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plasma lactate
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  血浆乳酸
     Oxygen consumption (VO2),pulmonary ventilation(VE),plasma lactate,plasma ammonia,blood pH,HCo3 and heart rate (HR)were obtaine during incremental exercise to exhaustion on a bicycle ergometer for 8 men under the following conditions:1)After the intake of NaHCo3(0. 3 g per 1kg body weight)-alkalosis Group (A).
     同时测定吸氧量(VO2)、通气量(VE)、血浆乳酸、血浆胺、血液pH、HCO3-及心率(HR)。 受试者在两种条件下进行实验:(1)每公斤体重摄取0.3aNaHCOd3(碱中毒组:A);
短句来源
     Moreover, plasma lactate level was lower in Xuebijing injection group than that in control group (P<0.05).
     对照组血浆乳酸浓度明显高于血必净组(P<0.05)。
短句来源
     Results There were statistic differences:(1)there were marked increased in plasma lactate level after aortic clamp for 5 minutes and before the end of CPB,compared with that before CPB in each group,(P<0.05,P<0.05);
     结果两组在主动脉阻断后5min和停CPB前的血浆乳酸水平均较CPB前明显升高(P均<0.05); A组在主动脉阻断后5min和停CPB前的血浆乳酸水平均明显高于B组(P均<0.05);
短句来源
     Moreover, plasma lactate levels were lower in NHF group than in HF group ( P <0.05).
     血浆乳酸浓度NHF组明显高于HF组。
短句来源
     Blood was sampled for detection of plasma lactate level before CPB, after aortic clamp for 5 minutes,before the end of CPB respectively.
     两组在体外循环(CPB)前、主动脉阻断后5min、停CPB前,分别采动脉血样检测血浆乳酸的水平。
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  乳酸
     Plasma lactate increased significantly in LPS group compared with normal control group (P<0.001), decreased sign ificantly in in tervention groups compared with LPS group (P<0.01).
     LPS组乳酸水平明显高于对照组(P<0.001); 两个UT干预组均明显低于LPS组(P均<0.01);
短句来源
     Oxygen consumption (VO2),pulmonary ventilation(VE),plasma lactate,plasma ammonia,blood pH,HCo3 and heart rate (HR)were obtaine during incremental exercise to exhaustion on a bicycle ergometer for 8 men under the following conditions:1)After the intake of NaHCo3(0. 3 g per 1kg body weight)-alkalosis Group (A).
     同时测定吸氧量(VO2)、通气量(VE)、血浆乳酸、血浆胺、血液pH、HCO3-及心率(HR)。 受试者在两种条件下进行实验:(1)每公斤体重摄取0.3aNaHCOd3(碱中毒组:A);
短句来源
     Moreover, plasma lactate level was lower in Xuebijing injection group than that in control group (P<0.05).
     对照组血浆乳酸浓度明显高于血必净组(P<0.05)。
短句来源
     Results There were statistic differences:(1)there were marked increased in plasma lactate level after aortic clamp for 5 minutes and before the end of CPB,compared with that before CPB in each group,(P<0.05,P<0.05);
     结果两组在主动脉阻断后5min和停CPB前的血浆乳酸水平均较CPB前明显升高(P均<0.05); A组在主动脉阻断后5min和停CPB前的血浆乳酸水平均明显高于B组(P均<0.05);
短句来源
     Moreover, plasma lactate levels were lower in NHF group than in HF group ( P <0.05).
     血浆乳酸浓度NHF组明显高于HF组。
短句来源
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  乳酸测定
     Application of plasma lactate measurement in children with acute abdominal pains
     血浆乳酸测定及其在儿科急腹症中的应用
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  “plasma lactate”译为未确定词的双语例句
     The changes of DAO levels were significantly correlated with plasma lactate, endotoxin and TNF levels in goats (r=0.872,p<0.01;r=0.842,p<0.01;r=0.817,p<0.01).
     血浆DAO的变化与血乳酸,TNF和LPS变化呈高度相关(r=0.872,0.842和0.817,p<0.01)。
短句来源
     Plasma lactate level was not significantly altered at the end of the test compared with the base line 〔from (1.41±0.05) mmol/L to (1.47±0.07) mmol/L〕.
     与基础值比较,试验结束时患者血乳酸浓度无明显变化〔(1.41±0.05)mmol/L比(1.47±0.07)mmol/L〕。
短句来源
     Plasma lactate levels in severe HIE neonates ( 9.11± 3.29 mmol/L) were higher than those of neonates with mild and moderate HIE ( 6.03± 2.66 and 6.56± 1.42 mmol/L) in the first day after birth (P< 0.05), but not in the following days.
     重度HIE组血浆Lac(9.11± 3.2 9mmol/L)与轻、中度HIE组 (6 .0 3±2 .6 6mmol/L ,6 .5 6± 1.4 2mmol/L)相比较仅在 0~ 2 4h明显升高 (均P <0 .0 5 )。
短句来源
     Results No significiant changes of CO,CI,SV,SI,RVSWI,HR and ECG tracing were observed during hypotension (P<0 05) SVR,PVR,Cpp,RPP and LVSWI were significantly decreased during hypotension,(P<0 05) During controlled hypotension DO 2 and S v O 2 decreased significiantly, ERO 2 increased significiantly and VO 2 and plasma lactate concentraion remained unchanged.
     结果 降压期间 CO、CI、SV、SI、RVSWI及 HR无明显变化 ,SVR、PVR、L VSWI、Cpp、RPP可逆性减少 (P<0 .0 5 )。 DO2 显著下降 ,ERO2 明显增加 ,VO2 无明显变化。
短句来源
     The heart preconditioned by specific inhibitor SB203580 of MLA and p38 was conducted ischemia/reperfusion at 24 h later to observe cardiac function, myocardial infarction and plasma lactate dehydrogenase (LDH) activity in each group. While p38 phosphorylation levels at once, at 10, 15 and 30 min after preconditioning with MLA and at 15 min after preconditioning with SB203580 and MLA were determined by Western blot.
     分别应用MLA、p38的特异性抑制剂SB203580预处理心脏,24h后对心脏进行缺血再灌注,观察各组心功能、心肌梗死范围、血浆乳酸脱氢酶(LDH)活性,同时用Western印迹法检测MLA预处理后即刻、10、15、30min、SB203580加单磷酰脂A预处理15min时p38磷酸化水平。
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  plasma lactate
Plasma lactate and oxidative capacity (VO2 and workload) were measured in 15 well-characterized MM patients during cycling.
      
Fourteen of 16 patients had impaired VO2max, 10/16 had elevated resting plasma lactate, and 10/11 that were investigated had impaired citrate synthase-corrected complex I activity.
      
Resting plasma lactate, pyruvate and lactate/pyruvate ratios were similar in all three groups.
      
After release, mean arterial pressure decreased, plasma lactate increased, and erythrocyte glutathione peroxidase decreased.
      
Effects of zinc deficiency and supplementation on the glycogen contents of liver and plasma lactate and leptin levels of rats pe
      
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The parameters of 797 measurements of electrolytes and blood gases in 191 patients during and after open heart surgery under cardio-pulmonary bypass were studied. It was found that both the anion gap (AG) and lactate levels were markedly elevated in the patients than in the normal controls. It is well known that increased plasma lactate due to hvpoxia is one of the main factors to result in anion gap acidosis, and the therapeutic measures should be directed to increase tissue oxygen supply rather than...

The parameters of 797 measurements of electrolytes and blood gases in 191 patients during and after open heart surgery under cardio-pulmonary bypass were studied. It was found that both the anion gap (AG) and lactate levels were markedly elevated in the patients than in the normal controls. It is well known that increased plasma lactate due to hvpoxia is one of the main factors to result in anion gap acidosis, and the therapeutic measures should be directed to increase tissue oxygen supply rather than administer alkali, which will be liable to induce alkalosis. Therefore, it is suggested that concomitant determinations of AG and blood gases bo valuable and helpful to the management of the postoperative cases of open heart surgery.

本文分析了191例797次体外循环心内直视手术患者的电解质及血气分析结果,发现本组病例AG值和乳酸值与正常人相比均有明显增高,从而提示在此组病例中,组织缺氧引起乳酸增高是引起高AG代谢性酸中毒的主要原因之一。其治疗应以改善组织缺氧为主,过多补碱可致医源性碱中毒。并提出对这类病人在血气监护的同时,动态观察AG是有价值的。

The protective effect of TGPs on acute liver damage induced by carbon tetrachloride ( CCl4 ) was investigated in mice . TGPs ( 40 or 80mg/kg·d × 3d, ip ) reduced the elevation of plasma or serume gl- utamic pyruvic transaminase ( GPT ) and plasma lactate dehydroge-nase ( LDH ) in CCl4-intoxicated mice, but if had no significant influence on the elevation of plasma glutamic oxalacetic transaminase ( GOT).TGPs (40mg/kg·d × 3d, ip ) showed a protective effect on the histopathologic changes of liver...

The protective effect of TGPs on acute liver damage induced by carbon tetrachloride ( CCl4 ) was investigated in mice . TGPs ( 40 or 80mg/kg·d × 3d, ip ) reduced the elevation of plasma or serume gl- utamic pyruvic transaminase ( GPT ) and plasma lactate dehydroge-nase ( LDH ) in CCl4-intoxicated mice, but if had no significant influence on the elevation of plasma glutamic oxalacetic transaminase ( GOT).TGPs (40mg/kg·d × 3d, ip ) showed a protective effect on the histopathologic changes of liver injury induced by CCl4. The results suggest that TGPs may a protect against hepatic damage.

白芍总甙(TGPs 40或80mg/kg·d~(-1)×3d,ip)可明显对抗四氯化碳(ig or sc)引起的小鼠血浆或血清谷丙转氨酶和血浆乳酸脱氢酶升高,但对谷草转氨酶的升高无明显影响。TGPs(40mg/kg·d~(-1)×3d,ip)对四氯化碳所致的肝病理组织改变有一定保护作用。提示TGPs可能具有保肝作用。

In this study,we evaluated the damage of hepatic cells and subcellular organelles induced by oxygen-de- rived free radicals in severe hemorrhage shack models in rats.Two hours after the onset of shock contents of malondialdehyde(MDA)in he sse epatic mito- chondria and lysosome suspensions were increased(P< 0.05~0.01 vs control valued),and the ivity of super- oxide dismutase(SOD)in hepatic tissue was compen- satively elevated(P<0.01)but the activity of SOD in hepatic mitochondria and lysosome suspensions were...

In this study,we evaluated the damage of hepatic cells and subcellular organelles induced by oxygen-de- rived free radicals in severe hemorrhage shack models in rats.Two hours after the onset of shock contents of malondialdehyde(MDA)in he sse epatic mito- chondria and lysosome suspensions were increased(P< 0.05~0.01 vs control valued),and the ivity of super- oxide dismutase(SOD)in hepatic tissue was compen- satively elevated(P<0.01)but the activity of SOD in hepatic mitochondria and lysosome suspensions were markedly decreased(P<0.05).Four hours after the on- set of shock,the activity of SOD in hepatic tissue,mito- chondria and lysosome suspensions was significantly de- creased(P<0.01).The results suggest that peroxidation injury mediated by oxygen-derived free radicals may be more severe in hepatic subcellular organelles than in hipatic tissue.In addition,plasma lactate,β-glucuronida- se,acid phosphatase and blood MDA and SOD appear to have changes to varying degrees.

本实验在大白鼠重度失血性休克模型上重点探讨氧自由基对肝脏细胞和亚细胞器的损伤作用。休克2°,肝组织、肝线粒体和溶酶体悬液 MDA 含量较对照明显升高(P<0.05~0.01),肝组织 SOD 活力呈代偿性升高(P<0.01),而肝线粒体和溶酶体悬液 SOD 活力明显下降(P<0.05)。休克4°肝组织、肝线粒体和溶酶体悬液 SOD 活力均明显下降(P<0.01)。结果提示氧自由基介导的脂质过氧化损伤在肝亚细胞器中较之肝组织要更重。此外血浆乳酸、溶酶体酶如β—葡萄糖醛酸酶和酸性磷酸酶以及血 MDA 和 SOD 在休克后均有不同程度改变。

 
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