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ischemic brain injury
相关语句
  缺血性脑损伤
    Study on the Changes of Transmitter Amino Acids in Hippocampi and Cortex of Ischemic Brain Injury Rats
    缺血性脑损伤大鼠海马皮层递质氨基酸变化的研究
短句来源
    C-FOS GENE EXPRESSION AND MEDICINAL REGULATION IN HYPOTHALAMUS OF RAT FOLLOWING ISCHEMIC BRAIN INJURY
    缺血性脑损伤大鼠下丘脑c-fos基因表达及药物调控
短句来源
    Mitochondria calcium overload and ischemic brain injury
    线粒体Ca~(2+)超载与缺血性脑损伤
短句来源
    Objective To study the pathogenic effect of platelet activating factor (PAF) on ischemic brain injury.
    目的研究血小板激活因子(platelet-activatingfactor,PAF)在缺血性脑损伤中的致病作用。
短句来源
    Conclusion: C-fos gene expression in hypothalamus increases after ischemic brain injury and Shenmai injection can cure the ischmic brain injury in a way.
    结论 :缺血性脑损伤时下丘脑中c -fos基因表达增多 ,而参麦注射液对缺血性脑损伤可能有一定治疗作用。
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  ischemic brain injury
Inhibition of neurotransmitter release may, in part, be responsible for the protective effect of pentobarbital in ischemic brain injury.
      
This study demonstrated that bradykinin postconditioning induces protection against ischemic brain injury and promotes neuronal survival.
      
Plasticity of Neurons and Glia Following Neonatal Hypoxic-Ischemic Brain Injury in Rats
      
We propose the monitoring of serine, asparagine, and threonine, together with excitatory amino acids, as an index of the degree of ischemic brain injury.
      
Research on ischemic brain injury has established a central role of mitochondria in neuron death (1-3).
      
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Objective This experiment was designed to establish a reversed phase HPLC method for determination of the levels of transmitter amino acids and to study on the changes of transmitter amino acids in hippocampi and cortex of ischemic brain injury rats. Method Prior to reversed phase HPLC analysis with fluorescence detection, the transmitter amino acids such as GABA、Asp、Glu、Tau、Gly in hippocampi and cortex of rats were pre colume derivatized with o phthalaldephde β mercaptoethanol. Result The method...

Objective This experiment was designed to establish a reversed phase HPLC method for determination of the levels of transmitter amino acids and to study on the changes of transmitter amino acids in hippocampi and cortex of ischemic brain injury rats. Method Prior to reversed phase HPLC analysis with fluorescence detection, the transmitter amino acids such as GABA、Asp、Glu、Tau、Gly in hippocampi and cortex of rats were pre colume derivatized with o phthalaldephde β mercaptoethanol. Result The method showed good linearity in the range of 50 pmol~2 nmol/L with r=0.9997. The average recoveries of the transmitter amino acids were between 87.75% and 108.53%. The coefficient of variation of peak areas was less than 4.73%. The levels of exciting amino acids in hippocampi and cortex of ischemic brain injury rats were significant higher than those of controls. Conclusions The method is simple, rapid, reproducible and very suitable for detection of the levels of transmission amino acids in hippocampi and cortex of rats. It provided a rapid analytical means for studying the mechanism of dysfunction of ischemic brain injury and the pharmacological research of traditional Chinese medicines for keeping the brain healthy. The studies on the changes of transmitter amino acids in hippocampi and cortex of ischemic brain injury rats provided some meaning for clues in making the mechanism of MID public from the point of neurobiochemistry.

目的 建立高效液相色谱分析法 ,研究缺血性脑损伤大鼠海马皮层递质氨基酸的变化。方法 用邻苯二甲醛 β 巯基乙醇柱前衍生 ,反相程序洗脱 ,荧光检测分析实验动物脑组织中的海马、皮层递质氨基酸即氨基丁酸 (GABA)、天门冬氨酸 (Asp)、谷氨酸 (Glu)、牛磺酸 (Tau)、甘氨酸 (Gly)的含量。 结果 该方法在 5 0 pmol~ 2nmol/L范围内具有良好的线性 ,线性相关系数平均为 0 .9997,平均回收率在 87.75 %~ 10 8 5 3 %之间 ,峰面积变异系数小于 4 73 %。缺血脑损伤大鼠皮层海马脑区的兴奋性氨基酸明显高于正常对照组。结论 为目前缺血性脑损伤功能障碍机理及健脑中药的药理研究提供了快速分析手段。为从神经生化角度揭示多发梗塞性痴呆的发生机制提供了有意义的线索和依据。

Objective: To explore the patho-physiologic machanism and the protective role of Shenmai injection following ischemic brain injury at the molecular level, and to provide some theoretical guidance for clinical pathologic study and treatment. Methods: 15 male Wistar rats were divided into control group, ischemia group and treatment group at random. C-fos gene expression and the protective role of Shenmai injection were studied in hypothalamic supraoptical nucleus(SON) and paraventricular nucleus(PVN) following...

Objective: To explore the patho-physiologic machanism and the protective role of Shenmai injection following ischemic brain injury at the molecular level, and to provide some theoretical guidance for clinical pathologic study and treatment. Methods: 15 male Wistar rats were divided into control group, ischemia group and treatment group at random. C-fos gene expression and the protective role of Shenmai injection were studied in hypothalamic supraoptical nucleus(SON) and paraventricular nucleus(PVN) following ischemic brain injury with immunohistochemical techniques, Results: In all rats following ischemic brain injury, c-fos gene expression was increased obviously in SON and PVN compared with normal rats, and it was decreased with the application of shenmai injection, but still higher than normal c-fos gene expression in hypothalamus. Conclusion: C-fos gene expression in hypothalamus increases after ischemic brain injury and Shenmai injection can cure the ischmic brain injury in a way.

目的 :从分子水平探讨缺血性脑损伤的病理生理改变和参麦注射液的保护作用。方法 :将 15只雄性wistar大鼠随机分为对照组、缺血组和治疗组 ,采用免疫组织化学方法观察了缺血性脑损伤时大鼠下丘脑视上核和室旁核内神经元c -fos基因的表达及参麦注射液的保护作用。结果 :缺血性脑损伤时大鼠视上核和室旁核内神经元c -fos基因的表达明显增多 (P <0 .0 5 ) ,而用参麦注射液后明显减少 ,但未减少至正常水平 (P <0 .0 5 )。结论 :缺血性脑损伤时下丘脑中c -fos基因表达增多 ,而参麦注射液对缺血性脑损伤可能有一定治疗作用。

Objective To investigate the influence of ischemic preconditioning on ischemic brain injury caused by subsequent middle cerebral artery occlusion (MCAO)in SD rats.Methods Male Sprage Dawley rats were divided three group.The first group(SI): Animals were only injected with saline water into right internal carotid artery ;The second group (BCAO):Animals were only treated by the method of both carotid artery occlusion;The third group(BS):Animals were given simultaneously two methods mentioned above.All...

Objective To investigate the influence of ischemic preconditioning on ischemic brain injury caused by subsequent middle cerebral artery occlusion (MCAO)in SD rats.Methods Male Sprage Dawley rats were divided three group.The first group(SI): Animals were only injected with saline water into right internal carotid artery ;The second group (BCAO):Animals were only treated by the method of both carotid artery occlusion;The third group(BS):Animals were given simultaneously two methods mentioned above.All experimental animals were treated three repeated procedures,3?min every time in an interval of 7?min.After 24?h,focal cerebral ischemia was made using the method of thread insertion for middle cerebral artery occlusion(MCAO).Brain water content was measured by dry wet method.Blood brain barrier permeability was determined with brain Evans blue (EB) content.Infarction size were measured by TTC staining.Results BS pretreatment decreased water content (P<0.01) and EB content (P<0.01)significantly 24?h and 48?h after MCAO.Cerebral infarction was decreased 72?h after MCAO in BS group as compared with other two groups (P<0.05).Conclusion BS pretreatment has protective effect on ischemic cerebral injury.

目的 观察缺血预处理对再次脑缺血损伤的保护作用。方法 将SD大鼠分为 3组 ,分别给予生理盐水右侧颈内动脉灌注 (SI)、双侧颈总动脉夹闭 (BCAO)和双侧颈总动脉夹闭并生理盐水右侧颈内动脉灌注 (BS)。每次持续 3min ,间隔 7min ,反复 3次。 2 4h后作经插线右大脑中动脉栓塞 (MCAO) ,观察MCAO后脑组织含水量、血脑屏障通透性 (以脑伊文思蓝含量表示 )和梗死体积。结果 BS组MCAO后 2 4h和 48h脑伊文思蓝 (EB)含量和脑含水量明显较其他两组低 (P <0 .0 1) ;MCAO 72hBS组脑梗死低于其它两组 (P <0 .0 5 )。结论 双侧颈总动脉夹闭并生理盐水右侧颈内动脉灌注对脑再次缺血有明显的保护作用。

 
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