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nerve
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  神经
    Information Detection of Peripheral Nerve and Nerve-signal Controlled Prosthesis: A Basic and Applied Experimental Study
    周围神经的信息检测及神经信息控制假肢的应用基础研究
短句来源
    Response to Spinal Cord Stimulation in Variants of the Spared Nerve Injury Model and Its Mechanism
    变异保留神经损伤模型对脊髓电刺激的反应及其作用机制的研究
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    Experimental Study on Electrophysiology Mechanism of Vagus Nerve Stimulation for Epilepsy and the Developing of New VNS System
    迷走神经刺激治疗癫痫的电生理机制及国产神经刺激系统研制的实验研究
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    VALUE OF NERVE CONDUCTION VELOCITY AND EMG STUDY IN THE DIAGNOSIS OF CARPAL TUNNEL SYNDROME AND SIMILAR CONDITIONS
    神经传导速度及肌电图研究对腕管综合征等的诊断价值
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    The Characteristics of Cerebral Cortex SEP m.n. by Median Nerve Stimulatin in Normal Adults
    我国正常成人平均正中神经体感诱发电位(SEPmn)的特征
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  听神经
    Skill for microsurgical removal and preservation of facial nerve and hearing for large acoustic neuromas
    大型听神经瘤显微手术切除及面听神经保留技巧
短句来源
    Microsurgical treatment of large acoustic neuromas and preservation of facial nerve and auditory nerve function
    大型听神经瘤显微外科手术中的面听神经功能保护
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    Auditory nerve impairment in the patients with multiple sclerosis
    多发性硬化患者的听神经损害
短句来源
    Microsurgical removal and preservation of facial nerve for large acoustic neuromas using the electromyography monitoring
    肌电图监护下大型听神经瘤的显微手术及面神经保留
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    Result: The incidence of acoustic nerval damage was 71. 4% in dose of 16. 1 Gy, trigeminal nerval damage was 42. 9% in 15. 4 Gy,the rate of preservation about acoustic nerve was 727% .
    结果:16.1Gy照射听神经造成听力减退发生率为71.4%,有效听力保留率为72.7%; 15.4Gy照射三叉神经造成三叉神经损害发生率为42.9%;
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  “nerve”译为未确定词的双语例句
    The Enzymatic Histocytochemistry Study of Oris Muscle after Facial Nerve Denervation
    面神经失神经支配后口轮匝肌酶组织化学研究
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    Proteomics of the Differentiation of PC12 Cells and Cos-7 Stable Cell Line Express TrkA under the Stimulation of Nerve Growth Factor and Protective Effect of Melatonin on Dopaminergic Neurons
    PC12细胞、稳定表达TrkA的Cos-7细胞在神经生长因子刺激下的蛋白质组学研究及褪黑素对多巴胺能神经元的保护作用
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    Experimental and Clinical Research on the Value of F Wave Assisted Electrophysiological Monitoring in the Protection of Facial Nerve Function during the Microoperation for Acoustic Neurinomas
    面肌F波辅助电生理监测对听神经瘤显微手术面神经保护价值的实验与临床研究
短句来源
    The Observations on the Effect of Acupoint-injection with Securinini Nitratis for 28 Cases of Peripheral Facial Nerve Paralysis
    硝酸—叶萩硷穴位注射治疗周围性面神经麻痹28例疗效观察
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    The Clinical Significance of Measurement of F Wave in the Diagnosis of Nerve Root Lesions
    F波测定对神经根病损诊断的临床意义
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  nerve
The compound 12 was found to be five times more potent than 11 in reducing twitch response to nerve stimulations, indicating the importance of extended interonium distances and 17-acetoxy function for potent antagonist activity.
      
Differential diagnosis of cervical nerve compression syndrome of the external intervertebral foramen
      
The aim of the present research is to study the mechanism of cervical nerve compression syndrome of the external intervertebral foramen and its differential diagnosis with cervical spondylosis.
      
Fifteen cases with cervical nerve compression syndrome of the external intervertebral foramen were healed by the diagnostic treatment.
      
Diagnostic treatment was propitious to differentiate cervical nerve compression syndrome of the external intervertebral foramen from cervical spondylosis.
      
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(1) The sciatic and medial nerves are stimulated by rectangular impulses with. a definite strength and duration(so called etectro acupuncture), and a narcotic state of animal is induced within 20-30 minutes, during which the animal shows no response to nociceptive stimuli. (2) The blood pressure rises immediately after, the application of electroacuptuncture, and then retnrns to about control level although the electro-acupuncture lasts. with the withdrawal of etecfro-acu Punoture, the bood pressure is...

(1) The sciatic and medial nerves are stimulated by rectangular impulses with. a definite strength and duration(so called etectro acupuncture), and a narcotic state of animal is induced within 20-30 minutes, during which the animal shows no response to nociceptive stimuli. (2) The blood pressure rises immediately after, the application of electroacuptuncture, and then retnrns to about control level although the electro-acupuncture lasts. with the withdrawal of etecfro-acu Punoture, the bood pressure is completely recovered, it is therefore shown that electro-acuPuncture does not exert undesirable action on the cardiovascular system. (3) The pressor reflex caused by occlusion of carotid artery shows an exaggeration during the course of elect ro-acupuncture and restores to control leel as the electro- acupuncture is discontinued. (4) During the course of electroacupuneture, the pressor reflex elicited by stimulation of central end of depressor nerve with various strength of Fa adic curreut reveals typical phasic changes of Wedensky s parabiotic inhibition. (5) On the basis of above observations it is suggested that the physiological mechanism of the electro-acupunetare narcosis is the development of parabiosis in the nerve centers as the consequence of prolonged actionof afferent impules from eletroacupuncture regions.

(一)用一定强度和时间的电针刺激家兔,能引起动物的麻醉状态,使动物对一般损伤性刺激不出现反应;停止电针后10——15分钟,动物对损伤性刺激的反应恢复。 (二)动脉血压在电针刚开始时突然增高,随后恢复至接近正常水平,电针停止后血压完全恢复,由此表明电针本身对心臟——血管系统并无不良影响。 (三)阻塞一侧颈总动脉所致的加压反射,在电针过程中表现加强,电针停止后又恢复正常。 (四)以感应电刺激减压神经向中段所致的减压反射,在电针过程中有特異性变化,表现为减压神经强刺激引起的减压效应较针前为弱,而弱刺激的效应反较针前为强,并出现了均等和反常的间生性效应。 (五)从本实验的结果来看,电针麻醉的机制是在于电针刺激的大量冲动,引起了神经中枢间生态的发展,并且这种发展取决于电针刺激的强度和时间,而在各动物之间有一定的个体差異。

Sustained nociceptive stimuli were applied to the hind limb of 8 monkeys (Macaca mulatta)in three ways:(1)fracturing the fibula;(2)applying a chemical irritant(ether extract of capsicum)to the sciatic nerve topically;(3)injecting hypertonic saline into the gas- trocnemius and its adjacent subcutaneous tissues.All these methods were found to be capable of producing,on the surface of the pinnae,small discrete spots tender to pressure.The topographical distribution of the spots was not regular;but,statistically,they...

Sustained nociceptive stimuli were applied to the hind limb of 8 monkeys (Macaca mulatta)in three ways:(1)fracturing the fibula;(2)applying a chemical irritant(ether extract of capsicum)to the sciatic nerve topically;(3)injecting hypertonic saline into the gas- trocnemius and its adjacent subcutaneous tissues.All these methods were found to be capable of producing,on the surface of the pinnae,small discrete spots tender to pressure.The topographical distribution of the spots was not regular;but,statistically,they often appeared near the tip of the triangular fossa and the antitragus of the pinna.They began to appear from 40 minutes to several hours after the noxious treatment,and persisted for a period from 3 days to a few months(7—10 days on average).Intravenous injection of drugs known to suppress the activity of the reticular forma- tion of the brain stem,such as chlorpromazine or nembutal,augmented the reaction to pressure and increased the number of spots.On the contrary,drugs,such as LSD_(25),amphetamine or adrenaline,which increase the excitability of the reticular formation,suppressed the reaction to pressure and decreased the number of spots.In other experiments,procaine solution was injected into the third ventricle of the brain;1—2 minutes after the injection the referred tender spots disap- peared and reappeared after 13—60 minutes.This fact suggests that the central mechanism con- cerned in the production of the referred tenderness involves neural structure or structures situated in the central portion of the rostral brain stem;and perhaps the mechanism is inhibited by impulses from the reticular formation,as indicated by the experiments of intravenous injection.

用以下三种方法在8只猴人工产生了牵涉性耳郭压痛点:(1)切断腓骨;(2)化学物质(辣椒的乙醚提取物)刺激坐骨神经;(3)下肢肌肉及皮下注射高渗盐水。压痛点可出现于耳郭各处,但比较集中于三角窝尖端及对耳屏附近。压痛点在损伤后40分钟至数小时出现,持续3天至数月(平均7—10天)。静脉注射药理学上认为对脑干网状结构有抑制性影响的药物(氯丙嗪及戊巴比妥)后,压痛点增多,压痛反应增强。注射对网状结构有兴奋性影响的药物(离麦角酸二乙胺、苯异丙胺或肾上腺素),效果相反。这暗示参与耳郭压痛现象的神经结构受来自网状结构的冲动的抑制。第三脑室注射普鲁卡因溶液可在1—2分钟内取消耳郭压痛点,至第13—60分钟又行恢复,表明参与牵涉性耳郭压痛反应的一个中枢环节在脑干前端的中央部分。

The experiments were made on guinea pigs. The results obtained demonstrate that after crushing the nerve the regeneration of the motor nerve endings in muscle poisoned by botulinum toxin can proceed with the same effectiveness as in unpoisoned muscle. At the same time they show that causing the poisoned nerve endings to degenerate and then regenerate may be employed as a means of accelerating the recovery of the poisoned muscle; for example, the very slow spontaneous recovery usually shown...

The experiments were made on guinea pigs. The results obtained demonstrate that after crushing the nerve the regeneration of the motor nerve endings in muscle poisoned by botulinum toxin can proceed with the same effectiveness as in unpoisoned muscle. At the same time they show that causing the poisoned nerve endings to degenerate and then regenerate may be employed as a means of accelerating the recovery of the poisoned muscle; for example, the very slow spontaneous recovery usually shown by a poisoned muscle requiring about a year for completion, can be shortened to about 11/2 months by crushing the nerve. The suggestion is made that the slow spontaneous recovery iust mentioned is probably the result of a process of slow spontaneous tissue renewal constantly occurring in the motor nerve endings. It is also pointed out that the experimental finding reported here constitutes a new piece of corroborative evidence for the conclusion that the site of action of botulinum toxin is confined to the motor nerve endings.

实驗在豚鼠进行,結果揭示,夹毁神经后运动神经末梢的再生在肉毒杆菌毒素中毒的肌肉与在正常肌肉完全一样地有效。用夹毁神经的办法,可以比较迅速地解除肉毒中毒肌肉的麻痹。本文结果,亦对肉毒的作用地点问题提供了新的佐証。

 
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