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syndrome
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  综合征
    Inflammation in Acute Coronary Syndrome and Anti-inflammation Mechanisms of Statin
    急性冠脉综合征炎症反应及他汀类降脂药的抗炎机制
短句来源
    The Study of Angiographic Evaluation on Tissue Level Perfusion in Patients with Non-ST-eleva-tion Acute Coronary Syndrome
    非ST段抬高型急性冠脉综合征患者组织微灌注的冠脉造影评价研究
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    Study of Diagnosis, Risk Stratification, and Epidemiology in Brugada Syndrome
    Brugada综合征诊断、危险分层和流行病学调查的研究
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    Clonality Analysis and Mutational Status of IGVH Gene in Richter's Syndrome
    Richter's综合征:IGVH基因克隆重排及突变分析
短句来源
    Association Study of Single Nucleotide Polymorphisms of SCN5A and HCN4 Genes and Sick Sinus Syndrome
    SCN5A与HCN4基因单核苷酸多态性与病态窦房结综合征的关联研究
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  “syndrome”译为未确定词的双语例句
    Experimental Study of the Effect of Aclacinomycin on the Cell Line from Myelodysplastic Syndrome (MDS-RAEB) in Vitro
    阿克拉霉素对骨髓增生异常综合征细胞株(RAEB型)细胞作用的体外实验研究
短句来源
    Expression and Significance of hTERTmRNA in Myelodysplastic Syndrome and the Effect of hTERT in Apoptosis of MUTZ-1 Cells Induced by Arsenic Trioxide
    hTERT基因在骨髓增生异常综合征中的表达及其在三氧化二砷诱导MUTZ-1细胞凋亡中的作用研究
短句来源
    Study on Apoptosis of Myelodysplastic Syndrome Marrow Cells
    骨髓增生异常综合征细胞凋亡的研究
短句来源
    Study on the Gene Transcription Pattern in Patients with Myelodysplastic Syndrome
    骨髓增生异常综合征患者的基因表达谱研究
短句来源
    MYELODYSPLASTIC SYNDROME: A HISTOCHEMICAL STUDY IN 38 CASES
    骨髓增生异常综合征 (3) 38例组织化学染色分析
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  syndrome
It is an essential enzyme required for replication of the acquired immunodeficiency syndrome (AIDS) virus.
      
Detection of a new mutation (T1140C) in a patient with Hunter syndrome from Guangdong, China
      
This study identified mutations of the idurnate-2-sulfatase (IDS) gene in a patient with Hunter syndrome, and established a basis for the diagnosis of the prenatal gene of Hunter syndrome.
      
It is possible that this mutation severely impairs enzymatic activity and is the underlying basis for the pathology seen in this patient with Hunter syndrome.
      
Association of metabolic syndrome with arterial compliance in children and adolescents
      
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Two cases of Bartter 's syndrome were reported and current view of clinical features, pathological changes, etiology, pathogenesis, diagnosis and treatment of this syndrome were briefly reviewed.The etiology of this syndrome is not clear yet. It may be familial and is probably inherited as a recessive trait, characterized by renal juxtaglo-merular hyperplasia and hypertrophy, hypokalemic and hypochloremic alka-losis, increased secretion of R-A-A system and Prostaglandins.Blood pressure is normal...

Two cases of Bartter 's syndrome were reported and current view of clinical features, pathological changes, etiology, pathogenesis, diagnosis and treatment of this syndrome were briefly reviewed.The etiology of this syndrome is not clear yet. It may be familial and is probably inherited as a recessive trait, characterized by renal juxtaglo-merular hyperplasia and hypertrophy, hypokalemic and hypochloremic alka-losis, increased secretion of R-A-A system and Prostaglandins.Blood pressure is normal or slightly decreased associated with marked resistance to the pressor action of infused angiotensin I and defect in chloride reabsorption of renal tubules which may be the essential cause of Bartter 's syndrome.There is no reliable and acceptable parameter for establishment of diagnosis. The diagnosis would be made mainly by clinical analysis of case history and results of clinical and laboratory examinations and exclusion of "pseudo-Bartter 's syndrome" .Restoration of body potassium by potassium loading, administration of aldo-sterone antagonist and prostaglandin synthetase inhibitor and β-blockade have been widely used in the treatment of the syndrome. Yet discontinuance of therapy is usually associated with return of clinical symptoms.

本文报告两例Bartter's综合征并结合文献简要介绍本病的临床特点、病理改变、病因、发病机理、诊断和治疗方法的近代观点.本病的特点是肾小球旁器增生肥大,低血钾、低血氯性碱中毒,R-A-A系统和P-Gs分泌增多,血压正常或偏低,对外源性AT-I的升压反应低下及肾小管回吸氯障碍;诊断主要靠综合分析临床特点和实验室各项检查结果,排除各种“假征”;尚无满意疗法,补钾及应用醛固酮拮抗剂、前列腺素合成酶抑制剂和β-阻断剂虽有一定疗效,但停药后症状复现.

Six cases of male patients, ages ranged from 3 to 8 years,with electrocardiographic and clinical evidences of sick sinus syndrome (SSS) are described.The common symptoms, were persistent bradycardin.fatigue and dizziness. X-ray examination demonstrated moderate cardiac enlargement in only one case. Symptoms related to myocarditis in 4, and one might be a case of congenital SSS.Although SSS occurs most frequently in children with congenital heart disease,particularly following correction cardiae surgery,...

Six cases of male patients, ages ranged from 3 to 8 years,with electrocardiographic and clinical evidences of sick sinus syndrome (SSS) are described.The common symptoms, were persistent bradycardin.fatigue and dizziness. X-ray examination demonstrated moderate cardiac enlargement in only one case. Symptoms related to myocarditis in 4, and one might be a case of congenital SSS.Although SSS occurs most frequently in children with congenital heart disease,particularly following correction cardiae surgery, it may occurs in acquired herat disease or otherwise normal individual. Recognition of the SSS in childhood is important and the treatment must he given according to the severity of symptoms.

本文报告小儿病态窦房结综合征6例,均系男孩。年龄3~8岁。病程最短10个月,最长已8年。主要临床表现有乏力、头晕及持续性心动过缓。X线检查仅1例心影中度增大。心电图表现:窦性心动过缓3例;结性心律5例;窦房传导阻滞3例;Ⅱ度房室传导阻滞1例;心房扑动及颤动1例。4例经阿托品试验为阳性。4例疑为病毒性心肌炎。2例病因不明,其中1例于胎儿期即有心动过缓,3岁时心电图示结性心律,心房扑动及颤动。文中介绍了本病的病因,临床表现,诊断及防治问题。

The clinical features of 120 patients with paroxysmal atrial fibrillation (PAF) were analyzed. Among them, 94 were male, 26 female. Their ages ranged from 20 to 74 years. Their etiologic diagnoses fell into 6 groups; coronary 41 cases, idiopathic 26, hypertensive 20, rheumatic 19, sick sinus syndrome 6, and miscellaneous 8. The average period of observation was 5 years with a range of 1-26 years. Nearly all patients complained of palpitation and feeling of irregular heart beats during the attacks of PAF;...

The clinical features of 120 patients with paroxysmal atrial fibrillation (PAF) were analyzed. Among them, 94 were male, 26 female. Their ages ranged from 20 to 74 years. Their etiologic diagnoses fell into 6 groups; coronary 41 cases, idiopathic 26, hypertensive 20, rheumatic 19, sick sinus syndrome 6, and miscellaneous 8. The average period of observation was 5 years with a range of 1-26 years. Nearly all patients complained of palpitation and feeling of irregular heart beats during the attacks of PAF; chest oppression and chest pain were only complained of in patients with organic heart disease. 26 cases (21.67%) had atrial premature beats before the attacks, 15 of them belonging to the coronary group. During the attacks, 32 cases (27%) had polyuria, a feature observed in 21 of the coronary and hypertensive groups. The amplitude of the f waves in lead V_1 during PAF was the highest in the rheumatic group (average 1.89 mm). No significant difference was found in the amplitude of the f wave between the other groups. Generally, the clinical status of the patients remained fairly stable during the period of observation. However, various complications were noted in 16 patients, which included cerebral embolism, heart failure, Adams-Stokes attacks. One patient with coronary heart disease died suddenly 3 years after his first attack of PAF. 22 cases turned to persistent atrial fibrillation after an average period of 5 years. These features were more common in the rheumatic, coronary and hypertensive groups and in the patients with the sick sinus syndrome, while in the idiopathic group only 2 patients developed persistent atrial fibrillation during the period of observation. It is concluded that clinically it is possible to distinguish the patients with the idiopathic form of PAF from those with coronary heart disease.

本文分析了阵发性心房颤动120例,按其病因分为六组:风湿性心脏病组、冠状动脉硬化性心脏病组、高血压组、特发性A组与B组、病态窦房结综合征组及其他组,对其病因、临床表现、心电图、并发症及预后作了初步分析与探讨,经过1~26年(平均5年)的随访观察,特发性组并发症最少,预后最佳.

 
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