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response
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  效应
    Structure of Recombinant Adenovirus Encoding Her-2/neu Gene of Extracellular and Transmembrane Domains Protein and Their Eliciting Immunological Response
    Her-2/neu膜外及跨膜区蛋白基因重组腺病毒的构建和诱导的免疫效应
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    The Construction of ERα Expression Vector Containing Vitamin D_3 Response Elements, and Its Effects on ER-negative Breast Cancer Cells in Combination with Tamoxifen
    1α,25二羟维生素D_3靶控ERα基因表达载体构建及其联合他莫西芬对ER阴性乳腺癌细胞抑制效应的研究
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    DOSE RESPONSE CURVE FOR MICRONUCLEUS OF CYTOKINESIS-BLOCK METHOD IN HUMAN LYMPHOCYTES AFTER ~(60)Co-GAMMA RAY EXPOSURE
    ~(60)Coγ射线诱发人淋巴细胞胞质分裂阻断法微核剂量效应关系的研究
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    EFFECT OF GYNOSTEMMA PENTAPHYLLUM ON IRRADIATION RESPONSE OF HUMAN GASTRIC ADENOCARCINOMA(SGC-7901)CELLS
    绞股蓝对人胃腺癌(SGC-7901)细胞~(60)Co照射效应的影响
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    The experimental research on antitumor immune response of tumor-bearing mice by S-180 tumor cells and BCG vaccines
    S-180瘤苗联用BCG治疗效应的研究
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    Results Of 29 cases after arterial perfusion, 2 cases were achieved complete response (CR), 18 cases were partial response (PR), 5 cases with stable disease (SD) and 4 cases with progressed disease (PD). The total response was 69.0%.
    结果29例患者经动脉灌注化疗后近期疗效为:完全缓解2例,部分缓解18例,无变化5例,进展4例,总有效率(PR+CR)为69.0%。
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    Results:The overall response rate was 60.87% including CR 3 cases and PR 11 cases,NC 5 cases,PD 4 cases. MST is 5.5 months.
    结果:23例患者近期治疗总有效率为60.87%,其中完全缓解(CR)3例,部分缓解(PR)11例,稳定(NC)5例,无效(PD)4例,中位生存时间(MST)为5.5月。
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    Results:Two patients achieved complete response(CR),14 patients partial response(PR),7 patients stable disease(SD),5 patients progression disease(PD).
    结果:全组28例均可评价,其中完全缓解(CR)2例,部分缓解(PR)14例,稳定(SD)7例,进展(PD)5例,总有效率(CR+PR)57.1%。
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    The blood supply of pulmonary matastasis was enhanced if the neopiasm was near to the inner region of the lung. Among 70 lesions in embolization group,30 obtained CR,25 PR,10 NC and 5 PD and the response rate was 78.6%.
    行支气管动脉栓塞后完全缓解(CR)30枚,部分缓解(PR)25枚,无变化(NC)10枚,进展(PD)5枚,总有效率78.6%。
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    Results Of the 28 patients,16 achieved CR,and 8 patients achieved PR,with a total response rate 85.7%. 1–year survival rate was 60.7%(17/28).
    结果肿瘤病灶完全缓解(CR)16例(57.1%),部分缓解(PR)8例(28.6%),总有效率(CR+PR)为85.7%,1年生存率为60.7%。
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  “response”译为未确定词的双语例句
    The Construction of Expression Vector of Fas/RAR Alpha Fusion Gene Containing Retinoic Acid Response Elemennt, and Its Effect Induced by Retinoic Acid on the Apoptosis of Tumor Cells
    视黄酸依赖Fas/RARα融合基因表达载体构建及其诱导肿瘤细胞凋亡的研究
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    The Study of Regional Cell Immune Response and Clinical Effect on Ultrasonically Guided Superantigen Injection with Microwave Coagulation Therapy for Hepatocellular Carcinoma
    超声引导瘤区注射高聚生协同微波治疗肝癌局部细胞免疫变化与临床疗效的研究
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    Prevent and Therapeutic Antitumor Response to Cervical Cancer in Mice Immunized with U14 Cell Vaccines Transfected with Costimulatory B7 Gene and HPV16E6/B7 DNA Recombinant Vaccines
    B7修饰的U14细胞疫苗和HPV16E6/B7重组基因疫苗预防及治疗宫颈癌实验研究
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    Study of Specific Antitumor Immune Response Induced by GST-MAGE-3
    GST-MAGE-3蛋白诱导特异性抗肿瘤免疫应答的实验研究
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    Study of Specific Antitumor Immune Response Induced by pcDNA3.1/HSP70-MAGE-3 Fusion DNA Vaccine
    pcDNA3.1/HSP70-MAGE-3融合DNA疫苗诱导特异性抗肿瘤免疫应答的实验研究
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  response
The compound 12 was found to be five times more potent than 11 in reducing twitch response to nerve stimulations, indicating the importance of extended interonium distances and 17-acetoxy function for potent antagonist activity.
      
in 1998, statistical curvature, and the local influence of the response vector perturbations are investigated in NRDM.
      
Existence of positive solution for a two-patches competition system with diffusion and time delay and functional response
      
The parameter estimation and the coefficient of contamination for the regression models with repeated measures are studied when its response variables are contaminated by another random variable sequence.
      
This paper is devoted to studying a free boundary problem modeling the effects of drug resistance and vasculature on the response of solid tumors to therapy.
      
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(1) A comparative study has been made on the induction of tryptophan pyrrolase by substrate and corticoids in liver tumor and the precancerous liver induced by 3'-MeDAB.(2) The liver tumor, contrary to normal liver, was inactive in response to both tryptophan and hydrocortisone for the induction of tryptophan pyrrolase, while the tissue adjacent to the tumor was active with respect to both substrate and hormonal induction.(3) A decrease in both substrate and hormonal induction was observed in the precancerous...

(1) A comparative study has been made on the induction of tryptophan pyrrolase by substrate and corticoids in liver tumor and the precancerous liver induced by 3'-MeDAB.(2) The liver tumor, contrary to normal liver, was inactive in response to both tryptophan and hydrocortisone for the induction of tryptophan pyrrolase, while the tissue adjacent to the tumor was active with respect to both substrate and hormonal induction.(3) A decrease in both substrate and hormonal induction was observed in the precancerous liver developed by feeding 3'-MeDAB for different time intervals. In experiments by injecting intraperitoneally the carcinogen into animals,for 25 hours, similar results were obtained as in the feeding experiments.(4) 2-MeDAB, a non-carcinogenic substance, caused the same effect on the induction of tryptophan pyrrolase in every case as did 3'-MeDAB. It thus appears that the effect of 3'-MeDAB on the enzyme induction may not be specific.(5) No inhibitors of tryptophan pyrrolase or activators of kynureninase were found in the cell sap of liver tumor and the liver of rats fed 3'-MeDAB or 2-MeDAB in the course of induction.(6) The cell sap from liver tumor (non-induced) contained only a small amount of enzyme protein, as shown by the fact that the enzyme activity being only slightly increased by the addition of either normal microsome or hematin, of which the level has not elevated by the administration of tryptophan or hydrocortisone.(7) Similar experiments have shown that the cell sap from the substrate and hormonal induced liver in the precancerous stage contained a decreased amount of enzyme protein as compared to that of the control. The same was true of the rats fed 2-MeDAB.(8) Microsomes from liver tumor have lost almost completely the ability of activating tryptophan pyrrolase in the cell sap. The ability of activation due to microsomes from the precancerous liver was remarkably reduced, though not yet completely lost, while the microsomes from the liver of rats fed 2-MeDAB were normal. It was in this respect that the effect of non-carcinogen (2-MeDAB) was found to be different from that of the carcinogen (3'-MeDAB).(9) From the results presented, it was concluded that the default of tryptophan pyrrolase induction observed in the liver tumor and the precancerous liver was mainly due to an inadequate amount of apoenzyme, rather than a deficiency of co-factor (hematin) or an increment of protein other than the enzyme. The possible cause of these effects was briefly discussed.

本文对大鼠肝癌及癌前期肝内TP的底物诱导和激素诱导作了比较研究。在3′-MeDAB诱发的肝癌中,TP活性很低,且不因注射色氨酸或氢可地松而升高,而癌周组织则仍保留对底物和激素诱导的能力。喂3′-MeDAB 13天、28天、90天大鼠肝内TP的底物诱导效应都较对照组为低。急性注射3′-MeDAB25小时,以100毫克/100克体重的L-色氨酸进行诱导,TP的诱导效应亦较对照组为低。氢可地松诱导的结果与底物诱导的相似,无论在喂或急性注射3′-MeDAB的情况下,TP的诱导效应都受到抑制。但在相同条件下,非致癌物,2-MeDAB,对TP的底物诱导和激素诱导(慢性的或急性的实验)也有相似的作用。诱导后肝癌组织或喂偶氮染料的肝组织中都未发现有TP的抑制物或狗尿酸酶的激活物。微粒体及正铁血红素与上清液的加合实验表明:(1)肝癌微粒体几乎完全不具有激活TP的活力;癌前期(3′-MeDAB组)肝微粒体已部分失去此种生化功能,但2-MeDAB组微粒体则否。3′-MeDAB对肝微粒体中辅助因子(正铁血红素)的结构并无破坏,而可能使辅助因子的含量减少。(2)微粒体对激活上清液TP的效果较自由的正铁血红素差,即使加入过量微粒体亦不能...

本文对大鼠肝癌及癌前期肝内TP的底物诱导和激素诱导作了比较研究。在3′-MeDAB诱发的肝癌中,TP活性很低,且不因注射色氨酸或氢可地松而升高,而癌周组织则仍保留对底物和激素诱导的能力。喂3′-MeDAB 13天、28天、90天大鼠肝内TP的底物诱导效应都较对照组为低。急性注射3′-MeDAB25小时,以100毫克/100克体重的L-色氨酸进行诱导,TP的诱导效应亦较对照组为低。氢可地松诱导的结果与底物诱导的相似,无论在喂或急性注射3′-MeDAB的情况下,TP的诱导效应都受到抑制。但在相同条件下,非致癌物,2-MeDAB,对TP的底物诱导和激素诱导(慢性的或急性的实验)也有相似的作用。诱导后肝癌组织或喂偶氮染料的肝组织中都未发现有TP的抑制物或狗尿酸酶的激活物。微粒体及正铁血红素与上清液的加合实验表明:(1)肝癌微粒体几乎完全不具有激活TP的活力;癌前期(3′-MeDAB组)肝微粒体已部分失去此种生化功能,但2-MeDAB组微粒体则否。3′-MeDAB对肝微粒体中辅助因子(正铁血红素)的结构并无破坏,而可能使辅助因子的含量减少。(2)微粒体对激活上清液TP的效果较自由的正铁血红素差,即使加入过量微粒体亦不能使TP活性增高到加入正铁血红素的水平;微粒体对3′-MeDAB组上清液的激活不如对2-MeDAB组及对照组上清液(底物或激素诱导)的激活显著,而正铁血红素对三组上清液都有显著激活。(3)肝癌细胞上清液只合有极少量的TP蛋白,且不因注射色氨酸或氢可地松而增加;癌前期肝细胞上清液的TP蛋白因底物或激素诱导而增加的量都较对照粗低。2-MeDAB组也有相似现象。以上结果表明,肝癌及癌前期肝内TP诱导的受损,主要是由于诱导后TP蛋白的缺少,而不是由于辅助因子(如正铁血红素)的不足,或非酶蛋白的增多。

In recent years, more and more insterest have been given to the relationship or immunology with Chinese Traditional Medicine. A recent survey of the literature centered on the reports on use of immunological parameters for the study of patients diagnosed to be suffering of low vitality by the criteria usually employed. In general, in many such patients cellular immune responses, both in vivo and in vitro, have been found to be lowered to quite a definite extent. However, in-depth and follow-up studies...

In recent years, more and more insterest have been given to the relationship or immunology with Chinese Traditional Medicine. A recent survey of the literature centered on the reports on use of immunological parameters for the study of patients diagnosed to be suffering of low vitality by the criteria usually employed. In general, in many such patients cellular immune responses, both in vivo and in vitro, have been found to be lowered to quite a definite extent. However, in-depth and follow-up studies have usualy not been carried out and further studies seem to be warranted, some suggestions for all-round studies have been presented for discussion and reference.

近年来国内临床工作者结合免疫学的实验,对于免疫同虚证的关系,已有一些报道。初步看来不少虚证病人的体内体外细胞免疫指标,有所降低。但进一步进行个例分析及治疗前后的动态观察尚不太多。在文中还提出几点值得注意的问题,供同道们参考及指正。

Recently,the dynamic changesof cytostatic and cytocidal effect ofspleen cells of tumor-bearing mice ontarget tumor cells were studied withthe technigue of ~(125)IUdR assay,Lym-phocyte transformation of similarspleens by Con A and PWM wasalso studied.In most experiments,Ehrlich ascitic tumor cells were usedand each C57BL mouse was inocula-ted intramuscularly with 5×10~6 viabletumor cells in the right thight as atumor bearer.In other experiments,Sarcoma 180 ascitic cells in C57BLmice and Hepatoma acitic cells...

Recently,the dynamic changesof cytostatic and cytocidal effect ofspleen cells of tumor-bearing mice ontarget tumor cells were studied withthe technigue of ~(125)IUdR assay,Lym-phocyte transformation of similarspleens by Con A and PWM wasalso studied.In most experiments,Ehrlich ascitic tumor cells were usedand each C57BL mouse was inocula-ted intramuscularly with 5×10~6 viabletumor cells in the right thight as atumor bearer.In other experiments,Sarcoma 180 ascitic cells in C57BLmice and Hepatoma acitic cells inTienjing No 1 albino mice were alsoemployed.Results of our experiments indic- ated that spleen cells of tumor-bearingmice inhibited strongly DNA syn-thesis of tumor cells in vitro.Suchcytostatic effect was already quitemanifested at the 7th day after tumorinoculation and increased henceforthin parallel with the growth of tumorand an enlargement of spleen.Afterthe surgical removal of tumor,cytost-atic effect decreased rapidly and couldno longer be detected on the 21thday after amputation and consequen-tly the spleen returned almost to itsnormal size.Moreover,we have alsoinoculated ascitic tumor cells intothymectomized,X-irrdiated,bone-marrow-reconstituted mice,and theirspleen cells were also shown to bestrongly cytostatic to tumor cells.On the other hand,our datapointed out that the spleen cells ofmice bearing large Ehrlich tumor couldnot kill in vitro ~(125)IUdR labelledEhrlich tumor cells as determined by~(125)IUdR release assay.Furthermore,these spleen cells were also found tobe suppressed with respect to theirresponse to lymphocyte mitogens ConA and PWM.In fact,spleen cells from micebearing Ehrlich tumor inhibited the~(125)IUdR uptake of Sarcoma 180cells and Hepatoma ascitic cells to asimilar extent.Similarly,the spleencells from mice bearing solid Hep-atoma and Sarcoma 180 also inhibitedstrongly the ~(125)IUdR uptake ofEhrlich tumor cells.Thus,cytostaticeffect of spleen cells of tumor-bearingmice against tumor cells in vitro isa kind of non-specific inhibition.Histopathologic studies showedthat the increase in the amount ofreticulum cells and macrophages inthe spleen of tumor-bearing mice hada good correlation with its increasein cytostatic effect on tumor cells.At the same time,there was a conco-mitant decrease in the number ofsmall lymphocytes and an incrase inthe number of immature haematopoie-tic cells.This latter fact might ex-plain the general absence of cytocidaleffect of spleen cells on tumor cellsas well as the deficient response ofspleen cells to lymphocyte mitogenslike ConA and PWM in the tumor-bearing mice.Taken together all the above re-sults,we tentatively suggest that theeffector cells of cytostatic effect intumor-bearing mouse spleen are notT、B lymphocytes.The exact natureof these suppsessor cells as well astheir possible mechanisms of actionare now under investigation.

应用5-~(125)碘-2’-脱氧尿嘧啶核苷(~(125)IUdR)参入抑制试验,证明带Ehrlich实体瘤的C57BL 小鼠脾脏细胞可在体外对同一移植肿瘤细胞的DNA 合成产生显著的抑制。随着肿瘤的增长,带瘤小鼠的脾脏进行性增大,脾细胞对肿瘤细胞的抑制效应也随之增强。这种对肿瘤细胞DNA合成的抑制作用是非特异的。肿瘤切除后,抑制效应也随之下降,到术后21天,细胞静止效应已不能测得。本文还应用~(125)IUdR技术同时测定了带瘤小鼠脾脏的T 杀伤细胞,脾脏淋巴细胞对致分裂原Con A 与PWM 的转化反应的动态变化,实验表明带瘤小鼠脾脏T、B 淋巴细胞的功能是受抑制的。组织病理学观察提示,带瘤动物脾脏的网织细胞和巨噬细胞的增多可能与细胞静止效应的增强有关。

 
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