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cerebellar atrophy
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  小脑萎缩
     Objective:To study the clinical feasibility of transplanting human neural stem cells in treatment of cerebellar atrophy (CA).
     目的 :探索人神经干细胞 (neural stem cell,NSC)移植治疗小脑萎缩 (cerebellar atrophy,CA)的有效性。
短句来源
     Clinical CT Diagnosis of Cerebellar Atrophy
     小脑萎缩的临床CT诊断
短句来源
     Results 6 patients from 4 SCA6 Chinese kindreds had abnormal SCA6 alleles with CAG repeat expanded to 25 and 26, respectively, of which 2.5% was about the positive rate, while CAG repeat of normal SCA6 allele ranged from 5 to 17. The basic characteristics of SCA6 patients were slowly progressive cerebellar ataxia and purely cerebellar atrophy.
     结果检测确定4个SCA6家系6例患者SCA6(CAG)n的重复数为25和26,正常人群SCA6(CAG)n的重复数为5~17,SCA6突变频率为本组SCA家系的2.5%。 SCA6患者临床仅表现为缓慢进展的小脑性共济失调,MRI显示单纯小脑萎缩
短句来源
     Clinical transplantation of human neural stem cells in treatment of cerebellar atrophy
     人神经干细胞移植治疗小脑萎缩
短句来源
     Pontine and cerebellar atrophy correlate with clinical disability in SCA2
     SCA2患者的临床残疾与脑桥-小脑萎缩的关系
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  “cerebellar atrophy”译为未确定词的双语例句
     Homoplastic Transplantation of Fetal Cerebellar Tissue on 30 Cases of Cerebellar Atrophy.
     胎脑移植术治疗小脑萎缩症30例
短句来源
     Severe infratentorial atrophy was demonstrated in 3 patients, together with cruciform high signal change at the pons in T2WI in 1 patient, and moderate cerebellar atrophy was shown in the other 2 patients.
     3例示桥脑及小脑严重萎缩,1例合并桥脑类“十”字形的T2像上高信号改变,另2例示小脑蚓部及半球中度萎缩。
短句来源
     CT scan showed cerebellar atrophy, Four of them in 4 to 17 years after onset died.
     4例发病后4~17年死亡。
短句来源
     Results When lesion was located in the spinal cord ventrally and olivo ponto OPCA cerebellar atrophy, the evoked potentials N30, P40 from C2 and occiput after stimulating tibial nerve showed simultaneously abnormal. C2 and occiput potentials from stimulating median nerve appeared normal when the potential from stimulating tibial nerve showed normal.
     结果 脊髓腹侧及小脑病变可致胫神经刺激,C2、枕点N30及P40电位出现同步性异常改变,且胫神经刺激出现异常者,正中神经刺激,C2、枕点电位却可保持正常。
短句来源
     Results Various degrees of cerebral and cerebellar atrophy were seen in 1 patient, and only the cerebral atrophy was found in another case with MRI and CT. The lesions in the pons, both caudate nuclei, thalamus, and claustra were hypointense to gray matter on T 1WI and hyperintense on T 2WI, and the lesions showed no enhancement after Gd DTPA injection in 1 case. The local abnormal signal lesions were seen on both thalamus on MRI in another case.
     结果 双侧大脑半球及小脑皮层轻度萎缩 1例 ,大脑半球轻度萎缩1例 ,MRI显示桥脑、双侧尾状核、丘脑及屏状核有特征性的局灶长T1、长T2 信号 ,无强化 1例 ,MRI示双侧丘脑局灶异常信号灶 1例。
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  相似匹配句对
     Clinical CT Diagnosis of Cerebellar Atrophy
     小脑萎缩的临床CT诊断
短句来源
     STUDY ON HEREDO-CEREBELLAR-ATROPHY-ATAXIA IN CALF
     犊牛遗传性小脑萎缩共济失调研究报告
短句来源
     Cerebellar infarction
     小脑梗塞
短句来源
     Cerebellar Hemorrhage
     小脑出血
短句来源
     atrophy of pancreas;
     胰腺萎缩;
短句来源
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  cerebellar atrophy
There is an extreme generalized cerebellar atrophy in one case.
      
Improvement of ataxia in alcoholic cerebellar atrophy through alcohol abstinence
      
The severity of cerebellar signs and the degree of cerebellar atrophy depicted by computed tomography (CT) were independently graded in 108 patients with cerebellar disorders.
      
In patients with involvement of the cerebellar hemispheres and anterior lobe, clinical signs tended to be more pronounced than the cerebellar atrophy revealed by CT.
      
Ataxia and cerebellar atrophy in chronic alcoholics
      
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16 patients with hereditary cerebellar atrophy were admitted to our hospital in the past three years. Computed tomography scans were performed on all of the patients. The CT scans in 2 cases were within normal limits,and The other 14 cases all showed remarkable cerebellar atrophy. The main positive findings were as following:1. The sulci of the cerebellar hemispheres and/or the vermis widened and could be seen clearly.2. The fourth ventricle was enlarged.3. The cisterns around, the cerebellum...

16 patients with hereditary cerebellar atrophy were admitted to our hospital in the past three years. Computed tomography scans were performed on all of the patients. The CT scans in 2 cases were within normal limits,and The other 14 cases all showed remarkable cerebellar atrophy. The main positive findings were as following:1. The sulci of the cerebellar hemispheres and/or the vermis widened and could be seen clearly.2. The fourth ventricle was enlarged.3. The cisterns around, the cerebellum and brain stem such as the quadrigeminal, superior cerebella, cerebellopontine angle and the prepontine cisterns were enlarged.4 In some cases the cerebral atrophy could be demonstrated. Thus, CT scan can provide the objective evidence for the diagnosis and differential diagnosis of the disease.

我院近3年来共收治小脑型遗传性共济失调患者16例,全部进行了CT脑扫描诊断。除2例未见明显阳性改变外,余14例均显示出明显的小脑萎缩性变化。主要阳性改变有以下几方面: 1.小脑半球和/或蚓部脑沟增宽,在CT片上清晰可见。 2.第四脑室扩大。 3.小脑及脑干周围脑池扩大,如回叠体池、上蚓池、桥小脑脚池、桥池等均可扩大。 4.部分病例可合并大脑萎缩。 CT扫描为本病的诊断和鉴别提供了客观依据。

This paper reports the condition of the sythetic study on calf heredoataxia cerebellaris and points out that the gradual ataxia such as shaking one's head, standing by pushing aside with both feet, walking shakily ect is pathognomonic symptom, that the cerebellar atrophy and purkinje-cell-storey degeneration, are pathognomonic changes in pathology. We should differ this disease from coenurosis, cerebellar neoplasma and paralysis nervi vestibularis ect. According to their pathological and clinical...

This paper reports the condition of the sythetic study on calf heredoataxia cerebellaris and points out that the gradual ataxia such as shaking one's head, standing by pushing aside with both feet, walking shakily ect is pathognomonic symptom, that the cerebellar atrophy and purkinje-cell-storey degeneration, are pathognomonic changes in pathology. We should differ this disease from coenurosis, cerebellar neoplasma and paralysis nervi vestibularis ect. According to their pathological and clinical characteristic, the author suggests that these diseases should be class into three types: cerebellar.spinoneural, cerebellar-spinoneural. These recessive genetic diseases occured in secondary and third filial of nearsighted generation. The attack rate of secondary filial was 6.29%. The score of healths and patients in F2 neared 15 to 1. The author explians this genetic phenomenon with freedom combination rule and multiple action principle of mendel. The report deuls really with the control mensurement established by author and it's subtantial effect. Eliminating carrier of harmful gene—the ox and prohibiting nearsighed generation bave been proved to be an effective measurement for eliminating this disease.

本文首次报导了犊牛遗传性运动失调症的综合研究情况。指出渐进性共济失调(摇头、叉开站立、行走不稳)为示病症状,小滴萎缩及蒲氏细胞层的变性为示病病变。诊断时尚须与脑包虫、小脑肿瘤、前庭神经麻痹等相鉴别、根据病理组织学和临床症状特点,作者建议将本病按小脑型、脊髓型、小脑脊髓型分类、本病发生于父母繁殖的二、三代,属于隐性遗传。本病发病率分别占该父女近亲繁殖二、三代总后裔的6.1%、11%,即级进二代健康后裔与患者的比数接近15∶1,9∶1。作者用孟德尔自由组合规律的重叠作用理解释了这一遗传现象,本报告真实地报导了作者拟定的防制措施及其在实践中取得的效益,证明淘汰有害基因携带者及可疑者禁止近亲繁殖是杜绝本病发生的有效措施。本文为临床遗传学的研究提供了罕见的素材。

The pathognomonic signs of calf heredo-ataxia-cerebellaris are those of gradual ataxia such as shaking of head, standing by stretching out all feet, walking shakily, etc. The pathognomonic changes in pathology are cerebellar atrophy and purkinjie-cell-layer degeneration. Should be differentiated.This disease from coenurosis, cerebellar neoplasma and paralysis nervi vestibularis, etc. It is suggested that, according to their pathological and clinical characteristic, these diseases should be classified...

The pathognomonic signs of calf heredo-ataxia-cerebellaris are those of gradual ataxia such as shaking of head, standing by stretching out all feet, walking shakily, etc. The pathognomonic changes in pathology are cerebellar atrophy and purkinjie-cell-layer degeneration. Should be differentiated.This disease from coenurosis, cerebellar neoplasma and paralysis nervi vestibularis, etc. It is suggested that, according to their pathological and clinical characteristic, these diseases should be classified into three types: cerebellar, soinoneural, and cerebellar-spinoneural. Thess recessive genetic diseases occurred in the inbred F2 and F3. The attack rate in F2 was 6.2%. The rate of healthy and diseased in F2 approached 15 to 1. The authors explain this genetic phenomenon with the rule of free combination and multiple action principle of Mendel. Control measures have been worked out by the authors. Eliminating the carriers of harmful genes, the bulls, and prohibiting inbreeding have been proved to be an effective measure for the control of this disease.

本文报道了犊牛遗传性运动失调症的综合研究情况,指出摇头、叉开站立、行走不稳等渐进性共济失调为示病症状。小脑萎缩及蒲氏细胞层的变性、坏死为示病病变,诊断时尚须与脑包虫、小脑肿瘤、前庭神经麻痹等相区别。根据病理组织学和临床症状特点,作者建议将本病按小脑型、脊髓型、小脑脊髓型分类、本病发生于西门塔尔种公牛与土种黄牛或荷杂近亲繁殖第二、三代,属于隐性遗传。其第二代发病率为6.2%,即F_2健康后裔与患畜的比数接近15:1,作者用孟德尔自由组合规律和重叠作用原理解释了这一遗传现象。本报告还报道了作者拟定的防制措施及其在实践中取得的效益,证明淘汰有害基因携带者种公牛,禁止近亲繁殖是杜绝本病发生的有效措施。本文为临床遗传学的研究提供了罕见的材料。

 
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