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ischemic brain injury
相关语句
  缺血性脑损伤的
    With the progress of molecular biology, recently it is found that the ischemic cascade reaction, called“ischemic cascade”, including ischemic-hypoxic energy metabolic dysfuntion,release of excitatory neuromediator, calcium over inflow, free radical reaction, cell apoptosis and so on, makes the central part of ischemic brain injury.
    随着分子生物学的发展,近年发现缺血、缺氧造成的能量代谢障碍—兴奋性神经介质释放—钙过量内流—自由基反应—细胞死亡等一系列缺血性连锁反应是导致缺血性脑损伤的中心环节,称之为缺血瀑布。 围绕这一理论,通过药物或其他手段干预阻断瀑布反应链中不同机制、不同环节的生化和代谢紊乱被称为神经保护治疗。
短句来源
    This suggests that intensifying the synthesis and release of ET may be one of the mechanisms for the involvement of monocytes in the pathologic process of ischemic brain injury during acute cerebral infarction. DXM can improve this process by inhibition of producing monocyte-derived ET. 
    提示:急性脑梗塞时,单核细胞增加ET的合成与释放可能是其参与缺血性脑损伤过程的机制之一,而DXM则抑制其合成与释放,具有改善缺血性脑损伤的作用
短句来源
    Objective To investigate the roles of peripheral blood mononuclear cells (PBMC) in the pathogenesis of ischemic brain injury.
    目的 探讨单个核细胞参与缺血性脑损伤的机制。
短句来源
    PBMC might be involved in the pathologic process of ischemic brain injury by secreting NO.
    单个核细胞可能通过分泌NO参与缺血性脑损伤的病理过程
短句来源
  血性脑损伤的
    With the progress of molecular biology, recently it is found that the ischemic cascade reaction, called“ischemic cascade”, including ischemic-hypoxic energy metabolic dysfuntion,release of excitatory neuromediator, calcium over inflow, free radical reaction, cell apoptosis and so on, makes the central part of ischemic brain injury.
    随着分子生物学的发展,近年发现缺血、缺氧造成的能量代谢障碍—兴奋性神经介质释放—钙过量内流—自由基反应—细胞死亡等一系列缺血性连锁反应是导致缺血性脑损伤的中心环节,称之为缺血瀑布。 围绕这一理论,通过药物或其他手段干预阻断瀑布反应链中不同机制、不同环节的生化和代谢紊乱被称为神经保护治疗。
短句来源
    This suggests that intensifying the synthesis and release of ET may be one of the mechanisms for the involvement of monocytes in the pathologic process of ischemic brain injury during acute cerebral infarction. DXM can improve this process by inhibition of producing monocyte-derived ET. 
    提示:急性脑梗塞时,单核细胞增加ET的合成与释放可能是其参与缺血性脑损伤过程的机制之一,而DXM则抑制其合成与释放,具有改善缺血性脑损伤的作用
短句来源
    Objective To investigate the roles of peripheral blood mononuclear cells (PBMC) in the pathogenesis of ischemic brain injury.
    目的 探讨单个核细胞参与缺血性脑损伤的机制。
短句来源
    PBMC might be involved in the pathologic process of ischemic brain injury by secreting NO.
    单个核细胞可能通过分泌NO参与缺血性脑损伤的病理过程
短句来源
  “ischemic brain injury”译为未确定词的双语例句
    To investigate the mechanism that monocytes participate in ischemic brain injury, endothelin (ET) levels in plasma, supernatants of cultured monocytes in vitro and those pretreated by dexamethason(DXM) were assayed by radioimmunoassay in 31 patients with acute cerebral infarction and 16 patients with hypertension (served as control).
    用放射免疫法测定31例急性脑梗塞患者和16例高血压患者血浆及体外诱生的单核细胞源性及地塞米松(DXM)干预后的内皮素(ET)水平。
短句来源
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  ischemic brain injury
Inhibition of neurotransmitter release may, in part, be responsible for the protective effect of pentobarbital in ischemic brain injury.
      
This study demonstrated that bradykinin postconditioning induces protection against ischemic brain injury and promotes neuronal survival.
      
Plasticity of Neurons and Glia Following Neonatal Hypoxic-Ischemic Brain Injury in Rats
      
We propose the monitoring of serine, asparagine, and threonine, together with excitatory amino acids, as an index of the degree of ischemic brain injury.
      
Research on ischemic brain injury has established a central role of mitochondria in neuron death (1-3).
      
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To investigate the mechanism that monocytes participate in ischemic brain injury, endothelin (ET) levels in plasma, supernatants of cultured monocytes in vitro and those pretreated by dexamethason(DXM) were assayed by radioimmunoassay in 31 patients with acute cerebral infarction and 16 patients with hypertension (served as control). Compared with the control group, the ET levels in supernatants of cultured monocytes increased and positively related with those in plasma and degree of neurological damage...

To investigate the mechanism that monocytes participate in ischemic brain injury, endothelin (ET) levels in plasma, supernatants of cultured monocytes in vitro and those pretreated by dexamethason(DXM) were assayed by radioimmunoassay in 31 patients with acute cerebral infarction and 16 patients with hypertension (served as control). Compared with the control group, the ET levels in supernatants of cultured monocytes increased and positively related with those in plasma and degree of neurological damage in the acute cerebral infarction group; while the ET levels in supernatants of cultured monocytes pretreated by DXM decreased. This suggests that intensifying the synthesis and release of ET may be one of the mechanisms for the involvement of monocytes in the pathologic process of ischemic brain injury during acute cerebral infarction. DXM can improve this process by inhibition of producing monocyte-derived ET.

用放射免疫法测定31例急性脑梗塞患者和16例高血压患者血浆及体外诱生的单核细胞源性及地塞米松(DXM)干预后的内皮素(ET)水平。结果发现:急性脑梗塞组体外诱生的单核细胞源性ET水平明显增高,并与血浆中ET水平及神经损伤程度正相关;DXM干预后,体外诱生的单核细胞源性ET水平降低。提示:急性脑梗塞时,单核细胞增加ET的合成与释放可能是其参与缺血性脑损伤过程的机制之一,而DXM则抑制其合成与释放,具有改善缺血性脑损伤的作用

Objective:To study the pathophysiologic role of glutamate (Glu) and adrenocorticotropic hormone (ACTH) in the stress response of central nervous system (CNS) in acute cerebral ischemia.Methods:Using highperformance liquid chromatography and radioimmunoassay,plasma and cerebrospinal fluid (CSF) Glu and ACTH concentrations were determined in 28 patients within 72 hours after acute cerebral infarction (ACI).Concomitantly,Canadian Stroke Scale (CSS) score and cerebral infarction volume (CIV) were also evaluated.Results:Plasma...

Objective:To study the pathophysiologic role of glutamate (Glu) and adrenocorticotropic hormone (ACTH) in the stress response of central nervous system (CNS) in acute cerebral ischemia.Methods:Using highperformance liquid chromatography and radioimmunoassay,plasma and cerebrospinal fluid (CSF) Glu and ACTH concentrations were determined in 28 patients within 72 hours after acute cerebral infarction (ACI).Concomitantly,Canadian Stroke Scale (CSS) score and cerebral infarction volume (CIV) were also evaluated.Results:Plasma and CSF Glu and ACTH concentrations in patients with ACI were enhanced significantly than control subjects 〔plasma Glu:(16929±3253)mmol/L vs.(12920±2815)mmol/L;plasma ACTH(1480±608)pmol/L vs.(858±081)pmol/L;CSF Glu:(1048±329)mmol/L vs.(309±148)mmol/L;CSF ACTH(388±129)pmol/L vs.(197±081)pmol/L, P<0001~005).Plasma or CSF Glu concentrations were positively correlated with ACTH levels (plasma:r=0493,P<001;CSF:r=0402,P<005).Conclusions:The excessive release of neuronal Glu may be involved in the pathogenesis of ACTH secretion during ACI.Glu concentrations in plasma or CSF may be a sensitive parameter to reflect ischemic brain injury,as well as to evaluate curative effect of Glu release inhibitor.

目的:探索谷氨酸(Glu)和促肾上腺皮质激素(ACTH)在急性脑缺血及其应激反应中的病理生理学意义。方法:应用高效液相色谱分析等方法检测了28例首次发病的急性脑梗死(ACI)患者发病72小时内的血浆和脑脊液(CSF)中Glu及ACTH浓度变化,并与同期检测的神经功能缺失程度(CSS积分)、CT测量脑梗死体积(CIV)进行相关分析;并另设同期住院的非心、脑血管病患者10例为对照组。结果:28例ACI患者的血浆Glu〔(169.29±32.53)mmol/L〕、ACTH〔(14.80±6.08)pmol/L〕和CSF中Glu〔(10.48±3.29)mmol/L〕、ACTH〔(3.88±1.29)pmol/L〕浓度均显著高于对照组〔血浆Glu(129.20±28.15)mmol/L,ACTH(8.58±0.81)pmol/L;CSF中Glu(3.09±1.48)mmol/L,ACTH(1.97±0.81)pmol/L,P<0.001~0.05〕;其血浆和CSF中Glu浓度分别与血浆和CSF中ACTH浓度呈非常显著或显著正相关(r分别为0.493和0.402,P<0.01和P<0.05)。结论:Glu参与了ACI?

Objective:To study the pattern of glutamate (Glu) elevation in patients with stable and progressive ischemic stroke.Methods:By highperformance liquid chromatography (HPLC),Glu concentrations both in plasma and cerebral spinal fluid (CSF) were measured in 128 patients with acute cerebral infarction (ACI) on days 1 to 7 after onset and 20 control subjects.At the same time,Canadian Stroke Scale (CSS) score and cerebral infarction volume (CTV) were also obtained and analyzed.Results:Mean plasma and CSF Glu concentrations...

Objective:To study the pattern of glutamate (Glu) elevation in patients with stable and progressive ischemic stroke.Methods:By highperformance liquid chromatography (HPLC),Glu concentrations both in plasma and cerebral spinal fluid (CSF) were measured in 128 patients with acute cerebral infarction (ACI) on days 1 to 7 after onset and 20 control subjects.At the same time,Canadian Stroke Scale (CSS) score and cerebral infarction volume (CTV) were also obtained and analyzed.Results:Mean plasma and CSF Glu concentrations in the patients significantly elevated compared with control subjects 〔plasma Glu:(136 29± 32 53)mmol/L vs.(129 20±28 15)mmol/L, P <0 05;CSF Glu:(6 48±3 29)mmol/L vs.(3 09± 1 48)mmol/L , P <0 01〕.Mean CSF Glu concentrations in 50 patients with progressive ischemic stroke significantly increased over that in 78 patients with stable ischemic stroke 〔(6 47±1 32)mmol/L vs.(5 32± 1 07)mmol/L , P <0 001〕.Mean Glu concentrations in plasma did not show significant changes in various time points during 7 days observation period in 78 patients with stable ischemic stroke,but it was markedly increased on day 4 after onset in 50 patients with progressive ischemic stroke.Moreover,mean CSF Glu concentrations in patients with CTV≥10 cm 3 were significantly higher than those with CTV<10 cm 3 〔(6 74± 1 37)mmol/L vs.(4 24± 1 04)mmol/L , P <0 001〕.In 50 patients with progressive ischemic stroke,Glu concentration in CSF was significantly positively correlated with CTV values ( r =0 303, P <0 05).Conclusions:The sustained elevation of Glu observed in progressive stroke suggests that the window to prevent neurological deterioration may be wider.Determination of Glu concentration in CSF may serve as an available index to judge the degree of ischemic brain injury and evaluate curative effect of Glu release inhibitors.

目的:探索稳定性和进展性急性缺血性卒中患者体液中谷氨酸( Glu)升高的时限。方法:应用高效液相色谱分析检测128 例首次发病的急性脑梗死( A C I)患者发病1~7 日的血浆 Glu、脑脊液( C S F) Glu 浓度的变化,并与同期检测的神经功能缺失程度( C S S积分)、脑梗死容积( C T V)进行相关分析。结果:128 例 A C I患者的平均血浆 Glu、 C S F Glu 浓度分别为(13629±3253)m m ol/ L和(648±329)m m ol/ L,均显著高于对照组〔(12920±2815)m m ol/ L和(309±148)m m ol/ L, P< 005 和 P< 0001〕;进展性 A C I组平均 C S F Glu 浓度为(647±132)m m ol/ L,极显著高于稳定性 A C I组〔(532±107)m m ol/ L, P< 0001〕;稳定性组 1~7 日各时间点的血浆 Glu 浓度无显著变化,而进展性 A C I组于发病 4 日内的血浆 Glu 浓度进行性升高; C T V≥10 cm 3 组平均 C S F...

目的:探索稳定性和进展性急性缺血性卒中患者体液中谷氨酸( Glu)升高的时限。方法:应用高效液相色谱分析检测128 例首次发病的急性脑梗死( A C I)患者发病1~7 日的血浆 Glu、脑脊液( C S F) Glu 浓度的变化,并与同期检测的神经功能缺失程度( C S S积分)、脑梗死容积( C T V)进行相关分析。结果:128 例 A C I患者的平均血浆 Glu、 C S F Glu 浓度分别为(13629±3253)m m ol/ L和(648±329)m m ol/ L,均显著高于对照组〔(12920±2815)m m ol/ L和(309±148)m m ol/ L, P< 005 和 P< 0001〕;进展性 A C I组平均 C S F Glu 浓度为(647±132)m m ol/ L,极显著高于稳定性 A C I组〔(532±107)m m ol/ L, P< 0001〕;稳定性组 1~7 日各时间点的血浆 Glu 浓度无显著变化,而进展性 A C I组于发病 4 日内的血浆 Glu 浓度进行性升高; C T V≥10 cm 3 组平均 C S F Glu 浓度为(674±137)m m ol/ L,极显著高于 C T V< 10 cm ?

 
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