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antagonistic
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  “antagonistic”译为未确定词的双语例句
    Antagonistic Effect of Propolis on Induced Tumor in Mice
    蜂胶对小鼠移植肿瘤的抑制作用
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    Methods We investigated the production of TGFβ 1,the biological effects of TGFβ and neutralizing antibody HCC cells. Activation of smad 2,3,4 induction of antagonistic smads(6,7),and promoter activities of two target genes, PAI-1 and P15.Results In human cell lines HCC-M and HCC-T, TGFβ accelerates their proliferation.
    方法 利用Northernblot、Westernblot、免疫组化等方法研究TGF - β1 的产生和生物学作用 ,特别是激活Smad2、3、4,诱导起相反作用的Smads(6,7)及其启动子的产生 ,并激活两个靶基因 ,即PAⅠ - 1、p15。 结果 肝癌细胞系HCC -M、HCC-T细胞中 ,TGF—β加速了它的增殖。
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    Methods:The effect of PKC activator phorbol-12-myristate-13-acetate (PMA) on cell adhesion and invasion in vitro of human colon carcinoma cell line HT-29 and the antagonistic effect of PKC inhibitor Staurosporine (SP) on the action induced by PMA were studied by means of an adherent to human umbilical vein endothelial cells (HUVECs) model,an established blood vessel invasive model and the membrane invasion culture system (MICS).
    方法采用体外细胞培养的方法,通过PKC激动剂佛波酯PMA和抑制剂Staurosporine(SP)对大肠癌HT-29细胞粘附人脐带静脉内皮细胞(HUVECs)能力、侵袭血管小块的形态学和侵袭人羊膜能力影响的研究,探讨PKC对HT-29细胞体外粘附、侵袭能力的调控作用。
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    4. Study on the inhibitory effects of HAbl8G/CD147 antagonistic peptides on invasion and metastasis of hepatocellular carcinoma in vitroThe anti-metastasis effects of 9 high affinity pep
    O)运动实验:AP-1~AP-9对HHCC运动能力的影响。 5.HAbl8G/CD147诱导明胶酶产生的信号转导机制第7 页
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    This antagonistic effect is irrespective of the proteasome sensitivity of p-catenin but requires the p-catenin-binding sites and catalytic domains on PCP-2. Expression of PCP-2 in SW480 colon cancer cells results in decreased free pool of P-catenin and increased cadherin-catenin complex stability, with a consequent reduction in p-catenin/TCF reporter gene activity and c-myc cellular levels.
    5.对该细胞系的进一步研究发现其p一catenin转录活性下调,E一cadherin表达水平 升高而游离的p一catenin下降,E一cadherin/p一catenin复合体稳定性提高,并伴随
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  antagonistic
All compounds were characterized by binding affinity determination for 5-HT2A and 5-HT2C subtypes and antagonistic activity for 5-HT2B receptor in rat stomach fundus.
      
None of the new compounds showed affinity for 5-HT2A and 5-HT2C subtypes, but some of them displayed antagonistic activity in rat stomach fundus at micromolar concentrations.
      
However, a combined antagonistic effect was observed in the treatment of K562 cells with hemin (30 μM, 48 h) followed by thermal stress (42°C, 30 min).
      
The results showed that Cd and As had an antagonistic effect on enhancing the growth of Rorippa globosa plants and Cd uptake and accumulation under the low concentration Cd and As treatments.
      
The antagonistic activity of these bacteria towardsEscherichia coli 08,E.
      
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A 1: 1 matched case--control study was conductedin 106 cases with clinical diagnosis of PLC and 106controls. The results are as follows: In the hair ofcases, the mean of selenium was significantly lowerthan that of controls. But copper was higher Themean of Mn and Zn were the same as in caes andcontrols. High intake of protein was a protective fac-tor. A close negative correlation was found between theamount of protein intake and the risk of PLC andthere was a dose response relationship. The resultsfurther provide...

A 1: 1 matched case--control study was conductedin 106 cases with clinical diagnosis of PLC and 106controls. The results are as follows: In the hair ofcases, the mean of selenium was significantly lowerthan that of controls. But copper was higher Themean of Mn and Zn were the same as in caes andcontrols. High intake of protein was a protective fac-tor. A close negative correlation was found between theamount of protein intake and the risk of PLC andthere was a dose response relationship. The resultsfurther provide the information to support thehypothesis that PLC was closely related to HBV infec-tion. There was a synergic interaction betweenHBV infection and low selenium high copper low pro-tein intake, but an antagonistic interaction appeared be-tween anti--HIBs and the factors mentioned above.

本文对106例确诊为原发性肝癌的病例进行了1:1配对病例-对照研究。结果表明:①病例组中硒低而铜高;锰、锌在病例与对照组间无显著差异。但低硒、高铜单独存在时其效应均较低。②蛋白质摄入量与肝癌发生呈密切负相关,并有明显剂量效应关系。③本文进一步证明了HBV感染与肝癌发病密切相关。④低硒、高铜、蛋白质摄入量低分别与HBV感染之间有协同作用,而与抗-HB_s之间有拮抗作用。

The effects of retinoic acid (RA) on the proliferation and differentiation of a newly established human osteosarcoma cell line, HOS-8603, were studied. The results showed that RA could inhibit the proliferation of HOS-8603 cells at different concentrations ranging from 10-6 to 10-10 mol/L, and the inhibitory effect of RA was in a dose-dependent manner. In addition, RA (10-8 mol/L) could also significantly suppress the clonal proliferation of HOS-8603 cells. The effects of RA on the alkaline phosphatase activity...

The effects of retinoic acid (RA) on the proliferation and differentiation of a newly established human osteosarcoma cell line, HOS-8603, were studied. The results showed that RA could inhibit the proliferation of HOS-8603 cells at different concentrations ranging from 10-6 to 10-10 mol/L, and the inhibitory effect of RA was in a dose-dependent manner. In addition, RA (10-8 mol/L) could also significantly suppress the clonal proliferation of HOS-8603 cells. The effects of RA on the alkaline phosphatase activity in HOS-8603 cells were also determined. It was found that the alkaline phosphatase activity could be induced 48 h after treatment with RA (10-7 mol/L). The induction of alkaline phosphatase activity and morphological changes in HOS-8603 cells by RA suggested that RA might play roles in the differentiating processes. The interactions between retinoic acid and dexmathasone (Dex), a synthetic glucocorticoid, in the control of HOS-8603 cell proliferation were also investigated, and it was found that RA at low concentrations (10-8 to 10-10 mol/L) had a synergistic effect, while high concentrations of RA (10-6 and 10-7 mol/L) had a antagonistic effect with Dex on the proliferation of HOS-8603 cells.

本文报道了视黄酸(Retinoic acid,RA)对人骨肉瘤细胞系HOS-8603细胞增殖分化的影响。研究结果表明,不同浓度(10~(-6)~10~(-10)mol/L)的RA均可不同程度地抑制HOS-8603细胞的增殖,而且具有明显的剂量依赖性特点:RA(10~(-8)mol/L)还可明显地抑制HOS-8603细胞的克隆增殖。碱性磷酸酶活性测定表明,RA可使HOS-8603细胞碱性磷酸酶活性提高,结合RA可使HOS-8603细胞形态发生改变,提示RA对HOS-8603细胞的分化过程也有一定影响。不同浓度的RA与10~(-8)mol/L地塞米松联合作用时,低浓度时呈现协同作用,但高浓度时则表现为拮抗作用。

Eighty patients with Non-Small cell lung cancer were treated by cisplatin (DDP) combined with scheme of chemotherapy (MAP,MVP,VP16+DDP,et al).A.B groups were divided with DDP 60mg/M2(Post-operated) and 90mg/M2(treated group) respectively. A group: d1 zofran 8mg+NS 50ml q8h or q 12h i.v. for 2 times, 15'each; DX 5mg iv(after zofran first time taken), then chemotherapy; d2~5 zofran 8mg tid po for four days.B group:d1 Metoclopramide 20mg q3h i.m. for four times,DX 5mg i.v.(after Metoclopramide first time taken),...

Eighty patients with Non-Small cell lung cancer were treated by cisplatin (DDP) combined with scheme of chemotherapy (MAP,MVP,VP16+DDP,et al).A.B groups were divided with DDP 60mg/M2(Post-operated) and 90mg/M2(treated group) respectively. A group: d1 zofran 8mg+NS 50ml q8h or q 12h i.v. for 2 times, 15'each; DX 5mg iv(after zofran first time taken), then chemotherapy; d2~5 zofran 8mg tid po for four days.B group:d1 Metoclopramide 20mg q3h i.m. for four times,DX 5mg i.v.(after Metoclopramide first time taken), Then chemotherapy;d2~5 Metoclopramide 10mg tid po for four days. The evaluation of therapeutic effect: in acnte stage(d1): zofran was 95%(38/40)and excelled the Metoclopramide 57.5%(23/40); in delay stage(d2~5): zofran was 77.5%(31/40) and Metoclopramide 80%(32/40). Side effects of zofran were abdominal distension, constipation, dry stool and increased intestinal peristalsis. Zofran is a strong, highly selective antagonistic drug for 5HT8-acceptor with minimal side effect. It can prevent gastro-intestinal reaction during chemotherapy.

自1991年1月~9月,选择以顺铂为主的联合化疗方案(MAP、MVP、VP16+DDP等)治疗80例非小细胞肺癌。分为A、B二组,即60mg/M2(术后组)和90mg/M2(治疗组)。疗效评定:急性期(d1)枢复宁为95%,优于胃复安(57.5%);延缓期(d2~5)枢复宁为77.5%,胃复安为80%。枢复宁副作用是腹胀、便闭、大便干燥和肠蠕动增强,经对症治疗后均好转,未出现椎体外系反应。

 
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