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chronic fluorosis
相关语句
  慢性氟中毒
     Methods:Chronic fluorosis model was formed by drinking water with 50 mg/L or 100mg/L of NaF for mice. The distribution and expression of Smad 4 in ameloblast of incisor teeth were detected by SABC immunohistochemical method in mice.
     方法 :分别用饮水加 5 0 mg/ L Na F、1 0 0 mg/ L Na F建立小鼠慢性氟中毒模型 ,SABC免疫组化法检测小鼠切牙成釉器细胞内 Smad4的分布与表达。
短句来源
     Fluoride injections were given in the rat with 0.25% sodium fluoride solution at the doses of 1-6 ml/kg body wt. The chronic fluorosis enamel appeared after 16-30 days.
     大鼠注射0.25%氟化钠溶液1~6毫升/千克体重持续到16~30天时,产生典型的慢性氟中毒
短句来源
     2 groups of Wistar rat with chronic fluorosis were duplicated with 2 concentrations of fluorine(F 1.58,2.63 mmol/L)in drinking water. At the same time,selenium 2.0mg/kg was supplemented in 2 groups of rat with fluorosis for 14 months.
     Wistar大鼠饮1.58、2.63mmol/L高氟水14个月造成慢性氟中毒,另2组大鼠在饮高氟水同时加饲2.0mg/kg硒饲料。
短句来源
     2 groups of Wistar rats with chronic fluorosis were duplicated with 30 and 50mg/L fluorine(F) in drinking water. 2.0mg/kg selenium (Se) in fodder was supplemented in the other 2 groups of rats with fluorosis.
     Wistar大鼠饮30和50mg/L高氟(F)水造成慢性氟中毒,同时另外两组大鼠饮高F水、饲2mg/kg加硒(Se)饲料。
短句来源
     Experimental study on antagonistic effects of Se-Zn agent on chronic fluorosis
     硒锌制剂拮抗慢性氟中毒的实验研究
短句来源
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  氟病
     Contents of cytokine in dairy goat with chronic fluorosis
     氟病奶山羊血清细胞因子含量的变化
短句来源
  “chronic fluorosis”译为未确定词的双语例句
     To study the effects of chronic fluorosis on cocks in pathologic change ,36 one-day cocks were randomly divided into 6 groups of 6 each. The cocks of test group drink water with 1.75 g/L(1/27LD50),0.875 g/L,0.438 g/L,0.219 g/L,0.109 g/L sodium fluoride respectively and the ones of control group drink running water,for 90 days.
     为了探讨氟对鸡的病理变化作用研究,选用1月龄蛋公鸡36只,第Ⅰ组为正常对照组,第Ⅱ~Ⅵ组分别饮水添加氟化钠1.75g/L(1/27LD50),0.875g/L,0.438g/L,0.219g/L,0.109g/L,试验周期为90d,每组6只。
短句来源
     Analysis of MBP,NSE,F~- content and CHE activity in brain tissue of rats with chronic fluorosis
     氟中毒大鼠脑组织MBP、NSE、氟含量及CHE活性分析
短句来源
     In order to observe the effect of high fluorine (F)and the protection of selenium(Se)on pituitary gland,thyroid and parathyroid,wistar rat of 2 groups with chronic fluorosis were duplicated with 2 concentration F in the drinking water (1. 58,2. 63 mmol/L)and 2 groups were fed with Se fodders (2. 024mg/kg)and high F for 14 months.
     为观察过量氟对大鼠垂体、甲状腺、甲状旁腺的影响及硒的保护作用,选用Wistar大鼠饮用30mg/L(1.58mmol/L)与50mg/L(2.63mmol/L)高氟水,同时设高氟水加富硒(se含量2.024mg/kg)饲料组。
短句来源
     The Relationship between Liver,Kidney Apoptosis and p53 in Chronic Fluorosis Rsts
     氟中毒大鼠肝肾细胞凋亡与p53的关系
短句来源
     Dynamically observed skeletal X-ray in rats with chronic fluorosis and effects of selenium
     氟中毒大鼠骨骼X线动态观察及硒的影响
短句来源
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  chronic fluorosis
The oxidative stress and modification of cellular membrane lipids may be involved in the pathogenesis of chronic fluorosis and provide a possible explanation for the gross system damage observed in the body, especially in soft tissues and organs.
      
An animal model of chronic fluorosis was produced by subjecting Wistar rats to high doses of fluoride in drinking water for a prolonged period.
      
It is concluded that chronic fluorosis exposure leads to a remarkable destruction in testes tissues of F1 and F2 rats via lipid peroxidation.
      
This experiment was designed to investigate the histological and lipid peroxidation effects of chronic fluorosis on testes tissues of first- and second-generation rats.
      
It is concluded that chronic fluorosis causes a marked destruction in kidney tissues of F1 and F2 rats by causing lipid peroxidation.
      
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This paper reports an outbreak and the diagnostic procedure of a disease which occurred in a herd of draft buffaloes belonging to a production brigade of a people's commune near a phosphate fer- tilizer plant in a county of Guangdong province. The geographic environment of the locality where this herd had been kept and the possible etio- logical factors were studied.clinical examinations were carried out on 75 animals of this herd.of these,2 were killed and autopsies performed.Samples of teeth and blood from...

This paper reports an outbreak and the diagnostic procedure of a disease which occurred in a herd of draft buffaloes belonging to a production brigade of a people's commune near a phosphate fer- tilizer plant in a county of Guangdong province. The geographic environment of the locality where this herd had been kept and the possible etio- logical factors were studied.clinical examinations were carried out on 75 animals of this herd.of these,2 were killed and autopsies performed.Samples of teeth and blood from these affected animals,grasses from pasture grazed by the animals and waste water from the plant were subjected to chemical analyses.As a result,a definite diagnosis of chronic fluorosis in the affected herd was made. 1)Post-mortem findings: The 2 animals autopsied revealed swollen pulmonary and mesenteric lymph nodes.On the cut surfaces of these lymph nodes,a large amount of evenly distributed black deposits could be found. In one case,on the upper part of the ribs somewhere near the longissimus dorsi,there was develop- ment of exostoses in the form of osteomas wherein osteoporesis of the ribs was also present. 2)Clinical symptoms: Affected animals in general were in poor condition and showed lowered draft ability.Oral exa- mination revealed that the table surfaces of some of the leeth had worn smooth.Malformation, uneven wear,excessive altrition of the teeth were also encountered.Staining of the enamel to a yellowish brown colour resulted in the presence of small foci of mottling.Black deposits accumula- ted at the gum line.Lameness in various degrees was observed in the affected herd.In severe cases, salivation with open mouth could be seen,and an epileptoid attack might sometimes be encountered. 3)Results of laboratory chemical analyses: Fluorine content of affected teeth was 4—5 times higher than normal(2260 ppm,while the normal average value is 418 ppm). Fluorine in the serum of affected animals was 7-8 times higher than normal(0.90-1.26 ppm, while the normal average is 0.13-0.15 ppm). Fluorine content of the grasses taken from the pastured area was 3-6 times higher than the ordi- nary mountain grass(namely,6 ppm,while the value recorded in literature is 1-2 ppm). Water in the ponds of pasturing area in question contained 0.27-0.62 ppm of fluorine,while or- dinary natural water contains 0.2-0.5 ppm. Waste water from the phosphate fertilizer plant contained 4.75 ppm and the tap water used in our eollege—0.2ppm.

本文是报道了广东省某县磷肥厂附近一个公社大队的一群水牛的发病情况和诊断经过。通过对该牛群所在地的地理环境和发病原因调查,并对该牛群的75头牛进行了临床检查,对其中二头进行了病理剖检,取回病牛的牙齿、血液,连同所在地的牧地草和工厂废水进行了化学分析,确诊该水牛群为慢性氟中毒。1.剖检所见:两头牛的肺门淋巴结和肠系膜淋巴结肿大,切面可见多量均匀点的黑色沉积物。一头在肋骨上部靠近背最长肌处有骨质增生,并呈骨瘤状,该处骨质疏松。2.临床症状:病牛普遍营养情况不良,耕作能力下降。口腔检查,齿面圆滑、畸型、磨灭不整,牙齿珐琅质呈黄褐色斑纹,齿龈部附着黑色块状物。牛群中可见有不同程度的跛行,严重的可见张口流涎,或呈癫痫样发作。3.实验室化学分析结果:病牛齿氟含量比正常平均值高4~5倍(2260ppm;正常平均值为418ppm)。病牛血清氟比正常平均值高7~8倍(0.90~1.26ppm;正常平均值为0.13~0.15ppm)。病牛牧区草含氟量比一般山草高3~6倍(大于6ppm;文献记载一般含量为1~2ppm)。病牛牧区池塘水含氟量为0.27~0.62ppm;一般自然水含氟量为0.2~0.5ppm。磷肥厂废水含氟量为4.75...

本文是报道了广东省某县磷肥厂附近一个公社大队的一群水牛的发病情况和诊断经过。通过对该牛群所在地的地理环境和发病原因调查,并对该牛群的75头牛进行了临床检查,对其中二头进行了病理剖检,取回病牛的牙齿、血液,连同所在地的牧地草和工厂废水进行了化学分析,确诊该水牛群为慢性氟中毒。1.剖检所见:两头牛的肺门淋巴结和肠系膜淋巴结肿大,切面可见多量均匀点的黑色沉积物。一头在肋骨上部靠近背最长肌处有骨质增生,并呈骨瘤状,该处骨质疏松。2.临床症状:病牛普遍营养情况不良,耕作能力下降。口腔检查,齿面圆滑、畸型、磨灭不整,牙齿珐琅质呈黄褐色斑纹,齿龈部附着黑色块状物。牛群中可见有不同程度的跛行,严重的可见张口流涎,或呈癫痫样发作。3.实验室化学分析结果:病牛齿氟含量比正常平均值高4~5倍(2260ppm;正常平均值为418ppm)。病牛血清氟比正常平均值高7~8倍(0.90~1.26ppm;正常平均值为0.13~0.15ppm)。病牛牧区草含氟量比一般山草高3~6倍(大于6ppm;文献记载一般含量为1~2ppm)。病牛牧区池塘水含氟量为0.27~0.62ppm;一般自然水含氟量为0.2~0.5ppm。磷肥厂废水含氟量为4.75ppm;本院自来水含氟0.2ppm。

By using the method of Fuzzy clustering analysis, a study on the source of fluorine in Kuitun Area, Xinjiang is presented. The authors believe that the source of fluorine in this area is connected with sediment and the following results have been obtained: 1.The hardness of the water is mainly depended on magnesium in the high morbidity district of endemic chronic fluorosis poisoning. The high morbidity in this district has relation with low density of magnesium of the water. 2.The size of pH affects the...

By using the method of Fuzzy clustering analysis, a study on the source of fluorine in Kuitun Area, Xinjiang is presented. The authors believe that the source of fluorine in this area is connected with sediment and the following results have been obtained: 1.The hardness of the water is mainly depended on magnesium in the high morbidity district of endemic chronic fluorosis poisoning. The high morbidity in this district has relation with low density of magnesium of the water. 2.The size of pH affects the quantity of the containing fluorine in the water, i.e. alkalinity is associated with morbidity. 3.In the north-western area of No. 129 Farm,Kuitun Area, the fluorine in deep well-water is higher than that in shallow one. However, in the south-eastern area of No. 129 Farm in deep well-water shows the same tendency of that in shallow one.

本文使用模糊聚类分析方法探讨了奎屯地区的氟源及其与其他元素空间分布的关系,并进行了地质学及医学上的解释。该区氟的来源主要是由于沉积物中夹带氟元素所致:(1)该区氟中毒病情的严重程度,在重病区与饮水中镁含量低有密切关系,在轻病区则与饮水中钙含量低有密切关系,防治工作中应注意补镁或钙;(2)该区氟中毒的病情程度与饮水的碱性呈正相关,改进膳食及防治过程中应注意之;(3)从129团起向西北方向一带的地下水中深井的含氟量明显高于浅井,这一带居民不宜饮用深井水,同时,也应禁用含氟高的井水灌溉食用的农作物。

31 rats with chronic fluorosis produced by long-term administering excessive fluorine through the drinking water containing 50 ppm fluorine and 24 rats drinking low-fluorine water served as control are studied on the DNA, RNA contents of brain.The results showed that in the test rats as compared with the control rats, the fluorine content of urine was higher; the brains were smaller and lighter; the fluorine contents of brain were increased; the RNA contents of brain were decreased, while the DNA not changed....

31 rats with chronic fluorosis produced by long-term administering excessive fluorine through the drinking water containing 50 ppm fluorine and 24 rats drinking low-fluorine water served as control are studied on the DNA, RNA contents of brain.The results showed that in the test rats as compared with the control rats, the fluorine content of urine was higher; the brains were smaller and lighter; the fluorine contents of brain were increased; the RNA contents of brain were decreased, while the DNA not changed. The findings suggest that(1) excessive fluorine in blood of the rats with chronic fluorosis can be transported through the blood-brain barrier into brain tissue,(2) excessive accumulation of fluorine in brain tissue can decrease the content and weight of brain by inhibiting RNA and protein synthesis, but can not influence the DNA content of brain.

研究了31只长期饲以含氟50ppm自来水而造成慢性氟中毒的Wistar大鼠大脑DNA、RNA含量的变化,以24只饲以正常饮食的大鼠作正常对照。结果显示:慢性氟中毒大鼠尿氟含量升高,出现氟斑牙,大脑组织氟含量增多,大脑重量减轻,大脑RNA含量降低、而DNA含量无明显改变。说明大脑氟量过多可抑制RNA合成,使其含量降低,并影响蛋白质合成,致大脑重量减轻。

 
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