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growth
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  增殖
    The Effect of Zilongjin Recipe on the Growth, Proliferation, Invasiveness and Correlative Gene Expression of Human Gastric Carcinoma BGC-823 Cells
    中药紫龙金对人胃癌BGC—823细胞生长、增殖、侵袭及相关基因表达的作用
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    Regulation of mTOR and MPF in Growth and Proliferation of Cell and Oral Tumor
    mTOR、MPF在细胞及口腔肿瘤生长与增殖中的调控作用
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    Expression of Survivin in Non-hodgkin's Lymphoma and Effects of Anti Survivin Oligonucleotides on Growth and Apoptosis of Lymphoma Cells
    Survivin在非霍奇金淋巴瘤中的表达及其反义寡核苷酸对淋巴瘤细胞增殖与凋亡的影响
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    EFFECT OF BETA-CAROTENE ON SPC-A-1 CELL DNA SYNTHESIS AND CELL GROWTH CYCLE PHASES BY USE OF ~3H-TdR AND FLOW CYTOMETRY
    β-胡萝卜素对人肺腺癌细胞株SPC-A-1增殖和~3H-TdR掺入DNA的影响
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    Expression of epidermal growth factor receptor and proliferating cell nuclear antigen io bile duct carciaoma
    胆管癌表皮生长因子受体、增殖细胞核抗原的表达
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  “growth”译为未确定词的双语例句
    Study on the Expression of Fas/FasL in Human Gliomas and the Inhibitory Effect of FasL cDNA on the Growth of Glioma Cells in Vitro as Well as in Vivo
    人脑胶质瘤Fas/FasL基因表达及FasL cDNA体内外抑瘤作用的研究
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    In Vitro and in Vivo Inhibitory Effect on Growth of Human Gliomas with P21~(WAF-1/CIP1) Gene Mediated by Targeted Non-viral Vector GE7 System
    靶向性非病毒载体GE7系统介导P21~(WAF-1/CIP1)基因治疗脑胶质瘤的实验研究
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    Correlation of Expression of Vascular Endothelial Cell Growth Factor and KDR Receptor to Invasion Potential in Acute Leukemia
    急性白血病VEGF及受体KDR的表达与侵袭潜能关系的研究
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    Investigation of Growth Inhibition of MCF-7 Cell Line by Tamoxifen Combined with Adriamycin、Cisplatin and Paclitaxol
    三苯氧胺联合阿霉素、顺铂及紫杉醇抑制MCF-7细胞株增殖的实验研究
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    The Mechanism of Vascular Endothelial Growth Factor (VEGF) in Micrometastasis of Lewis Lung Carcinoma
    VEGF在Lewis肺癌微转移灶形成中的作用及机制研究
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  growth
Complexity of Homogeneous Spaces and Growth of Multiplicities
      
We give a representation-theoretic interpretation of this number as the exponent of growth for multiplicities of simple G-modules in the spaces of sections of homogeneous line bundles on G/H.
      
A necessary and sufficient geometric condition on the growth of the boundary of approximate tiles is reduced to a problem in Fourier analysis that is shown to have an elegant simple solution in dimension one.
      
We prove a Tauberian theorem of the form $\phi * g (x)\sim p(x)w(x)$ as $x \to \infty,$ where p(x) is a bounded periodic function and w(x) is a weighted function of power growth.
      
A complex Radon measure μ on ?n is said to be of at most exponential-quadratic growth if there exist positive constants C and α such that.
      
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A comparative study of phosphate-activated glutaminase activity has been conducted in some normal and tumour tissues. It was found that:(1) There was a wide range of difference in the glutaminase activity among the tumours so far investigated, which was however not as wide as that observed in the normal tissues. The highest level of glutaminase activity was found in the transplanted thymolymphosarcoma, which was about seven times higher than that in the normal thymus.(2) During the growth of transplanted...

A comparative study of phosphate-activated glutaminase activity has been conducted in some normal and tumour tissues. It was found that:(1) There was a wide range of difference in the glutaminase activity among the tumours so far investigated, which was however not as wide as that observed in the normal tissues. The highest level of glutaminase activity was found in the transplanted thymolymphosarcoma, which was about seven times higher than that in the normal thymus.(2) During the growth of transplanted solid tumours, the glutaminase activity was found to vary with the weight of tumour tissue. An approximate parallelism between the increase in glutaminase activity and the weight of tumour has been observed in some, but not all of the tumours so far studied.(3) The enzyme activity in the regenerating rat liver was lower than that in the normal liver.(4) The possible significance of glutaminase to the growth of some tumours has been discussed.

(1) 本实验比较了一些正常组织与癌瘤组织中谷氨酰胺酶活性。结果发现移植性癌瘤的酶活性以淋巴肉瘤最高。肿瘤间酶活性的变动范围不如正常组织间的变动范围大。诱发性淋巴肉瘤的酶活性较其来源组织为高,但皮肤癌则较正常表皮低。(2) 在肿瘤生长过程中,谷氨酰胺酶活性随肿瘤的重量而有所变化,如艾氏腹水癌(实质型)、脑瘤22、胸腺淋巴肉瘤等的谷氨酰胺酶活性有随肿瘤生长而升高的趋势,但这并不是肿瘤的一般规律,如脑瘤53、Walker癌、肉瘤180等则无此现象。(3) 再生肝的谷氨酰胺酶活性较正常肝低。(4) 讨论了谷氨酰胺酶与某些肿瘤生长的可能关系。

In the 3'-MeDAB induced liver tumour, it was found that the activities of glucoses-6-phosphate dehydrogenase, 6-phosphogluconic dehydrogenase and dipeptidases were higher than those in the normal liver, while the activities of glutamic dehydrogenase, glutaminase(phosphate-activated), ornithine carbamyl transferase, tryptophan pyrrolase, threonine dehydrase and tyrosine transaminase were lower or even absent.Changes in most enzyme activities were observed in the precancerous stage and the pattern of these changes...

In the 3'-MeDAB induced liver tumour, it was found that the activities of glucoses-6-phosphate dehydrogenase, 6-phosphogluconic dehydrogenase and dipeptidases were higher than those in the normal liver, while the activities of glutamic dehydrogenase, glutaminase(phosphate-activated), ornithine carbamyl transferase, tryptophan pyrrolase, threonine dehydrase and tyrosine transaminase were lower or even absent.Changes in most enzyme activities were observed in the precancerous stage and the pattern of these changes followed that in the liver tumour. Thus in the course of carcinogenesis, the activities of glucose-6-phosphate dehydrogenase, 6-phosphogluconic dehydrogenase and dipeptidase(glycylglycine as substrate)increased while those of glutamic dehydrogenase, glutaminase and ornithine carbamyl transferase decreased. However, there was no significant change in the activity of dipeptidase when DL-alanylglycine was used as substrate. Changes in activity of tryptophan pyrrolase and tyrosine transaminase were not pronounced as compared with those of the control. The activity of glutathione reductase in the liver tumour was similar to that of normal liver, but it increased from the 4th to 13th week of feeding of the carcinogen. Threonine dehydrase was remarkably influenced by the nutritive factor of the basal diet so that the effect of 3'-MeDAB on threonine dehydrase could not be observed during carcinogenesis.A comparative study has been made with 2-MeDAB, a non-carcinogenic substance. It had no apparent effect on the above enzymes except that it caused the activity of glutamic dehydrogenase to be higher than that of the control.Further experiments have shown that the specific activities of glutamic dehydrogenase and glutaminase in the mitochondria of liver tumour were lower than those of the control. In the precancerous liver the specific activity of glutaminase in the mitochondria was lower than that of the control, while there was no significant change in the case of glutamic dehydrogenase.From these results together with those from other laboratories a possible biochemical mechanism of the carcinogenesis induced by 3'-MeDAB was proposed(Fig. 12). It was suggested that the change in enzyme activity during carcinogenesis may possibly have resulted from the carcinogen being first metabolized in the liver, leading to a higher activity of the oxidative metabolism of glucose-6-phosphate and exhibiting influences on liver enzymes by the metabolites of 3'-MeDAB through different mechanisms. Consequently there produced an abnormal growth and differentiation of liver cell which became neoplastic with the formation of liver cancer, 2-MeDAB may be metabolized in a different way from that of 3'-MeDAB thus producing different effects on most enzyme activities. It is therefore evident that the specific effect of 3'-MeDAB on the liver enzymesmay be closely related to its property of carcinogenisity.

在3'-MeDAB誘发的肝癌組織中,G-6-PD、6-PGD、二肽酶的活性都較正常肝高,而另一些酶如GDH、GMA、OCT、TP、TD、TTA的活性則較正常肝低或甚至測不出来。大多数酶活性都在癌前期即有明显的变化,其变化情况多趋向于癌的特征,如肝癌組織中活性較高的酶,在引癌过程中其活性較对照組有升高趋势,如G-6-PD、6-PGD、二肽酶(以甘氨酰甘氨酸为底物);肝癌組織中活性降低的酶,在引癌过程中其活性有降低趋势,如GDH、GMA、OCT。但以丙氨酰甘氨酸为底物的二肽酶活性的变化則与对照組基本相似。癌前期TP及TTA活性較对照組都无明显差异。肝癌組織中GSSGR活性与正常肝相似,但在引癌过程中(4—13周)則有升高趋势。苏氨酸去水酶受基础食料中营养因素的影响較大,癌前期看不出3'-MeDAB对它的影响。非致癌物,2-MeDAB,除了使GDH活性升高外,对上述其他酶活性都无明显的影响。肝癌綫粒体內GMA和GDH比活性都較对照組及正常肝綫粒体为低。癌前期肝綫粒体GMA比活性較对照組显著降低,而GDH比活性則无明显改变。根据本实驗及其他实驗室結果,我們认为:3'-MeDAB所引起的肝脏酶活性变化,可能是由于它在肝...

在3'-MeDAB誘发的肝癌組織中,G-6-PD、6-PGD、二肽酶的活性都較正常肝高,而另一些酶如GDH、GMA、OCT、TP、TD、TTA的活性則較正常肝低或甚至測不出来。大多数酶活性都在癌前期即有明显的变化,其变化情况多趋向于癌的特征,如肝癌組織中活性較高的酶,在引癌过程中其活性較对照組有升高趋势,如G-6-PD、6-PGD、二肽酶(以甘氨酰甘氨酸为底物);肝癌組織中活性降低的酶,在引癌过程中其活性有降低趋势,如GDH、GMA、OCT。但以丙氨酰甘氨酸为底物的二肽酶活性的变化則与对照組基本相似。癌前期TP及TTA活性較对照組都无明显差异。肝癌組織中GSSGR活性与正常肝相似,但在引癌过程中(4—13周)則有升高趋势。苏氨酸去水酶受基础食料中营养因素的影响較大,癌前期看不出3'-MeDAB对它的影响。非致癌物,2-MeDAB,除了使GDH活性升高外,对上述其他酶活性都无明显的影响。肝癌綫粒体內GMA和GDH比活性都較对照組及正常肝綫粒体为低。癌前期肝綫粒体GMA比活性較对照組显著降低,而GDH比活性則无明显改变。根据本实驗及其他实驗室結果,我們认为:3'-MeDAB所引起的肝脏酶活性变化,可能是由于它在肝內进行代謝引起G-6-P旁路代謝的活跃,以及3'-MeDAB代謝产物通过各种不同机制对酶的影响所致。这些酶活性的变化可能导致肝細胞的异常生长和异常分化因而形成肝癌(图12)。非致癌物,2-MeDAB,可能与3'-MeDAB的代謝途径不同,因而产生不同的影响,而3'-MeDAB所产生的特殊影响則可能与其致癌作用有关。

Observations on the localization of AFP in rat liver tissue in correlation to serum AFP dynamics during 3'-MeDAB carcinogenesis were carried out by the immunoenzyme technique and the radio-rocket-electrophoresis autography.The following results were obtained:(1) During the precancerous stage, AFP was found in a few liver cells in the pseudolobules, in the cells of the basophilio anaplastic regenerative nodules and in some of the "survival" liver cells and "transitional" cells. Most of these cells showed basophilio...

Observations on the localization of AFP in rat liver tissue in correlation to serum AFP dynamics during 3'-MeDAB carcinogenesis were carried out by the immunoenzyme technique and the radio-rocket-electrophoresis autography.The following results were obtained:(1) During the precancerous stage, AFP was found in a few liver cells in the pseudolobules, in the cells of the basophilio anaplastic regenerative nodules and in some of the "survival" liver cells and "transitional" cells. Most of these cells showed basophilio cytoplasm and appeared dedifferentiated.(2) AFP was not detected in the bile duct carcinomas nor in most of the liver cell carcinomas of low grade malignancy, but was demonstrated in the poorly differentiated liver cell carcinomas. The intensity of AFP synthesis in liver cell carcinomas increased in coincidence with their activity in cell growth and cell proliferation but decreased with cell differentiation. AFP synthesis was not directly related to whether or not the cell was in the mitotic stage.(3) In the liver cell carcinomas, the extent of AFP positive reaction as visualized by the immunoenzyme technique basically paralleled the serum AFP level.The possible mechanism and the histologic basis of the serum AFP "saddle shaped" curve during 3'-MeDAB carcinogenesis are discussed, and a preliminary postulation on the histogenesis of liver cancer so induced is suggested.

本工作采用放射火箭电泳自显术和免疫酶标记定位技术,对大鼠3'-MeDAB诱癌过程血清和肝组织甲胎蛋白(AFP)的动态变化和定位情况进行了观察。发现(1)肝硬化假小叶内的少数肝细胞、嗜碱性间变再生结节细胞、少数“幸存肝细胞”及少数“过渡性细胞”,具有合成AFP的能力。未见肝内其他类型细胞合成AFP。合成AFP的细胞大多具有胞浆嗜碱性、生长活跃和形态上去分化的特点。(2)未见胆管癌细胞合成AFP。肝细胞癌分化好的癌细胞绝大多数也未见合成AFP,分化差的癌细胞合成AFP的能力,基本上与其生长活跃程度呈正相关,与分化程度呈负相关,而与癌细胞是否处于核分裂阶段关系不大。(3)肝癌组织AFP酶标的强度和范围与血清AFP水平之间基本上有平行关系。此外,本文还就什么细胞合成AFP、血清AFP“马鞍型”变化的成因及病理组织学基础等进行了讨论,并根据实验结果,对大鼠3'-MeDAB肝癌的组织发生提出了初步的设想。

 
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