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blood
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  血液
    The Study on Gene Mutations of Two Inherited Blood Diseases in Yunnan Province
    云南省两种遗传性血液疾病的基因突变研究
短句来源
    INFLUENCE OF ACETATE-GOSSYPOL UPON HUMAN PERIPHERAL BLOOD LYMPHOCYTE'S CHROMOSOMES
    醋酸棉酚对人体周围血液淋巴细胞染色体影响的观察
短句来源
    Cytochemical Identification of Hnman T Lympocytes in Peripheral Blood in 248 Cancer Patients
    用细胞化学法检测周围血液T淋巴细胞——附248例癌瘤患者检测结果
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    Acute Myocardial Infarct and Blood Viscosity
    急性心肌梗塞与血液粘度
短句来源
    A Biochemical and Histological Investigation on G6PD Deficiency in Blood
    G6PD缺陷症血液生化及组织学研究
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  血压
    Experimental Study on the Mechanism of Blood Pressure Elevation in the Insulin-Resistant State
    胰岛素抵抗血压升高机制的实验研究
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    Blood Pressure Distribution in Normal Children and Evaluation of Diagnostic Criteria of Hypertension in Children
    正常儿童血压的分布及儿童高血压诊断标准的探讨
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    Experiments on the Accuracy of Measuring the Blood Pressure of Children
    关于准确测量儿童血压的实验观察
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    An Investigation of the Relationship between the Concentration of Sodium and Potassium in Erythrocytes and Plasma and Blood Pressure in 270 Normal Children and Adolescents in Wuhan
    武汉市270名儿童少年红细胞内外钠、钾浓度及其与血压关系的调查
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    The Study of the Relationship Between Body Weight and Blood Pressure
    体重与血压关系的研究
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  “blood”译为未确定词的双语例句
    The Study on Vulnerable Blood in the Elderly Male Patients with CAD and Effect of Hcy on the Expression of TM and TFPI in ECV304 Cells
    老年男性冠心病患者脆性血液的临床研究及Hcy对内皮细胞表达TM和TFPI的影响
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    Mechanisms of ABCA1 Gene on Atherosclerogenosis in Blood Vessel Cells
    血管细胞ABCA1基因在动脉粥样硬化发生中作用机制的研究
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    Effects of Apolipoprotein A5-1131T>C Polymorphism on Blood Lipids and Coronary Heart Disease and Strokes
    载脂蛋白A5-1131T>C基因多态性与血脂及心脑血管疾病
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    A Case of Am Blood Group Family
    一例正常献血员Am家系
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    Study on the Activity of rRNA Genes in Peripheral Blood Lymphocyte of the Normal Middle-aged and Old People
    正常中、老年组外周血淋巴细胞中rRNA基因活性的研究
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  blood
A slightly elevated fasting blood glucose profile, 1.5 fold higher serum insulin levels and impaired glucose tolerance was prevalent in ethanol treated rats.
      
Picroliv significantly reverted most of the above said altered blood and hepatic parameters in the alcohol-fed male and female rats to almost normal levels.
      
Investigations of their biodistribution in mice showed all five pseudo-peptide chelators (MGQ, MGGQ, MAGQ, MVGQ, MFGQ) are rapidly cleared from blood, mainly through renal clearance.
      
99mTc-MGGQ and 99mTc-MVGQ had high kidney uptake, quick blood clearance and high activity ratios of kidney to blood, thus showing potential application as renal imaging agents.
      
Anilines 6a, b, d, and h demonstrated nonselective inhibition of COX-1 and -2 in human whole blood.
      
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Cardiac output was determined according to Fick's principle, while renaI blood flow (RBF) and glomerular filtration rate (GFR) were measured from the clearances of PAH and creatinine. One hour after an i.v. injection of guanethidine 5 mg/kg, normal dogs showed a very slight hypotension, which was relatively more noticeable in renal hypertensive dogs. The cardiac output, stroke volume, and ventricular work tended to decrease tn normotensive dogs, but tended to augment in hypertensive dogs. These alterations,...

Cardiac output was determined according to Fick's principle, while renaI blood flow (RBF) and glomerular filtration rate (GFR) were measured from the clearances of PAH and creatinine. One hour after an i.v. injection of guanethidine 5 mg/kg, normal dogs showed a very slight hypotension, which was relatively more noticeable in renal hypertensive dogs. The cardiac output, stroke volume, and ventricular work tended to decrease tn normotensive dogs, but tended to augment in hypertensive dogs. These alterations, however, were statistically insignificant. The total peripheral resistance was not much influenced by guanethidine in normal dogs, yet markedly diminished in hypertensive dogs. Diuresis was evident during the 1-hour postmedication period in both models. In hypertensive dogs, i.v. administrations of guanethidine caused a conspicuous rise of RBF and GFR, but a great reduction of the renal vascular resistance. In normal rats, following an i.p. injection of 20 mg/kg, the circulating blood volume (studied with I~(131)-labeled serum protein) exhibited a slight but non-significant increase within 3 hours, and returned to the original level at 24 hours. No significant change was found in DOCA-treated rats. Normal rats receiving guanethidine revealed an increase of blood volumes in all organs, especially spleen and kidney. These results demonstrate that the hypotensive action of guanethidine is not via the reduction of blood volume, but by way of a vasodilator action, which particularly amends the ischemic state of kidney and thus alleviates the pathologic process of renal hypertension.

本文用正常血压及腎型高血压狗研究胍乙啶对心和腎的血流动力作用,并用大白鼠試驗其对循环及器官血容量的影响。根据Fick氏原則測定心輸出量,同时从腎脏对于对氨基馬尿酸及肌酐的清除率計算腎血流量及腎小球滤过率。靜脉注射胍乙啶5毫克/公斤1小时后,正常狗血压略有降低,高血压狗降压則較为明显。胍乙啶对正常血压狗的心輸出量、心搏量及心室作功量有減 低的趋向,而对腎型高血压狗則略有增加,但均不显著。胍乙啶不影响正常狗的外周血管阻力,而能显著降低腎型高血压狗的总外周阻力,扩张其外周血管。注射胍乙啶后,正常血压及腎型高血压狗的尿量增加均非常显著。腎型高血压狗的腎小球滤过率及腎血流量都非常显著地低于正常狗,腎血管阻力則大大地高于正常狗,靜脉注射胍乙啶后,滤过率及血流量均明显增加,而血管阻力則大大降低。正常大白鼠腹腔注射胍乙啶20毫克/公斤后,3小时內循环血量略有增加,但不显著,6小时后开始恢复,2斗小时后回至原水平。对注射DOCA的大白鼠的循环血量則沒有明显影响。正常大白鼠注射胍乙啶后,各器官及組織內合血量普遍有增加趋势,其中以脾及腎的血容量增加最为明显,說明胍乙啶对这些部位的血管有扩张作用。上述資料証明:胍乙啶的降压作用并非通...

本文用正常血压及腎型高血压狗研究胍乙啶对心和腎的血流动力作用,并用大白鼠試驗其对循环及器官血容量的影响。根据Fick氏原則測定心輸出量,同时从腎脏对于对氨基馬尿酸及肌酐的清除率計算腎血流量及腎小球滤过率。靜脉注射胍乙啶5毫克/公斤1小时后,正常狗血压略有降低,高血压狗降压則較为明显。胍乙啶对正常血压狗的心輸出量、心搏量及心室作功量有減 低的趋向,而对腎型高血压狗則略有增加,但均不显著。胍乙啶不影响正常狗的外周血管阻力,而能显著降低腎型高血压狗的总外周阻力,扩张其外周血管。注射胍乙啶后,正常血压及腎型高血压狗的尿量增加均非常显著。腎型高血压狗的腎小球滤过率及腎血流量都非常显著地低于正常狗,腎血管阻力則大大地高于正常狗,靜脉注射胍乙啶后,滤过率及血流量均明显增加,而血管阻力則大大降低。正常大白鼠腹腔注射胍乙啶20毫克/公斤后,3小时內循环血量略有增加,但不显著,6小时后开始恢复,2斗小时后回至原水平。对注射DOCA的大白鼠的循环血量則沒有明显影响。正常大白鼠注射胍乙啶后,各器官及組織內合血量普遍有增加趋势,其中以脾及腎的血容量增加最为明显,說明胍乙啶对这些部位的血管有扩张作用。上述資料証明:胍乙啶的降压作用并非通过循环血量的減少,而是由于外周血管的扩张所致。胍乙啶能扩张腎血管,改善腎缺血状态,从而緩解腎型高血压的病理生理过程。

(1)In the chloralose-urethanized or curarized rabbits,infusion of saline or blood into the right auricle in amount of 8—10 ml/kg at the rate of 0.5—1.0 ml/sec induced bradycardia and hypotension. (2)Section of sino-aortic nerves and bilateral vagotomy did not alter the negative chronotropic effect due to auricular infusion,thus indicating that a local mechanism was responsible for the effect. (3)Distension of the right auricle by inflating an indwelling balloon with various pressures(20—70 mm Hg)also evoked...

(1)In the chloralose-urethanized or curarized rabbits,infusion of saline or blood into the right auricle in amount of 8—10 ml/kg at the rate of 0.5—1.0 ml/sec induced bradycardia and hypotension. (2)Section of sino-aortic nerves and bilateral vagotomy did not alter the negative chronotropic effect due to auricular infusion,thus indicating that a local mechanism was responsible for the effect. (3)Distension of the right auricle by inflating an indwelling balloon with various pressures(20—70 mm Hg)also evoked bradyeardia which persisted after cutting the vagi.Plotting the change in heart rate against pressure within the balloon,a non- linear relationship was revealed.The bradycardia observed could not be attributed to anoxemia resulting from reduced venous return,since the temporary occlusion of superior and inferior venae cavae failed to evoke bradycardia. (4)The blockage of sino-auricular node by local novocaine infiltration as well as by local or general hypothermia(26—28℃)abolished the negative chronotropio res- ponse to the atrial infusion.Therefore it was suggested that such a response occurred as a result of alteration in the functional state of sino-auricular node. (5)Under our experimental conditions,the cardiac acceleration reflex originally found by Bainbridge could not be demonstrated in the rabbits.

(一)在氯醛糖和氨基甲酸乙酯麻醉的兔或未麻醉的箭毒化兔,右心房内输液(输入量为8—10毫升/公斤,输入速度为0.5—1.0毫升/秒),可规律地引起心率减慢和动脉血压下降。(二)右心房输液引起的心率变化,在切断两对缓冲神经和迷走神经后并不消失,说明此种输液效应并非反射作用。(三)经颈外静脉向右心房内引入一小气囊,以不同压力扩张后,也可导致心率减慢。此时气囊内压值与心率变化之间的关系不是完全呈直线式的。切断迷走神经后,效应仍存在;且此效应也不是由于气囊扩张阻断血液回流所致。(四)以奴佛卡因溶液阻断窦房结区后,右心房输液不再引起心率减慢。全身低温或选择性心脏低温(26—28℃)后,大多数实验中的心率在输液后不再变慢,由此说明右心房内输液对心率的影响是刺激直接作用于窦房结而改变其机能活动的结果。(五)在我们的实验条件下,从兔身上不能证示有 Bainbridge 反射的存在。

Intracardiac phonocardiography was studied both in human beings and animals.The local- made microphone sealed in the tip of a Courn- and's cardiac catheter was used.The phonocar- diogram was recorded through N.E.P.mul- tichannel physiological recorder. Ten noncardiac patients were subjected to the study.To each of them a No.7 phonoca- theter was inserted through the peripheral vein to the right heart.Heart sounds were recorded in the pulmonary artery,the right ventricle and the right auricle.Ordinary phonocardiogram,...

Intracardiac phonocardiography was studied both in human beings and animals.The local- made microphone sealed in the tip of a Courn- and's cardiac catheter was used.The phonocar- diogram was recorded through N.E.P.mul- tichannel physiological recorder. Ten noncardiac patients were subjected to the study.To each of them a No.7 phonoca- theter was inserted through the peripheral vein to the right heart.Heart sounds were recorded in the pulmonary artery,the right ventricle and the right auricle.Ordinary phonocardiogram, limb leads electrocardiogram and carotid pulse tracing were recorded simultaneously.In the animal study,ten dogs were used.Under ex- perimental condition,eight dogs had their hearts exposed through open-chest,No.4 or No.7 phonocatheter was inserted in each heart cham- ber,the ascending aorta and the pulmonary artery either by direct puncture or through in- cision.Additional tracings were recorded when aortic stenosis and pulmonary stenosis had been artificially created.In the remaining two dogs, the phonocatheter being inserted through jugular vein,tracings from right heart were recorded only. All the intracardiac phonocardiographic tracings were carefully analyzed and compared with those ordinary phonocardiograms.The results are presented in detail. The authors believe that:(1) intracardiac phonocardiogram which has been obtained in each heart chamber or great vessel reflects the heart sound produced there;(2) the 2nd com- ponent of the 1st heart sound is likely produced by closure of the atrioventricular valves,while the 3rd component is produced by opening of the semilunar valves;(3) there is always a systolic murmur in the pulmonary artery;(4)cardiac murmur is transmitted in the direction of blood flow,and is not transmitted in the opposite direction of blood flow;(5)when intracardiac phonocardiogram is recorded care is taken not to include the sound produced by friction bet- ween microphone and endocardial surface of the heart.

本文报告应用国产微型心音图微音器,通过心脏插管检查,进行人及狗心腔内心音图检查的结果。认为此微音器可以记录人的一侧心脏腔内的心音,对杂音的起源有定位作用,是诊断心脏病中的一项有价值的工具。文中对10例正常人右心各腔与10只狗左右心各腔的心音图进行分析,并就正常人右心各腔心昔图的特点、第一音的第二、三部分的发生机制、心腔内杂音的传播情况以及本检查应注意的事项等方面,加以讨论。

 
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