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effect
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  药效
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    Analysis of the fixed effect model by relative operating characteristic curve
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    深部真菌感染治疗药物药效比较研究
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  保护作用
    Protective Effect of ONO-1078 and Related Molecule Changes after Globral Cerebral Ischemia and NMDA-induced Injury in Rats
    大鼠全脑缺血及NMDA脑损伤中ONO-1078保护作用及相关分子变化特点
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    Ischemic-like and Excitotoxicity-induced Injuries Activate 5-lipoxygenase in PC12 Cells and the Protective Effect of Minocycline
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    Evaluation of Nitroglycerin Delay Protection Effect on Myocardial Stuning Using MCE and IBS
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    Protective Effect and Pharmacological Significance of Na~+-Ca~(2+) Exchanger in Heart/Brain Ischemia and β-amyloid Peptide Neurotoxicity
    Na~+-Ca~(2+)交换体在心、脑缺血和β-淀粉样肽损伤中的保护作用及药理学意义
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    Studies on the Neuroprotective Effect and Mechanism of Salidroside, DL0108 and AV
    红景天苷、DL0108及AV的神经保护作用及机制研究
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  “effect”译为未确定词的双语例句
    Study on the Antitumor Effect and Toxicity of F951, a Novel Antisense Phosphorothioate Oligodeoxynucleotide to Bcl-2, on AML, Both in Vitro and in Mice
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    Study of the Protective Effect of Lovastatin on Hypoxia-Reoxygenated Cardiomyocytes
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    The Experimental Study on the Analgesic Effect and Mechanisms of Agmatine on the Inflammatory Pain
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  effect
Finally, we consider the effect on the rate of convergence of not sampling enough local maxima.
      
Finally, we consider the effect on the rate of convergence of not sampling enough local maxima.
      
The method we use is a combination of the smoothing effect of the operator ?t + ?x(2j+1) and a gauge transformation performed on a linear system, which allows us to consider initial data with arbitrary size.
      
Our studies demonstrated an in vivo cardioprotection effect of (N-(3,4,-dimethoxy-2-chlorobenzylideneamino)-guanidine: ME10092) in ischaemic reperfusion injury in the rodent.
      
The compounds were fully characterized by spectral and elemental analyses, and were tested for their effect on gross behavior, antireserpine and anorexigenic activity.
      
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Observations were made to compare the effect of HCl introduced into the small intestine of dogs on pancreatic secretion in the following three conditions: (1) Acute experiments with barbiturates(pentothal sodium or pentobarbital sodium) as an(?)sthesia; (2) Chronic experimental dogs with Thomas pancreatic fistula; (3) Chronic dogs in(2) an(?)sthetized by means of the same barbiturates as in the acute experiments. The results indicated that in the acute experiments, the latent period of pancreatic secretion...

Observations were made to compare the effect of HCl introduced into the small intestine of dogs on pancreatic secretion in the following three conditions: (1) Acute experiments with barbiturates(pentothal sodium or pentobarbital sodium) as an(?)sthesia; (2) Chronic experimental dogs with Thomas pancreatic fistula; (3) Chronic dogs in(2) an(?)sthetized by means of the same barbiturates as in the acute experiments. The results indicated that in the acute experiments, the latent period of pancreatic secretion was about 3-5 minutes and the amount secreted was usually below 20 drops in the first 30 minutes after the introduction of the acid. But in the case of chronic experiments, the results were quite different. The latent period was shorter, usually less than 2 minutes and the amount of pancreatic secretion was usually 10 times as much as in the acute experiments. Moreover, the pancreatic secretion of the chronic fistula dogs was not changed either in latent period or in amount when under the barbiturate an(?)sthesia. From the above results, it is quite natural to assume that the influence of the barbiturate an(?)sthetics, pentothal sodium and pentobarbital sodium on pancreatic secretion induced by HCl in the intestine of dogs is negligible. The great difference in the pancreatic secretion between the acute and the chronic experiments might, therefore, be due to the operative trauma which is unavoidable in acute experiments. Another observation was made to determine the effect of atropine on pancreatic secretion induced by HCl in chronic fistula dogs. The result showed that atropine distinctly reduced the response of pancreas to the acid. The latent period was increased and the amount of pancreatic secretion was decreased as compared with the result obtained in normal chronic fistula dogs. Hence it is quite suggestive that there might exist a nervous component in the mechanism of pancreatic secretion induced by HCl in the small intestine. We wish to express our gratitude to Comrade for his valuable advice and to professors T. C. Shen and T. F. Liu for their encouragement throughout this work.

本實驗比較急性實驗狗、慢性胰瘻狗和經過麻醉的慢性胰屢狗對於鹽酸注入小腸所引起的胰液分泌量和潛伏期,結果證明: (1)在急性實驗情况下,狗胰腺對鹽酸刺激小腸所引起的胰液分泌量遠較在慢性實驗時為少,且潛伏期較長。 (2)巴比妥類麻醉劑:硫賁妥鈉(sodium pentothal)和戊烷巴比妥鈉(sodiumpentobarbital)對鹽酸所引起的胰液分泌量及潛伏期影響極微。 (3)在急性實驗情况下,由鹽酸所引起的胰液分泌量的減少和潛伏期的加長,似乎不是由於巴比妥類麻醉劑的作用,而可能是由於手術創傷的影響。 (4)注射阿托平後,胰腺對於鹽酸刺激小腸所引起的反應顯著减小,故推测在鹽酸引起胰液分泌的機制中可能有神經反射作用的參與。本工作在进行過程中,承蘇聯專家同志親切地給予指導,并承沈(?)淇、劉曾復二教授关懷和支持,(?)此誌謝。

Acute toxicity experiment consisted of one single intraperitoneal injection, while subacute toxicity experiment consisted of one injection daily for 14 days, of tartar emetic and a subsequent 3-day holding period. The mortality of mice from tartar emetic was observed after concomitant injections of 5 testing drugs (procaine-HCl, sodium phenyl acetate, sodium α α'-dimercaptoadipate, sodium mercaptosuccinate and sodium thiosulfate). The effective detoxicants were mixed up with tartar emetic and then injected intraperitoneally,...

Acute toxicity experiment consisted of one single intraperitoneal injection, while subacute toxicity experiment consisted of one injection daily for 14 days, of tartar emetic and a subsequent 3-day holding period. The mortality of mice from tartar emetic was observed after concomitant injections of 5 testing drugs (procaine-HCl, sodium phenyl acetate, sodium α α'-dimercaptoadipate, sodium mercaptosuccinate and sodium thiosulfate). The effective detoxicants were mixed up with tartar emetic and then injected intraperitoneally, once daily for 14 days, to infected mice. The mice were killed after a holding period of another 14 days. Basing on the average number of worms remained in each mouse, the effects of detoxicants on the antibilharzial activity of tartar emetic were compared. The results were as follows: (1) In mice the acute and subacute LD_(50) after intraperitoneal injection of tartar emetic were found to be 38 and 35 mg/kg/day respectively. (2) The mortality of mice from tartar emetic could be markedly reduced by simultaneous injection of procaine, sodium phenyl acetate, sodium α α'-dimercaptoadipate and sodium mercaptosuccinate, while sodium thiosulfate did not afford any protection. (3) Procaine, sodium phenyl acetate, sodium α α'-dimercaptoadipate and sodium mercaptosuccinate did not decrease the therapeutic. activity of tartar emetic, and, moreover, procaine could significantly augment its therapeutic activity against schistosomiasis japonica.

本文叙述了5种药物(盐酸普鲁卡因、苯乙酸钠、aa′-二巯基己二酸钠、巯基丁二酸钠及硫代硫酸钠)对小白鼠腹腔注射吐酒石急性(注射1次,观察3天)及亚急性(注射14天,观察3天)中毒死亡的影响;并将其中有效解毒药分别和吐酒石混合後腹腔注射小鼠14天以治疗日本血吸虫病,然後停药14天解剖,根据平均每鼠余存虫数比较各药对於吐酒石疗效的影响。结果如下: (一)小白鼠腹腔注射吐酒石急性及亚急性LD_(50)分别为38及35毫克/千克/天。 (二)普鲁卡因、苯乙酸钠、aa′-二巯基己二酸钠及巯基丁二酸钠能使小鼠腹腔注射吐酒石急性及亚急性中毒死亡率明显地减低,而硫代硫酸钠则无效。 (三)普鲁卡因、苯乙酸钠、aa′-二巯基己二酸钠及巯基丁二酸钠4种解毒药并不减低吐酒石之疗效,其中普鲁卡因尚能提高吐酒石治疗日本血吸虫病之疗效。

Acetylcholine(I;X=OH,n=1)is a substance possessing intense physiological activity toward the vegetative nerves.It would be interesting to see the variation of physiological activity in the homologous series of choline esters(I). C_nH_(2n+1)·CO·OCH_2·CH_2·N~+(CH_3)_3X~- (I) The literature methods for the preparation of choline esters either by the interaction of β-halogen-substituted ethyl esters with trimethylamine or by the acylation of choline salts are not satisfactory in the purification of the products....

Acetylcholine(I;X=OH,n=1)is a substance possessing intense physiological activity toward the vegetative nerves.It would be interesting to see the variation of physiological activity in the homologous series of choline esters(I). C_nH_(2n+1)·CO·OCH_2·CH_2·N~+(CH_3)_3X~- (I) The literature methods for the preparation of choline esters either by the interaction of β-halogen-substituted ethyl esters with trimethylamine or by the acylation of choline salts are not satisfactory in the purification of the products. Loury prepared a few choline ester salts by the acylation of 2-dimethylaminoetha- nol and the subsequent quaternization of the tertiary amines so obtained,but he did not give the details including the physical constants and the analyses of the products.In the present work this method has been extended,a series of fatty acid esters of choline iodide has been synthesized.The products are well-defined crystalline solids.The lower members of the series are soluble in water,methanol or ethanol;the higher members are only slightly soluble,and the solid substances are greasy and the solutions are soap-like.The higher members gave two melting points,heating to high temperature caused the decomposition of these compounds, trimethylamine hydroiodide was isolated in several cases. When these compounds were used on the isolated rabbits' heart the lower members produced an acetylcholine-like effect,i.e.a slowing of the heart rate, the octanoyl compound was more or less inactive,and from then onwards with increasing chain-length an increasing degree of inhibition of the response to acetyl- choline was produced.From the decanoyl compound onwards with increasing chain length they inhibited spontaneous gastric secretion and the secretion of the vagotomised stomach stimulated by prostigmine in the rats as well if administered intramuscularly.

2-二甲氨基乙醇用相应的脂醯氯醯化后得到的叔胺再用碘甲烷季铵化,合成了一系列晶体的胆碱脂酸酯氢碘酸盐,其中并包括了两个不饱和脂酸酯.在这系列化合物中,短链的化合物对神经系统的作用与乙醯胆碱相仿,但长链化合物却抑制了乙醯胆碱的作用.致谢:本文大部分实验工作系在英国 Birmingham 大学药理系所作,作者对 A.C.Frazer教授的鼓励与协助致谢.

 
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