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hypoxic ischemic
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  缺氧缺血
     Conclusion:① the ratios of NAA/Cho,Lac/NAA,Lac/Cr,Lac/Cho,Lac/ (Cr+ Cho) were the most sensitive indicators of HIE,they quantitatively reflected the biochemical alterations of cerebral hypoxic ischemic injury.
     结论:①NAA/Cho、Lac/NAA、Lac/Cr、Lac/Cho和Lac/(Cr+Cho)是定量反映脑缺氧缺血生化改变的最敏感指标;
短句来源
     Group C7 was scanned at 0,1, 3, 6, 12, 18, 24 hours after hypoxic ischemic, and thedata was used to study the dynamic changes of MRI appearances in HIE.
     C_7组分别在缺氧缺血后0、1、3、6、12、18、24小时行MRI扫描得到7组连续性MRI资料(用于缺血灶影像学动态变化研究)。
短句来源
     Objective To study the effects of 17β estradiol (17β E 2) on extracellular glutamate(Glu) level post hypoxic ischemic brain damage(HIBD) in neonatal rats and to explore whether 17β E 2 has some protective effects on HIBD.
     目的 研究 17β 雌二醇 (17β E2 )对新生大鼠缺氧缺血脑损伤 (HIBD)后谷氨酸 (Glu)释放的影响 ,探讨 17β E2 是否对HIBD有保护作用。
短句来源
     Methods Seven day old Sprague Dawley (SD) rats were randomly divided into 3 groups:sham operated group ( n = 12), hypoxic ischemic brain damage (HIBD) group ( n = 15), BC group ( n = 13).
     方法 新生 7日龄SD大鼠随机分为假手术组 (n =12 )、缺氧缺血组 (HIBD组 ,n =15 )、黄芩苷治疗组 (BC组 ,n =13 )。
短句来源
     [Conclusion] Glutathione inhibits effectively neuronal cell apoptosis of hippocampal CA1 in hypoxic ischemic brain damage.
     【结论】 谷胱甘肽可有效抑制缺氧缺血损伤引起的海马部位神经细胞凋亡 ,可有效对抗氧化张力在缺氧缺血损伤中引起的细胞凋亡。
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  缺氧缺血性
     Expression of NgR mRNA and NgR Protein in Brain Tissues of Newborn Rats with Hypoxic Ischemic Brain Damage
     新生鼠缺氧缺血性脑损伤脑组织NgR mRNA和NgR蛋白表达及意义
短句来源
     Results TCa and Ca 2+ i levels of hypoxic ischemic group were much higher than those of normal control group ( P <0.01). TCa and Ca 2+ i levels of MK 801 group were much lower than those of hypoxic ischemic group ( P <0.01).
     结果缺氧缺血性脑损伤组TCa、Ca2+i明显高于正常对照组(P<0.01),MK801治疗组TCa、Ca2+i明显低于缺氧缺血性脑损伤组(P<0.01)。
短句来源
     AIM: To study the expression of HSP70 and pathologic damage in hippocampal CA 1 after hypoxic ischemic brain damage(HIBD)in neonatal rats and the effect of monosialotetrahexosylganglioside(GM 1)on it .
     目的 :研究新生大鼠缺氧缺血性脑损伤 (HIBD)后海马CA1区 70ku热休克蛋白 (HSP70 )表达、病理学损伤变化和外源性单唾液酸四已唐神经节苷脂 (GM1)对HSP70表达及病理学损伤的影响 .
短句来源
     Protective effects of the MK801 on the hypoxic ischemic brain injury
     MK801对缺氧缺血性脑损伤的保护作用
短句来源
     A Dynamic Observation on Interleukin 1 β Gene Transcription in Neonate Rats with Hypoxic Ischemic Brain Damage
     新生大鼠缺氧缺血性脑损伤后白细胞介素-1β基因转录的动态观察
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  “hypoxic ischemic”译为未确定词的双语例句
     Application of Large Dosage of Salvia Miltiorrhiza Compositae in Treating 23 Patients of Newborn Hypoxic Ischemic Encephalopathy
     Application of Large Dosage of Salvia Miltiorrhiza Compositae in Treating 23 Patients of Newborn Hypoxic Ischemic Encephalopathy
短句来源
     Effect of hyperbaric oxygenation on plasma levels of ET, CGRP, 6 keto PGF 1α and TXB 2 in neonates with hypoxic ischemic encephalopathy
     高压氧对新生儿缺氧缺血性脑病血浆ET、CGRP、6-keto-PGF_(1α)及TXB_2浓度的影响
短句来源
     Detection and significance of caspase-1 and IL-1β in serum of newborns with hypoxic ischemic encephalopathy
     新生儿缺氧缺血性脑病血清caspase-1和IL-1β测定及意义
短句来源
     ① The neuron-specific enolase and interleukin-6 in serum were obviously higher in the hypoxic ischemic encephalopathy group than in the control group [(23.904±8.432), (7.292 ±2.940) μg/L, P < 0.05; (180.269±78.570), (75.912±19.160) ng/L, P < 0.05].
     ①新生儿缺氧缺血性脑病组血清神经元特异性烯醇化酶及细胞介素6明显高于正常对照组(23.904±8.432,7.292±2.940μg/L,P<0.05); (180.269±78.570,75.912±19.160ng/L,P<0.05)。
短句来源
     Detection and Significance of NSE、IL-6 in Serum of Newborns with Hypoxic Ischemic Encephalopathy
     新生儿缺氧缺血性脑病血清NSE、IL-6测定及意义
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  hypoxic ischemic
In particular, we identified the peak representing the neurons and astrocytes and showed that they maintain different basal pH (6.95 and 7.05, respectively) and behave differently during hypoxic/ischemic episodes.
      
The hypoxic-induced elevation of these two neurotransmitters during normoxia may be important in the production of hypoxic/ischemic-induced cell damage.
      
Normal brains of four term or near-term neonates and two with hypoxic/ischemic lesions were also studied.
      
These include bronchopulmonary dysplasia retinopathy of prematurity necrotising enterocoltics patient ductus arteriosus and neuronal injury of hypoxic ischemic encephalopathy.
      
Levene's classification is a useful clinical tool for grading the severity of hypoxic ischemic encephalopathy.
      
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Severe neonatal acidosis (pH 7 or less) occurred in 103 cases (1.9%)among 6,509 cases admitted from 1978 to 1985. Of these cases, 50 were term babies, 48 preterm, 3 post-term and 2 small-for-date. There were 20 primary diseases causing severe neonatal acidosis, They were respiratory tract diseases, including meconium aspiration, pneumonia. respiratory distress syndrome and pulmonary dysplasia (53 cases, 51.5%); septicemia (18 cases, 17.5%); gastrointestinal disorders, including diarrhea, necrotizing enterocolitis,...

Severe neonatal acidosis (pH 7 or less) occurred in 103 cases (1.9%)among 6,509 cases admitted from 1978 to 1985. Of these cases, 50 were term babies, 48 preterm, 3 post-term and 2 small-for-date. There were 20 primary diseases causing severe neonatal acidosis, They were respiratory tract diseases, including meconium aspiration, pneumonia. respiratory distress syndrome and pulmonary dysplasia (53 cases, 51.5%); septicemia (18 cases, 17.5%); gastrointestinal disorders, including diarrhea, necrotizing enterocolitis, perforation of stomach and intestinal obstruction (14 cases. 13.5%) and others as hypoxic ischemic encephalopathy and/or intracranial hemorrhage, congenital cynotic heart disease, sclerema neonatorium and late acidosis in pretcrm babies (18 cases, 17.5%). The major symptoms were respiratory distress (90.3%), apnca (48.5%), cynosis (86.4%), grey face (78.6%) and hypothermia (77.7%). The mortality was high (73.8%), especially in those babies with respiratory tract diseases (84.9%) or in babies less than 7 days of age (87.5%). 16 of the lethal cases were complicated with massive pulmonary hemorrhage. The prognosis was grave for both term and preterm babies.

1979~1985年住院新生儿患者中血pH≤7.0有103例,占同期住院新生儿1.9%。导致重度酸中毒的原发疾病有20种,以呼吸系统疾病占首位(51.5%),败血症第二位(17.5%),消化系疾病第三位(13.6%)。治愈好转率仅24.3%,呼吸系统疾病,败血症,缺血缺氧性脑病,颅内出血,先天性青紫型心脏病预后差。而消化系统疾病预后较好,早产儿晚期酸中毒预后好。生后一周内发病率与病死率均高,血PCO_2升高时预后亦差。对新生儿重度酸中毒的防治问题进行了讨论。

Ultrasonographic scans were performed in 115 newborn babies with Apgar score≤7 at 1 minute after birth. The results were normal in 48 cases (41.7%); hypoxic-ischemic encephalopathy (HIE) in 36 (31.3%); intracranial hemorrhage (ICH) in 26 (22.6%) and ventriculomegaly in 5 (4.4%). The normal rate of ultrasonographic scans was 82.9% in those with low Apgar score but without clinical symptoms and signs. However the abnormal rate was 81.1% in those having clinical symptoms and signs, either irritable...

Ultrasonographic scans were performed in 115 newborn babies with Apgar score≤7 at 1 minute after birth. The results were normal in 48 cases (41.7%); hypoxic-ischemic encephalopathy (HIE) in 36 (31.3%); intracranial hemorrhage (ICH) in 26 (22.6%) and ventriculomegaly in 5 (4.4%). The normal rate of ultrasonographic scans was 82.9% in those with low Apgar score but without clinical symptoms and signs. However the abnormal rate was 81.1% in those having clinical symptoms and signs, either irritable and hyperalert appearance or apathetic and hypotonic state. The ventricles were often enlarged following HIE and ICH. Among 26 cases, 14 had mild to moderate enlargement while 3 were progressively dilated. The babies with blood pH<7.25 and/or PCO_2>6.7 kPa were more common in ICH group. Therefore it is important to correct the acid-base imbalance, especially in respiratory acidosis.

对115例出生时Apgar评分≤7分的新生儿进行B型超声检查。结果有48例超声检查正常(41.7%),缺氧缺血性脑病36例(31.3%),颅内出血26例(22.6%)。脑室扩大5例(4.4%)。Apgar评分低不伴有神经系统症状、体征者超声检查正常率为82.9%,而伴神经系统症状、体征者81.9%患儿超声检查发现异常。超声随访发现缺氧缺血性脑病、颅内出血者脑室扩大者多:轻到中度脑室扩大53.8%(14/26),进行性脑室扩大11.5%(3/16)。对72例临床及实验室资料回顾性分析发现,pH<7.25,PCO_2>6.7kPa,颅内出血组明显高于正常组,因此在抢救窒息新生儿时,加强呼吸管理及维持内环境稳定很重要。

The animal model of postnatal cerebral hypoxia-ischemia was prepared in 7 days old rats by permanent ligation of right common carotid artery combined with a temporary systemic hypoxia at 37℃. The early effects of hypoxia-ischemia on the striatal cholinergic system were investigated 24 h after lesion by ACh RIA-determination and AChE-histochemical image analysis. The striatal ACh content and average optical density of the AChE-positive cell bodies declined by 28 % and 12% respectively in the hypoxic-ischemic...

The animal model of postnatal cerebral hypoxia-ischemia was prepared in 7 days old rats by permanent ligation of right common carotid artery combined with a temporary systemic hypoxia at 37℃. The early effects of hypoxia-ischemia on the striatal cholinergic system were investigated 24 h after lesion by ACh RIA-determination and AChE-histochemical image analysis. The striatal ACh content and average optical density of the AChE-positive cell bodies declined by 28 % and 12% respectively in the hypoxic-ischemic group as compared with the normal control group (p<0.01, p<0.05). There was no difference in the values between the ligated and non-ligated sides in the hypoxic-ischemic group. The striatal ACh content in the animals of the hypoxic group, which was prepared by temporary exposure to hypoxia at 37℃ with the carotid arteries intact, was also found to be reduced to the same extent as in the hypoxic-ischemic group. The density of AChE-positive cell profiles in the caudate-putamen remained unchanged on both sides in the hypoxic-ischemic group. The results indicate that hypoxia-ischemia, at its early phase, brings about cell damage rather than cell death and, as a result, leads to the weakening of the neuron markers, ACh and AChE in this case of the striatal cholinergic system of the young rat. It is suggested that developing cholinergic system is susceptible to hypoxia-ischemia, probably with anoxia as the primary pathogeny.

采用7日龄大鼠右侧颈总动脉结扎合并高温、低氧环境制作新生动物脑低氧缺血模型,观察了脑低氧缺血对新生动物纹状体胆碱能系统的影响。乙酰胆碱(ACh)放射免疫测定结果表明,低氧缺血损伤后24h,两侧纹状体ACh含量均比正常对照组明显下降。乙酰胆碱酯酶(AChE)组织化学图象定量提示,脑低氧缺血后24h,纹状体内拟胆碱能神经元数量未见减少,而胞体内AChE染色强度略有下降。胆碱能递质和该标志酶在新生鼠脑低氧缺血早期的一致改变,证明发育中纹状体胆碱能系统对低氧缺血敏感。鉴于动物不结扎动脉仅作低氧处理者双侧纹状体ACh含量出现与低氧缺血组相同的改变,故提示缺氧可能是造成胆碱能系统早期损伤的直接原因。

 
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