The morbidity rates of the therapeutic and control group were15.24% and 5.15% separately. There were 215organs experiencing dysfunction in therapeutic group with 133 cases and the average number was 2.05per case; on the contrary,there were 133 organs experiencing dysfunction in control group with 97 cases and the average number was 1.27 per case;
there were significant differences in the severity of gastric emptying dysfunction between spleen asthenia with phlegm-turbidity, spleen asthenia with phlegm blood-stasis and spleen asthenia with qi stagnation (P<0.001), no difference between the former two syndromes (P>05).
Study on the action of resistin-induced human umbilical vein endothelial cell dysfunction
Adipokine resistin exerted a direct effect in promoting HUVEC dysfunction by promoting ICAM-1 and ET-1 expression.
This review focuses on presently known biochemical mechanisms of cell response to thrombin and their role in endothelial barrier dysfunction.
The hypothesis is based on the fact that both diabetes and carcinogenesis cause dysfunction of the vital cellular signal system regulated by the protein kinase C (PKC) family.
Mitochondrial dysfunction including induction of the mitochondrial permeability transition (MPT) and inhibition of mitochondrial respiration have been implicated as major determinants in the pathogenesis of DOX cardiotoxicity.