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   oral dysplasia 的翻译结果: 查询用时:0.18秒
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口腔科学
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oral dysplasia
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  异常增生
     bcl-2 was rarely expressed in negative control,and there was up-regulation in oral dysplasia and OSCC(χ2=19.433,P<0.05).
     bcl-2在阴性对照组黏膜多不表达,随上皮异常增生程度加重表达上调,重度异常增生时达最高峰,鳞癌组表达降低,与异常增生组显著差异,而与阴性对照组相比,差异有显著性(χ2=19.433,P<0.05)。
短句来源
     Oral dysplasia and SCC M demonstrated higher Bcl-2 positive rate than the other lesions (P<0. 05).
     口腔粘膜异常增生Bcl-2蛋白阳性表达率显著高于口腔粘膜单纯增生和鳞癌Ⅰ、Ⅱ级(P<0.05),鳞癌Ⅲ级Bcl-2蛋白阳性表达率显著高于鳞癌Ⅰ级(P<0.05)。
短句来源
     Methods: Immunohistochemical staining with SP methods was used to detect theexpression of PTEN, PIP3, MAPK, Cyclin Dl and CDK4 in 63 OSCC cases, 11 oral dysplasia cases, 11 oral simple hyperplasia cases and 12 normal oral mucosa cases. Statistic and correlation analyses were applied to analyze the results.
     方法:应用免疫组织化学SP法检测63例OSCC,29例单纯增生,11例异常增生以及12例正常口腔黏膜中PTEN、PIP3、MAPK、Cyclin D1、CDK4蛋白的表达水平,对结果进行统计和相关性分析。
短句来源
     Conclusion: 1) PsD-007 mediated Krypton Laser PDT is effective for the oral dysplasia of hamster.
     结论:1)氪激光匹配静脉型光敏剂血啉甲醚(PsD- 007)的PDT作用于金黄地鼠颊囊粘膜异常增生具有良好的效果。
     Objective:To study the involvement of Fas. FasL and Bcl-2 protein in oral dysplasia epithelia and carcinomas.
     目的:研究凋亡相关基因Fas、FasL及Bcl-2蛋白在口腔黏膜异常增生上皮及鳞癌组织中变化规律。
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  口腔异常增生
     The study of apoptosis, PCNA, Bcl-2 and p53 in oral dysplasia epithelia and carcinomas
     口腔异常增生上皮及鳞癌组织细胞凋亡和增殖的研究
短句来源
     The clinical evaluation of the expression of the PCNA and cell nuclear area in the different layers of oral dysplasia epithelium
     口腔异常增生上皮PCNA及细胞核面积分层计数的临床意义
短句来源
     Objective To study the involvement of apoptosis, PCNA, Bcl-2 and p53 gene expression in oral dysplasia epithelia and SCC.
     目的 了解细胞凋亡、增殖及p53、Bcl-2在口腔异常增生上皮及鳞癌组织中的变化规律。
短句来源
  “oral dysplasia”译为未确定词的双语例句
     2) The dose of 10mg/kg PsD-007 could obtained effective result in Krypton Laser PDT for oral dysplasia of hamster.
     2)10mg/kg 浓度的PsD-007剂量已达到氪激光PDT 有效处理异常增生的初始剂量。
     2) The dose of 10mg/kg PsD-007 could obtained effective result in Krypton Laser PDT for oral dysplasia of hamster.
     2)10mg/kg 浓度的PsD-007剂量已达到氪激光PDT有效处理异常增生的初始剂量。
     The Expression of Bcl-2,Fas Protein in Oral Dysplasia and SCC
     口腔粘膜癌前病变和鳞癌组织凋亡相关蛋白Bcl-2和Fas的表达
短句来源
     Use of allelic loss to predict malignant risk for early oral dysplasia
     口腔黏膜早期不典型性增生等位基因缺失的检测
短句来源
     Objective To explore Chinese medicine herbs for break or turn oral dysplasia and to evaluate effect about the prevention and cure of oral leukoplakia with PSP.
     目的 探寻能阻断或逆转口腔上皮异常增生的中草药 ,评价PSP对白斑癌变的防治效果。
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  oral dysplasia
These findings taken together suggest that BM collagen-IV α chains undergo remodeling where selective increase and loss of these molecules are probably early and late events, respectively, during progression of oral dysplasia to cancer.
      
We addressed this question with FDG and TYR in an oral dysplasia animal model.
      
It appeared that, for the detection of oral dysplasia, the tissue hyperplasia was more important than malig nant features of dysplastic mucosa.
      
Furthermore, we are embarking upon another study focusing on patients with oral dysplasia.
      
Eighty-nine patients with a prior history of oral cancer being followed prospectively in the Oral Dysplasia Clinic were used.
      
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To investigate the role of Bcl-2 protein in the carcinogenesis of human oral squamous cell carcinoma(SCC). Methods: The expression of Bcl-2 protein in 16 cases of simple hyperplasia, 23 of dysplasia and 46 cases of SCC was examined with immunohistochemistry technic. The health tissues of oral mucosa were used the control. Results:None of normal controls showed Bcl-2 positive staining. The Bcl-2 positive rates in simple hyperplasia,dysplasia,SCC Ⅰ,SCC Ⅱ and SCC Ⅲ were 18. 75 % (3/6),...

To investigate the role of Bcl-2 protein in the carcinogenesis of human oral squamous cell carcinoma(SCC). Methods: The expression of Bcl-2 protein in 16 cases of simple hyperplasia, 23 of dysplasia and 46 cases of SCC was examined with immunohistochemistry technic. The health tissues of oral mucosa were used the control. Results:None of normal controls showed Bcl-2 positive staining. The Bcl-2 positive rates in simple hyperplasia,dysplasia,SCC Ⅰ,SCC Ⅱ and SCC Ⅲ were 18. 75 % (3/6), 52. 17 % (12/23), 18. 18% (4/22), 22. 22 % (2/9) and 53. 33 % (8/15) respectively. Oral dysplasia and SCC M demonstrated higher Bcl-2 positive rate than the other lesions (P<0. 05). Conclusion: Bcl-2 protein expression may be as- sociated with the differentiation status of oral mucosa lesions. SCCs with high expression of Bcl-2 may be highly aggressive.

目的:研究Bcl-2蛋白表达在口腔鳞状细胞癌发生发展过程中的作用及可能的临床意义。方法:对85例病变标本采用鼠抗人Bcl-2蛋白单克隆抗体进行免疫组织化学染色。结果:正常口腔粘膜Bcl-2免疫组化染色均为阴性,口腔上皮单纯增生、异常增生、鳞癌Ⅰ、Ⅱ、Ⅲ级Bcl-2蛋白阳性表达率分别为18.75%、52.17%、18.18%、22.22%和53.33%。口腔粘膜异常增生Bcl-2蛋白阳性表达率显著高于口腔粘膜单纯增生和鳞癌Ⅰ、Ⅱ级(P<0.05),鳞癌Ⅲ级Bcl-2蛋白阳性表达率显著高于鳞癌Ⅰ级(P<0.05)。结论:Bcl-2蛋白表达与组织分化程度相关,Bcl-2阳性表达的口腔鳞癌具有更高的侵袭能力。

Objective To evaluate the malignant potential of oral leukoplakia(OLK)through the expression of p53 protein、p16 protein and PCNA in OLK.Methods With immunohistochemistry technic.Results None of normal controls showed positive p53 protein staining,but all of them were stained positive for p16 protein and PCNA.Expression rate of p53 protein was significantly higher in dysplasia OLK than in hyperplasis OLK (58.16%, 12.50%, P <0 05).There was a significant difference in p53 positive index between dysplasia...

Objective To evaluate the malignant potential of oral leukoplakia(OLK)through the expression of p53 protein、p16 protein and PCNA in OLK.Methods With immunohistochemistry technic.Results None of normal controls showed positive p53 protein staining,but all of them were stained positive for p16 protein and PCNA.Expression rate of p53 protein was significantly higher in dysplasia OLK than in hyperplasis OLK (58.16%, 12.50%, P <0 05).There was a significant difference in p53 positive index between dysplasia OLK and oral squamous cell carcinoma ( P <0 05).Additionally,It was found that oral dysplasia OLK mainly exhibited the superbasal positive PCNA staining pattern.This significantly differed from the basal positive PCNA staining pattern in most of the oral hyperplasia OLK ( P <0 01).PCNA positive index in dysplasia OLK was significantly higher than in simple hyperplasia OLK ( P <0 01).Conclusions Expression of p53 protein plays an important role in the process in which hyperplasia transforms to dysplasia.PCNA positive index may well indicate cell proliferation activity of oral mucosa lesions.Superbasal positive pattern of PCNA staining is a good indicator for dysplasia in OLK.Expression rate of p53 protein、PCNA positive index and PCNA staining pattern can serve as useful indicators to assess the malignent potential of OLK and select out the most dangerous OLK.This can offer grounds for different clinical treatments.

目的 利用p5 3蛋白、p16蛋白及增殖细胞核抗原 (PCNA)在口腔粘膜白斑 (OralLeukoplakia ,OLK)中的表达 ,评估OLK的恶变潜能。方法 免疫组化技术。结果 正常口腔粘膜p5 3蛋白表达阴性 ,p16、PCNA表达阳性。口腔不典型增生性白斑 (dysplasialeukoplakia ,DLK)p5 3阳性表达率 (5 8 16 % )显著高于单纯增生性白斑 (simplehyperplasialeukoplakia ,SHLK) (12 5 0 % ,P <0 0 5 )。口腔DLK与口腔鳞癌组间p5 3蛋白阳性指数差异有显著性 (P <0 0 5 )。PCNA在口腔DLK中表达分布主要为基底上层模式 ,与正常口腔粘膜及SHLK的基底层分布模式间差异有显著性 (P <0 0 1) ,其PCNA表达阳性指数显著较SHLK为高 (P <0 0 1)。结论 p5 3蛋白表达在OLK由单纯增生向不典型增生转化过程中起主要作用。PCNA阳性指数能较好的反映口腔粘膜病变的增殖水平。PCNA基底上层表达模式能较好反映口腔粘膜白斑的不典型增生。通过对这些基因产物检测 ,可评价OLK的恶变...

目的 利用p5 3蛋白、p16蛋白及增殖细胞核抗原 (PCNA)在口腔粘膜白斑 (OralLeukoplakia ,OLK)中的表达 ,评估OLK的恶变潜能。方法 免疫组化技术。结果 正常口腔粘膜p5 3蛋白表达阴性 ,p16、PCNA表达阳性。口腔不典型增生性白斑 (dysplasialeukoplakia ,DLK)p5 3阳性表达率 (5 8 16 % )显著高于单纯增生性白斑 (simplehyperplasialeukoplakia ,SHLK) (12 5 0 % ,P <0 0 5 )。口腔DLK与口腔鳞癌组间p5 3蛋白阳性指数差异有显著性 (P <0 0 5 )。PCNA在口腔DLK中表达分布主要为基底上层模式 ,与正常口腔粘膜及SHLK的基底层分布模式间差异有显著性 (P <0 0 1) ,其PCNA表达阳性指数显著较SHLK为高 (P <0 0 1)。结论 p5 3蛋白表达在OLK由单纯增生向不典型增生转化过程中起主要作用。PCNA阳性指数能较好的反映口腔粘膜病变的增殖水平。PCNA基底上层表达模式能较好反映口腔粘膜白斑的不典型增生。通过对这些基因产物检测 ,可评价OLK的恶变潜能 ,筛选高危白斑 ,为临床治疗提供依据。

Aim:To investigate the relationship between expression of P 21wafl and genesis of oral squamous cell carcinoma Methods:A retrospective study on paraffin-embedded specimens with immunohistochemical technique.Results:P 21wafl -positive rate in oral simple hyperplasia was 75%(12/16)which was significantly higher than in oral dysplasia(26.09%,6/23)(p<0 05) and in oral squamous cell carcinoma(28.26%,13/46)(p<0 05).P 21wafl -positive cell counting was positively associated with the malignancy...

Aim:To investigate the relationship between expression of P 21wafl and genesis of oral squamous cell carcinoma Methods:A retrospective study on paraffin-embedded specimens with immunohistochemical technique.Results:P 21wafl -positive rate in oral simple hyperplasia was 75%(12/16)which was significantly higher than in oral dysplasia(26.09%,6/23)(p<0 05) and in oral squamous cell carcinoma(28.26%,13/46)(p<0 05).P 21wafl -positive cell counting was positively associated with the malignancy progressior of oral mucosa lesions.Conclusion:The down-regulation of P 21wafl expression is an early event in carcinogenesis of and mucosa,P 21wafl -positive rate oral P 21wafl -positive cell counting combined can serve as a indicator for malignant transition of oral precancerous lesions. [WT5”FZ]

目的 :了解P2 1wafl表达与口腔鳞状细胞癌发生发展的关系。方法 :利用免疫组化技术 ,进行石蜡切片回顾性研究。结果 :P2 1wafl蛋白在口腔上皮单纯增生的表达率为 75 % (12 / 16 ) ,显著高于口腔不典型增生 (2 6 0 9% ,6 / 2 3) ((p <0 0 5 )和鳞状细胞癌 (2 8 2 6 % ,13/ 46 ) (p<0 0 5 ) ;P2 1wafl阳性标本的阳性细胞计数与组织恶性程度成正相关。结论 :P2 1wafl表达的下调是口腔鳞癌发生发展过程中较早期事件 ,P2 1wafl表达率结合P2 1wafl阳性细胞计数在口腔粘膜癌前病变监视方面是一项有用的指标。

 
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