (LC), a nd to investigate effect of LC on accumulation of antitumor drugs in regional ly mph nodes through comparing with the effect of intravenous chemotherapy (VC).
     本研究检测乳腺癌患者淋巴化疗(lymphaticchemotherapy,LC)后腋窝淋巴结内的药物浓度,并与静脉化疗(intravenouschemotherapy,VC)作对比,以确定LC能否有效提高区域淋巴结内抗癌药物的聚积。
短句来源
     Anticancer drug-induced apoptosis in killing cancer cells plays an important role in drug treatment of malignant lymphoma. The effects of antitumor drugs will change inevitably if apoptosis- related genes alter in malignant lymphoma.
     在恶性淋巴瘤的药物治疗中,抗癌药物的重要机制是通过诱导肿瘤细胞的凋亡而发挥杀灭作用,如果调控恶性淋巴瘤细胞凋亡相关基因发生改变,必然会引起抗癌药物效应的变化。
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  “antitumor drugs”译为未确定词的双语例句
     AIM:To study the effect of etoposide (VP16) on the death and apoptosis of SHG 44 glioma cell which expresses beta 1, 4 galactosyltransferase Vβ 1,4 GalT V), so as to establish basis for further exploration of the relationship between β 1,4 GalT V and sensitivity of anti tumor drugs.
     目的:研究鬼臼乙叉甙(Etoposide,VP16)对表达β-1,4-半乳糖基转移酶V(beta-1,4-galactosyltransferaseV,β-1,4-GalTV)的SHG-44胶质瘤细胞死亡和凋亡的影响,为进一步探讨β-1,4-GalTV与化疗药物敏感性的关系奠定基础。
短句来源
     METHOD: Using MTT method, we tested the inhibition rates of 17 antitumor drugs in 86 cancer patients, and also analysed the P53 gene status with polymerase chain reactionsingle stranded conformation polymorphism in it.
     方法:用MTT法测定17种抗癌药对86例恶性肿瘤的抑制率,并用聚合酶链反应———单链构象多态分析技术检测其P53基因状况。
短句来源
     RESULTS: The inhibition rates of majority antitumor drugs in the P53 gene normal group were significantly higher than in the P53 gene abnormal group.
     结果:统计比较抗癌药在P53基因正常组和P53基因异常组中的抑制率,发现多数抗癌药抑制率前者显著地大于后者。
短句来源
     With the increase of the concentration of rh--TNFα, its cytotoxic activity increased progressively (P<0. 05),especially on 213 cell and C1184 cell,and the effects of rh--TNFα combined with antitumor drugs were significantly enhanced, as comparedwith rh--TNFα alone.
     其中尤以对213细胞和C1184细胞的作用为明显(P<0.05); 当rh—TNFα与5—Fu或PYM或MMC合用时,其细胞毒作用明显增强(P<0.05)。
短句来源
     Utilization of Antitumor Drugs in Our Hospital During the Period 2001~2005
     我院2001~2005年抗肿瘤药利用分析
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  antitumor drugs
Integrin expression was investigated in MCF-7 human breast adenocarcinoma line and in the MCF-7Dox line, which was selected from MCF-7 by a resistance to multiple antitumor drugs (MDR).
      
The combined use of cytoskeleton inhibitors, which are antitumor drugs, with the recombinant TRAIL preparations may be efficient in therapy of tumors resistant to traditional chemotherapy.
      
The constructs were proposed for generating reporter cell lines from various cultures in order to identify the genetic or chemical factors that modulate the p53 activity and may be employed in new antitumor drugs.
      
The characteristics and the possible mechanisms of action of cytotoxic ribonucleases (RNases), promising antitumor drugs, are described.
      
Geranylgeranyl diphosphate synthase (GGPPS) [EC 2.5.1.29] catalyzes the biosynthesis of geranylgeranyl diphosphate (GGPP), which is a key precursor for diterpenes and, in particular, Taxol, one of the most potent antitumor drugs.
      
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Tenacigenin A was obtained from crude glucoside of Marsdenia tenacissima (Roxb.)Wight et Arn., a folk antitumor drug in Yunnan, by treatment with acid and base. This compound C_(21)H_(32)O_5, mp.255—260℃, belonging to C_(21)-steroidal type, readily yielded a diacetate(Ⅲ)and under forcing condition a triacetate(Ⅳ).Two of the three hydroxyl groups were able to be oxidized to diketone(Ⅵ), which were atta- ched to the six-membered rings.The other two oxygen—atoms existed as in the form of epoxy.Tenacigenin...

Tenacigenin A was obtained from crude glucoside of Marsdenia tenacissima (Roxb.)Wight et Arn., a folk antitumor drug in Yunnan, by treatment with acid and base. This compound C_(21)H_(32)O_5, mp.255—260℃, belonging to C_(21)-steroidal type, readily yielded a diacetate(Ⅲ)and under forcing condition a triacetate(Ⅳ).Two of the three hydroxyl groups were able to be oxidized to diketone(Ⅵ), which were atta- ched to the six-membered rings.The other two oxygen—atoms existed as in the form of epoxy.Tenacigenin A afforded 3β, 12β, 14β-triacetoxy-5α-pregn-7(8), 9(11)-diene- 20-one(Ⅸ), which was hydrogenized with Pt in AcOH to(Ⅹ), by successive treatment with Ac_2O and PTS at 100℃ for 30 hr. Accoring to the above chemical reactions(Fig.3)and data of IR, UV, NMR (Fig.1)and MS(Fig.2), the structure of tenacigenin A has been suggested as(Ⅰ).

通光藤[Marsdenia tenacissima(Roxb.)Wight et Arn.]为云南民间抗癌药用植物。从其藤茎提取的粗甙经水解、皂化后得到一个新的主要甙元,称通光藤甙元甲(tenacigenin A)。通光藤甙元甲分子式 C_(21)H_(32)O_5,熔点255—260℃,属 C_(21)-甾体化合物,能用常法制得二乙酰化合物(Ⅲ),三乙酰化物(Ⅳ)需在剧烈条件下才能得到。三个羟基中有两个可被氧化成六元环酮(Ⅵ)。另两个氧原子则以环氧的形式存在。通光藤甙元甲用醋酐和对甲苯磺酸于100℃加热30小时后得到3β、12β、14β-三乙酰-5α-甾烷-7(8)、9(11)-二烯-20-酮(Ⅸ),后者用铂黑在醋酸中氢化得(Ⅹ)。据上述化学反应和红外光谱、紫外光谱、核磁共振谱及质谱数据,作者提出通光藤甙元甲的结构为(Ⅰ)。

In order to sort out more effective antitumor drugs in the clinic, we tried to employ the method of ampule culture, taking the H7922 cell line (the representative of normal cell line) and Raji cell line (the representative of malignant cell line) as the objects in this experiment. The effect of inhibition of eight antitumor drugs on these two cell lines was observed. This inhibition effect was represented by the 50% growing inhibitory dosage (50% GID), i.e. the dosage of each drug used when...

In order to sort out more effective antitumor drugs in the clinic, we tried to employ the method of ampule culture, taking the H7922 cell line (the representative of normal cell line) and Raji cell line (the representative of malignant cell line) as the objects in this experiment. The effect of inhibition of eight antitumor drugs on these two cell lines was observed. This inhibition effect was represented by the 50% growing inhibitory dosage (50% GID), i.e. the dosage of each drug used when the cell numbers of the test tube after 72 hours cultivation were 50% of the control.

本文用安瓿培养法,以淋巴母细胞样细胞系H7922(代表正常细胞)和Raji细胞(代表恶变细胞)作为试验对象,初步观察了8种抗癌药物对以上二种细胞的不同抑制效应。为临床在选择抗癌药物品种及确定治疗剂量时,提供了简便的实验手段。

Effects of five antitumor drugs on the murine normal hemopoietic and P_(388) leukemic colony-forming cells were studied and compared by means of spleen colony assay. Dose-survival curves for the normal spleen colony-forming units (NCFU-S) and the leukemic spleen colony-forming units (LCFU-S) exposed to harringtonine (H), partial synthetic harringtonine (PSH), homoharringtonine (HH), and cyclophosphamide (CP)exhibited exponential forms. The value of efficacy ratios of the four drugs was 5.15, 6.01,...

Effects of five antitumor drugs on the murine normal hemopoietic and P_(388) leukemic colony-forming cells were studied and compared by means of spleen colony assay. Dose-survival curves for the normal spleen colony-forming units (NCFU-S) and the leukemic spleen colony-forming units (LCFU-S) exposed to harringtonine (H), partial synthetic harringtonine (PSH), homoharringtonine (HH), and cyclophosphamide (CP)exhibited exponential forms. The value of efficacy ratios of the four drugs was 5.15, 6.01, 7.96, and 9.98 in the following order for H, PSH, HH, and CP, respectively. All dosage levels of arabinosylcytosine (Ara-C)used showed no distinct effect on the NCFU-S, but exhibited a strong effect on the LCFU-S with increasing dosage; the dose-survival curve for LCFU-S was decreased to a plateau value of 0.2% at the dosage of 500-1000 mg/kg. No obvious effect of H, HH, and PSH on the dose-survival curves was observed when the doses used were less than 0.3, 0.28 and 0.75 mg/kg, respectively.

用脾集落形成法比较了5种抗癌药对小鼠造血干细胞(NCFU-S)和P_(388)白血病干细胞(LCFU-S)的作用。三尖杉酯碱、半合成三尖杉酯碱、高三尖杉酯碱和环磷酰胺对两类干细胞作用的剂量—反应曲线呈指数形,效能比依次为5.15,6.01,7.96和9.98。阿糖胞苷对NCFU-S无明显作用,但对LCFU-S杀伤作用强,大剂量时剂量—反应曲线趋于水平。当三尖杉酯碱、高三尖杉酯碱和半合成三尖杉酯碱的剂量分别低于0.30,0.28和0.75mg/kg时,对LCFU-S无明显作用,剂量—反应曲线上出现“肩形”,提示LCFU-S在小剂量三尖杉酯类生物碱作用下,有一受亚致死性损伤后修复过程。

 
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