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chronic hepatitis     
相关语句
  慢性肝炎
     Study on the Relationship between Hepatitis C Virus and the Development of Chronic Hepatitis、Liver Cirrhosis、Primary Hepatic Carcinoma
     丙型肝炎病毒与慢性肝炎、肝硬变、原发性肝癌关系的研究
短句来源
     Immunotherapy of Chronic Hepatitis
     慢性肝炎的免疫治疗探讨
短句来源
     CLINICAL PRELIMINARY OBSERVATIONS ON THE RELATION BETWEEN LIVER-SPECIFIC MEMBRANE LIPOPROTEIN (LSP), CHRONIC HEPATITIS AND CIRRHOSIS
     临床初步观察特异性肝膜脂蛋白和慢性肝炎肝硬变的关系
短句来源
     The Relationship between Suppressor T Cells and the Pathogenesis of Chronic Hepatitis
     抑制性T细胞功能和慢性肝炎病因学的关系
短句来源
     The Significance of Circulating Immune Complexes in the Diagnosis of Chronic Hepatitis
     循环免疫复合物对慢性肝炎的诊断意义
短句来源
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  慢性乙型肝炎
     Clinical and Experimental Study of Acupuncture Treatment in Patients with Chronic Hepatitis B
     针刺治疗慢性乙型肝炎的临床与实验研究
短句来源
     1.The Clinic Study of N-Acetylcysteine Injection Therapy to Chronic Hepatitis B 2.The Study of N-Acetylcysteine-Restrined Apoptosis of Human Hepatocyte in Vitro
     1.乙酰半胱氨酸治疗慢性重度乙型肝炎临床研究  2.高效复合干扰素对慢性乙型肝炎的治疗研究
短句来源
     1. The Experimental Study of rALR on Immunologic Response Induced by Exogenous Antigen in Rat 2. Analysis on Cause of Death in 115 Patients with Hepatic Failure Induced by Viral Hepatitis 3. The Primary Clinical Study of Peginterferon Alfa-2a in the Treatment the Patients with HBeAg-Positive Chronic Hepatitis B and with Drug Fast to Lamivudine
     1.肝再生增强因子对外原性抗原引起机体免疫应答影响的初步研究 2.病毒性肝炎肝衰竭患者死亡原因分析 3.聚乙二醇化干扰素α-2a治疗HBeAg阳性的慢性乙型肝炎及其应用于拉米夫定耐药的初步临床观察
短句来源
     Study on Clinical Characteristics and Occult Mechanism of Chronic Hepatitis B with Occult Syndromes
     隐证型慢性乙型肝炎的临床特点及其隐证机制研究
短句来源
     Immunologic, Pathologic and Clinical Observation of Treatment of Chronic Hepatitis B by HBsAg-iRNA
     乙型肝炎特异性免疫核糖核酸(HBsAg—iRNA)治疗慢性乙型肝炎的临床、免疫及病理观察——附5例报告
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  慢性肝病
     In 5 groups of chronic hepatitis, the difference of positive rate of group A was significant(χ2=23.17, P<0.01), which of group B was not significant(χ2=3.72, P>0.05) and which of group C was significant(χ2=25.74, P<0.01).
     5组慢性肝病间比较,A组阳性率有显著性差异(χ2=23.17,P<0.01),B组阳性率无显著性差异(χ2=3.72,P>0.05),C组阳性率有显著性差异(χ2=25.74,P<0.01)。
短句来源
     The results indicated that serum HA and PⅢ P in chronic hepatitis were significantly higher than in acute hepatitis (P < 0. 05 - 0. 01 ).
     结果表明:慢性肝病各组的HA、PⅢP除慢迁肝的HA外均高于急性肝炎组(P<0.01,P<0.05),慢性肝病组间存在显著性差异。
短句来源
     The PSSI for controls was 0.192±0.068,chronic hepatitis was 0.246±0.057,cirrhosis was 0.541±0.082,cirrhosis Child-pugh A 0.384±0.052,B 0.523±0.07,C 0.680±0.081.If the threshold for cirrhosis was set at PSSI>0.36,the sensitivity was 91.7%,superior to the above other methods.
     PSSI值非肝病对照组为 0 .192± 0 .0 6 8,慢性肝病组为 0 .2 46± 0 .0 5 7,肝硬化门脉高压组为0 .5 41± 0 .0 82。 按肝硬化 Child A、B、C分级 ,三组分别为 0 .384± 0 .0 5 2、0 .5 2 3± 0 .0 72、0 .6 80± 0 .0 81。
短句来源
     Results: The results showed that the levels of serum HA, Un and PⅢ P in chronic hepatitis were significantly higher than that in acute hepatitis (P<0.05, P<0.01).
     结果:慢性肝病各组的HA、PⅢP和Un均高于急性肝炎组(P<0.01,P<0.05),各种慢性肝病组间差异有显著性意义,以活动性肝硬化为最明显。
短句来源
     Methods 30 cases of HCC and corresponding noncancerous tissues, 10 cases of liver cirrhosis, 5 cases of mild chronic hepatitis and 4 cases of normal liver tissues were stained with CD34, CoⅣ and LM by immunohistochemistry.
     方法 对 30例人HCC及相应癌旁组织 ,10例肝硬化 ,5例轻度慢性肝炎和 4例正常肝组织进行了CD34、CoⅣ和LM的免疫组化检测 ,行半定量计数 ,系统地观察SEC及基膜成份在正常、慢性肝病和肝癌中的形态和数量的改变 ,并结合肝癌的临床病理资料进行分析。
短句来源
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  慢性乙肝
     Expression of CD69 in CD4~+ and CD8~+ T lymphocytes in chronic hepatitis
     CD69在慢性乙肝患者CD4~+、CD8~+T淋巴细胞的表达
短句来源
     It was in factor that the high level of IL-6, IL-8 and TNF-α in chronic hepatitis B could be down-regulated to (250.5±33.3) pg/ml, (153.4±22.2) pg/ml, (232.6±21.2) pg/ml by Lamivudine, which was more obvious than that of routine medicine (P<0.01).
     慢性乙肝患者IL-6、IL-8、TNF-α表达水平较高,拉米夫定可显著下调IL-6、IL-8、TNF-α至(250.5±33.3)pg/ml、(153.4±22.2)pg/ml、(232.6±21.2)pg/ml,与常规治疗组相比差异有统计学意义(P<0.01)。
短句来源
     (2)The CD8+/CD45RA+/CD28+ in chronic hepatitis B (10.99±7.33%) group was significantly higher than that of control, while the CD8+/CD45RA+/CD28+ in chronic hepatitis B and ASC group were lower than that of control.
     ②慢性乙肝组CD8+/CD45RO+/CD28+的表达(10.99±7.33%)明显高于正常对照组,而慢性乙肝组和慢性HBV携带组CD8+/CD45RA+/CD28+表达率均明显低于正常对照组;
短句来源
     Expression and research of intrahepatic CD4~+,CD8~+,CD25~+ T lymphocytes in patients with chronic hepatitis B.
     慢性乙肝患者肝组织内CD4~+、CD8~+和CD25~+ T淋巴细胞的表达和研究
短句来源
     The elevation of CD8+/CD28+CD45RO+ subtype was probably related to the progress of chronic hepatitis B and the serum HBeAg conversion.
     CD8+/CD45RO+/CD28+亚型淋巴细胞数的升高与慢性乙肝的病情进展及血清HBeAg转换有关。
短句来源
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  chronic hepatitis
Clinical features of chronic hepatitis B patients with YMDD mutation after lamivudine therapy
      
Objective: To study the clinical features of chronic hepatitis B (CHB) patients with tyrosine-methionine-aspartate-aspartate (YMDD) mutation after lamivudine therapy.
      
In addition, liver cancer is often followed by a procession of chronic hepatitis or cirrhosis, so that hepatic function is damaged obviously on these bases, which may significantly influence lipid and lipoprotein metabolism in vivo.
      
Neurological manifestations of chronic hepatitis C
      
Hemiballism with insular infarction as first manifestation of Takayasu's arteritis in association with chronic hepatitis B
      
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Transfer factor (TP) is a dialysable (ultrafilterable) extract of sensitised leukocytes. It transfers cellular immunity from a skin test positive donor to a skin test negative recipient. Currently TF is recognized as one of the most potent immunological reagents. In this paper the detailed method for preparation of transfer factor from normal blood bank donors has been described and some physico-chemical and biological properties of the TF preparations have been examined. The production of TF is briefly as follows:...

Transfer factor (TP) is a dialysable (ultrafilterable) extract of sensitised leukocytes. It transfers cellular immunity from a skin test positive donor to a skin test negative recipient. Currently TF is recognized as one of the most potent immunological reagents. In this paper the detailed method for preparation of transfer factor from normal blood bank donors has been described and some physico-chemical and biological properties of the TF preparations have been examined. The production of TF is briefly as follows: After separation of the plasma (for plasma products), the buffy coats are harvested and pooled. The contaiminating red blood cells are disrupted by tris buffered or isotonic NH_4Cl solution. The pooled leukocytes having been washed iwice with cold normal saline are then alternately frozen and thawed ten times with dry ice in acetone and a 37℃ water bath. The cell lysate is placed in dialysis tubing and dialysed in the cold for 36~48 hours against running pyrogenfree deionized water. The dialysate is lyophilized. The yellowish white powder is saved and redissolved in the desired volume of pyrogen-free deionized water, and passed through a Seitz filter (EKS). 2ml of the sterile TF solution (equivalent to the dialysate of 4×10~8 leukocytes) are placed in each ampoule. The final product is stored at -20℃.The TF preparation is protein-free as determined by protein precipitating reagents. It contains ca. 148μg of peptides and 14μg of ribose per mg of dry powder. The ultraviolet absorption curve gives a peak at 250~251nm. Adenine, guanine and uracil are present in the paper chromatogram of the TF acid hydrolysate. Silica- gel thin layer chromatography reveals four spots stained with ninhydrin. Sephadex G-25 filtration gives reproducibly a characteristic elution pattern yielding 3 main peaks and several small peaks with many of the eluted peaks beyond the total volume of the column. The results of animal experiments show that the TF preparations are non-toxic, non-anaphylactic and non-antigenic.So far over 160 patients (variously afflicted with herpes zoster, chronic hepatitis B, systemic lupus erythematosus, primary carcinoma of the liver, carcinoma of the lung and leukemia) have been treated with the TF preparations. The results will be published elsewhere.

转移因子是致敏白细胞中的可透析物质,能将一个有免疫能力的人的某些细胞免疫力转移给无免疫力的人。所以供给转移因子可能是一种有效的免疫治疗措施。本文详细介绍综合利用血源制备正常人转移因子的方法,并分析其某些理化性质和生物学性质。收集分离血浆后的白细胞层,用三羟甲基氨基甲烷缓冲的或等渗的氯化铵溶液破坏其中污染的红细胞,白细胞经生理盐水洗涤后进行10次冻融,白细胞匀浆对流动去离子水透析,透析液冷冻干燥、除菌分装。每安瓿2毫升转移因子注射液相当4×10~8个白细胞的透析物,保存于-20℃备用。本制剂蛋白质定性检查阴性;每毫克干粉约含多肽148微克、核糖14微克;在250~251毫微米有吸收高峰;转移因子酸水解液硷基纸层析有三种硷基;硅胶薄板层析呈四个茚三酮显色点;葡聚糖G-25凝胶过滤呈可重复的特征性洗脱图谱,具有3个主要峰及若干个小峰,主要洗脱部分在V_t之后。动物实验表明此制剂无毒性、无过敏性和抗原性。本制剂已用于临床治疗带状疱疹、系统性红斑狼疮、慢性乙型肝炎、原发性肝癌、肺癌及白血病等一百六十余例,结果将另文发表。

Recent studies on viral hepatitis showed that the pathogenesis and the out-come of viral hepatitis are closely related to the immunologic reactions of the patients.Since 1974 we have observed the immunologic states of normal subject, acute hepatitis, chronic hepatitis and cirrhosis with 7 different immunologic tests including those of cellular immunity, humoral immunity and auto-immunity. The results were as follows: Acute hepatitis had a relative good immunologic conditions. While that of chronic...

Recent studies on viral hepatitis showed that the pathogenesis and the out-come of viral hepatitis are closely related to the immunologic reactions of the patients.Since 1974 we have observed the immunologic states of normal subject, acute hepatitis, chronic hepatitis and cirrhosis with 7 different immunologic tests including those of cellular immunity, humoral immunity and auto-immunity. The results were as follows: Acute hepatitis had a relative good immunologic conditions. While that of chronic hepatitis were rather complex (effect of virus, formation of antigen-antibody complex, auto immune reactions, immuno-pathologic lesions act to different extents on the mechanism and pathogenesis of chronic hepatitis.) Cirrhosis showed depression of cellular immunity and disterbances of humoral immunity.

近年来关于病毒性肝炎的研究表明,其发病机理及转归与机体的免疫反应密切相关。五年来我们应用七项免疫学检查指标(包括细胞免疫、体液免疫、自家免疫),对正常人、急性肝炎、慢性肝炎,肝硬化患者的机体免疫状态进行了观察。其结果:急性肝炎患者机体免疫状况尚好,慢性肝炎患者机体免疫情况较复杂。病毒的作用,免疫复合物的产生,自身免疫反应,免疫病理损害不同程度地参与慢性肝炎的发病;肝硬化患者是细胞免疫低下,体液免疫失调。

Ganoderma has been considered to be a precious drug and a panacea for thousands of years in Chinese medicine. In recent years, Ganoderma was found to be useful for the treatment of chronic hepatitis, angina pectoris, neurasthenia and certain other diseases. Based on the clinical clues, we have studied the pharmacological actions of Ganoderma on mouse liver. The alcoholic extracts of Ganoderma lucidum and G. japonicum were shown to impede significantly the elevation of SGPT and the accumulation of lipids...

Ganoderma has been considered to be a precious drug and a panacea for thousands of years in Chinese medicine. In recent years, Ganoderma was found to be useful for the treatment of chronic hepatitis, angina pectoris, neurasthenia and certain other diseases. Based on the clinical clues, we have studied the pharmacological actions of Ganoderma on mouse liver. The alcoholic extracts of Ganoderma lucidum and G. japonicum were shown to impede significantly the elevation of SGPT and the accumulation of lipids in the liver induced by injection of GCl_4 Moreover, the extracts were found to inhibit the infiltration of lipids in the liver caused by dl-ethionine and to decrease the mortality rate of mice given high doses of digitoxin and indomethacin. They were also shown to shorten pentobarbital sleeping time and promote regeneration of the liver in partially hepatectomized mice. All these pharmacological activities of the Ganodermas may be partially responsible for its use as tonics.

紫芝和赤芝酒精提取物对于四氯化碳引起的小鼠血清谷丙转氨酶活力和肝脏甘油三酯含量的升高均有降低作用,并且能减轻乙硫氨酸引起的小鼠肝脏脂肪的蓄积,减少小鼠因大剂量洋地黄毒甙和消炎痛中毒引起的死亡,提高小鼠肝脏代谢戊巴比妥钠的能力,促进部分切除肝脏小鼠的肝脏再生。上述的这些结果可能与灵芝的“扶正培本”作用有一定关系。

 
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