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injury
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    Protective effects of Panax notoginsenoside Rb_1 and Rg_1 preconditioning on angiotensin Ⅱ induced hypertrophied neonatal rat myocytes against hypoxia-reoxygenation injury and effect of Panax notoginsenoside Rg_1 on thansduction from BMSCs to cardi
    参三七皂甙Rb_1、Rg_1预适应对AngⅡ致肥厚心肌细胞H/R损伤的保护作用及Rg_1对5-AZ诱导大鼠骨髓基质细胞向心肌细胞转化的影响
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    Study of Astragalosides' Mechanisms on Protective Effect in Myocardial Injury
    黄芪总皂甙保护心肌损伤的机制研究
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    Protective Effects of Allicin on Cerebral Ischemia-Reperfusion Injury in Rats and Its Mechanism
    大蒜素对大鼠局灶性脑缺血-再灌注损伤的保护作用及机制
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    Studies on Neuroprotective Mechanisms of Resveratrol Against Cerebral Ischemia Reperfusion Injury in Mice
    白藜芦醇对小鼠脑缺血再灌注损伤的保护作用机制研究
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    Toll-like Receptor 4 Mediated Mechanism of Bacillus Calmette Guerin and Lipopolysaccharide Inducing Immunological Liver Injury and Role of Paeoniflorin
    Toll样受体4介导BCG/LPS诱导免疫性肝损伤的机制及芍药苷的作用
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    Neuroprotective Effect of 20(S)-Ginsenoside Rg_3 Against Injury on Cerebral Ischemia
    20(S)-人参皂苷Rg_3抗脑缺血作用研究
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    Studies on the Protective Effect of Salvianolic Acid B on the Mitochondrial Injury & Neuronal Apoptosis and Its Action Mechanisms
    丹酚酸B保护线粒体和抗神经细胞凋亡作用及其作用机制研究
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    Protective Effects and Mechanisms of WuLongDan Against Neuronal Injury in Rat Focal Cerebral Ischemia Model
    乌龙丹对大鼠局灶性脑缺血模型神经细胞保护效应及其机制的研究
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    Study on the Effect and Mechanism of Tongxinluo in Proliferation and Differentiation of Neural Stem Cells after Ischemia Reperfusion Injury in Rats
    通心络对缺血再灌注鼠脑神经干细胞增殖分化的作用及机理研究
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    RCR AND ADP/O RATIO CHANGES OF RAT LIVER MITOCHONDRIA DURING EARLY PHASE OF BURN INJURY
    大鼠烫伤后早期肝脏线粒体RCR和ADP/O的变化
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  injury
Our studies demonstrated an in vivo cardioprotection effect of (N-(3,4,-dimethoxy-2-chlorobenzylideneamino)-guanidine: ME10092) in ischaemic reperfusion injury in the rodent.
      
Diclofenac (25 mg kg-1) and meloxicam (7.5 mg kg-1) produced 9.3 and 19.6 mm2 ulcer areas in stomachs of rats, respectively, when used alone, but when combined with nimesulide diclofenac and meloxicam did not cause any injury in rat stomachs.
      
These proteins may be involved in cardiovascular injury, and could play an important role in the treatment of coronary heart disease.
      
SOD and POD activities declined correspondingly, followed by significant increases of MDA and MP, and leaf injury was finally observed.
      
Hepatocellular injury was the predominant type in these cases (132 cases, 48%).
      
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Qinghaosu (Artemisine) suspension in oil was injected im to 30 rhesus monkeys qd×14 d. Damages to several organs and tissues were noticed in 96 and 192 mg/kg/d groups on d 3 after the last injection. They were mainly erythroblastopenia, myeloblastope-nia and megakaryocytosis in bone marrow, degeneration and necrosis of myocardium, degeneration of hepatic cells and epithelia of proximal convoluted tubule of the kidney, and soft tissue injury at the injection site. Bone marrow and myocardial damages were...

Qinghaosu (Artemisine) suspension in oil was injected im to 30 rhesus monkeys qd×14 d. Damages to several organs and tissues were noticed in 96 and 192 mg/kg/d groups on d 3 after the last injection. They were mainly erythroblastopenia, myeloblastope-nia and megakaryocytosis in bone marrow, degeneration and necrosis of myocardium, degeneration of hepatic cells and epithelia of proximal convoluted tubule of the kidney, and soft tissue injury at the injection site. Bone marrow and myocardial damages were more prominent than those in other organs. Only mild lesions or no damage could be found on d 35 after the last injection. Therefor, it is concluded that qinghaosu suspension in oil in doses over 96 mg/kg/d might induce transient organ or tissue damages which disappear within 5 weeks. 3 of 4 rhesus monkeys in 192 mg/kg/d dose group died of severe myocardial damage within 1-3 days after the last injection.

恒河猴30只,经连续14天肌注青蒿素油悬剂,于停药后3天,主要在每天肌注96和192mg/kg组中引起多种脏器组织的损伤。表现为:骨髓红系和粒系细胞数减少,成熟发育障碍,巨核细胞增生,心肌细胞变性和灶性坏死(以超微结构病变为主);肝、肾营养不良性改变;淋巴组织萎缩;注射部位的损伤等,其中以骨髓和心肌损伤较为明显。于停药后35天,上述病变明显减轻或消失,表明青蒿素的毒副作用是可逆性的。每天肌注青蒿素24和48mg/kg为轻微中毒剂量;96mg/kg为严重中毒剂量;192mg/kg为致死剂量。每天肌注192mg/kg组的4只猴,有3只于末次给药后1~3天死于严重心肌损伤。

Injection of typhoid endotoxin into the right internal carotid artery inducedbrain edema in experimental rabbits,and normal saline injection was used as acontrol.65 rabbits divided into three groups were used in this investigation.Water,sodium,and potassium content and Evan's blue discoloration ofbrain tissue were assessed in the first randomly designed experiment using 40rabbits,in order to evaluate the presence of edema and blood brain barrier injury.Evan's blue discoloration was demonstrated on the...

Injection of typhoid endotoxin into the right internal carotid artery inducedbrain edema in experimental rabbits,and normal saline injection was used as acontrol.65 rabbits divided into three groups were used in this investigation.Water,sodium,and potassium content and Evan's blue discoloration ofbrain tissue were assessed in the first randomly designed experiment using 40rabbits,in order to evaluate the presence of edema and blood brain barrier injury.Evan's blue discoloration was demonstrated on the toxin injected side in allrabbits,but the saline injected controls only one showed slight bluish discolora-tion.Average water content of brain samples in experimental rabbits was muchhigher than that of the control,which showed a very significant diference(rt.ant.81.98±0.41,vs 78.79±0.13,P<0.001;rt.post.81.95±0.05,vs 78.16±0.13,p<0.001).Less significant was the sodium content(rt.ant.323.8±27.6,vs 275.4±10.4,P>0.05;rt.post.354.4±30.3,vs 258.6±9.5,P<0.01).Potassium content showed a trend to decrease but without statistical significance(rt.ant.406.5±21.7,vs 424.5±12.5,P>0.05;rt.post.355.5±20.9 vs 388.5±15.1,P>0.05).The data indicated that after injection of typhoid toxin bloodbrain barrier was injured,water and sodium content of brain increased.Thisproved that brain edema had been induced. Seventeen rabbits were used in the second experiment for morphological andhistochemical controlled investigation under light microscope,and in the thirdexperiment 8 rabbits were used to investigate kinetically under electron micros-cope.Acc(?)mpanied with the progressing vascular endotheliopathy and increasingof capillary permeability,the brain tissue began to show the swelling of theastrocytes initially and then the necrotic nerve ceils could be seen.These obser-vations indicated that this typhoid toxin induced both the vasogenic as well ascytotoxic brain edema,which belongs to a mixed type of brain edema.This study provided data as reference to clinical work.This animal modelof brain edema has much resemblance to brain edema complicated in severe infec-tions seen in pediatric clinics.

向右颈内动脉内注射伤寒内毒素,造成了实验兔脑水肿模型;同样途径注射生理盐水作为对照。分别测定实验组和对照组兔脑的水、钠、钾含量及伊文思兰染,以确认脑水肿的存在和血脑屏障的损伤,在光镜下作形态学和组织化学研究,以及在电镜下对脑水肿病变作动态观察。随着血管内皮细胞病变和毛细血管通透性增加的进展,最初出现星状胶质细胞的肿胀,然后看到神经细胞的坏死。提示这是一种既有血管源性又有细胞毒性原因的混合型脑水肿。这种兔脑水肿模型成功率高,动物一般可存活6小时以上,与儿科临床中并发于严重感染的脑水肿有较多相似之处。

Combined effects of α-chlorohydrin and glycidol on the ultrastructure of theinitial segment and the tail region of the rat epididymis have been studied by ultra-thin sections under electronmicroscope.Individual effects of the both drugs on thesame tissues were also examined simultaneously.The major results are as follows.1.Numbers of swollen and degenerated mitochondria were observed in principalcells,clear cells,apical cells and basal cells of epididymal epithelium,as well as insmooth muscle cells of the ductus...

Combined effects of α-chlorohydrin and glycidol on the ultrastructure of theinitial segment and the tail region of the rat epididymis have been studied by ultra-thin sections under electronmicroscope.Individual effects of the both drugs on thesame tissues were also examined simultaneously.The major results are as follows.1.Numbers of swollen and degenerated mitochondria were observed in principalcells,clear cells,apical cells and basal cells of epididymal epithelium,as well as insmooth muscle cells of the ductus epididymidis and in endothelial cells of the capil-laries in epididymal interstitium during a period of 3~30 days after administrationof low dose of α-chlorohydrin combined with glycidol(5 mg α-chlorohydrin plus75mg glycidol/kg/day×2),which previously has been proved to be effective inmale rat antifertility.Additionally,Golgi vacuoles,pinocytotic vesicles,multivesicularbodies,stereocilia and smooth endoplasmic reticulum in the apical cytoplasm of someprincipal cells occasionally were found to decrease in number.No Morphologicalchanges of the nuclei were found in these affected cells.The number of morphologi-cally unchanged mitochondria appears to become more with extended time after drugswithdrawal.2.In the group treated with the large combined dose of the drugs(20 mg α-chlorohydrin plus 100 mg glycidol/kg/day×2),apart from the changes mentionedabove,necrosis and exfoliation of some epididymal epithelial cells could be found.Some lesions of the sperm,such as the destruction of the cytoplasmic membrane,swelling of mitochondria and pyknosis of nucleus were present in a few specimenstaken from this group.3.Effects similiar to that of the combined use of the drugs were also found ingroups using α-chlorohydrin or glycidol alone,but a less degree of lesions revealed.These observations suggested that the antifertile effects of α-chlorohydrin andglycidol either used in combination or alone could result injury of mitochondria inepididymal epithelial cells,which should lead to the decrease of the function ofepididymis,hence to interfere with the maturation of the sperm in it.

本研究用电镜(辅以半薄切片的光镜检查)观察了3-氯丙二醇及2,3-氧丙醇合并用药对大白鼠附睾起始部及尾部超微结构的影响。同时,对二者单独使用时的作用进行了对比观察。结果表明:在较低有效抗生育剂量合并用药(α-ch 5mg+gly 75mg/kg/天×2)3天后,多数动物附睾管的主细胞、顶端细胞、基底细胞及亮细胞,管壁平滑肌细胞以及间质微血管内皮细胞的线粒体出现了不同程度的肿胀和退变。部分动物附睾上皮主细胞的高尔基大泡,吞饮小泡、多泡小体、不动纤毛及顶部滑面内质网亦有减少。细胞核未见明显改变。随着停药时间的延长,正常形态的线粒体的数量有相对增加的趋向,其它因药物作用而变化的细胞器亦趋向恢复。在高剂量合并用药组(α-ch 20mg+gly 100 mg/kg/天×2),除见有上述变化外,在部分动物的少数附睾管断面见到上皮细胞坏死、脱落,精子的质膜破裂、线粒体肿胀及核固缩等改变。单独使用3-氯丙二醇及2,3-氧丙醇对附睾超微结构的影响与合并用药相似。但二者影响的程度不及合并用药明显。这类药物在较低有效抗生育剂量下引起的附睾上皮细胞线粒体的损伤,主细胞高尔基复合体、滑面内质网及吞饮小泡的减少或功能降低,可能导致附睾正...

本研究用电镜(辅以半薄切片的光镜检查)观察了3-氯丙二醇及2,3-氧丙醇合并用药对大白鼠附睾起始部及尾部超微结构的影响。同时,对二者单独使用时的作用进行了对比观察。结果表明:在较低有效抗生育剂量合并用药(α-ch 5mg+gly 75mg/kg/天×2)3天后,多数动物附睾管的主细胞、顶端细胞、基底细胞及亮细胞,管壁平滑肌细胞以及间质微血管内皮细胞的线粒体出现了不同程度的肿胀和退变。部分动物附睾上皮主细胞的高尔基大泡,吞饮小泡、多泡小体、不动纤毛及顶部滑面内质网亦有减少。细胞核未见明显改变。随着停药时间的延长,正常形态的线粒体的数量有相对增加的趋向,其它因药物作用而变化的细胞器亦趋向恢复。在高剂量合并用药组(α-ch 20mg+gly 100 mg/kg/天×2),除见有上述变化外,在部分动物的少数附睾管断面见到上皮细胞坏死、脱落,精子的质膜破裂、线粒体肿胀及核固缩等改变。单独使用3-氯丙二醇及2,3-氧丙醇对附睾超微结构的影响与合并用药相似。但二者影响的程度不及合并用药明显。这类药物在较低有效抗生育剂量下引起的附睾上皮细胞线粒体的损伤,主细胞高尔基复合体、滑面内质网及吞饮小泡的减少或功能降低,可能导致附睾正常生理功能的改变,从而造成不利于精子成熟的环境而产生抗生育作用。此外,药物对附睾管壁平滑肌细胞的线粒体及附睾微血管内皮细胞线粒体均引起不同程度的损伤,其程度严重者可能导致精液囊肿的产生。

 
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