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n-methyl-d-aspartate
n-甲基-d-天门冬氨酸
Objective To investigate the effect of intraspinal grafting of adenovirus- mediated BDNF ex vivo transgene myoblasts and GM- 1 gangliosides on N- methyl- D- aspartate (NMDA) receptors after spinal cord injury (SCI) in rats. 目的研究脑源性神经营养因子(BDNF)转基因细胞移植和神经节苷脂(GM-1)对大鼠脊髓损伤N-甲基-D-天门冬氨酸(NMDA)受体的影响。 短句来源 Effect of intraspinal grafting of adenovirus- mediated BDNF ex vivo transgene myoblasts and GM- 1 gangliosides on N- methyl- D- aspartate (NMDA) receptors after cervical spinal cord contusion in rats 脑源性神经营养因子转基因细胞移植和神经节苷脂对大鼠脊髓损伤N-甲基-D-天门冬氨酸受体的影响 短句来源 Objective: To investigate the effects of dextromethorphan (DM) which is an antitussive and a N-methyl-D-aspartate (NMDA) receptor antagonist as well. It is intramuscularly (I.M.) injected in perioperative period, reduces pain and decreases the requirements of opioid analgesic after radical mastectomy (RM). 目的探讨围术期使用N-甲基-D-天门冬氨酸(NMDA)受体拮抗剂右美沙芬(Dextromethor-phan,DM)缓解乳腺根治术后疼痛的效应及对术后阿片类镇痛药物用量的影响。 短句来源 n-甲基-d-天门冬氨酸
Objective To investigate the effect of intraspinal grafting of adenovirus- mediated BDNF ex vivo transgene myoblasts and GM- 1 gangliosides on N- methyl- D- aspartate (NMDA) receptors after spinal cord injury (SCI) in rats. 目的研究脑源性神经营养因子(BDNF)转基因细胞移植和神经节苷脂(GM-1)对大鼠脊髓损伤N-甲基-D-天门冬氨酸(NMDA)受体的影响。 短句来源 Effect of intraspinal grafting of adenovirus- mediated BDNF ex vivo transgene myoblasts and GM- 1 gangliosides on N- methyl- D- aspartate (NMDA) receptors after cervical spinal cord contusion in rats 脑源性神经营养因子转基因细胞移植和神经节苷脂对大鼠脊髓损伤N-甲基-D-天门冬氨酸受体的影响 短句来源 Objective: To investigate the effects of dextromethorphan (DM) which is an antitussive and a N-methyl-D-aspartate (NMDA) receptor antagonist as well. It is intramuscularly (I.M.) injected in perioperative period, reduces pain and decreases the requirements of opioid analgesic after radical mastectomy (RM). 目的探讨围术期使用N-甲基-D-天门冬氨酸(NMDA)受体拮抗剂右美沙芬(Dextromethor-phan,DM)缓解乳腺根治术后疼痛的效应及对术后阿片类镇痛药物用量的影响。 短句来源 “n-methyl-d-aspartate”译为未确定词的双语例句
(2) Reginal water and ion contents of rat spinal cord tissue were measured with the atomic absorption spectrophotometry after administering intrathecally different dosage of 3-(2-carboxypiperazin-4-yl)-propyl-l-phosphonic acid (CPP),a specific antagonist of N-methyl-D-aspartate (NMDA) receptor,to rats at 48 h posttrauma. （2）给予不同剂量N－甲基－D－天门冬氨酸（NMDA）受体拮抗剂4－（3－磷酸基丙基）－2－哌嗪磷酸（CPP），48h后取伤段脊髓以原子吸收光谱法测定其离子改变及水含量。 短句来源 Effect of intraspinal grafting of adenovirus-mediated BDNF ex vivo transgene myoblasts cells and methylprednisolone on N-methyl-D-aspartate(NMDA) receptors after cervical spinal cord contusion in rats 转基因细胞移植对损伤脊髓天门冬氨酸受体的影响 短句来源 Ketamine is an antagonist of glutamate N-methyl-d-aspartate receptors (NMDA-R) which takes effect on the PCP site of NMDA-R. As a non-competitive antagonist, its effects can not be reversed even by high dose of NMDA or glutamic (Glu). 氯胺酮是 N-甲基-（d）-天（门）冬氨酸受体（NMDA—R）的拮抗剂，作用于NMDA—R的苯环哌啶（PCP）受点，由于大剂量的NMDA、Glu都不能逆转其作用，故称NMDA—R非竞争性拮抗剂。 短句来源 Abstract The protective effect of D-AP7 (D-(-)-2-Amino-7-Phosphono-heptanoic Acid). a specific antagonist of N-Methyl-D-Aspartate (NMDA) receptor, was evaluated in cat with traumatic brain edema D-AP7 (15μg/kg) was administered through cisterma cerebellomedullaris. 20 min after head injury in one group (n=9). 本实验观察了竞争性N－甲基－D－天门冬氨酸（NMDA）受体拮抗剂D－（－）－2－氨基－7－磷庚酸酯（D－AP7）对猫创伤性脑水肿的影响。 短句来源 N-methyl-D-aspartate receptor(NMDAR)in cat cerebral 本实验采用放射性配基受体结合分析法，测定了猫脑外伤时大脑皮质和海马N─甲基─D─天冬氨酸受体（NMDAR）的变化。 并用氨基酸自动分析仪观察了猫大脑皮质兴奋性氨基酸含量变化。 短句来源 更多 
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The effects of intracerebroventricular (icv) agonists and antagonists of N-methyl-D-aspartate (NMDA) and alpha-amino-3-hydroxy-5-methyl-4-isoxazole-propionic acid (AMPA) receptors on the general anesthesia of propofol were studied.
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It has been demonstrated that the key target of these peptides are N-methyl-D-aspartate glutamate receptors.
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Activation of rat cerebellum granule cells by N-methyl-D-aspartate (NMDA, 10-4-10-3 M) results in progressive increase in reactive oxygen species (ROS) and suppression of the ouabain-sensitive part of Na/K-ATPase activity.
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Objective: To study the effect of glycine site/NMDA (N-methyl-D-aspartate) receptor antagonist MRZ2/576 on the conditioned place preference (CPP) and locomotor activity induced by morphine in mice.
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Background and purpose A low-affinity, use-dependent N-Methyl-D-Aspartate (NMDA) antagonist AR-R15896AR has neuroprotective properties in animal models of ischaemic stroke.
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Abstract The protective effect of D-AP7 (D-(-)-2-Amino-7-Phosphono-heptanoic Acid).a specific antagonist of N-Methyl-D-Aspartate (NMDA) receptor, was evaluated in cat with traumatic brain edema D-AP7 (15μg/kg) was administered through cisterma cerebellomedullaris. 20 min after head injury in one group (n=9).treated with artificial cerebrospinal fluid in another group (ACSF,n=9) and same operation (n=9) controls.After 6 hours all cats were sac... 本实验观察了竞争性Ｎ－甲基－Ｄ－天门冬氨酸（ＮＭＤＡ）受体拮抗剂Ｄ－（－）－２－氨基－７－磷庚酸酯（Ｄ－ＡＰ７）对猫创伤性脑水肿的影响。结果发现Ｄ－ＡＰ７可明显减轻脑含水量，改善脑Ｎａ＋、Ｋ＋、ｃａ＋＋、Ｍｇ＋＋含量，提示兴奋性氨基酸与创伤性脑水肿有关，其中ＮＭＤＡ受体可能起主要作用，本文探讨了ＮＭＤＡ受体拮抗剂对创伤性脑水肿的保护机理。 文摘来源 In the present study,Sprague-Dawley rats were subjected to traumatic spinal cord injury using Allen's method.At different periods posttrauma:(1) Reginal concentrations of excitatory amino acid (EAA) were assessed with high-performance liquid chromatography;(2) Reginal water and ion contents of rat spinal cord tissue were measured with the atomic absorption spectrophotometry after administering intrathecally different dosage of 3-(2-carboxypiperazin-4-yl)-propyl-l-phosphonic acid (CPP... 本实验以Ａｌｌｅｎ法造成脊髓损伤（ＳＣＩ）模型，伤后不同时间分别：（１）取伤段脊髓，采用高效液相色谱法检测脊髓组织谷氨酸（Ｇｌｕ）、天门冬氨酸（Ａｓｐ）的含量变化；（２）给予不同剂量Ｎ－甲基－Ｄ－天门冬氨酸（ＮＭＤＡ）受体拮抗剂４－（３－磷酸基丙基）－２－哌嗪磷酸（ＣＰＰ），４８ｈ后取伤段脊髓以原子吸收光谱法测定其离子改变及水含量。结果发现ＳＣＩ后１０～３０ｍｉｎＧｌｕ、Ａｓｐ较对照组明显升高，其幅度与ＳＣＩ程度呈正相关；损伤组Ｎａ＋、Ｃａ（２＋）和水含量增多，Ｋ＋、Ｍｇ（２＋）减少，ＣＰＰ处理组各种离子和水的改变则较轻，其效果与给药剂量和时间呈正相关。ＣＰＰ的拮抗作用提示ＳＣＩ时的离子和水含量改变与ＮＭＤＡ的过度激活有关。表明ＳＣＩ后急性期兴奋性氨基酸的过度升高很可能参与了脊髓创伤后的继发性损伤过程。 文摘来源 N-methyl-D-aspartate receptor(NMDAR)in cat cerebral 本实验采用放射性配基受体结合分析法，测定了猫脑外伤时大脑皮质和海马Ｎ─甲基─Ｄ─天冬氨酸受体（ＮＭＤＡＲ）的变化。并用氨基酸自动分析仪观察了猫大脑皮质兴奋性氨基酸含量变化。结果表明，伤后２和６ｈ两侧大脑皮质和海马ＮＭＤＡＲ的最大结合容量明显降低，伤后２ｈ以海马变化最大，并以伤后６ｈ伤侧大脑皮质中降低最为显著；而大脑皮质兴奋性氨基酸含量伤后５ｍｉｎ即显著升高，然后呈下降趋势，且以伤侧大脑皮质变化为大。提示：脑外伤后ＮＭＤＡＲ的下调与兴奋性氨基酸的大量释放有关，可能在兴奋毒性脑损伤中起重要作用。 文摘来源 << 更多相关文摘 
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